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1. Full Text Forkhead box O3 plays a role in skeletal muscle atrophy through expression of E3 ubiquitin ligases MuRF-1 and atrogin-1 in Cushing’s syndrome
by Kang, Seol-Hee and Lee, Hae-Ahm and Kim, Mina and Lee, Eunjo and Sohn, Uy Dong and Kim, Inkyeom
American Journal of Physiology - Endocrinology and Metabolism, ISSN 0193-1849, 06/2017, Volume 312, Issue 6, pp. E495 - E507
Cushing’s syndrome is caused by overproduction of the adrenocorticotropic hormone (ACTH), which stimulates the adrenal grand to make cortisol. Skeletal muscle... 
Skeletal muscle atrophy | Cushing’s syndrome | Forkhead box O3 | Glucocorticoid receptor
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2. Full Text Histone deacetylase inhibition ameliorates hypertension and hyperglycemia in a model of cushing’s syndrome
by Lee, Hae-Ahm and Kang, Seol-Hee and Kim, Mina and Lee, Eunjo and Cho, Hyun-Min and Moon, Eun-Kyung and Kim, Inkyeom
American Journal of Physiology - Endocrinology and Metabolism, ISSN 0193-1849, 01/2018, Volume 314, Issue 1, pp. E39 - E52
Cushing’s syndrome (CS) caused by hypercortisolism is occasionally accompanied by metabolic disorders such as hypertension, diabetes mellitus (DM),... 
Hypertension | Histone deacetylashyperglycemia | Cushing’s syndrome | Glucocorticoid receptor
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3. Forkhead box O^sub 3^ plays a role in skeletal muscle atrophy through expression of E^sub 3^ ubiquitin ligases MuRF-1 and atrogin-1 in Cushing's syndrome
by Seol-Hee Kang and Hae-Ahm Lee and Mina Kim and Eunjo Lee and Uy Dong Sohn and Inkyeom Kim
American Journal of Physiology, ISSN 0193-1849, 06/2017, Volume 312, Issue 6, p. E495
Cushing's syndrome is caused by overproduction of the adrenocorticotropic hormone (ACTH), which stimulates the adrenal grand to make cortisol. Skeletal muscle... 
Ubiquitin | Transcription | Glucocorticoids | Hormones | Hydrocortisone | Atrophy | Proteins | Infusion | Rodents | Forkhead protein | FOXO3 protein | Enzymes | Nervous system diseases | Pol II | Dexamethasone | Diabetes mellitus | Muscles | Cushing's syndrome | Skeletal muscle | Musculoskeletal system | Adrenocorticotropic hormone | Pituitary | Weight reduction | Protein expression | Sepsis
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4. Full Text Comparative molecular analysis of endurance exercise in vivo with electrically stimulated in vitro myotube contraction
by Son, Young Hoon and Lee, Seung-Min and Lee, Seol Hee and Yoon, Jong Hyeon and Kang, Jae Sook and Yang, Yong Ryoul and Kwon, Ki-Sun
Journal of Applied Physiology, ISSN 8750-7587, 10/2019
Exercise has positive effects on health and improves a variety of disease conditions. An in vitro model of exercise has been developed to better understand its... 
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5. Histone deacetylase inhibition ameliorates hypertension and hyperglycemia in a model of Cushing's syndrome
by Hae-Ahm Lee and Seol-Hee Kang and Mina Kim and Eunjo Lee and Hyun-Min Cho and Eun-Kyung Moon and Inkyeom Kim
American Journal of Physiology, ISSN 0193-1849, 01/2018, Volume 314, Issue 1, p. E39
Cushing's syndrome (CS) caused by hypercortisolism is occasionally accompanied by metabolic disorders such as hypertension, diabetes mellitus (DM),... 
Histone deacetylase | Regulators | Chromatin | Immunoprecipitation | Transcription | Glucocorticoids | Genes | Dyslipidemia | Genomes | Glucose | Blood | Hyperglycemia | Rodents | Pituitary gland | Blood pressure | Inhibition | Acetylation | Gluconeogenesis | Hypertension | Nervous system diseases | Kidneys | Dexamethasone | Diabetes mellitus | Rats | Cushing's syndrome | Gene expression | Patients | Morbidity | Glucose tolerance | Intolerance | Adrenocorticotropic hormone | Pituitary | Cardiovascular diseases | Metabolic disorders
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6. Full Text Linalool elicits vasorelaxation of mouse aortae through activation of guanylyl cyclase and K+ channels
by Kang, Purum and Seol, Geun Hee
Journal of Pharmacy and Pharmacology, ISSN 0022-3573, 05/2015, Volume 67, Issue 5, pp. 714 - 719
Objectives The aim of this study was to investigate the cardiovascular relaxing properties of monoterpene alcohol (‐)‐linalool (LIN), a principal component of... 
K+ channel | (‐)‐linalool | soluble guanylyl cyclase | vasorelaxation | channel | (-)-linalool | Vasorelaxation | Soluble guanylyl cyclase | K + channel | SMOOTH-MUSCLE | POTASSIUM CHANNELS | ESSENTIAL OIL | NITRIC-OXIDE | PHARMACOLOGY & PHARMACY | STRESS | HYPERTENSIVE-RATS | BLOOD-PRESSURE | NG-Nitroarginine Methyl Ester - pharmacology | Tetraethylammonium - pharmacology | Vasodilator Agents - pharmacology | Calcium - metabolism | Aorta - drug effects | Endoplasmic Reticulum - metabolism | Guanylate Cyclase - metabolism | Male | Aorta - metabolism | Enzyme Activation - drug effects | Dose-Response Relationship, Drug | Potassium Channels - metabolism | Quinoxalines - pharmacology | Calcium Chloride - pharmacology | Monoterpenes - pharmacology | Animals | Oxadiazoles - pharmacology | Calcium Chloride - antagonists & inhibitors | Dinoprost - antagonists & inhibitors | Mice | Monoterpenes - antagonists & inhibitors | Vasodilation - drug effects | Dinoprost - pharmacology | Blood vessels | Dilatation | Nitric oxide
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7. Full Text Histone deacetylase inhibition attenuates cardiac hypertrophy and fibrosis through acetylation of mineralocorticoid receptor in spontaneously hypertensive rats
by Kang, Seol-Hee and Kang, Seol-Hee and Seok, Young M.i and Seok, Young Mi and Song, Min-ji and Song, Min-Ji and Lee, Hae-Ahm and Lee, Hae-Ahm and Kurz, Thomas and Kurz, Thomas and Kim, InKyeom and Kim, InKyeom
Molecular pharmacology, ISSN 0026-895X, 05/2015, Volume 87, Issue 5, pp. 782 - 791
Inhibition of histone deacetylases (HDACs) by valproic acid (VPA) attenuates inflammatory, hypertrophic, and fibrotic responses in the hearts of spontaneously... 
ANDROGEN RECEPTOR | TRANSCRIPTIONAL ACTIVITY | GLUCOCORTICOID-RECEPTOR | HINGE REGION | TRANSACTIVATION | P300 | ALPHA | PHARMACOLOGY & PHARMACY | ANGIOTENSIN-II | ALDOSTERONE | BINDING | Receptors, Mineralocorticoid - genetics | Fibrosis - drug therapy | Gene Expression - drug effects | Gene Expression - genetics | Receptors, Mineralocorticoid - metabolism | Rats, Inbred WKY | Male | RNA Polymerase II - metabolism | Fibrosis - metabolism | Promoter Regions, Genetic - drug effects | Promoter Regions, Genetic - genetics | Chromatin Immunoprecipitation - methods | Rats, Inbred SHR | Fibrosis - genetics | Histone Deacetylases - genetics | Cardiomegaly - drug therapy | Rats | Histone Deacetylases - metabolism | Acetylation - drug effects | Animals | Histones - genetics | Histone Deacetylase Inhibitors - pharmacology | RNA Polymerase II - genetics | Histones - metabolism | Cardiomegaly - genetics | Cardiomegaly - metabolism
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8. Histone deacetylase inhibition, but not a mineralocorticoid receptor antagonist spironolactone, attenuates atypical transcription by an activating mutant MR (MR sub(S) sub(810L))
by Kang, Seol-Hee and Lee, Hae-Ahm and Lee, Eunjo and Kim, Mina and Kim, Inkyeom
Clinical and Experimental Pharmacology and Physiology, ISSN 0305-1870, 10/2016, Volume 43, Issue 10, pp. 995 - 1003
A mutation in the mineralocorticoid receptor (MR sub(S) sub(810L)) leads to early-onset hypertension, which is markedly exacerbated during pregnancy. The... 
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9. Full Text Histone deacetylase inhibition ameliorates hypertension and hyperglycemia in a model of Cushing's syndrome
by Lee, HA and Kang, SH and Kim, M and Lee, E and Cho, HM and Moon, EK and Kim, I
AMERICAN JOURNAL OF PHYSIOLOGY-ENDOCRINOLOGY AND METABOLISM, ISSN 0193-1849, 01/2018, Volume 314, Issue 1, pp. E39 - E52
Cushing's syndrome (CS) caused by hypercortisolism is occasionally accompanied by metabolic disorders such as hypertension, diabetes mellitus (DM),... 
glucocorticoid receptor | TRANSCRIPTIONAL ACTIVITY | SODIUM | PHYSIOLOGY | REDUCES INSULIN-RESISTANCE | TYPE-2 DIABETIC-RAT | HDAC INHIBITION | Cushing's syndrome | VALPROIC ACID | GLUCOCORTICOID RESPONSE ELEMENT | histone deacetylase | MINERALOCORTICOID RECEPTOR | hyperglycemia | GLUCOSE-HOMEOSTASIS | GENE | ENDOCRINOLOGY & METABOLISM | hypertension | Hyperglycemia - prevention & control | Cushing Syndrome - physiopathology | Humans | Hyperglycemia - complications | Rats | Male | Rats, Sprague-Dawley | Hypertension - physiopathology | Blood Glucose - drug effects | Hypertension - blood | Animals | Cushing Syndrome - pathology | Hyperglycemia - blood | HEK293 Cells | Hypertension - complications | Hypertension - prevention & control | Cushing Syndrome - blood | Histone Deacetylase Inhibitors - pharmacology | Blood Pressure - drug effects | Histone Deacetylase Inhibitors - therapeutic use | Blood Glucose - metabolism | Hyperglycemia - physiopathology | Cushing Syndrome - drug therapy | Disease Models, Animal
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10. Full Text Forkhead box O3 plays a role in skeletal muscle atrophy through expression of E3 ubiquitin ligases MuRF-1 and atrogin-1 in Cushing's syndrome
by Kang, SH and Lee, HA and Kim, M and Lee, E and Sohn, UD and Kim, I
AMERICAN JOURNAL OF PHYSIOLOGY-ENDOCRINOLOGY AND METABOLISM, ISSN 0193-1849, 06/2017, Volume 312, Issue 6, pp. E495 - E507
Cushing's syndrome is caused by overproduction of the adrenocorticotropic hormone (ACTH), which stimulates the adrenal grand to make cortisol. Skeletal muscle... 
glucocorticoid receptor | PHYSIOLOGY | PROTEIN | ANTAGONIST | skeletal muscle atrophy | COMPONENTS | Cushing's syndrome | AUTOPHAGY | FOXO TRANSCRIPTION FACTORS | HYPERTROPHY | GLUCOCORTICOID-RECEPTOR | ENDOCRINOLOGY & METABOLISM | GENE-EXPRESSION | forkhead box O3 | CROSSTALK | UP-REGULATION | Receptors, Glucocorticoid - antagonists & inhibitors | Cushing Syndrome - physiopathology | SKP Cullin F-Box Protein Ligases - genetics | Ubiquitin-Protein Ligases - antagonists & inhibitors | Male | Muscle, Skeletal - metabolism | Receptors, Glucocorticoid - metabolism | Muscle Fibers, Skeletal - drug effects | Muscle Fibers, Skeletal - metabolism | Cushing Syndrome - metabolism | Promoter Regions, Genetic - drug effects | Receptors, Glucocorticoid - agonists | Forkhead Box Protein O3 - genetics | Chromatin Immunoprecipitation | Cushing Syndrome - pathology | RNA Interference | Muscle, Skeletal - drug effects | Muscle Proteins - metabolism | Forkhead Box Protein O3 - antagonists & inhibitors | Muscle Proteins - antagonists & inhibitors | Muscular Atrophy - etiology | Disease Models, Animal | Cell Line | Tripartite Motif Proteins - antagonists & inhibitors | Ubiquitin-Protein Ligases - metabolism | Tripartite Motif Proteins - agonists | Tripartite Motif Proteins - genetics | SKP Cullin F-Box Protein Ligases - metabolism | SKP Cullin F-Box Protein Ligases - antagonists & inhibitors | Rats, Sprague-Dawley | Forkhead Box Protein O3 - metabolism | Hormone Antagonists - pharmacology | Forkhead Box Protein O3 - agonists | Gene Expression Regulation - drug effects | Muscle Proteins - genetics | Response Elements - drug effects | Muscle Proteins - agonists | Animals | Active Transport, Cell Nucleus - drug effects | Muscle Fibers, Skeletal - pathology | Glucocorticoids - pharmacology | Genes, Reporter - drug effects | Tripartite Motif Proteins - metabolism | Muscle, Skeletal - pathology | Ubiquitin-Protein Ligases - genetics | Muscles | Physiological aspects | Atrophy, Muscular | Cushing syndrome
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11. Full Text Antioxidant activity of linalool in patients with carpal tunnel syndrome
by Seol, Geun-Hye and Kang, Purum and Lee, Hui Su and Seol, Geun Hee
BMC Neurology, ISSN 1471-2377, 02/2016, Volume 16, Issue 1, p. 17
Background: Carpal tunnel syndrome (CTS) is a common peripheral neuropathy and ischemic-reperfusion injury. Oxidative stress is considered a major cause of... 
Carpal tunnel syndrome | Antioxidative activity | Linalool | OXIDATIVE STRESS | INFLAMMATION | RATS | MICE | CLINICAL NEUROLOGY | Double-Blind Method | Humans | Middle Aged | Administration, Inhalation | Male | Heart Rate - drug effects | Antioxidants - therapeutic use | Adult | Female | Aged | Blood Pressure - drug effects | Carpal Tunnel Syndrome - drug therapy | Monoterpenes - therapeutic use | Antioxidants | Complications and side effects | Care and treatment | Reactive oxygen species | Influence | Research
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12. Full Text Histone deacetylase inhibition attenuates hepatic steatosis in rats with experimental Cushing's syndrome
by Kim, Mina and Lee, Hae-Ahm and Cho, Hyun-Min and Kang, Seol-Hee and Lee, Eunjo and Kim, In Kyeom
The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology, ISSN 0372-1582, 2018, Volume 22, Issue 1, pp. 23 - 33
Cushing's syndrome (CS) is a collection of symptoms caused by prolonged exposure to excess cortisol. Chronically elevated glucocorticoid (GC) levels contribute... 
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13. Full Text Combined oral contraceptive synergistically activates mineralocorticoid receptor through histone code modifications
by Igunnu, Adedoyin and Seok, Young-Mi and Olatunji, Lawrence A and Kang, Seol-Hee and Kim, Inkyeom
European Journal of Pharmacology, ISSN 0014-2999, 12/2015, Volume 769, pp. 48 - 54
Clinical studies have shown that the use of combined oral contraceptive in pre-menopausal women is associated with fluid retention. However, the molecular... 
Oral contraceptive | Mineralocorticoid receptor | Histone code | Gene transcription | SYSTEM | TRANSCRIPTIONAL ACTIVITY | HEART-FAILURE | RATS | BLOOD-PRESSURE | DROSPIRENONE | PHARMACOLOGY & PHARMACY | ALDOSTERONE | ETHINYLESTRADIOL | BLOCKADE | FLUID-RELATED SYMPTOMS | Kidney Cortex - drug effects | Kidney Cortex - metabolism | Receptors, Mineralocorticoid - metabolism | Protein-Serine-Threonine Kinases - genetics | Rats | Norgestrel - pharmacology | Protein Transport - drug effects | RNA Polymerase II - metabolism | Transcription Factors - genetics | Ethinyl Estradiol - pharmacology | Promoter Regions, Genetic - drug effects | Rats, Sprague-Dawley | Drug Synergism | Gene Expression Regulation - drug effects | Animals | Immediate-Early Proteins - genetics | Histone Code - drug effects | Base Sequence | Contraceptives, Oral, Combined - pharmacology | Female | Receptors, Mineralocorticoid - chemistry | Norgestrel | Medical colleges | Corticosteroids | RNA | Analysis | Histones | Ethinyl estradiol | Oral contraceptives | Adenosine triphosphatase | Steroids
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14. Full Text Histone deacetylase 3 and 4 complex stimulates the transcriptional activity of the mineralocorticoid receptor
by Lee, Hae-Ahm and Song, Min-Ji and Seok, Young-Mi and Kang, Seol-Hee and Kim, Sang-Yeob and Kim, Inkyeom
PLoS ONE, ISSN 1932-6203, 08/2015, Volume 10, Issue 8, p. e0136801
Histone deacetylases (HDACs) act as corepressors in gene transcription by altering the acetylation of histones, resulting in epigenetic gene silencing. We... 
PROTEIN-KINASE-A | SYSTEM | LOCALIZATION | ACETYLATION | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | CLASS-I | MUSCLE | ALDOSTERONE | INHIBITORS | CAMP | Cyclic AMP-Dependent Protein Kinases - metabolism | Promoter Regions, Genetic | Receptors, Mineralocorticoid - genetics | Histone Deacetylases - genetics | Receptors, Mineralocorticoid - metabolism | Humans | Phosphoprotein Phosphatases - metabolism | Histone Deacetylase Inhibitors - administration & dosage | Repressor Proteins - genetics | Histone Deacetylases - metabolism | Gene Expression Regulation, Enzymologic | Oxazoles - administration & dosage | Transcription, Genetic | RNA, Small Interfering | Acetylation | Repressor Proteins - metabolism | Epigenetic inheritance | Mineralocorticoids | Genetic transcription | Research | Protein kinase A | Histone deacetylase | Chromatin | Phosphorylation | Transcription factors | Immunoprecipitation | Genes | Aldosterone | Kinases | Phosphatase | DNA-directed RNA polymerase | Proteins | Transcription activation | Histones | Physiology | Calyculin A | Translocation | Pharmacology | siRNA | Gene expression | Nuclear transport | Medicine | Polymerase | Gene silencing | Ribonucleic acids | Lysine | Plasmids | RNA polymerase II
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15. Full Text Self-assembled metalla-rectangles bearing azodipyridyl ligands: Synthesis, characterization and antitumor activity
by Vajpayee, Vaishali and Lee, Sunmi and Kim, Seol-Hee and Kang, Se Chan and Cook, Timothy R and Kim, Hyunuk and Kim, Dong Wook and Verma, Shashi and Lah, Myoung Soo and Kim, In Su and Wang, Ming and Stang, Peter J and Chi, Ki-Whan
Dalton Transactions, ISSN 1477-9226, 01/2013, Volume 42, Issue 2, pp. 466 - 475
Sixteen arene-Ru based molecular-rectangles were self-assembled in high yields by the equimolar mixing of arene-Ru acceptors (Aa-Ad) with various azopyridyl... 
MOLECULAR-RECTANGLES | MULTI-CARBOXYLATE ANIONS | ANTICANCER ACTIVITY | IN-VITRO | INHIBITION | ARENE COMPLEXES | CANCER-CHEMOTHERAPY | SERUM-ALBUMIN | RUTHENIUM | CELL-DEATH | CHEMISTRY, INORGANIC & NUCLEAR | Chemistry Techniques, Synthetic | Apoptosis - drug effects | Pyridines - chemistry | Antineoplastic Agents - chemical synthesis | Humans | Molecular Conformation | Models, Molecular | Antineoplastic Agents - chemistry | Antineoplastic Agents - metabolism | Azo Compounds - chemistry | Biological Transport | Organometallic Compounds - metabolism | Organometallic Compounds - chemical synthesis | Cell Line, Tumor | Ligands | Organometallic Compounds - chemistry | Antineoplastic Agents - pharmacology | Cell Cycle - drug effects | Organometallic Compounds - pharmacology
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16. Self-assembled metalla-rectangles bearing azodipyridyl ligands: synthesis, characterization and antitumor activity
by Vajpayee, Vaishali and Lee, Sunmi and Kim, Seol-Hee and Kang, Se Chan and Cook, Timothy R and Kim, Hyunuk and Kim, Dong Wook and Verma, Shashi and Lah, Myoung Soo and Kim, In Su and Wang, Ming and Stang, Peter J and Chi, Ki-Whan
Dalton Transactions, ISSN 1477-9226, 12/2012, Volume 42, Issue 2, pp. 466 - 475
Sixteen arene-Ru based molecular-rectangles were self-assembled in high yields by the equimolar mixing of arene-Ru acceptors (Aa-Ad) with various azopyridyl... 
Human | Cellular | Diffraction | Fluorescence | Ligands | Position (location) | Apoptosis | Cancer
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17. Full Text A deep subwavelength cavity formed by total external reflection of surface plasmon polariton
by Seol, Kang Hee and Lee, Kwang-Geol and Song, Seok Ho
Journal of Applied Physics, ISSN 0021-8979, 05/2015, Volume 117, Issue 17, p. 173104
We numerically analyze the characteristics of a nanocavity in surface plasmon polariton (SPP) modes confined by total external reflection (TER) at deep... 
PHYSICS, APPLIED | PHOTOLUMINESCENCE | LASERS | WAVE-GUIDES | CONFINEMENT | ABSORPTION | SCALE | ZNSE | PROPAGATION | METAL-FILMS | Polaritons | Research | Plasmons (Physics) | Analysis | Photonics
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18. The essential oil of Citrus bergamiaRisso induces vasorelaxation of the mouse aorta by activating K+ channels and inhibiting Ca2+ influx
by Purum Kang and Suk Hyo Suh and Sun Seek Min and Geun Hee Seol
Journal of Pharmacy and Pharmacology, ISSN 0022-3573, 05/2013, Volume 65, Issue 5, p. 745
  Objectives The aim of this study was to explore the effect of the essential oil of Citrus bergamiaRisso (bergamot) on mouse blood vessels and to analyse the... 
Proteins | Rodents | Blood vessels | Endothelium
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19. Full Text Low body mass index can be associated with the risk and poor outcomes of neuromyelitis optica with aquaporin-4 immunoglobulin G in women
by Baek, Seol-Hee and Kim, Ji-Sun and Jang, Myoung-jin and Kim, Yoo Hwan and Kwon, Ohyun and Oh, Jung-Hwan and Kang, Sa-Yoon and Kang, Ji-Hoon and Park, Kee Hong and Park, Yong-Shik and Park, Kyung Seok and Shin, Dong Wook and Kim, Byung-Jo and Kim, Sung-Min
Journal of Neurology, Neurosurgery & Psychiatry, ISSN 0022-3050, 11/2018, Volume 89, Issue 11, pp. 1228 - 1228
The demographic data and clinical characteristics of patients with low BMImin-1yr (<18.5 kg/m2) and normal BMImin-1yr (≥18.5 kg/m2) are shown in table 5 in the... 
neuroimmunology | SURGERY | PSYCHIATRY | CLINICAL NEUROLOGY | Studies | Neurology | Body mass index | Immunoglobulins | Statistical analysis | Disease | Womens health | Aquaporins | Rheumatoid arthritis | Mortality | Regression analysis | Age | 1506 | PostScript
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20. Full Text Histidine residues in the IS3–IS4 loop are critical for nickel‐sensitive inhibition of the Cav2.3 calcium channel
by Kang, Ho-Won and Moon, Hyung-Jo and Joo, Seol-hee and Lee, Jung-Ha
FEBS Letters, ISSN 0014-5793, 12/2007, Volume 581, Issue 30, pp. 5774 - 5780
We recently reported that a histidine (H191) in the S3–S4 loop of domain I is critical for nickel inhibition of the Cav3.2 T‐type Ca2+ channel. As in Cav3.2,... 
Cav2.3 channel | Nickel inhibition | S3–S4 loop | Histidine | Voltage clamping | 2.3 channel | S3-S4 loop | Xenopus | Amino Acid Sequence | Cation Transport Proteins - antagonists & inhibitors | Protein Structure, Secondary | Humans | Molecular Sequence Data | Mutant Proteins - metabolism | Structure-Activity Relationship | Histidine - metabolism | Dose-Response Relationship, Drug | Sequence Alignment | Animals | Cation Transport Proteins - metabolism | Mutant Proteins - chemistry | Female | Calcium Channels, R-Type - chemistry | Cation Transport Proteins - chemistry | Nickel - pharmacology | Calcium Channels, R-Type - metabolism | Ion Channel Gating - drug effects
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