X
Search Filters
Format Format
Format Format
X
Sort by Item Count (A-Z)
Filter by Count
Journal Article (260) 260
Publication (33) 33
Patent (9) 9
Conference Proceeding (7) 7
Magazine Article (7) 7
Newspaper Article (3) 3
Book / eBook (1) 1
Book Chapter (1) 1
more...
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
index medicus (75) 75
animals (46) 46
humans (42) 42
cancer (28) 28
oncology (28) 28
analysis (27) 27
article (27) 27
pancreatic cancer (26) 26
pancreatic neoplasms - pathology (23) 23
mice (22) 22
life sciences (21) 21
gene expression (20) 20
microbiology (19) 19
proteins (18) 18
biochemistry & molecular biology (17) 17
cell line, tumor (17) 17
expression (17) 17
pancreatic neoplasms - metabolism (16) 16
apoptosis (15) 15
female (15) 15
tumors (15) 15
cell proliferation (14) 14
pancreatic neoplasms - genetics (14) 14
research (14) 14
genes (13) 13
genetic aspects (13) 13
phylogeny (13) 13
growth (12) 12
multidisciplinary sciences (12) 12
biotechnology & applied microbiology (11) 11
carcinoma, pancreatic ductal - pathology (11) 11
gene (11) 11
health aspects (11) 11
signal transduction (11) 11
biotechnology (10) 10
cell biology (10) 10
chemistry (10) 10
escherichia-coli (10) 10
genomes (10) 10
identification (10) 10
plant sciences (10) 10
research article (10) 10
adenocarcinoma (9) 9
biology (9) 9
disease models, animal (9) 9
genetics & heredity (9) 9
male (9) 9
metabolism (9) 9
metabolites (9) 9
molecular sequence data (9) 9
mutation (9) 9
plant genetics & genomics (9) 9
wheat (9) 9
agronomy (8) 8
bacteria (8) 8
cells (8) 8
genetic research (8) 8
metastasis (8) 8
pancreatic ductal adenocarcinoma (8) 8
studies (8) 8
virology (8) 8
adenocarcinoma - genetics (7) 7
adenocarcinoma - pathology (7) 7
autophagy (7) 7
base sequence (7) 7
carcinoma, pancreatic ductal - genetics (7) 7
carcinoma, pancreatic ductal - metabolism (7) 7
care and treatment (7) 7
gemcitabine (7) 7
gene expression profiling (7) 7
gene expression regulation, neoplastic (7) 7
genomics (7) 7
homeostasis (7) 7
medicine (7) 7
metallurgy (7) 7
microbial genetics and genomics (7) 7
organic chemistry (7) 7
pancreatic neoplasms - drug therapy (7) 7
prognosis (7) 7
resistance (7) 7
salmonella (7) 7
agriculture (6) 6
amino acid sequence (6) 6
cell growth (6) 6
china (6) 6
dna (6) 6
ductal adenocarcinoma (6) 6
enzymes (6) 6
fibroblasts (6) 6
geese (6) 6
immunization (6) 6
immunogenicity (6) 6
medical research (6) 6
medicine, research & experimental (6) 6
mice, nude (6) 6
models (6) 6
pathway (6) 6
phosphorylation (6) 6
physiological aspects (6) 6
plant physiology (6) 6
more...
Library Location Library Location
Language Language
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


Nature, ISSN 0028-0836, 02/2017, Volume 542, Issue 7639, pp. 119 - 123
The genome of pancreatic ductal adenocarcinoma (PDAC) frequently contains deletions of tumour suppressor gene loci, most notably SMAD4, which is homozygously... 
SREBP ACTIVITY | PATHWAYS | AMINO-ACID-METABOLISM | ACTIVATED PROTEIN-KINASE | GLUTAMINE | GENE | ADENOCARCINOMA | MULTIDISCIPLINARY SCIENCES | BIOLOGY | GROWTH | TARGETS | Mitochondria - enzymology | AMP-Activated Protein Kinases - metabolism | Reactive Oxygen Species - metabolism | Amino Acids, Branched-Chain - metabolism | Humans | Pregnancy Proteins - genetics | Male | Carcinoma, Pancreatic Ductal - genetics | Malate Dehydrogenase - genetics | Carcinoma, Pancreatic Ductal - psychology | Gene Deletion | Minor Histocompatibility Antigens - genetics | NADP - metabolism | Ketoglutaric Acids - metabolism | Sterol Regulatory Element Binding Protein 1 - metabolism | Transaminases - biosynthesis | Biocatalysis | Transaminases - genetics | Pancreatic Neoplasms - pathology | Pancreatic Neoplasms - enzymology | Pancreatic Neoplasms - genetics | Carcinoma, Pancreatic Ductal - therapy | Malate Dehydrogenase - deficiency | Mitochondria - pathology | NADP - biosynthesis | Pregnancy Proteins - biosynthesis | Carcinoma, Pancreatic Ductal - enzymology | Minor Histocompatibility Antigens - biosynthesis | Animals | Mice | Pancreatic Neoplasms - therapy | Enzymes | Complications and side effects | Pancreatic cancer | Genetic aspects | Chromosome deletion | Health aspects | Risk factors | Cell growth | Mitochondria | Metabolites | Homeostasis | Collateral | Biosynthesis | Tumors | Cancer | Apoptosis
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2015, Volume 10, Issue 3, pp. e0121082 - e0121082
Background HNF1A (Hepatocyte nuclear factor 1 alpha) is a transcription factor that is known to regulate pancreatic differentiation and maintain homeostasis of... 
TRANSCRIPTION FACTORS | INACTIVATION | FACTOR 1-ALPHA | GENE | HNF-1-ALPHA | HNF1-ALPHA | MULTIDISCIPLINARY SCIENCES | ACTIVATED AKT | DOMINANT-NEGATIVE SUPPRESSION | EXPRESSION | BETA-CELL FUNCTION | Immunohistochemistry | Phosphorylation | Adenocarcinoma - pathology | Cell Proliferation | Pancreatic Neoplasms - metabolism | Hepatocyte Nuclear Factor 1-alpha - genetics | TOR Serine-Threonine Kinases - metabolism | Humans | Pancreatic Neoplasms - pathology | Gene Expression Regulation, Neoplastic | Pancreatic Neoplasms - genetics | Signal Transduction - genetics | Hepatocyte Nuclear Factor 1-alpha - metabolism | Blotting, Western | Gene Knockdown Techniques | Cell Cycle | Cell Line, Tumor | Adenocarcinoma - genetics | Proto-Oncogene Proteins c-akt - metabolism | Genes, Tumor Suppressor | Apoptosis | RNA, Small Interfering - metabolism | Proteins | RNA | Analysis | Pancreatic cancer | Genomics | Genomes | Cellular signal transduction | Genetic aspects | Gene expression | Cancer | Adenocarcinoma | Cell proliferation | Biotechnology | Flow cytometry | G2 phase | Homeostasis | AKT protein | Oncology | Kinases | Tissues | Carcinogenesis | Gene sequencing | Liver cancer | Signal transduction | Carcinogens | Cell growth | Pancreatic carcinoma | Rodents | Cell cycle | Pancreas | Growth factors | Hepatocyte nuclear factor 1 | Rapamycin | Tumor cell lines | Polymerase chain reaction | Cytometry | Signaling | S phase | Cell lines | Tumor suppressor genes | HNF1a gene | Diabetes | Mutation | Tumors | Index Medicus
Journal Article
当代县域经济, ISSN 2095-6371, 2017, Issue 10, pp. 70 - 72
Journal Article
Cancer Letters, ISSN 0304-3835, 09/2017, Volume 403, pp. 296 - 304
The mechanism for improved therapeutic efficacy of the combination therapy with nanoparticle albumin-bound paclitaxel (nAb-PTX) and gemcitabine (gem) for... 
nAb-paclitaxel | Multistage nanovectors | Gemcitabine | Drug resistance | Transport | Pancreatic cancer | PLUS GEMCITABINE | SYSTEMIC DELIVERY | BREAST-CANCER THERAPY | BIOLOGICAL BARRIERS | PHASE-III TRIAL | NUDE-MOUSE MODEL | ONCOLOGY | MESOPOROUS SILICON PARTICLES | CAVEOLIN-1 GENE | Up-Regulation | Pancreatic Neoplasms - metabolism | Apoptosis - drug effects | Humans | Carcinoma, Pancreatic Ductal - metabolism | Deoxycytidine - pharmacology | Drug Resistance, Neoplasm | Male | Drug Carriers | Nanoparticles | Pancreatic Neoplasms - drug therapy | Time Factors | Drug Compounding | Antimetabolites, Antineoplastic - pharmacology | Antineoplastic Agents, Phytogenic - metabolism | Albumin-Bound Paclitaxel - metabolism | Pancreatic Neoplasms - pathology | Antineoplastic Agents, Phytogenic - chemistry | Carcinoma, Pancreatic Ductal - pathology | Caveolin 1 - metabolism | Xenograft Model Antitumor Assays | Carcinoma, Pancreatic Ductal - drug therapy | Animals | Tumor Burden - drug effects | Mice, Nude | Cell Line, Tumor | Albumin-Bound Paclitaxel - chemistry | Cell Proliferation - drug effects | Antineoplastic Agents, Phytogenic - pharmacology | Deoxycytidine - analogs & derivatives | Adenocarcinoma | Chemotherapy | Analysis | Albumin | Cancer | Encapsulation | Cell proliferation | Caveolin-1 | Silicon wafers | Metastasis | Cancer therapies | Accumulation | Biomedical materials | Paclitaxel | Pancreas | Polyclonal antibodies | Caveolin | Data processing | Breast cancer | Patients | Nanotechnology | Apoptosis | Tumors | nab-paclitaxel | gemcitabine | multistage nanovectors | drug resistance | transport
Journal Article
by Li, Zheng and Allingham, R. Rand and Nakano, Masakazu and Jia, Liyun and Chen, Yuhong and Ikeda, Yoko and Mani, Baskaran and Chen, Li-Jia and Kee, Changwon and Garway-Heath, David F and Sripriya, Sarangapani and Fuse, Nobuo and Abu-Amero, Khaled K and Huang, Chukai and Namburi, Prasanthi and Burdon, Kathryn and Perera, Shamira A and Gharahkhani, Puya and Lin, Ying and Ueno, Morio and Ozaki, Mineo and Mizoguchi, Takanori and Krishnadas, Subbiah Ramasamy and Osman, Essam A and Lee, Mei Chin and Chan, Anita S.Y and Tajudin, Liza-Sharmini A and Do, Tan and Goncalves, Aurelien and Reynier, Pascal and Zhang, Hong and Bourne, Rupert and Goh, David and Broadway, David and Husain, Rahat and Negi, Anil K and Su, Daniel H and Ho, Ching-Lin and Blanco, Augusto Azuara and Leung, Christopher K.S and Wong, Tina T and Yakub, Azhany and Liu, Yutao and Nongpiur, Monisha E and Han, Jong Chul and Hon, Do Nhu and Shantha, Balekudaru and Zhao, Bowen and Sang, Jinghong and Zhang, NiHong and Sato, Ryuichi and Yoshii, Kengo and Panda-Jonas, Songhomita and Ashley Koch, Allison E and Herndon, Leon W and Moroi, Sayoko E and Challa, Pratap and Foo, Jia Nee and Bei, Jin-Xin and Zeng, Yi-Xin and Simmons, Cameron P and Bich Chau, Tran Nguyen and Sharmila, Philomenadin Ferdinamarie and Chew, Merwyn and Lim, Blanche and Tam, Pansy O.S and Chua, Elaine and Ng, Xiao Yu and Yong, Victor H.K and Chong, Yaan Fun and Meah, Wee Yang and Vijayan, Saravanan and Seongsoo, Sohn and Xu, Wang and Teo, Yik Ying and Cooke Bailey, Jessica N and Kang, Jae H and Haines, Jonathan L and Cheng, Ching Yu and Saw, Seang-Mei and Tai, E.-Shyong and Richards, Julia E and Ritch, Robert and Gaasterland, Douglas E and Pasquale, Louis R and Liu, Jianjun and Jonas, Jost B and Milea, Dan and George, Ronnie and Al-Obeidan, Saleh A and Mori, Kazuhiko and Macgregor, Stuart and Hewitt, Alex W and Girkin, Christopher A and Zhang, Mingzhi and Sundaresan, Periasamy and Vijaya, Lingam and Mackey, David A and Wong, Tien Yin and Craig, Jamie E and ... and ICAARE-Glaucoma Consortium and NEIGHBORHOOD Consortium
Human Molecular Genetics, ISSN 0964-6906, 07/2015, Volume 24, Issue 13, pp. 3880 - 3892
Primary open angle glaucoma (POAG), a major cause of blindness worldwide, is a complex disease with a significant genetic contribution. We performed Exome... 
NUMBER | RISK-FACTORS | GENE | BIOCHEMISTRY & MOLECULAR BIOLOGY | SUSCEPTIBILITY | GENETICS & HEREDITY | LOCI | PEOPLE | IDENTIFICATION | LINKAGE | CANCER | GENOME-WIDE ASSOCIATION | Life Sciences | Human health and pathology | Association Studies
Journal Article
Laboratory Investigation, ISSN 0023-6837, 07/2018, Volume 98, Issue 7, pp. 947 - 956
Journal Article
International Journal of Biological Sciences, ISSN 1449-2288, 01/2018, Volume 14, Issue 1, pp. 100 - 110
Journal Article
by Aung, T and Ozaki, M and Lee, M.C and Schlotzer-Schrehardt, U and Thorleifsson, G and Mizoguchi, T and Igo, R.P., Jr and Haripriya, A and Williams, S.E and Astakhov, Y.S and Orr, A.C and Burdon, K.P and Nakano, S and Mori, K and Abu-Amero, K and Hauser, M and Li, Z and Prakadeeswari, G and Bailey, J.N and Cherecheanu, A.P and Kang, J.H and Nelson, S and Hayashi, K and Manabe, S.I and Kazama, S and Zarnowski, T and Inoue, K and Irkec, M and Coca-Prados, M and Sugiyama, K and Jarvela, I and Schlottmann, P and Lerner, S.F and Lamari, H and Nilgun, Y and Bikbov, M and Park, K.H and Cha, S.C and Yamashiro, K and Zenteno, J.C and Jonas, J.B and Kumar, R.S.S and Perera, S.A and Chan, A.S.Y and Kobakhidze, N and George, R and Vijaya, L and Do, T and Edward, D.P and Juan Marcos, L. de and Pakravan, M and Moghimi, S and Ideta, R and Bach-Holm, D and Kappelgaard, P and Wirostko, B and Thomas, S and Gaston, D and Bedard, K and Greer, W.L and Yang, Z and Chen, X and Huang, L and Sang, J and Jia, H and Jia, L and Qiao, C and Zhang, H and Liu, X and Zhao, B and Wang, Y.X and Xu, L and Leruez, S and Reynier, P and Chichua, G and Tabagari, S and Uebe, S and Zenkel, M and Berner, D and Mossbock, G and Weisschuh, N and Hoja, U and Welge-Luessen, U.C and Mardin, C and Founti, P and Chatzikyriakidou, A and Pappas, T and Anastasopoulos, E and Lambropoulos, A and Ghosh, A and Shetty, R and Porporato, N and Saravanan, V and Venkatesh, R and Shivkumar, C and Kalpana, N and Sarangapani, S and Kanavi, M.R and Beni, A.N and Yazdani, S and ...
Nature Genetics, ISSN 1061-4036, 2017, Volume 49, Issue 7, pp. 993 - 1004
Exfoliation syndrome (XFS) is the most common known risk factor for secondary glaucoma and a major cause of blindness worldwide. Variants in two genes, LOXL1... 
INDIVIDUALS | RISK LOCI | PSEUDOEXFOLIATION SYNDROME | INFLAMMATORY-BOWEL-DISEASE | METAANALYSIS | POLYMORPHISMS | COMMON SEQUENCE VARIANTS | GENETICS & HEREDITY | GLAUCOMA | BLOOD-PRESSURE | GENOME-WIDE ASSOCIATION | Haplotypes | Humans | Asian Continental Ancestry Group - genetics | Extracellular Matrix - metabolism | Male | Gene Expression Profiling | Mutation, Missense | Amino Acid Oxidoreductases - genetics | Exfoliation Syndrome - ethnology | RNA, Messenger - biosynthesis | Aged, 80 and over | Female | Spheroids, Cellular | Calcium Channels - genetics | Molecular Chaperones - biosynthesis | Eye - metabolism | Genetic Predisposition to Disease | Genome-Wide Association Study | Amino Acid Oxidoreductases - physiology | Molecular Chaperones - genetics | Cell Adhesion | Point Mutation | Alleles | Exfoliation Syndrome - genetics | Amino Acid Substitution | Exfoliatins | Disease susceptibility | Genetic aspects | Analysis | Risk factors | Glaucoma | Genes | Principal components analysis | Cardiovascular disease | Genomes | Biology | Loci | Exfoliation | Studies | Collaboration | Blindness | Alzheimers disease | Life Sciences | Populations and Evolution | Microbiology and Parasitology | Parasitology | Quantitative Methods | Computer Science | Genetics | Santé publique et épidémiologie | Bioinformatics | Human genetics | Virology
Journal Article
Cancer Research, ISSN 0008-5472, 07/2018, Volume 78, Issue 13 Supplement, pp. 1927 - 1927
Journal Article
Cancer Research, ISSN 0008-5472, 07/2016, Volume 76, Issue 14 Supplement, pp. 4215 - 4215
Pancreatic cancer, the most lethal of solid tumors is an aggressive disease that develops relatively symptom-free. The absence of early symptoms has created a... 
Journal Article
Nature, ISSN 0028-0836, 04/2013, Volume 496, Issue 7443, pp. 101 - 105
Cancer cells have metabolic dependencies that distinguish them from their normal counterparts(1). Among these dependencies is an increased use of the amino... 
GLUCOSE | MULTIDISCIPLINARY SCIENCES | MASS-SPECTROMETRY | ADDICTION | TRANSFORMED-CELLS | Care and treatment | Pancreatic cancer | Physiological aspects | Development and progression | Research | Health aspects | Guanosine triphosphatase | Glutamine | Cancer | Enzymes | Oxidative stress | Glucose | Metabolites | Cancer therapies
Journal Article