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index medicus (11) 11
hypertension (10) 10
humans (9) 9
peripheral vascular disease (9) 9
animals (8) 8
blood-pressure (7) 7
male (7) 7
proteinuria (7) 7
blood pressure - drug effects (6) 6
mechanisms (6) 6
hypertension - chemically induced (5) 5
rats (5) 5
rats, inbred wky (5) 5
vascular endothelial growth factor (5) 5
angiogenesis inhibition (4) 4
endothelin-1 (4) 4
endothelin-1 - metabolism (4) 4
female (4) 4
hypertension - physiopathology (4) 4
indoles - pharmacology (4) 4
nitric-oxide (4) 4
physics, applied (4) 4
pyrroles - pharmacology (4) 4
angiogenesis inhibitors - pharmacology (3) 3
carcinoma, renal cell - drug therapy (3) 3
endothelial growth-factor (3) 3
expression (3) 3
hypertension - drug therapy (3) 3
hypertension - metabolism (3) 3
indoles - adverse effects (3) 3
kidney neoplasms - drug therapy (3) 3
middle aged (3) 3
molecular-beam epitaxy (3) 3
nitric oxide - metabolism (3) 3
oxidative stress (3) 3
photoluminescence (3) 3
physics, condensed matter (3) 3
pyrroles - adverse effects (3) 3
sunitinib (3) 3
vascular endothelial growth factor a - antagonists & inhibitors (3) 3
vascular endothelial growth factor a - metabolism (3) 3
78.55.cr (2) 2
activation (2) 2
aged (2) 2
angiogenesis (2) 2
antineoplastic agents - adverse effects (2) 2
bradykinin - pharmacology (2) 2
chemical-vapor-deposition (2) 2
complications and side effects (2) 2
coronary circulation - drug effects (2) 2
cubic gan (2) 2
dose-response relationship, drug (2) 2
endocrinology & metabolism (2) 2
endothelin 1 (2) 2
epitaxy (2) 2
gaas (2) 2
gastrointestinal stromal tumors - drug therapy (2) 2
growth (2) 2
health aspects (2) 2
indoles - therapeutic use (2) 2
indoles - toxicity (2) 2
inhibitor sunitinib (2) 2
kidney - drug effects (2) 2
kidney - metabolism (2) 2
kidney diseases - chemically induced (2) 2
kidney diseases - metabolism (2) 2
nadph oxidase (2) 2
optical-properties (2) 2
phase epitaxy (2) 2
preeclampsia (2) 2
preeclampsia-like syndrome (2) 2
pyrroles - therapeutic use (2) 2
pyrroles - toxicity (2) 2
quantum dots (2) 2
rarefaction (2) 2
rat (2) 2
receptor (2) 2
receptors, vascular endothelial growth factor - antagonists & inhibitors (2) 2
receptors, vascular endothelial growth factor - metabolism (2) 2
renal-cell carcinoma (2) 2
research (2) 2
signal transduction - drug effects (2) 2
tyrosine kinase inhibitors (2) 2
vascular endothelial growth factor a (2) 2
vascular endothelial growth factor a - blood (2) 2
vegf (2) 2
71.35.–y (1) 1
78.47.+p (1) 1
78.67.de (1) 1
81.07.ta (1) 1
abridged index medicus (1) 1
absorption (1) 1
absorptionskante (1) 1
acute kidney injury - chemically induced (1) 1
acute kidney injury - drug therapy (1) 1
acute kidney injury - physiopathology (1) 1
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1. Full Text Hypertension Induced by the Tyrosine Kinase Inhibitor Sunitinib Is Associated With Increased Circulating Endothelin-1 Levels
by Kappers, Mariëtte H.W and van Esch, Joep H.M and Sluiter, Wim and Sleijfer, Stefan and Danser, A.H. Jan and van den Meiracker, Anton H
Hypertension, ISSN 0194-911X, 10/2010, Volume 56, Issue 4, pp. 675 - 681
Angiogenesis inhibition with sunitinib, a multitarget tyrosine kinase inhibitor of the vascular endothelial growth factor receptor, is associated with... 
endothelin | endothelial growth factors | experimental | hypertension | angiogenesis | MYOCARDIUM | HUMAN PLASMA | RATS | CARDIOTOXICITY | PROTEINURIA | MECHANISMS | PREECLAMPSIA | BLOOD-PRESSURE | PERIPHERAL VASCULAR DISEASE | GROWTH-FACTOR | Heart - physiology | Humans | Middle Aged | Rats, Inbred WKY | Male | Hypertension - blood | Heart Rate - drug effects | Hypertension - chemically induced | Pyrroles - adverse effects | Receptor Protein-Tyrosine Kinases - antagonists & inhibitors | Female | Indoles - pharmacology | Blood Pressure - drug effects | Carcinoma, Renal Cell - drug therapy | Pyrroles - therapeutic use | Bradykinin - pharmacology | Angiotensin II - pharmacology | Endothelial Cells - metabolism | Endothelin-1 - metabolism | Cells, Cultured | Vascular Endothelial Growth Factor A - blood | Rats | Renin - blood | Hypertension - physiopathology | Endothelin-1 - blood | Pyrroles - pharmacology | Animals | Indoles - adverse effects | Endothelial Cells - cytology | Gastrointestinal Stromal Tumors - drug therapy | Heart - drug effects | Indoles - therapeutic use | Aged | Coronary Circulation - drug effects | In Vitro Techniques | Kidney Neoplasms - drug therapy | Endothelial Cells - drug effects | Index Medicus
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2. Treatment options for resistant hypertension: from pseudoresistant to refractory hypertension
by Versmissen, Jorie and Hendriks, Kelly and van den Meiracker, Anton H and Kappers, Mariëtte H W
Nederlands tijdschrift voor geneeskunde, ISSN 0028-2162, 2015, Volume 160, p. A9937
Resistant hypertension is defined as blood pressure above the target level despite treatment with 3 classes of antihypertensive drugs, including a diuretic. A... 
Blood Pressure | Humans | Diet, Sodium-Restricted | Diuretics - therapeutic use | Hypertension - drug therapy | Hypertension - therapy | Antihypertensive Agents - therapeutic use | Blood Pressure Determination
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3. Full Text Treatment of hypertension and renal injury induced by the angiogenesis inhibitor sunitinib preclinical study
by Lankhorst, Stephanie and Kappers, Mariette and Esch, Joep and Smedts, Frank and Sleijfer, Stefan and Mathijssen, Ron and Baelde, H.J and Danser, Jan and Meiracker, Anton
Hypertension, ISSN 0194-911X, 01/2014, Volume 64, Issue 6, pp. 1282 - 1289
textabstractCommon adverse effects of angiogenesis inhibition are hypertension and renal injury. To determine the most optimal way to prevent these adverse... 
Hypertension | Sunitinib | Acute renal injury | Vascular endothelial growth factor A | Endothelin-1 | Therapeutics | sunitinib | acute renal injury | therapeutics | endothelin-1 | hypertension | vascular endothelial growth factor A | RECEPTOR BLOCKADE | FACTOR-A | SILDENAFIL | ATRASENTAN | PROTEINURIA | MECHANISMS | BLOOD-PRESSURE | NITRIC-OXIDE | TYROSINE KINASE INHIBITOR | PERIPHERAL VASCULAR DISEASE | ENDOTHELIAL GROWTH-FACTOR | Follow-Up Studies | Acute Kidney Injury - drug therapy | Rats | Rats, Inbred WKY | Hypertension - drug therapy | Indoles - toxicity | Treatment Outcome | Acute Kidney Injury - physiopathology | Hypertension - physiopathology | Amlodipine - therapeutic use | Glomerular Filtration Rate - drug effects | Animals | Angiogenesis Inhibitors - toxicity | Sulfonamides - therapeutic use | Pyrimidines - therapeutic use | Pyrroles - toxicity | Hypertension - chemically induced | Acute Kidney Injury - chemically induced | Blood Pressure - drug effects | Drug Therapy, Combination | Endothelin A Receptor Antagonists - therapeutic use | Disease Models, Animal
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4. Full Text Mechanism of hypertension and proteinuria during angiogenesis inhibition: Evolving role of endothelin-1
by Lankhorst, Stephanie and Kappers, Mariëtte H. W and Van Esch, Joep H. M and Danser, A. H. Jan and Van Den Meiracker, Anton H
Journal of Hypertension, ISSN 0263-6352, 2013, Volume 31, Issue 3, pp. 444 - 454
Angiogenesis inhibition by blocking vascular endothelial growth factor (VEGF)-mediated signalling with monoclonal antibodies or tyrosine kinase inhibitors has... 
Hypertension | Soluble vascular endothelial growth factor type 1 receptor | Nitric oxide | Endothelin-1 | Rarefaction | Vascular endothelial growth factor | Proteinuria | nitric oxide | proteinuria | NITRIC-OXIDE SYNTHASE | PREECLAMPSIA-LIKE SYNDROME | SKIN MICROVASCULAR DENSITY | THROMBOTIC MICROANGIOPATHY | RENAL-CELL CARCINOMA | BLOOD-PRESSURE | TYROSINE KINASE INHIBITORS | GROWTH-FACTOR VEGF | SIGNALING PATHWAY | soluble vascular endothelial growth factor type 1 receptor | vascular endothelial growth factor | SALT-SENSITIVE HYPERTENSION | PERIPHERAL VASCULAR DISEASE | endothelin-1 | rarefaction | hypertension | Receptors, Vascular Endothelial Growth Factor - physiology | Signal Transduction | Humans | Angiogenesis Inhibitors - pharmacology | Vascular Endothelial Growth Factor A - metabolism | Endothelin-1 - physiology | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Vascular Endothelial Growth Factor A - physiology | Hypertension - physiopathology | Proteinuria - physiopathology
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5. Full Text Sunitinib-Induced Systemic Vasoconstriction in Swine Is Endothelin Mediated and Does Not Involve Nitric Oxide or Oxidative Stress
by Kappers, Mariëtte H.W and de Beer, Vincent J and Zhou, Zhichao and Danser, A.H Jan and Sleijfer, Stefan and Duncker, Dirk J and van den Meiracker, Anton H and Merkus, Daphne
Hypertension, ISSN 0194-911X, 01/2012, Volume 59, Issue 1, pp. 151 - 157
Angiogenesis inhibition with agents targeting tyrosine kinases of vascular endothelial growth factor receptors is an established anticancer treatment, but is,... 
endothelin 1 | hypertension | pulmonary hypertension | vascular endothelial growth factor | oxidative stress | RAREFACTION | NADPH OXIDASE | CELLS | CORONARY | RECEPTOR | ANGIOGENESIS INHIBITION | BLOOD-PRESSURE | TREADMILL EXERCISE | PERIPHERAL VASCULAR DISEASE | EXPRESSION | Motor Activity - physiology | Reactive Oxygen Species - metabolism | Coronary Circulation - physiology | Endothelin-1 - metabolism | Oxidative Stress - physiology | Angiogenesis Inhibitors - pharmacology | Male | Hemodynamics - physiology | Vasoconstriction - drug effects | Pulmonary Circulation - physiology | Rest - physiology | Vasoconstriction - physiology | Pyrroles - pharmacology | Animals | Female | Indoles - pharmacology | Blood Pressure - drug effects | Blood Pressure - physiology | Hemodynamics - drug effects | Pulmonary Circulation - drug effects | Coronary Circulation - drug effects | Nitric Oxide - metabolism | Sus scrofa | Index Medicus
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6. Full Text Sunitinib-Induced Hypothyroidism Is due to Induction of Type 3 Deiodinase Activity and Thyroidal Capillary Regression
by Kappers, Mariëtte H. W and van Esch, Joep H. M and Smedts, Frank M. M and de Krijger, Ronald R and Eechoute, Karel and Mathijssen, Ron H. J and Sleijfer, Stefan and Leijten, Frank and Danser, A. H. Jan and van den Meiracker, Anton H and Visser, Theo J
The Journal of Clinical Endocrinology & Metabolism, ISSN 0021-972X, 10/2011, Volume 96, Issue 10, pp. 3087 - 3094
Context: Anticancer treatment with the tyrosine kinase inhibitor sunitinib causes thyroid dysfunction. Objective: Our objective was to investigate the time... 
TYROSINE KINASE INHIBITORS | MANAGEMENT | RAT | DISEASE | ENDOCRINOLOGY & METABOLISM | MECHANISMS | DYSFUNCTION | HYPOXIA | GASTROINTESTINAL STROMAL TUMORS | RENAL-CELL CARCINOMA | Hypothyroidism - chemically induced | Prospective Studies | Humans | Middle Aged | Body Weight - drug effects | Rats, Inbred WKY | Thyroxine - blood | Capillaries - drug effects | Male | Antineoplastic Agents - therapeutic use | Thyroid Gland - enzymology | Thyroid Gland - blood supply | Antineoplastic Agents - adverse effects | Pyrroles - adverse effects | Female | Hypothyroidism - enzymology | Retrospective Studies | Carcinoma, Renal Cell - drug therapy | Kidney Neoplasms - complications | Pyrroles - therapeutic use | Thyrotropin - blood | Stomach Neoplasms - complications | Rats | Stomach Neoplasms - drug therapy | Regional Blood Flow | Triiodothyronine - blood | Regression Analysis | Animals | Indoles - adverse effects | Gastrointestinal Stromal Tumors - drug therapy | Gastrointestinal Stromal Tumors - complications | Indoles - therapeutic use | Aged | Carcinoma, Renal Cell - complications | Kidney Neoplasms - drug therapy | Thyroid Gland - metabolism | Iodide Peroxidase - biosynthesis | Protein-Tyrosine Kinases - antagonists & inhibitors
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7. Full Text Greater Sensitivity of Blood Pressure Than Renal Toxicity to Tyrosine Kinase Receptor Inhibition with Sunitinib
by Lankhorst, Stephanie and Baelde, H.J and Kappers, Mariette and Smedts, Frank and Hansen, Alastair and Clahsen-van Groningen, Marian and Sleijfer, Stefan and Mathijssen, Ron and Danser, Jan and Meiracker, Anton
Hypertension, ISSN 0194-911X, 01/2015, Volume 66, Issue 3, pp. 543 - 549
textabstractHypertension and renal injury are off-target effects of sunitinib, a tyrosine kinase receptor inhibitor used for the treatment of various tumor... 
sunitinib | endothelin-1 | hypertension | vascular endothelial growth factor A | proteinuria | FACTOR VEGF | ACTIVATION | NEPHRIN | MECHANISMS | PODOCYTES | NITRIC-OXIDE | PERIPHERAL VASCULAR DISEASE | EXPRESSION | ENDOTHELIAL GROWTH-FACTOR | Kidney - drug effects | Podocytes - metabolism | Vascular Endothelial Growth Factor A - blood | Rats | Rats, Inbred WKY | Indoles - toxicity | Male | Antineoplastic Agents - toxicity | Dose-Response Relationship, Drug | Kidney - metabolism | Pyrroles - pharmacology | Animals | Podocytes - drug effects | Pyrroles - toxicity | Kidney Diseases - chemically induced | Protein Kinase Inhibitors - toxicity | Indoles - pharmacology | Antineoplastic Agents - pharmacology | Blood Pressure - drug effects | Protein Kinase Inhibitors - pharmacology | Kidney Diseases - metabolism | Index Medicus
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8. Cumulative dose of bevacizumab associates with albuminuria rather than podocyturia in cancer patients
by Lankhorst, Stephanie and Baelde, Hans J and Verstijnen, Jose A.M.C and ten Tije, Albert J and Thelen, Marc H.M and Danser, A.H. Jan and van den Meiracker, Anton H and Kappers, Mariëtte H.W
Journal of the American Society of Hypertension, ISSN 1933-1711, 11/2018, Volume 12, Issue 11, pp. e1 - e7
Angiogenesis inhibition with bevacizumab, a monoclonal antibody against vascular endothelial growth factor A (VEGF-A), is an anticancer treatment associated... 
angiogenesis inhibition | nephrinuria | Albuminuria | INHIBITION | THERAPY | VEGF | SUNITINIB | PROTEINURIA | PERIPHERAL VASCULAR DISEASE | PREECLAMPSIA-LIKE SYNDROME | HYPERTENSION
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9. New insights into the pathogenesis of pre-eclampsia: the role of angiogenesis-inhibiting factors
by Verdonk, Koen and Visser, Willy and Steegers, Eric A P and Kappers, Mariëtte H W and Danser, A H Jan and van den Meiracker, Anton H
Nederlands tijdschrift voor geneeskunde, ISSN 0028-2162, 2011, Volume 155, p. A2946
The pathogenesis of pre-eclampsia is biphasic. The first phase is characterised by insufficient placentation and the second phase by an increased placental... 
Antigens, CD - biosynthesis | Humans | Solubility | Vascular Endothelial Growth Factor Receptor-1 - biosynthesis | Pre-Eclampsia - enzymology | Placenta - metabolism | Pregnancy | Endoglin | Angiogenesis Inducing Agents - antagonists & inhibitors | Female | Pre-Eclampsia - metabolism | Receptors, Cell Surface - biosynthesis | Endothelium, Vascular
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10. Nieuwe inzichten in pathogenese van pre-eclampsie. De rol van angiogeneseremmende factoren
by Verdonk, Koen and Visser, Willy and Steegers, Eric A P and Kappers, Mariëtte H W and Danser, A H Jan and van den Meiracker, Anton H
Nederlands tijdschrift voor geneeskunde, 2011, Volume 155, p. A2946
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11. Full Text Cardiovascular and renal toxicity during angiogenesis inhibition: clinical and mechanistic aspects
by Kappers, Mariëtte HW and van Esch, Joep HM and Sleijfer, Stefan and Danser, AH Jan and van den Meiracker, Anton H
Journal of Hypertension, ISSN 0263-6352, 12/2009, Volume 27, Issue 12, pp. 2297 - 2309
Inhibition of angiogenesis with humanized monoclonal antibodies to vascular endothelial growth factor (VEGF) or with tyrosine kinase inhibitors targeting VEGF... 
Hypertension | Vvascular endothelial growth factor | Renal disease | Angiogenesis inhibition | Cardiac disease | Proteinuria | SUNITINIB TREATMENT | DEPENDENT RELAXATION | proteinuria | COHERENCE TOMOGRAPHY FINDINGS | cardiac disease | BEVACIZUMAB AVASTIN(R) | TYROSINE-KINASE INHIBITOR | renal disease | BLOOD-PRESSURE | INTRAVITREAL INJECTION | angiogenesis inhibition | PHASE-I TRIAL | vascular endothelial growth factor | PERIPHERAL VASCULAR DISEASE | hypertension | ENDOTHELIAL GROWTH-FACTOR | MOLECULAR-MECHANISMS | Kidney Diseases - physiopathology | Humans | Antibodies, Monoclonal - adverse effects | Endothelium, Vascular - drug effects | Vascular Endothelial Growth Factor A - metabolism | Protein Kinase Inhibitors - adverse effects | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Vascular Endothelial Growth Factor A - genetics | Hypertension - physiopathology | Hypertension - metabolism | Gene Expression Regulation - drug effects | Receptors, Vascular Endothelial Growth Factor - metabolism | Signal Transduction - drug effects | Endothelium, Vascular - metabolism | Hypertension - chemically induced | Kidney Diseases - chemically induced | Receptors, Vascular Endothelial Growth Factor - antagonists & inhibitors | Angiogenesis Inhibitors - adverse effects | Kidney Diseases - metabolism | Index Medicus
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12. Full Text Hypertension During Vascular Endothelial Growth Factor Inhibition: Focus on Nitric Oxide, Endothelin-1, and Oxidative Stress
by Lankhorst, Stephanie and Kappers, Mariëtte H.W and van Esch, Joep H.M and Danser, A.H. Jan and van den Meiracker, Anton H
Antioxidants & Redox Signaling, ISSN 1523-0864, 01/2014, Volume 20, Issue 1, pp. 135 - 145
Significance: Angiogenesis inhibition with humanized antibodies targeting vascular endothelial growth factor (VEGF) or orally active small tyrosine kinase... 
Forum Review Articles | NADPH OXIDASE | SYNTHASE EXPRESSION | BEVACIZUMAB | VEGF | BIOCHEMISTRY & MOLECULAR BIOLOGY | ARTERIAL-HYPERTENSION | ENDOCRINOLOGY & METABOLISM | RECEPTOR | CELL-PROLIFERATION | MECHANISMS | TUMOR ANGIOGENESIS | BLOOD-PRESSURE | Animals | Receptors, Vascular Endothelial Growth Factor - metabolism | Oxidative Stress | Signal Transduction - drug effects | Endothelin-1 - metabolism | Humans | Receptors, Vascular Endothelial Growth Factor - antagonists & inhibitors | Hypertension - drug therapy | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Nitric Oxide - metabolism | Hypertension - metabolism | Hypertension | Complications and side effects | Development and progression | Angiogenesis inhibitors | Vascular endothelial growth factor | Health aspects
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13. Full Text The Vascular Endothelial Growth Factor Receptor Inhibitor Sunitinib Causes a Preeclampsia-Like Syndrome With Activation of the Endothelin System
by Kappers, Mariëtte H.W and Smedts, Frank M.M and Horn, Thomas and van Esch, Joep H.M and Sleijfer, Stefan and Leijten, Frank and Wesseling, Sebastiaan and Strevens, Helena and Jan Danser, A.H and van den Meiracker, Anton H and Department of Obstetrics and Gynaecology (Lund) and Lund University and Obstetrik och gynekologi, Lund and Lunds universitet
Hypertension, ISSN 0194-911X, 08/2011, Volume 58, Issue 2, pp. 295 - 302
Angiogenesis inhibition is an established treatment for several tumor types. Unfortunately, this therapy is associated with adverse effects, including... 
endothelin 1 | renal toxicity | Angiogenesis inhibition | preeclampsia | OXIDATIVE STRESS | INJURY | PROTEINURIA | HOMOZYGOUS REN-2 RATS | BLOOD-PRESSURE | angiogenesis inhibition | PREGNANT RATS | PERIPHERAL VASCULAR DISEASE | CHRONIC KIDNEY-DISEASE | HYPERTENSION | BLOCKADE | EXPRESSION | Rats, Inbred SHR | Vascular Endothelial Growth Factor Receptor-1 - antagonists & inhibitors | Kidney - drug effects | Kidney - pathology | Endothelin-1 - metabolism | Angiogenesis Inhibitors - pharmacology | Rats | Rats, Inbred WKY | Male | Hypertension - pathology | Hypertension - metabolism | Dose-Response Relationship, Drug | Kidney - metabolism | Pyrroles - pharmacology | Animals | Hypertension - chemically induced | Indoles - pharmacology | Blood Pressure - drug effects | Oxidative Stress - drug effects | Nitric Oxide - metabolism | Index Medicus | Kardiologi | Clinical Medicine | Cardiac and Cardiovascular Systems | Medical and Health Sciences | Klinisk medicin | Medicin och hälsovetenskap
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14. New insights into the pathogenesis of preeclampsia: The role of angiogenesis-inhibiting factors
by Verdonk, Koen and Visser, Willy and Steegers, Eric A. P and Kappers, Mariëtte H. W and Danser, A. H. Jan and Van Den Meiracker, Anton H
Nederlands Tijdschrift voor Geneeskunde, ISSN 0028-2162, 04/2011, Volume 155, Issue 16, pp. 742 - 747
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15. Full Text Community-acquired necrotizing fasciitis due to Pseudomonas aeruginosa
by Kappers, Mariëtte H. W and van der Klooster, Johan M and Ouwendijk, Rob J. Th and Dees, Ad
Intensive Care Medicine, ISSN 0342-4642, 7/2006, Volume 32, Issue 7, pp. 1093 - 1094
Pediatrics | Pain Medicine | Pneumology/Respiratory System | Emergency Medicine | Medicine & Public Health | Necrotizing fasciitis | Intensive / Critical Care Medicine | Pseudomonas aeruginosa | Anesthesiology | immunocompromised host | Immunocompromised host | necrotizing fasciitis | CRITICAL CARE MEDICINE | Community-Acquired Infections - diagnosis | Diagnosis, Differential | Pseudomonas aeruginosa - isolation & purification | Humans | Middle Aged | Male | Pseudomonas Infections - diagnosis | Pseudomonas Infections - microbiology | Pseudomonas aeruginosa - pathogenicity | Fasciitis, Necrotizing - microbiology | Fatal Outcome | Fasciitis, Necrotizing - diagnosis | Community-Acquired Infections - microbiology | Causes of | Pseudomonas aeruginosa infections | Complications and side effects
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16. Full Text Light-induced vs. bradykinin-induced relaxation of coronary arteries: Do S-nitrosothiols act as endothelium-derived hyperpolarizing factors?
by Batenburg, Wendy W and Kappers, Mariette H.W and Eikmann, Melissa J and Ramzan, Serge N.A and De Vries, René and Danser, Ah Jan
Journal of Hypertension, ISSN 0263-6352, 08/2009, Volume 27, Issue 8, pp. 1631 - 1640
Background Light-induced relaxation depends on S-nitrosothiols. S-Nitrosothiols may also serve as endothelium-derived hyperpolarizing factors, mediating the... 
Nitric oxide synthase | Photorelaxation | S-nitrosothiols | Bradykinin | Endothelium-derived hyperpolarizing factor | BKCA CHANNELS | ACTIVATION | RAT | photorelaxation | INTERNAL MAMMARY ARTERY | bradykinin | ESSENTIAL HYPERTENSIVE PATIENTS | endothelium-derived hyperpolarizing factor | VASCULAR SMOOTH-MUSCLE | RECOGNITION SITES | NITRIC-OXIDE | PERIPHERAL VASCULAR DISEASE | nitric oxide synthase | EPOXYEICOSATRIENOIC ACIDS | NG-Nitroarginine Methyl Ester - pharmacology | Potassium Channels, Calcium-Activated - physiology | Sodium-Potassium-Exchanging ATPase - physiology | Coronary Vessels - physiology | Biological Factors - physiology | Animals | Swine | Light | Nitric Oxide Synthase Type III - physiology | Pyridines - pharmacology | S-Nitrosothiols | Vasodilation - drug effects | Cyclic GMP - biosynthesis | Bradykinin - pharmacology | Pyrazoles - pharmacology
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17. Full Text Re: Hypertension as a Biomarker of Efficacy in Patients With Metastatic Renal Cell Carcinoma Treated With Sunitinib
by van den Meiracker, Anton H and Danser, A. H. Jan and Sleijfer, Stefan and Kappers, Mariette H. W
Journal of the National Cancer Institute, ISSN 0027-8874, 10/2011, Volume 103, Issue 20, pp. 1557 - 1557
DENSITY | ONCOLOGY | INHIBITOR SUNITINIB | Indoles - adverse effects | Antineoplastic Agents - adverse effects | Humans | Hypertension - chemically induced | Pyrroles - adverse effects | Male | Blood Pressure - drug effects | Carcinoma, Renal Cell - drug therapy | Kidney Neoplasms - drug therapy
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18. Hypertension induced by antiangiogenic therapy: Clinical and pathophysiological aspects
by van den Meiracker, Anton H and Lankhorst, Stephanie and van Esch, Joep H.M and Jan Danser, A.H and Kappers, Mariëtte H.W
European Journal of Hospital Pharmacy, ISSN 2047-9956, 2012, Volume 19, Issue 3, pp. 327 - 329
Angiogenesis inhibition with humanised monoclonal antibodies to vascular endothelial growth factor (VEGF) or with VEGF receptor tyrosine kinase inhibitors... 
ANGIOGENESIS | BIOMARKER | NITRIC-OXIDE | RESISTANCE | PHARMACOLOGY & PHARMACY | MECHANISMS | INHIBITOR SUNITINIB | ENDOTHELIN-1 | CANCER
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19. Full Text Dynamics of single InGaN quantum dots
by Taylor, R.A and Robinson, J.W and Rice, J.H and Jarjour, A and Smith, J.D and Oliver, R.A and Briggs, G.A.D and Kappers, M.J and Humphreys, C.J and Arakawa, Y
Physica E: Low-dimensional Systems and Nanostructures, ISSN 1386-9477, 2004, Volume 21, Issue 2, pp. 285 - 289
Decay dynamics for single InGaN quantum dots are presented using time-resolved photoluminescence. The recombination is shown to be characterized by a single... 
InGaN | Single quantum dots | Time-resolved luminescence | time-resolved luminescence | PHYSICS, CONDENSED MATTER | single quantum dots | PHOTOLUMINESCENCE
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20. Full Text InGaN quantum dots grown by MOVPE via a droplet epitaxy route
by Rice, J.H and Oliver, R.A and Robinson, J.W and Smith, J.D and Taylor, R.A and Briggs, G.A.D and Kappers, M.J and Humphreys, C.J and Yasin, S
Physica E: Low-dimensional Systems and Nanostructures, ISSN 1386-9477, 2004, Volume 21, Issue 2, pp. 546 - 550
A novel method for the growth of InGaN quantum dots (QDs) by metal-organic vapour phase epitaxy (MOVPE) is described. A thin InGaN epilayer is grown on a GaN... 
MOVPE growth | InGaN photoluminescence | Quantum dots | quantum dots | PHYSICS, CONDENSED MATTER | THRESHOLD | PHOTOLUMINESCENCE | GAN
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