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1. Full Text Novel genetic loci associated with hippocampal volume
by Hibar, Derrek P and Adams, Hieab H. H and Jahanshad, Neda and Chauhan, Ganesh and Stein, Jason L and Hofer, Edith and Renteria, Miguel E and Bis, Joshua C and Arias-Vasquez, Alejano and Ikram, M. Kamran and Desrivières, Sylvane and Vernooij, Meike W and Abramovic, Lucija and Alhusaini, Saud and Amin, Najaf and Andersson, Micael and Arfanakis, Konstantinos and Aribisala, Benjamin S and Armstrong, Nicola J and Athanasiu, Lavinia and Axelsson, Tomas and Beecham, Ashley H and Beiser, Alexa and Bernard, Manon and Blanton, Susan H and Bohlken, Marc M and Boks, Marco P and Bralten, Janita and Brickman, Adam M and Carmichael, Owen and Chakravarty, M. Mallar and Chen, Qiang and Ching, Christopher R. K and Chouraki, Vincent and Cuellar-Partida, Gabriel and Crivello, Fabrice and den Braber, Anouk and Doan, Nhat Trung and Ehrlich, Stefan and Giddaluru, Sudheer and Goldman, Aaron L and Gottesman, Rebecca F and Grimm, Oliver and Griswold, Michael E and Guadalupe, Tulio and Gutman, Boris A and Hass, Johanna and Haukvik, Unn K and Hoehn, David and Holmes, Avram J and Hoogman, Martine and Janowitz, Deborah and Jia, Tianye and Jørgensen, Kjetil N and Karbalai, Nazanin and Kasperaviciute, Dalia and Kim, Sungeun and Klein, Marieke and Kraemer, Bernd and Lee, Phil H and Liewald, David C. M and Lopez, Lorna M and Luciano, Michelle and Macare, Christine and Marquand, Ane F and Matarin, Mar and Mather, Karen A and Mattheisen, Manuel and McKay, David R and Milaneschi, Yuri and Muñoz Maniega, Susana and Nho, Kwangsik and Nugent, Allison C and Nyquist, Paul and Loohuis, Loes M. Olde and Oosterlaan, Jaap and Papmeyer, Martina and Pirpamer, Lukas and Pütz, Benno and Ramasamy, Adaikalavan and Richards, Jennifer S and Risacher, Shannon L and Roiz-Santiañez, Roberto and Rommelse, Nanda and Ropele, Stefan and Rose, Emma J and Royle, Natalie A and Rundek, Tatjana and Sämann, Philipp G and Saremi, Arvin and Satizabal, Claudia L and Schmaal, Lianne and Schork, Anew J and Shen, Li and Shin, Jean and Shumskaya, Elena and Smith, Albert V and Sprooten, Emma and Strike, Lachlan T and Teumer, Alexander and ... and Medicinska fakulteten and Institutionen för integrativ medicinsk biologi (IMB) and Umeå centrum för funktionell hjärnavbildning (UFBI) and Umeå universitet
Nature communications, ISSN 2041-1723, 2017, Volume 8, Issue 1, p. 13624
The hippocampal formation is a brain structure integrally involved in episodic memory, spatial navigation, cognition and stress responsiveness. Structural... 
BRAIN-REGIONS | COMMON VARIANTS | METAANALYSIS | TEMPORAL-LOBE EPILEPSY | MEMORY | ALZHEIMERS-DISEASE | MULTIDISCIPLINARY SCIENCES | SUSCEPTIBILITY LOCI | BIPOLAR DISORDER | SUBFIELDS | GENOME-WIDE ASSOCIATION | Glycoproteins - genetics | Alzheimer Disease - physiopathology | Genetic Predisposition to Disease | Genome-Wide Association Study | Microtubule-Associated Proteins - genetics | Humans | Middle Aged | Protein-Serine-Threonine Kinases - genetics | Organ Size | Dipeptidyl Peptidase 4 - genetics | Male | Genetic Loci | Methionine Sulfoxide Reductases - genetics | Nerve Tissue Proteins - genetics | Young Adult | Adolescent | Aged, 80 and over | Adult | Female | Aged | Alzheimer Disease - genetics | Child | Hippocampus - growth & development | Cohort Studies | Neuroscience | Cognitive science | Basic Medicine | Medical Genetics | Medicinsk genetik | Biological Sciences | Naturvetenskap | Medical and Health Sciences | Medicin och hälsovetenskap | Biologiska vetenskaper | Medicinska och farmaceutiska grundvetenskaper | Natural Sciences | Bioinformatik och systembiologi | Bioinformatics and Systems Biology
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2. Full Text Genetic Differences in the Immediate Transcriptome Response to Stress Predict Risk-Related Brain Function and Psychiatric Disorders
by Arloth, Janine and Bogdan, Ryan and Weber, Peter and Frishman, Goar and Menke, Andreas and Wagner, Klaus V and Balsevich, Georgia and Schmidt, Mathias V and Karbalai, Nazanin and Czamara, Darina and Altmann, Andre and Trümbach, Dietrich and Wurst, Wolfgang and Mehta, Divya and Uhr, Manfred and Klengel, Torsten and Erhardt, Angelika and Carey, Caitlin E and Conley, Emily Drabant and Ripke, Stephan and Wray, Naomi R and Lewis, Cathryn M and Lewis, Glyn and Hamilton, Steven P and Weissman, Myrna M and Breen, Gerome and Byrne, Enda M and Blackwood, Douglas H.R and Boomsma, Dorret I and Cichon, Sven and Heath, Andrew C and Holsboer, Florian and Lucae, Susanne and Madden, Pamela A.F and Martin, Nicholas G and McGuffin, Peter and Muglia, Pierandrea and Noethen, Markus M and Penninx, Brenda P and Pergadia, Michele L and Potash, James B and Rietschel, Marcella and Lin, Danyu and Müller-Myhsok, Bertram and Shi, Jianxin and Steinberg, Stacy and Grabe, Hans J and Lichtenstein, Paul and Magnusson, Patrik and Perlis, Roy H and Preisig, Martin and Smoller, Jordan W and Stefansson, Hreinn and Stefansson, Kari and Uher, Rudolf and Kutalik, Zoltan and Tansey, Katherine E and Teumer, Alexander and Viktorin, Alexander and Barnes, Michael R and Bettecken, Thomas and Binder, Elisabeth B and Binder, Elisabeth B and Breuer, René and Castro, Victor M and Churchill, Susanne E and Coryell, William H and Craddock, Nick and Craig, Ian W and De Geus, Eco J and Degenhardt, Franziska and Farmer, Anne E and Fava, Maurizio and Frank, Josef and Gainer, Vivian S and Gallagher, Patience J and Gordon, Scott D and Goryachev, Sergey and Gross, Magdalena and Guipponi, Michel and Henders, Anjali K and Herms, Stefan and Hickie, Ian B and Hoefels, Susanne and Hoogendijk, Witte and Hottenga, Jouke Jan and Iosifescu, Dan V and Ising, Marcus and Jones, Lisa and Jones, Ian and Jung-Ying, Tzeng and Knowles, James A and Kohane, Isaac S and Kohli, Martin A and Korszun, Ania and Landen, Mikael and Lawson, William B and MacIntyre, Donald and Maier, Wolfgang and Mattheisen, Manuel and ... and Psychiat Genomics Consortium PGC and Major Depressive Disorder Working Group of the Psychiatric Genomics Consortium PGC and Major Depressive Disorder Working Group of the Psychiatric Genomics Consortium (PGC)
Neuron, ISSN 0896-6273, 06/2015, Volume 86, Issue 5, pp. 1189 - 1202
Depression risk is exacerbated by genetic factors and stress exposure; however, the biological mechanisms through which these factors interact to confer... 
LIFE EVENTS | EXPRESSION | NEUROSCIENCES | Gene Regulatory Networks - genetics | Humans | Mice, Inbred C57BL | Risk Factors | Male | Transcriptome - genetics | Brain - physiology | Forecasting | Stress, Psychological - diagnosis | Mental Disorders - genetics | Stress, Psychological - genetics | Animals | Polymorphism, Single Nucleotide - genetics | Genetic Variation - genetics | Mice | Mental Disorders - diagnosis | Cohort Studies | Animal experimentation | Medicine, Experimental | Brain | Medical research | Corticosteroids | Depression, Mental | Neurosciences | Neural circuitry | Genomics | Genetic research | Single nucleotide polymorphisms | Genetic transcription | Attention deficit disorder | Working groups | Gene expression | Risk factors | Binding sites
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3. Full Text Solid organ transplantation in patients with inflammatory bowel diseases (IBD): Analysis of transplantation outcome and IBD activity in a large single center cohort
by Schnitzler, Fabian and Friedrich, Matthias and Stallhofer, Johannes and Schönermarck, Ulf and Fischereder, Michael and Habicht, Antje and Karbalai, Nazanin and Wolf, Christiane and Angelberger, Marianne and Olszak, Torsten and Beigel, Florian and Tillack, Cornelia and Göke, Burkhard and Zachoval, Reinhart and Denk, Gerald and Guba, Markus and Rust, Christian and Grüner, Norbert and Brand, Stephan
PLoS ONE, ISSN 1932-6203, 08/2015, Volume 10, Issue 8, p. e0135807
Background Currently, limited data of the outcome of inflammatory bowel disease (IBD) in patients after solid organ transplantation (SOT) are available. We... 
HEMOLYTIC-UREMIC SYNDROME | RISK-FACTORS | CROHNS-DISEASE | KIDNEY-TRANSPLANTATION | ORTHOTOPIC LIVER-TRANSPLANTATION | IMMUNOSUPPRESSION | MULTIDISCIPLINARY SCIENCES | ULCERATIVE-COLITIS | CASE SERIES | CLINICAL-COURSE | PRIMARY SCLEROSING CHOLANGITIS | Liver Transplantation - adverse effects | Humans | Immunosuppressive Agents - therapeutic use | Middle Aged | Heart Transplantation - adverse effects | Male | Survival Rate | Treatment Outcome | Colitis, Ulcerative - pathology | Tacrolimus - therapeutic use | Immunosuppression | Crohn Disease - pathology | Crohn Disease - drug therapy | Adult | Female | Aged | Colitis, Ulcerative - drug therapy | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Kidney Transplantation - adverse effects | Heart | Care and treatment | Immunotherapy | Analysis | Patient outcomes | Liver | Transplantation | Liver cirrhosis | Ulcerative colitis | Therapy | Inflammatory bowel diseases | Nephrology | Transplants & implants | Syngeneic grafts | Clinical trials | Immunosuppressive agents | Intestine | Hepatology | Gastroenterology | Remission | Tumor necrosis factor-TNF | Liver diseases | Heart transplantation | Cholangitis | Disease control | Patients | Crohns disease | Inflammatory bowel disease | Heart rate | Medicine | Studies | Cirrhosis | Tacrolimus | Tumor necrosis factor | Surgeons | Liver transplantation | Kidney transplantation
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4. Full Text PTPN2 gene variants are associated with susceptibility to both Crohn's disease and ulcerative colitis supporting a common genetic disease background
by Glas, Jürgen and Wagner, Johanna and Seiderer, Julia and Olszak, Torsten and Wetzke, Martin and Beigel, Florian and Tillack, Cornelia and Stallhofer, Johannes and Friedrich, Matthias and Steib, Christian and Göke, Burkhard and Ochsenkühn, Thomas and Karbalai, Nazanin and Diegelmann, Julia and Czamara, Darina and Brand, Stephan
PLoS ONE, ISSN 1932-6203, 03/2012, Volume 7, Issue 3, p. e33682
Background: Genome-wide association studies identified PTPN2 (protein tyrosine phosphatase, non-receptor type 2) as susceptibility gene for inflammatory bowel... 
INSULIN-RECEPTOR | INFLAMMATORY-BOWEL-DISEASE | ATG16L1 | IL23R | MULTIDISCIPLINARY SCIENCES | LOCI | CARD15 | IDENTIFICATION | PROTEIN-TYROSINE-PHOSPHATASE | GENOME-WIDE ASSOCIATION | CELIAC-DISEASE | Tyrosine | Phosphatases | Analysis | Gastrointestinal diseases | Genetic aspects | Disease susceptibility | DNA binding proteins | Colitis | Inflammatory bowel diseases | Transcription factors | Epithelial cells | Epistasis | Genomes | Single-nucleotide polymorphism | CCAAT/enhancer-binding protein | Kinases | Phosphatase | Autophagy | Small intestine | Proteins | Genotype & phenotype | Intestine | Trends | Deoxyribonucleic acid--DNA | NOD2 protein | NF-κB protein | Cytokines | Crohn's disease | Dentistry | Gene expression | Patients | Crohns disease | Inflammatory bowel disease | Disease prevention | Medicine | Studies | Celiac disease | GATA-3 protein | Gene loci | Diabetes | Binding sites | Ulcerative colitis | Protein-tyrosine-phosphatase | PTPN2 protein | Deoxyribonucleic acid | DNA
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5. Full Text A Genome-Wide Association Study Suggests Novel Loci Associated with a Schizophrenia-Related Brain-Based Phenotype
by Hass, Johanna and Walton, Esther and Kirsten, Holger and Liu, Jingyu and Priebe, Lutz and Wolf, Christiane and Karbalai, Nazanin and Gollub, Randy and White, Tonya and Roessner, Veit and Müller, Kathrin U and Paus, Tomas and Smolka, Michael N and Schumann, Gunter and Scholz, Markus and Cichon, Sven and Calhoun, Vince and Ehrlich, Stefan and IMAGEN Consortium
PLoS ONE, ISSN 1932-6203, 06/2013, Volume 8, Issue 6, p. e64872
Patients with schizophrenia and their siblings typically show subtle changes of brain structures, such as a reduction of hippocampal volume. Hippocampal volume... 
HUMAN CEREBRAL-CORTEX | GENE | DATA QUALITY-CONTROL | HIPPOCAMPAL ATROPHY | ALZHEIMERS-DISEASE | MULTIDISCIPLINARY SCIENCES | QUANTITATIVE TRAIT | STRUCTURAL MRI | PSYCHOTIC DISORDERS | VERBAL MEMORY | BIPOLAR DISORDER | Genome-Wide Association Study | Schizophrenia - metabolism | Humans | Gene Expression Regulation | Male | Gene Expression Profiling | Genetic Loci | Schizophrenia - diagnostic imaging | Hippocampus - diagnostic imaging | Hippocampus - metabolism | Schizophrenia - genetics | Adolescent | Female | Polymorphism, Single Nucleotide | Brain | Nervous system diseases | Genetic markers | Genomics | Genes | Schizophrenia | Genetic aspects | Genomes | Single nucleotide polymorphisms | Gene expression | Chromosome 19 | Neuroimaging | Adolescence | Mental disorders | Pathogenesis | Memory | Cognitive ability | Single-nucleotide polymorphism | Epidemiology | Consortia | Genotype & phenotype | Genetics | Neuropsychology | Adolescents | Siblings | Brain architecture | Medical imaging | Computer engineering | Interdisciplinary aspects | Chromosome 1 | Studies | Brain research | Hospitals | Alzheimers disease | Health informatics | Hippocampus | Child & adolescent psychiatry
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6. Full Text The challenges of genome-wide interaction studies: Lessons to learn from the analysis of HDL blood levels
by Leeuwen, Elisa and Smouter, Françoise A.S and Kam-Thong, Tony and Karbalai, Nazanin and Smith, G.D and Harris, Tamara and Launer, Lenore and Sitlani, Colleen and Li, Guo and Brody, Jennifer and Bis, Joshua and White, Charles and Jaiswal, Alok and Oostra, Ben and Hofman, Albert and Rivadeneira Ramirez, Fernando and Uitterlinden, Ané and Boerwinkle, Eric and Ballantyne, Christie and Gudnason, Vilmundur and Psaty, Bruce and Cupples, Aienne and Jarvelin, Marjo-Riitta and Ripatti, Samuli and Isaacs, Aaron and Müller-Myhsok, B and Karssen, Lennart and Duijn, Cock
PLoS ONE, ISSN 1932-6203, 10/2014, Volume 9, Issue 10, p. e109290
textabstractGenome-wide association studies (GWAS) have revealed 74 single nucleotide polymorphisms (SNPs) associated with high-density lipoprotein cholesterol... 
RISK | LOCI | DNA METHYLATION | CORONARY-HEART-DISEASE | MULTIDISCIPLINARY SCIENCES | Genome-Wide Association Study | Humans | Middle Aged | Cholesterol, HDL - blood | Female | Male | Polymorphism, Single Nucleotide | Genomes | Single nucleotide polymorphisms | Chromosomes | Analysis | Genomics | Cholesterol | Biometrics | Heart | Demography | Laboratories | Graphics processing units | Lipids | Cardiovascular disease | Single-nucleotide polymorphism | Epidemiology | Blood | Research methodology | DNA methylation | Lipoproteins (high density) | Public health | Deoxyribonucleic acid--DNA | Medical research | Departments | Filtration | Internal medicine | Regression analysis | Blood levels | Medicine | Studies | Gene loci | Software | Psychiatry | Deoxyribonucleic acid | DNA
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7. Full Text GLIDE: GPU-Based Linear Regression for Detection of Epistasis
by Kam-Thong, Tony and Azencott, Chloé-Agathe and Cayton, Lawrence and Pütz, Benno and Altmann, André and Karbalai, Nazanin and Sämann, Philipp G and Schölkopf, Bernhard and Müller-Myhsok, Bertram and Borgwardt, Karsten M
Human Heredity, ISSN 0001-5652, 09/2012, Volume 73, Issue 4, pp. 220 - 236
Due to recent advances in genotyping technologies, mapping phenotypes to single loci in the genome has become a standard technique in statistical genetics.... 
Original Paper | Epistasis | Genome-wide interaction analysis | Graphics processing units | Computational biology | SUSCEPTIBILITY | HIPPOCAMPAL | LOCI | MAJOR DEPRESSION | IDENTIFICATION | GENES | GENETICS & HEREDITY | DIVERSITY | COMPLEX DISEASES | ASSOCIATION | EXPRESSION | Computational Biology - methods | Genetic Predisposition to Disease | Genome-Wide Association Study | Reproducibility of Results | Humans | Organ Size | Linear Models | Genetic Loci | Bipolar Disorder - diagnosis | Bipolar Disorder - genetics | Phenotype | Genetics, Population - methods | Time Factors | Epistasis, Genetic | Chromosome Mapping - methods | Hippocampus - anatomy & histology | Polymorphism, Single Nucleotide | Bipolar Disorder - epidemiology | Databases, Factual | Genotype & phenotype | Gene loci | Heredity | Disease | Genomics
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8. Full Text Restless Legs Syndrome-Associated intronic common variant in Meis1 alters enhancer function in the developing telencephalon
by Spieler, Derek and Kaffe, Maria and Knauf, Franziska and Bessa, José and Tena, Juan J and Giesert, Florian and Schormair, Barbara and Tilch, Erik and Lee, Heekyoung and Horsch, Marion and Czamara, Darina and Karbalai, Nazanin and Toerne, Christine Von and Waldenberger, Melanie and Gieger, Christian and Lichtner, Peter and Claussnitzer, Melina and Naumann, Ronald and Müller-Myhsok, Bertram and Torres, Miguel and Garrett, Lillian and Rozman, Jan and Klingenspor, Martin and Gailus-Durner, Valérie and Fuchs, Helmut and De Angelis, Martin Hrabě and Beckers, Johannes and Hölter, Sabine M and Meitinger, Thomas and Hauck, Stefanie M and Laumen, Helmut and Wurst, Wolfgang and Casares, Fernando and Gómez-Skarmeta, Jose Luis and Winkelmann, Juliane
Genome Research, ISSN 1088-9051, 2014, Volume 24, Issue 4, pp. 592 - 603
Genome-wide association studies (GWAS) identified the MEIS1 locus for Restless Legs Syndrome (RLS), but causal single nucleotide polymorphisms (SNPs) and their... 
VISUALIZATION | TRANSCRIPTION FACTOR-BINDING | NONCODING SEQUENCES | BIOCHEMISTRY & MOLECULAR BIOLOGY | LABEL-FREE | CONSERVATION | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | MOTOR RESTLESSNESS | DNA | DISEASE | GENETICS & HEREDITY | CHROMATIN INTERACTIONS | GENOME-WIDE ASSOCIATION | Genome-Wide Association Study | Introns | Basal Ganglia - metabolism | Basal Ganglia - pathology | Homeodomain Proteins - genetics | Restless Legs Syndrome - genetics | Animals | Enhancer Elements, Genetic | Telencephalon - growth & development | Telencephalon - pathology | Alleles | Mice | Polymorphism, Single Nucleotide | Myeloid Ecotropic Viral Integration Site 1 Protein | Neoplasm Proteins - genetics | Disease Models, Animal | Restless legs syndrome | Genetic variation | Telencephalon | Physiological aspects | Genetic aspects | Research | Risk factors
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9. Full Text GWAS for executive function and processing speed suggests involvement of the CADM2 gene
by Ibrahim-Verbaas, Carla and Bressler, Jan and Debette, Stéphanie and Schuur, Maaike and Smith, A.V and Bis, Joshua and Davies, Gail and Trompet, Stella and Smith, J.A and Björnsson, Asgeir and Chibnik, Lori and Liu, Y and Vitart, Veronique and Kirin, Mirna and Petrovic, Katja and Polasek, Ozren and Zgaga, Lina and Fawns-Ritchie, C and Hoffmann, Per and Karjalainen, Juha and Lahti, J and Llewellyn, D.J and Schmidt, Carsten O and Mather, R and Chouraki, Vincent and Sun, Q and Resnick, S.M and Rose, Lynda and Oldmeadow, Christopher and Stewart, M and Smith, B.H and Gudnason, Vilmundur and Yang, Qiong and Mirza, Saira and Jukema, J.W and DeJager, Philip L and Harris, Tamara and Liewald, David C and Amin, Najaf and Coker, Laura and Stegle, Oliver and Lopez, O.L and Schmidt, R and Teumer, Alexander and Ford, I and Karbalai, Nazanin and Becker, James and Jonsdottir, Maria K and Au, Rhoda and Fehrmann, Rudolf and Herms, Stefan and Nalls, Michael and Zhao, W and Turner, Stephen and Yaffe, K and Lohman, Kurt and Swieten, John and Kardia, Sharon L.r and Knopman, David and Meeks, William and Heiss, Gerardo and Holliday, Elizabeth and Schofield, P.W and Tanaka, Toshiko and Stott, David J and Wang, Jing and Ridker, Paul and Gow, A.J and Pattie, Alison and Starr, John and Hocking, L.J and Armstrong, Nicola and McLachlan, Stela and Shulman, J.M and Pilling, Luke and Eiriksdottir, Gudny and Scott, Rodney and Kochan, Nicole A and Palotie, Aarno and Hsieh, Yi-Chen and Eriksson, Johan G and Penman, Alan and Gottesman, Rebecca and Oostra, Ben and Yu, Lei and DeStefano, Anita and Beiser, Alexa and Garcia, M and Rotter, Jerome I and Nöthen, Markus M and Hofman, Albert and Slagboom, Eline and Westendorp, R.G.J and Buckley, Brendan M and Wolf, P.A and Uitterlinden, Ané and Psaty, Bruce and Grabe, Hans Jörgen and Bandinelli, Stefania and Chasman, Daniel and ... and Generation Scotland and on behalf of Generation Scotland
Molecular Psychiatry, ISSN 1359-4184, 02/2016, Volume 21, Issue 2, pp. 189 - 197
textabstractTo identify common variants contributing to normal variation in two specific domains of cognitive functioning, we conducted a genome-wide... 
COMMON VARIANTS | METAANALYSIS | PSYCHIATRY | PERFORMANCE | BIOCHEMISTRY & MOLECULAR BIOLOGY | NEUROSCIENCE | SCHIZOPHRENIA | LOCI | NEUROSCIENCES | COGNITIVE FUNCTION | LINKAGE ANALYSIS | EXPRESSION | GENOME-WIDE ASSOCIATION | gamma-Aminobutyric Acid | Cell Adhesion Molecules - genetics | European Continental Ancestry Group - genetics | Genome-Wide Association Study | Cell Adhesion Molecules - physiology | Genetic Association Studies | Introns | Genomics | Humans | Middle Aged | Male | Executive Function - physiology | Neuropsychological Tests | Aged, 80 and over | Cognition - physiology | Female | Aged | Genetic Variation - genetics | Polymorphism, Single Nucleotide | Cohort Studies | GABA | Physiological aspects | Genetic aspects | Single nucleotide polymorphisms | Research | Diagnosis | Dementia
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