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The Journal of Physiology, ISSN 0022-3751, 07/2016, Volume 594, Issue 13, pp. 3539 - 3552
Journal Article
Glia, ISSN 0894-1491, 01/2019, Volume 67, Issue 1, pp. 125 - 145
Following spinal cord injury (SCI), the population of mature oligodendrocytes undergoes substantial cell death; promoting their preservation and replacement is... 
cell differentiation | leukocyte common antigen‐related receptor | chondroitin sulfate proteoglycans | myelination | apoptosis | protein tyrosine phosphatase‐sigma receptor | oligodendrocytes | rat spinal cord injury | neural precursor cells | leukocyte common antigen-related receptor | protein tyrosine phosphatase-sigma receptor | Animals, Newborn | Amino Acid Sequence | Oligodendroglia - metabolism | Spinal Cord Injuries - drug therapy | Spinal Cord Injuries - metabolism | Mice, Inbred C57BL | Cells, Cultured | Rats | Peptide Fragments - pharmacology | Rats, Sprague-Dawley | Spinal Cord Injuries - pathology | Oligodendroglia - pathology | Animals | Oligodendroglia - drug effects | Receptor-Like Protein Tyrosine Phosphatases, Class 2 - metabolism | Female | Mice | Peptide Fragments - therapeutic use | Chondroitin Sulfate Proteoglycans - biosynthesis | Peptide Fragments - genetics | Receptor-Like Protein Tyrosine Phosphatases, Class 4 - metabolism | Regulators | Spinal cord | Peptides | Recovery of function | Spinal cord injury | Caspase-3 | Glial stem cells | Proteins | Receptors | Precursors | Oligodendrocytes | Repair | Injuries | Tyrosine | Proteoglycans | Preservation | Myelin | Caspase | Substantia alba | Progenitor cells | White blood cells | Myelination | Molecular modelling | Cell death | Cells (biology) | Neural stem cells | Chondroitin sulfate | Spinal cord injuries | Differentiation | Apoptosis | Protein-tyrosine-phosphatase | Integrity
Journal Article
Glia, ISSN 0894-1491, 07/2017, Volume 65, Issue 7, pp. 1152 - 1175
Spinal cord injury (SCI) results in glial activation and neuroinflammation, which play pivotal roles in the secondary injury mechanisms with both pro‐ and... 
spinal cord injury | IL‐10 | neuregulin‐1 | functional recovery | glial scar | neuroinflammation | neuregulin-1 | IL-10 | PRO-INFLAMMATORY CYTOKINES | CHONDROITIN SULFATE PROTEOGLYCANS | MICROGLIAL CELLS | NEUROSCIENCES | OLIGODENDROCYTE DIFFERENTIATION | TOLL-LIKE RECEPTORS | NITRIC-OXIDE | CENTRAL-NERVOUS-SYSTEM | ACTIVATED MACROPHAGES | NEURAL PRECURSOR CELLS | Recovery of Function - drug effects | Spinal Cord Injuries - complications | Arginase - metabolism | Culture Media, Conditioned - pharmacology | Neuregulin-1 - therapeutic use | Glial Fibrillary Acidic Protein - metabolism | Neuroglia - drug effects | Spinal Cord Injuries - pathology | Time Factors | Interleukin-10 - metabolism | Recovery of Function - physiology | Female | Disease Models, Animal | Locomotion - drug effects | Animals, Newborn | Lipopolysaccharides - toxicity | Mice, Inbred C57BL | Cells, Cultured | Nervous System Diseases - drug therapy | Neuroglia - physiology | Gene Expression Regulation - physiology | Rats | Neuregulin-1 - pharmacology | Gene Expression Regulation - drug effects | Animals | Signal Transduction - drug effects | Neuregulin-1 - metabolism | Nervous System Diseases - etiology | Mice | Tyrosine | Disabled persons | Interleukins | Analysis | Nitric oxide | Spinal cord injuries | Sulfates | Neuroprotection | Phosphorylation | Spinal cord | Matrix metalloproteinases | Neuregulin 1 | Downstream effects | Interleukin | Recovery of function | Arginase | Spinal cord injury | Dosage | Neuronal-glial interactions | Proteins | Recovering | Receptors | Rodents | Interleukin 1 | Toll-like receptors | Neuregulin | Immune system | Proteoglycans | Preservation | Immunomodulation | Stat3 protein | Extracellular signal-regulated kinase | Inflammation | ErbB-2 protein | Gelatinase A | Injury prevention | Gliosis | Molecular modelling | Interleukin 10 | MyD88 protein | Chondroitin sulfate | Sulfate | In vitro methods and tests
Journal Article
Glia, ISSN 0894-1491, 03/2018, Volume 66, Issue 3, pp. 538 - 561
Oligodendroglial cell death and demyelination are hallmarks of neurotrauma and multiple sclerosis that cause axonal damage and functional impairments.... 
lysolecithin demyelination | IL‐10 | neuregulin‐1 | neural precursor cells | oligodendrogenesis | remyelination | neuregulin-1 | IL-10 | FUNCTIONAL RECOVERY | NEUROLOGICAL RECOVERY | CHONDROITIN SULFATE PROTEOGLYCANS | NEUROTROPHIC FACTOR | NEUROSCIENCES | SCHWANN-CELL | OLIGODENDROCYTE DIFFERENTIATION | NEURAL STEM/PROGENITOR CELLS | CENTRAL-NERVOUS-SYSTEM | TERMINAL DIFFERENTIATION | PRECURSOR CELLS | Demyelinating Diseases - therapy | Humans | Extracellular Matrix - immunology | Male | Drug Carriers | Neural Stem Cells - immunology | Oligodendroglia - immunology | Spinal Cord - pathology | Spinal Cord Diseases - therapy | Demyelinating Diseases - immunology | Female | Neuregulin-1 - administration & dosage | Demyelinating Diseases - pathology | Neuroprotective Agents - administration & dosage | Disease Models, Animal | Ganglia, Spinal - immunology | Spinal Cord Diseases - immunology | Immunomodulation | Lactic Acid | Polyglycolic Acid | Cells, Cultured | Chondroitin Sulfate Proteoglycans - metabolism | Neural Stem Cells - pathology | Rats, Sprague-Dawley | Recombinant Proteins - administration & dosage | Oligodendroglia - pathology | Spinal Cord - immunology | Animals | Remyelination - physiology | Ganglia, Spinal - pathology | Spinal Cord Diseases - pathology | Extracellular Matrix - pathology | Medical colleges | Multiple sclerosis | Glycosaminoglycans | Interleukins | Cell death | Analysis | Sulfates | Spinal cord | Proteoglycans | Maturation | Myelin | Interleukin | Inflammatory response | Bioavailability | Axons | Rodents | Glycolic acid | Choline | Lesions | Recombinant | Immune system
Journal Article