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Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 06/2016, Volume 36, Issue 6, pp. 1220 - 1229
OBJECTIVE—Left ventricular (LV) remodeling after acute myocardial infarction still remains an important issue in cardiovascular medicine. We have recently... 
caveolin-1 | integrins | myocardial ischemia | caveolae | myocardial infarction | KINASE | IN-VITRO | CELL-MIGRATION | SHOCK-WAVE THERAPY | IMPROVES | PERIPHERAL VASCULAR DISEASE | GENE-THERAPY | MICE | HEMATOLOGY | EXPRESSION | MECHANOTRANSDUCTION | Phosphorylation | Human Umbilical Vein Endothelial Cells - metabolism | Ventricular Function, Left | Humans | Middle Aged | Male | Vascular Endothelial Growth Factor A - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Vascular Endothelial Growth Factor A - genetics | Case-Control Studies | Myocardial Infarction - therapy | Ventricular Remodeling | Transfection | RNA Interference | Time Factors | Autopsy | Myocardial Infarction - pathology | Myocardium - metabolism | Ventricular Dysfunction, Left - pathology | Female | Myocardial Infarction - physiopathology | Nitric Oxide Synthase Type III - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Disease Models, Animal | Ultrasonic Waves | Mice, Inbred C57BL | Cells, Cultured | Caveolin 1 - genetics | Caveolin 1 - deficiency | Genotype | Myocardium - pathology | Myocardial Infarction - metabolism | Ventricular Dysfunction, Left - physiopathology | Ventricular Dysfunction, Left - prevention & control | Mice, Knockout | Caveolin 1 - metabolism | Integrin beta1 - metabolism | Ventricular Dysfunction, Left - metabolism | Mechanotransduction, Cellular | Phenotype | Animals | Aged | Integrin beta1 - genetics | Neovascularization, Physiologic
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