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1. Diagnosis and treatment of genitourinary malignancies
by Pienta, Kenneth J
1996, Cancer treatment and research, ISBN 9780792341642, vi, 272
diagnosis | Urogenital Neoplasms | Genitourinary organs | Molecular aspects | therapy | Cancer
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2. Full Text Selection‐free method reveals phenotypic diversity among prostate cancer circulating tumour cells
by Chalfin, Heather J and Pienta, Kenneth J and Gorin, Michael A
BJU International, ISSN 1464-4096, 11/2017, Volume 120, Issue 5B, pp. E4 - E4
UROLOGY & NEPHROLOGY | BLOOD | Prostate cancer | Methods | Metastasis | Prostate | Tumors
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3. Full Text Imaging and Characterization of Macrophage Distribution in Mouse Models of Human Prostate Cancer
by Copeland, Ben T and Shallal, Hassan and Shen, Chentian and Pienta, Kenneth J and Foss, Catherine A and Pomper, Martin G
Molecular Imaging and Biology, ISSN 1536-1632, 12/2019, Volume 21, Issue 6, pp. 1054 - 1063
Prostate carcinoma consists of tumor epithelium and malignant stroma. Until recently, diagnostic and therapeutic efforts have focused exclusively on targeting... 
Prognosis | Fluorescence imaging | NIRF | Medicine & Public Health | Imaging / Radiology | DPA-713 | Prostate carcinoma | Phenotypes | Animal models | Medical imaging | Xenotransplantation | Stroma | Fluorescence | Cytotoxicity | Inflammation | Macrophages | Epithelium | Sectioning | Genotype & phenotype | Infrared imaging | Xenografts | Extracellular matrix | Mice | Diagnostic systems | Prostate cancer | Prostate | Cancer | Tumors | Antigens
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4. Full Text Human prostate cancer metastases target the hematopoietic stem cell niche to establish footholds in mouse bone marrow
by Shiozawa, Yusuke and Pedersen, Elisabeth A and Havens, Aaron M and Jung, Younghun and Mishra, Anjali and Joseph, Jeena and Kim, Jin Koo and Patel, Lalit R and Ying, Chi and Ziegler, Anne M and Pienta, Michael J and Song, Junhui and Wang, Jingcheng and Loberg, Robert D and Krebsbach, Paul H and Pienta, Kenneth J and Taichman, Russell S
Journal of Clinical Investigation, ISSN 0021-9738, 04/2011, Volume 121, Issue 4, pp. 1298 - 1312
HSC homing, quiescence, and self-renewal depend on the bone marrow HSC niche. A large proportion of solid tumor metastases are bone metastases, known to usurp... 
PROGENITOR CELLS | MEDICINE, RESEARCH & EXPERIMENTAL | ADHESION | MOBILIZATION | GROWTH | MICE | INDUCTION | IDENTIFICATION | RECEPTORS | EXPRESSION | LINEAGE | Neoplasm Transplantation | Prostatic Neoplasms - pathology | Humans | Hematopoietic Stem Cells - pathology | Male | Transplantation, Heterologous | Mice, SCID | Bone Marrow Neoplasms - secondary | Osteoblasts - pathology | Animals | Bone Marrow Neoplasms - pathology | Models, Biological | Bone Marrow Transplantation | Cell Line, Tumor | Mice, Inbred NOD | Mice | Tissue Donors | Bone marrow | Genetic aspects | Metastasis | Research | Properties | Health aspects | Prostate cancer | Hematopoietic stem cells
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5. Full Text Targeting the tumour stroma to improve cancer therapy
by Valkenburg, Kenneth C and De Groot, Amber E and Pienta, Kenneth J
Nature Reviews Clinical Oncology, ISSN 1759-4774, 06/2018, Volume 15, Issue 6, pp. 366 - 381
Cancers are not composed merely of cancer cells alone; instead, they are complex 'ecosystems' comprising many different cell types and noncellular factors. The... 
GENE-EXPRESSION SIGNATURE | GROWTH-FACTOR-BETA | PANCREATIC STELLATE CELLS | CARCINOMA-ASSOCIATED FIBROBLASTS | RESISTANCE CAM-DR | ONCOLOGY | PROSTATE-CANCER | ADULT HUMAN FIBROBLASTS | FIBROBLAST ACTIVATION PROTEIN | MESENCHYMAL STEM-CELLS | METASTATIC COLON-CANCER | Care and treatment | Research | Gene mutations | Analysis | Cancer cells | Cancer | Stroma | Metastasis | Patients | Clinical outcomes | Metastases
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6. Proposal for a Structured Reporting System for Prostate-Specific Membrane Antigen–Targeted PET Imaging: PSMA-RADS Version 1.0
by Steven P Rowe and Kenneth J Pienta and Martin G Pomper and Michael A Gorin
The Journal of Nuclear Medicine, ISSN 0161-5505, 03/2018, Volume 59, Issue 3, p. 479
As medical imaging has grown progressively more complicated and subspecialized in recent decades, several standardized reporting systems have been proposed for... 
Antigens | Relaying | Medical imaging | Standardization | Positron emission | Reporting | Studies | Urea | Reporting requirements | Data collection | Tomography | Lesions | Prostate cancer | Prostate
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7. Full Text Cooperation among cancer cells: applying game theory to cancer
by Archetti, Marco and Pienta, Kenneth J
Nature Reviews Cancer, ISSN 1474-175X, 02/2019, Volume 19, Issue 2, pp. 110 - 117
Cell cooperation promotes many of the hallmarks of cancer via the secretion of diffusible factors that can affect cancer cells or stromal cells in the tumour... 
PUBLIC-GOODS | EVOLUTIONARY-GAME | ONCOLOGY | NATURAL-SELECTION | SOCIAL DILEMMAS | MICROENVIRONMENTAL REGULATION | GROWTH-FACTOR PRODUCTION | CLONAL EVOLUTION | DRUG-RESISTANCE | TUMOR-GROWTH | VOLUNTEERS DILEMMA | Cell interaction | Care and treatment | Research | Health aspects | Oncology, Experimental | Cancer | Secretion | Game theory | Cooperation | Stromal cells | Tumors
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8. Full Text Circulating Tumor Cells Predict Survival Benefit from Treatment in Metastatic Castration-Resistant Prostate Cancer
by Johann S. de Bono and Howard I. Scher and R. Bruce Montgomery and Christopher Parker and M. Craig Miller and Henk Tissing and Gerald V. Doyle and Leon W.W.M. Terstappen and Kenneth J. Pienta and Derek Raghavan
Clinical Cancer Research, ISSN 1078-0432, 10/2008, Volume 14, Issue 19, pp. 6302 - 6309
Purpose: A method for enumerating circulating tumor cells (CTC) has received regulatory clearance. The primary objective of this prospective study was to... 
circulating tumor cells | prostate cancer | METIS-251923 | BREAST-CANCER | SURROGATE END-POINT | ONCOLOGY | DISEASE | RECEPTOR | PROGRESSION | ANTIGEN | Prostatic Neoplasms - pathology | Prognosis | Humans | Middle Aged | Proportional Hazards Models | Neoplastic Cells, Circulating - metabolism | Drug Resistance, Neoplasm | Male | Treatment Outcome | Disease Progression | Prostatic Neoplasms - mortality | Neoplasm Metastasis | Algorithms | Castration | Time Factors | Aged, 80 and over | Prostatic Neoplasms - blood | Aged
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9. Full Text Polyclonal breast cancer metastases arise from collective dissemination of keratin 14-expressing tumor cell clusters
by Kevin J. Cheung and Veena Padmanaban and Vanesa Silvestri and Koen Schipper and Joshua D. Cohen and Amanda N. Fairchild and Michael A. Gorin and James E. Verdone and Kenneth J. Pienta and Joel S. Bader and Andrew J. Ewald
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2/2016, Volume 113, Issue 7, pp. E854 - E863
Recent genomic studies challenge the conventional model that each metastasis must arise from a single tumor cell and instead reveal that metastases can be... 
Keratin 14 | Breast cancer | Collective dissemination | Polyclonal metastasis | Collective invasion | MIGRATION | MATRIX | MULTIDISCIPLINARY SCIENCES | breast cancer | MODEL | CLUMPS | INVASION | keratin 14 | MICROENVIRONMENT | polyclonal metastasis | collective invasion | EXPRESSION | PROGRESSION | collective dissemination | STEPS | Neoplasm Metastasis - genetics | Animals | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Humans | Mice | Keratin-14 - genetics | Disease Models, Animal | Keratin | Genetic aspects | Metastasis | Gene expression | Observations | Health aspects | Biological Sciences | PNAS Plus
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10. Full Text The mutational landscape of lethal castration-resistant prostate cancer
by Grasso, Catherine S and Wu, Yi-Mi and Robinson, Dan R and Cao, Xuhong and Dhanasekaran, Saravana M and Khan, Amjad P and Quist, Michael J and Jing, Xiaojun and Lonigro, Robert J and Brenner, J. Chad and Asangani, Irfan A and Ateeq, Bushra and Chun, Sang Y and Siddiqui, Javed and Sam, Lee and Anstett, Matt and Mehra, Rohit and Prensner, John R and Palanisamy, Nallasivam and Ryslik, Gregory A and Vandin, Fabio and Raphael, Benjamin J and Kunju, Lakshmi P and Rhodes, Daniel R and Pienta, Kenneth J and Chinnaiyan, Arul M and Tomlins, Scott A
Nature, ISSN 0028-0836, 07/2012, Volume 487, Issue 7406, pp. 239 - 243
Characterization of the prostate cancer transcriptome and genome has identified chromosomal rearrangements and copy number gains and losses, including ETS gene... 
FOXA1 | ANDROGEN RECEPTOR | NETWORK | FUSIONS | BINDING | MULTIDISCIPLINARY SCIENCES | Prostatic Neoplasms - pathology | Cell Proliferation | Signal Transduction | Humans | Receptors, Androgen - metabolism | Cells, Cultured | Molecular Sequence Data | Male | Hepatocyte Nuclear Factor 3-alpha - genetics | Orchiectomy | Sequence Alignment | Prostatic Neoplasms - genetics | Mutation | Development and progression | Genetic aspects | Research | Gene mutations | Health aspects | Prostate cancer | Genetics | Kinases | Genomics | Deoxyribonucleic acid--DNA
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11. Full Text Computational modeling of the crosstalk between macrophage polarization and tumor cell plasticity in the tumor microenvironment
by Li, Xuefei and Jolly, Mohit Kumar and George, Jason T and Pienta, Kenneth J and Levine, Herbert
Frontiers in Oncology, ISSN 2234-943X, 2019, Volume 9, p. 10
Tumor microenvironments contain multiple cell types interacting among one another via different signaling pathways. Furthermore, both cancer cells and... 
M1-/M2-polarized macrophages | Multi-stability | EMT-epithelial-to-mesenchymal transition | Interaction network | MET-mesenchymal-to-epithelial transition | CANCER-CELLS | STATES | multi-stability | PHENOTYPE | EMT | interaction network | ZEB1 | ONCOLOGY | PROGRESSION | PARACRINE | Cancer cells | Physiological aspects | Models | Cellular signal transduction | Macrophages | Carcinogenesis | Health aspects | MET–mesenchymal-to-epithelial transition | EMT–epithelial-to-mesenchymal transition
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12. Full Text Abstract IA15: Targeting oligometastatic disease in prostate cancer
by Pienta, Kenneth J
Cancer Research, ISSN 0008-5472, 08/2018, Volume 78, Issue 16 Supplement, pp. IA15 - IA15
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13. Full Text CXCL12 / CXCR4 / CXCR7 chemokine axis and cancer progression
by Sun, Xueqing and Cheng, Guangcun and Hao, Mingang and Zheng, Jianghua and Zhou, Xiaoming and Zhang, Jian and Taichman, Russell S and Pienta, Kenneth J and Wang, Jianhua
Cancer and Metastasis Reviews, ISSN 0167-7659, 12/2010, Volume 29, Issue 4, pp. 709 - 722
Chemokines, small pro-inflammatory chemoattractant cytokines that bind to specific G-protein-coupled seven-span transmembrane receptors, are major regulators... 
Biomedicine | Oncology | Cancer Research | Cancer progression | CXCL12 / CXCR4 / CXCR7 chemokine axis | Biomedicine general | Chemokines | PROTEIN COUPLED RECEPTOR | STEM-CELLS | CXCR4 EXPRESSION | TRANSENDOTHELIAL MIGRATION | FACTOR-I | COMPLEMENT COMPONENT C3 | BREAST-CANCER | chemokine axis | CELL-DERIVED FACTOR-1 | ONCOLOGY | RHABDOMYOSARCOMA CELLS | CXCL12 / CXCR4 / CXCR7 | TUMOR-GROWTH | Neoplasms - metabolism | Animals | Chemokine CXCL12 - metabolism | Receptors, CXCR - metabolism | Humans | Neoplasms - pathology | Disease Progression | Neoplasms - drug therapy | Receptors, CXCR4 - metabolism | Development and progression | Care and treatment | Metastasis | Cancer
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