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Clinical Cancer Research, ISSN 1078-0432, 06/2016, Volume 22, Issue 12, pp. 2908 - 2918
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2012, Volume 7, Issue 7, p. e40378
During progression of melanoma, malignant melanocytes can be reprogrammed into mesenchymal-like cells through a process similar to epithelial-mesenchymal... 
PROTEIN | SIGNALING PATHWAY | MULTIDISCIPLINARY SCIENCES | KINASE | GROWTH | REPRESSES E-CADHERIN | TUMOR | MALIGNANT-MELANOMA | TRANSCRIPTION FACTOR SLUG | EXPRESSION | PROGRESSION | Cadherins - metabolism | Humans | Gene Expression Regulation, Neoplastic | Melanocytes - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Epithelial-Mesenchymal Transition - genetics | Cell Movement - genetics | Proto-Oncogene Proteins c-akt - genetics | Gene Knockdown Techniques | RNA Interference | Cell Transformation, Neoplastic - genetics | Melanoma - genetics | Tumor Suppressor Proteins - genetics | Osteonectin | Cadherins - genetics | Proto-Oncogene Proteins c-akt - metabolism | Snail Family Transcription Factors | Melanoma - metabolism | Tumor Suppressor Proteins - metabolism | Signal Transduction | Neoplasm Invasiveness | Tumor Suppressor Protein p53 - metabolism | Melanoma - pathology | Transcription Factors - genetics | Cell Transformation, Neoplastic - metabolism | Cell Line, Tumor | Metastasis | RNA | Stem cells | Melanoma | Transcription factors | Motility | Leukocyte migration | Mesenchyme | Melanocytes | AKT protein | Kinases | E-cadherin | Metastases | Cell adhesion & migration | Skin cancer | Proteins | Genotype & phenotype | Biomedical materials | Cell growth | Extracellular matrix | Biocompatibility | Inhibition | Autocrine signalling | Pharmacology | Breast cancer | Metabolism | 1-Phosphatidylinositol 3-kinase | Collaboration | Medical prognosis | Mutation | Snail protein | Cell migration | Apoptosis | Tumors | Life Sciences | Genetics
Journal Article
Experimental Dermatology, ISSN 0906-6705, 11/2019
Journal Article
Experimental dermatology, ISSN 0906-6705, 08/2019
We developed an artificial intelligence algorithm (AIA) for smartphones to determine the severity of facial acne using the GEA scale and to identify different... 
Journal Article
NATURE COMMUNICATIONS, ISSN 2041-1723, 08/2019, Volume 10, Issue 1, pp. 3648 - 8
Journal Article
Acta dermato-venereologica, ISSN 0001-5555, 08/2019, Volume 99, Issue 12, pp. 1186 - 1187
Abstract is missing (Short communication) 
melanoma | tumor infiltrating lymphocytes | indoleamine 2,3-dioxygenase
Journal Article
Acta Dermato-Venereologica, ISSN 0001-5555, 02/2018, Volume 98, Issue 2, pp. 262 - 267
Propionibacterium acnes, a major member of normal skin microbiota, is subdivided into 6 phylotypes: IA1, IA2, IB, IC, II and III. This study investigated P.... 
P. acnes phylotypes | SLST-types | Acne | Clonal complexes | STRAINS | INFECTIONS | PATHOGEN | PHYLOGENETIC ANALYSIS | SUSCEPTIBILITY | MICROBIOME | DERMATOLOGY | RESISTANT | IN-VITRO | acne | SURFACE | clonal complexes | SKIN | Life Sciences | Cancer | acne,P.acnesphylotypes | clonalcomplexes
Journal Article
Experimental Dermatology, ISSN 0906-6705, 08/2019, Volume 28, Issue 8, pp. 961 - 967
Acne is the most common inflammatory skin disease, affecting up to 85% of the 11‐30 years old world population. Skin microbiota appears as a key player... 
microbiology | inflammatory skin diseases | acne | innate immunity | microbiome | PATHOGENESIS | IMMUNITY | DISEASE | PROPIONIBACTERIUM-ACNES | HEALTH | DERMATOLOGY | Microbiota (Symbiotic organisms) | Acne | Skin | Homeostasis | Skin diseases | Microbiota | Commensals
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2016, Volume 11, Issue 11, p. e0167237
Journal Article
International Journal of Dermatology, ISSN 0011-9059, 05/2017, Volume 56, Issue 5, pp. 527 - 533
Journal Article
European Journal of Immunology, ISSN 0014-2980, 07/2016, Volume 46, Issue 7, pp. 1770 - 1782
We previously demonstrated an accumulation of tumor‐reactive CD4+CD8+ double positive (DP) T cells within melanoma‐infiltrating lymphocytes, supporting their... 
IL‐9R | IL‐9 | CD4+CD8+ double‐positive T lymphocyte | TIL | Melanoma | CD4 | IL-9R | CD8 | double-positive T lymphocyte | IL-9 | TH9 CELLS | CD8(+) | INDUCED APOPTOSIS | INTERLEUKIN-9 | RECEPTOR | PERIPHERAL-BLOOD | IMMUNOLOGY | CYTOKINE EXPRESSION | IN-VIVO | CD4(+) CD8(+) double-positive T lymphocyte | GROWTH-FACTOR | LYMPHOCYTES | T-Lymphocyte Subsets - immunology | Apoptosis - drug effects | Humans | CD4-Positive T-Lymphocytes - immunology | Lymphocyte Activation - immunology | Interleukin-9 - pharmacology | Melanoma - genetics | T-Lymphocyte Subsets - drug effects | CD8-Positive T-Lymphocytes - metabolism | Lymphocytes, Tumor-Infiltrating - metabolism | Cytotoxicity, Immunologic - drug effects | Melanoma - metabolism | Receptors, Interleukin-9 - metabolism | Gene Expression | Cytokines - metabolism | CD4-Positive T-Lymphocytes - metabolism | Cells, Cultured | Lymphocytes, Tumor-Infiltrating - drug effects | Melanoma - pathology | Cell Survival - immunology | Apoptosis - immunology | Receptors, Interleukin-9 - genetics | Lymphocyte Activation - drug effects | Melanoma - immunology | T-Lymphocyte Subsets - metabolism | CD8-Positive T-Lymphocytes - drug effects | CD8-Positive T-Lymphocytes - immunology | Interleukin-9 - immunology | CD4-Positive T-Lymphocytes - drug effects | Lymphocytes, Tumor-Infiltrating - immunology | Parrots | T cells | Tumors | Life Sciences | Cancer
Journal Article