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1988, ISBN 088167401X, xiii, 274 p., [1] leaf of plates
Book
Neurochemistry International, ISSN 0197-0186, 2004, Volume 45, Issue 2, pp. 191 - 202
Journal Article
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Journal of Neurochemistry, ISSN 0022-3042, 2001, Volume 77, Issue 2, pp. 530 - 541
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Journal Article
Experimental Neurology, ISSN 0014-4886, 2008, Volume 212, Issue 1, pp. 44 - 52
Accumulating evidence indicates that the polyphenol resveratrol (trans-3, 5, 4”-trihydroxystibene, RVT) potently protects against cerebral ischemia neuronal... 
TO-PRO-3 iodide | Oxygen-glucose deprivation | Resveratrol | sEPSC | BK channels | Antioxidant | AMPA/NMDA | LARGE-CONDUCTANCE | antioxidant | ISCHEMIA/REPERFUSION INJURY | BRAIN-INJURY | CA2+-ACTIVATED K+ CHANNELS | ANOXIC DEPOLARIZATION | MECHANISMS | oxygen-glucose deprivation | NEUROSCIENCES | ISCHEMIA-REPERFUSION INJURY | DETERMINES | CA1 | RED WINE | resveratrol | Pyramidal Cells - metabolism | Large-Conductance Calcium-Activated Potassium Channels - metabolism | Nerve Degeneration - physiopathology | Large-Conductance Calcium-Activated Potassium Channels - antagonists & inhibitors | Hippocampus - drug effects | Stilbenes - pharmacology | Carbocyanines | Time Factors | Nerve Degeneration - prevention & control | Pyramidal Cells - drug effects | Cell Death - drug effects | Action Potentials - drug effects | Hypoxia-Ischemia, Brain - metabolism | Organ Culture Techniques | Potassium Channel Blockers - pharmacology | Membrane Potentials - drug effects | Hypoxia-Ischemia, Brain - drug therapy | Rats | Antioxidants - pharmacology | Membrane Potentials - physiology | Action Potentials - physiology | Hippocampus - metabolism | Animals | Cell Death - physiology | Hippocampus - physiopathology | Nerve Degeneration - drug therapy | Hypoxia-Ischemia, Brain - physiopathology | Antioxidants | Neurons | Index Medicus | AMPA | NMDA
Journal Article
Experimental Neurology, ISSN 0014-4886, 2007, Volume 204, Issue 2, pp. 819 - 827
Journal Article
Experimental Neurology, ISSN 0014-4886, 2008, Volume 210, Issue 2, pp. 514 - 520
Previous studies have indicated that volume regulated anion channels (VRACs) may be involved in the pathology of the ischemic brain cortical penumbra due to... 
Neuroprotection | Cerebral ischemia | Reversible focal middle cerebral artery occlusion | Microdialysis | Rat | Volume regulated anion channels (VRACs) | Excitotoxicity | 4-(2-butyl-6,7-dichloro-2-cyclopentyl-indan-1-on-5-yl) oxobutyric acid (DCPIB) | Infarct size | Behavior | rat | MECHANISM | TAMOXIFEN | 4-(2-butyl-6,7-dichloro-2-cyclopentyl-indan-1-on-5-yl) oxobutyric | NEUROSCIENCES | EXCITATORY AMINO-ACIDS | IDENTITY | volume regulated anion channels (VRACs) | infarct size | microdialysis | CORTEX | cerebral ischemia | neuroprotection | reversible focal middle cerebral artery occlusion | CEREBRAL-ARTERY OCCLUSION | CULTURED ASTROCYTES | BRAIN INJURY | excitotoxicity | behavior | acid (DCPIB) | Drug Administration Routes | Rats | Dialysis - methods | Male | Chromatography, High Pressure Liquid | Infarction, Middle Cerebral Artery - metabolism | Rats, Sprague-Dawley | Tetrazolium Salts | Brain Infarction - etiology | Animals | Infarction, Middle Cerebral Artery - complications | Statistics, Nonparametric | Aspartic Acid - metabolism | Fluorenes - administration & dosage | Glutamic Acid - metabolism | Indans - administration & dosage | Infarction, Middle Cerebral Artery - drug therapy | Neuroprotective Agents - administration & dosage | Brain Infarction - drug therapy | Cyclopentanes - administration & dosage | Disease Models, Animal | Glutamate | Amino acids | Index Medicus | 4-(2-butyl-6, 7-dichloro-2-cyclopentyl-indan-1-on-5-yl) oxobutyric acid (DCPIB)
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2008, Volume 3, Issue 10, pp. e3543 - e3543
A variety of physiological and pathological factors induce cellular swelling in the brain. Changes in cell volume activate several types of ion channels, which... 
HIPPOCAMPAL SLICES | SENSITIVE ANION CHANNELS | CHLORIDE CHANNELS | ISCHEMIC CORTICAL PENUMBRA | MOLECULAR IDENTITY | BIOLOGY | SH-SY5Y NEUROBLASTOMA-CELLS | CULTURED ASTROCYTES | REGULATORY VOLUME DECREASE | GLIAL-CELLS | CEREBRAL-CORTEX | Astrocytes - drug effects | Synaptosomes - drug effects | Water-Electrolyte Balance - physiology | Brain Edema - metabolism | Sodium Chloride - pharmacology | Synaptosomes - physiology | Brain Edema - pathology | Cells, Cultured | Rats | Male | Water-Electrolyte Balance - drug effects | Osmosis - drug effects | Synaptosomes - metabolism | Rats, Sprague-Dawley | Brain - drug effects | Brain - metabolism | Animals | Osmolar Concentration | Osmosis - physiology | Excitatory Amino Acids - metabolism | Neurons - metabolism | Neurons - drug effects | Taurine - metabolism | Astrocytes - metabolism | Taurine | Brain | Aspartate | Product introduction | Glutamate | Neurosciences | Physiological effects | Hydrogen peroxide | Traumatic brain injury | Amino acids | Cytotoxicity | Exocytosis | Anions | Ischemia | Anion channels | Rodents | Extracellular levels | Ion channels | Synaptosomes | Excitation | Localization | Cadmium | Swelling | Astrocytes | Cortex | Permeability | Sodium chloride | Properties (attributes) | Sensitivity | Neurotransmitters | Brain research | Microdialysis | Perfusion | Excitatory amino acids | In vivo methods and tests | Glutamic acid transporter | Cell size | Laboratory animals | Transporter | Index Medicus
Journal Article