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International journal of molecular sciences, ISSN 1422-0067, 07/2019, Volume 20, Issue 15, p. 3737
Diabetes mellitus is one of the major risk factors for cardiovascular disease and is an important health issue worldwide. Long-term diabetes causes endothelial... 
Biochemistry & Molecular Biology | Physical Sciences | Chemistry | Life Sciences & Biomedicine | Chemistry, Multidisciplinary | Science & Technology | Gap Junctions - metabolism | Cardiovascular Diseases - drug therapy | Diabetes Mellitus, Type 2 - genetics | Humans | Biological Factors - genetics | Myocytes, Smooth Muscle - pathology | Diabetes Mellitus, Type 1 - metabolism | Endothelium, Vascular - drug effects | Diabetes Mellitus, Type 1 - complications | Diabetes Mellitus, Type 2 - metabolism | Cardiovascular Diseases - genetics | Potassium Channels, Calcium-Activated - genetics | Potassium Channels, Calcium-Activated - metabolism | Biological Factors - metabolism | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | Diabetes Mellitus, Type 2 - complications | Hypoglycemic Agents - therapeutic use | Membrane Potentials - drug effects | Cardiovascular Diseases - etiology | Gap Junctions - pathology | Cardiovascular Diseases - metabolism | Signal Transduction | Risk Factors | Gene Expression Regulation | Insulin Resistance | Diabetes Mellitus, Type 1 - genetics | Diabetes Mellitus, Type 1 - drug therapy | Animals | Endothelium, Vascular - metabolism | Endothelium, Vascular - pathology | Vasodilation - drug effects | Diabetes Mellitus, Type 2 - drug therapy | Gap Junctions - drug effects | Lipoproteins (low density) | Retinopathy | Cardiomyopathy | Impairment | Smooth muscle | Neuropathy | Kinases | Arteries | Calcium influx | Lysophosphatidylcholine | Incidence | Calcium signalling | Proteins | Rodents | Aorta | Ion channels | Blood pressure | Calcium channels | Calcium (intracellular) | Diabetes mellitus | Cell membranes | Endothelial cells | Endothelium | Nephropathy | Nitric oxide | Insulin resistance | Protein expression | Microvasculature | Diabetes | Intracellular | Potassium | Calcium (extracellular) | Hyperpolarization | Calcium ions | Veins & arteries | Index Medicus | endothelial function | endothelium-dependent hyperpolarization | reactive oxygen species | Ca2+-activated K+ channel | diabetes mellitus | gap junction | antidiabetic agent | endothelium-derived hyperpolarizing factor
Journal Article
Journal Article
American journal of physiology. Renal physiology, ISSN 1522-1466, 06/2014, Volume 306, Issue 12, pp. F1418 - F1428
Hyperphosphatemia contributes to increased cardiovascular mortality through vascular calcification (VC) in patients with chronic kidney disease (CKD).... 
Vascular calcification | Chronic kidney disease | Inflammation | Malnutrition-inflammation-atherosclerosis syndrome | Malnutrition | Phosphate | Physiology | Life Sciences & Biomedicine | Urology & Nephrology | Science & Technology | Inflammation - chemically induced | Tumor Necrosis Factor-alpha - metabolism | Muscle, Smooth, Vascular - metabolism | Humans | Oxidative Stress - physiology | Uremia - physiopathology | Malnutrition - metabolism | Male | Uremia - metabolism | Hepatocytes - metabolism | Dose-Response Relationship, Drug | Renal Insufficiency, Chronic - metabolism | Vascular Calcification - metabolism | Hepatocytes - cytology | Inflammation - metabolism | Adenine - adverse effects | Malnutrition - chemically induced | Blood Pressure - drug effects | Blood Pressure - physiology | Phosphates - pharmacology | Hepatocytes - drug effects | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Acute-Phase Proteins - metabolism | Cells, Cultured | Rats | Muscle, Smooth, Vascular - cytology | Renal Insufficiency, Chronic - physiopathology | Animals | Phosphates - adverse effects | Vascular Calcification - chemically induced | Oxidative Stress - drug effects | Renal Insufficiency, Chronic - chemically induced | Phosphates | Medical research | Physiological aspects | Calcification | Medicine, Experimental | Research | Uremia | Health aspects | Cardiovascular disease | Kidney diseases | Mortality | Risk factors | Index Medicus
Journal Article
Scientific reports, ISSN 2045-2322, 01/2019, Volume 9, Issue 1, pp. 1043 - 1043
... tokumoto1, Kosuke Masutani5, Hiroaki ooboshi 1, toshiaki Nakano 3, Kazuhiko tsuruya6 and takanari Kitazono3 there has been limited data discussing the relationship... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Hypertension | Stroke | Hemodialysis | Data processing | Cardiovascular disease | Hemorrhage | Patients | Coronary artery disease | Ischemia | Blood pressure | Cardiovascular diseases | Health risk assessment | Heart diseases | Index Medicus
Journal Article
Circulation journal : official journal of the Japanese Circulation Society, ISSN 1347-4820, 2016, Volume 80, Issue 10, pp. 2165 - 2172
Journal Article
Cancer science, ISSN 1347-9032, 01/2017, Volume 108, Issue 1, pp. 108 - 115
We previously reported that celecoxib, a selective COX‐2 inhibitor, strongly inhibited human colon cancer cell proliferation by suppressing the Wnt/β‐catenin... 
TCF7L2 | 2,5‐dimethylcelecoxib | Wnt/β‐catenin signaling pathway | celecoxib | colon cancer | 2,5-dimethylcelecoxib | Wnt/β-catenin signaling pathway | Life Sciences & Biomedicine | Oncology | Science & Technology | Transcription, Genetic - drug effects | Pyrazoles - therapeutic use | Humans | Body Weight - drug effects | Male | Wnt Proteins - metabolism | Transcription Factor 7-Like 2 Protein - metabolism | TCF Transcription Factors - metabolism | DNA Glycosylases - deficiency | Proteolysis - drug effects | Sulfonamides - blood | Pyrazoles - blood | Female | Pyrazoles - pharmacology | Intestinal Neoplasms - metabolism | DNA Glycosylases - genetics | Celecoxib - blood | Celecoxib - pharmacology | Celecoxib - therapeutic use | Sulfonamides - pharmacology | beta Catenin - metabolism | Blood Cell Count | Intestinal Neoplasms - pathology | Animals | Wnt Signaling Pathway - drug effects | Sulfonamides - therapeutic use | beta Catenin - antagonists & inhibitors | Cell Line, Tumor | Mice | Oxidative Stress - drug effects | Intestinal Neoplasms - drug therapy | Cell proliferation | Drugs | Plasma | Oxidative stress | Carcinoma | Wnt protein | Toxicity | Genes | Colorectal cancer | Cyclin D1 | Drug development | Signal transduction | Cell growth | Colon cancer | Antitumor agents | Intestine | Cell cycle | Catenin | Oral administration | Celecoxib | Survivin | Tumor cell lines | Plasmids | Antitumor activity | Mutation | Apoptosis | Tumors | Index Medicus | β‐catenin signaling pathway | Wnt | Original
Journal Article
Drug delivery system, ISSN 0913-5006, 12/2015, Volume 30, Issue 4, pp. 274 - 274
Journal Article
Journal Article
Journal Article