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Journal of Biological Chemistry, ISSN 0021-9258, 02/2007, Volume 282, Issue 7, pp. 4524 - 4532
The stability and transcriptional activity of the hypoxia-inducible factors (HIFs) are regulated by two oxygen-dependent events that are catalyzed by three HIF... 
Gene Expression Regulation, Enzymologic - drug effects | Transcription, Genetic - drug effects | Vascular Endothelial Growth Factor A - biosynthesis | Fibroblasts - enzymology | Fumarate Hydratase - chemistry | Mixed Function Oxygenases - metabolism | Fumarate Hydratase - deficiency | Mixed Function Oxygenases - chemistry | Succinate Dehydrogenase - chemistry | Procollagen-Proline Dioxygenase - metabolism | Dose-Response Relationship, Drug | Fumarate Hydratase - metabolism | Neoplasms - chemistry | Neoplasms - genetics | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Procollagen-Proline Dioxygenase - antagonists & inhibitors | Hypoxia-Inducible Factor 1, alpha Subunit - chemistry | Erythropoietin - biosynthesis | Fibroblasts - chemistry | Cell Line | Enzyme Inhibitors - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Citric Acid Cycle - drug effects | Neoplasms - enzymology | Mixed Function Oxygenases - antagonists & inhibitors | Fibroblasts - pathology | Succinate Dehydrogenase - deficiency | p300-CBP Transcription Factors - metabolism | Citric Acid Cycle - genetics | Procollagen-Proline Dioxygenase - genetics | Gene Expression Regulation, Enzymologic - genetics | Succinate Dehydrogenase - metabolism | Mixed Function Oxygenases - genetics | Neoplasms - pathology | Procollagen-Proline Dioxygenase - chemistry
Journal Article
International Journal of Biochemistry and Cell Biology, ISSN 1357-2725, 2010, Volume 42, Issue 7, pp. 1189 - 1200
The hypoxia-inducible transcription factors (HIFs) play a central role in the response of cells to hypoxia. HIFs are αβ dimers, the human α subunit having... 
Hypoxia-inducible factor | Alternative splicing | Hypoxia-inducible factor isoform 3 | Hypoxia response | Cancer cells
Journal Article
EMBO Journal, ISSN 0261-4189, 03/1997, Volume 16, Issue 6, pp. 1173 - 1180
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 08/2003, Volume 278, Issue 33, pp. 30772 - 30780
Journal Article
FASEB Journal, ISSN 0892-6638, 08/2005, Volume 19, Issue 10, pp. 1308 - 1310
Journal Article
Molecular and Cellular Biology, ISSN 0270-7306, 08/2013, Volume 33, Issue 16, pp. 3321 - 3329
Journal Article
INTERNATIONAL JOURNAL OF BIOCHEMISTRY & CELL BIOLOGY, ISSN 1357-2725, 07/2010, Volume 42, Issue 7, pp. 1189 - 1200
The hypoxia-inducible transcription factors (HIFs) play a central role in the response of cells to hypoxia. HIFs are alpha beta dimers, the human a subunit... 
Alternative splicing | BIOCHEMISTRY & MOLECULAR BIOLOGY | ALPHA | Hypoxia-inducible factor isoform 3 | NEGATIVE REGULATOR | FAMILY | CELL BIOLOGY | OXYGEN | Cancer cells | Hypoxia-inducible factor | GENE-EXPRESSION | PROLINE HYDROXYLATION | PROLYL 4-HYDROXYLASES | HIF-1-ALPHA | VHL | Hypoxia response | PAS DOMAIN PROTEIN
Journal Article
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 04/2016, Volume 36, Issue 4, pp. 608 - 617
OBJECTIVE—Small-molecule hypoxia-inducible factor prolyl 4-hydroxylase (HIF-P4H) inhibitors are being explored in clinical studies for the treatment of anemia.... 
atherosclerosis | hypoxia | cholesterol | inflammation | immunology | LIPID-METABOLISM | FACTOR-I | HYDROXYLASE DOMAIN PROTEIN-2 | OBESITY | PATHWAY | ROLES | GLUCOSE | LIVER | DISEASE | SREBPS | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | Atherosclerosis - genetics | Autoantibodies - blood | Cholesterol - blood | Male | Hypoxia-Inducible Factor-Proline Dioxygenases - antagonists & inhibitors | Basic Helix-Loop-Helix Transcription Factors - metabolism | Liver - drug effects | Time Factors | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Aortic Diseases - prevention & control | Weight Gain - drug effects | Lipoproteins, LDL - metabolism | Disease Models, Animal | Atherosclerosis - pathology | Adiposity - drug effects | Atherosclerosis - immunology | Aortic Diseases - immunology | Enzyme Inhibitors - pharmacology | Hypoxia-Inducible Factor-Proline Dioxygenases - genetics | Aorta - immunology | Plaque, Atherosclerotic | Macrophages - enzymology | Mice, Knockout | Aorta - pathology | Atherosclerosis - blood | Aortic Diseases - genetics | Lipoproteins, LDL - immunology | Liver - enzymology | Aortic Diseases - blood | Atherosclerosis - enzymology | Receptors, LDL - deficiency | Aortic Diseases - enzymology | Protein Stability | Aorta - enzymology | Aortic Diseases - pathology | Receptors, LDL - genetics | Immunity, Innate - drug effects | Aorta - drug effects | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation | Inflammation Mediators - blood | Insulin Resistance | Animals | Hypoxia-Inducible Factor-Proline Dioxygenases - metabolism | Adipose Tissue, White - enzymology | Macrophages - drug effects | Adipose Tissue, White - drug effects | Atherosclerosis - prevention & control | Index Medicus
Journal Article
Hypertension, ISSN 0194-911X, 2014, Volume 63, Issue 6, pp. 1235 - 1240
Connective tissue growth factor (CTGF) is involved in the pathogenesis of various fibrotic disorders. However, its role in the heart is not clear. To... 
Connective tissue growth factor | Ventricular remodeling | Hypertrophy, left ventricular | Ventricular dysfunction, left | Fibrosis | TGF-BETA | MYOCYTES | hypertrophy, left ventricular | ventricular remodeling | MATRICELLULAR PROTEINS | ventricular dysfunction, left | MECHANISMS | ANGIOTENSIN | connective tissue growth factor | FIBROBLAST ACTIVATION | MYOCARDIAL FIBROSIS | CARDIAC-HYPERTROPHY | PERIPHERAL VASCULAR DISEASE | fibrosis | ENDOTHELIN-1 | EXPRESSION | Gene Expression - drug effects | Connective Tissue Growth Factor - immunology | Heart Failure - physiopathology | Male | Constriction, Pathologic - physiopathology | Collagen Type I - genetics | Myocardium - metabolism | Aorta, Thoracic - drug effects | Plasminogen Activator Inhibitor 1 - genetics | Antibodies, Monoclonal - immunology | Aorta, Thoracic - pathology | Fibroblasts - metabolism | Heart - physiopathology | Heart Failure - complications | Angiotensin II - pharmacology | Matrix Metalloproteinase 2 - metabolism | Antibodies, Monoclonal - pharmacology | Connective Tissue Growth Factor - antagonists & inhibitors | Mice, Inbred C57BL | Aorta, Thoracic - metabolism | Ventricular Dysfunction, Left - etiology | Myocardium - pathology | Reverse Transcriptase Polymerase Chain Reaction | Pressure | Ventricular Dysfunction, Left - prevention & control | Weight-Bearing - physiology | Matrix Metalloproteinase 2 - genetics | Animals | Myocytes, Cardiac - drug effects | Fibroblasts - drug effects | Heart - drug effects | Myocytes, Cardiac - metabolism | Connective Tissue Growth Factor - genetics | Mice | Ventricular Remodeling - drug effects | Index Medicus
Journal Article