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Nature Medicine, ISSN 1078-8956, 2015, Volume 21, Issue 2, pp. 140 - 149
Journal Article
Circulation, ISSN 0009-7322, 2017, Volume 136, Issue 19, pp. 1809 - 1823
Background: Clinical trials of bone marrow cell-based therapies after acute myocardial infarction (MI) have produced mostly neutral results. Treatment with... 
Myocardial infarction | Bone marrow | Angiogenesis | Inflammation | Monocyte | TRIALS | CARDIAC & CARDIOVASCULAR SYSTEMS | PHOSPHORYLATION | angiogenesis | myocardial infarction | MONOCYTES | ENDOTHELIAL-CELL MIGRATION | IDENTIFICATION | bone marrow | CARDIAC REPAIR | DETERMINES | HEART-DISEASE | THERAPY | inflammation | monocyte | PERIPHERAL VASCULAR DISEASE | Angiogenic Proteins - genetics | Myocardial Infarction - genetics | Neovascularization, Physiologic - drug effects | Monocytes - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Membrane Proteins - deficiency | Time Factors | Myocardial Infarction - pathology | Myocardium - metabolism | Bone Marrow Transplantation | Membrane Proteins - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Wound Healing - drug effects | Protein-Serine-Threonine Kinases - metabolism | Disease Models, Animal | Angiogenic Proteins - deficiency | Signal Transduction | Angiogenic Proteins - administration & dosage | Endothelial Cells - metabolism | Membrane Proteins - genetics | Mice, Inbred C57BL | Cells, Cultured | Genotype | Myocardium - pathology | Myocardial Infarction - metabolism | p21-Activated Kinases - metabolism | Mice, Knockout | Membrane Proteins - administration & dosage | Macrophages - metabolism | Phenotype | Animals | Myocardial Infarction - drug therapy | Monomeric GTP-Binding Proteins - metabolism | Angiogenic Proteins - metabolism | Bone Marrow Cells - metabolism | Index Medicus | Abridged Index Medicus
Journal Article
Circulation Research, ISSN 0009-7330, 02/2008, Volume 102, Issue 4, pp. 439 - 447
Journal Article
Circulation, ISSN 0009-7322, 10/2017
BACKGROUND—Clinical trials of bone marrow cell (BMC)-based therapies after acute myocardial infarction (MI) have produced mostly neutral results. Treatment... 
Journal Article
Circulation Research, ISSN 0009-7330, 03/2010, Volume 106, Issue 4, pp. 695 - 704
RATIONALE:We previously discovered the human 10T→C (Trp4Arg) missense mutation in exon 2 of the muscle LIM protein (MLP, CSRP3) gene. OBJECTIVE:We sought to... 
Heart failure | Mechanosensation | Genetics | Mechanotransduction | Cardiomyopathy | Circulation | CARDIAC-FUNCTION | heart failure | CARDIAC & CARDIOVASCULAR SYSTEMS | FAMILIAL HYPERTROPHIC CARDIOMYOPATHY | MYOCARDIAL-INFARCTION | mechanotransduction | HEART-FAILURE | DILATED CARDIOMYOPATHY | SKELETAL-MUSCLE | genetics | circulation | GENE-EXPRESSION | PERIPHERAL VASCULAR DISEASE | TROPONIN-T | mechanosensation | cardiomyopathy | IONIC-STRENGTH | HEMATOLOGY | POLYACRYLAMIDE-GEL | Connectin | Age Factors | Ventricular Function, Left | Heart Failure - physiopathology | Muscle, Skeletal - metabolism | Cardiomyopathy, Hypertrophic - complications | Mutation, Missense | RNA, Messenger - metabolism | Recombinant Fusion Proteins - metabolism | Transfection | Muscle Proteins - metabolism | Aging | Disease Models, Animal | Myocardial Contraction | Cardiomyopathy, Hypertrophic - genetics | LIM Domain Proteins | Cardiomyopathy, Hypertrophic - metabolism | Mice, Inbred C57BL | Cells, Cultured | Rats | Genotype | Heart Failure - genetics | Mice, Transgenic | Heart Failure - metabolism | Gene Knock-In Techniques | Muscle Proteins - genetics | Homozygote | Myocytes, Cardiac - pathology | Phenotype | Animals | Cardiomyopathy, Hypertrophic - physiopathology | Fibrosis | Myocytes, Cardiac - metabolism | Heterozygote | Mice | Polymorphism, Single Nucleotide | Muscle, Skeletal - pathology | Index Medicus
Journal Article
Circulation research, 08/2019
Mechanistic insight into the inflammatory response after acute myocardial infarction (MI) may inform new molecularly-targeted treatment strategies to prevent... 
Journal Article
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