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Leukemia, ISSN 0887-6924, 11/2017, Volume 31, Issue 11, pp. 2398 - 2406
Journal Article
JOURNAL OF HEPATOCELLULAR CARCINOMA, ISSN 2253-5969, 2019, Volume 6, pp. 93 - 103
Journal Article
PLoS Pathogens, ISSN 1553-7366, 06/2018, Volume 14, Issue 6, p. e1007126
The human fungal pathogen, Cryptococcus neoformans, dramatically alters its cell wall, both in size and composition, upon entering the host. This cell wall... 
CHITIN SYNTHASE | MICROBIOLOGY | VIRULENCE | SACCHAROMYCES-CEREVISIAE | SEXUAL CYCLE | CYTOKINE PRODUCTION | CANDIDA-ALBICANS | TITAN CELLS | VIROLOGY | CRYPTOCOCCUS-NEOFORMANS | C3H/HEJ MICE | PARASITOLOGY | WHEAT-GERM LECTIN | Cell Wall - immunology | Cryptococcosis - microbiology | Cryptococcus neoformans - enzymology | Receptors, Pattern Recognition - immunology | Dendritic Cells - immunology | Humans | Dendritic Cells - pathology | Male | Cell Wall - enzymology | Host-Pathogen Interactions - immunology | Cryptococcus neoformans - pathogenicity | Female | beta-Glucans - immunology | Dendritic Cells - metabolism | Macrophages - immunology | Cryptococcosis - pathology | Macrophages - pathology | Mice, Inbred C57BL | Cells, Cultured | Fungal Proteins - genetics | Cryptococcosis - immunology | Protein Transport | Macrophages - metabolism | Animals | Mice | Mice, Inbred BALB C | Immune Evasion - immunology | Fungal Proteins - metabolism | Host-bacteria relationships | Analysis | Cryptococcus | Physiological aspects | Immune recognition | Genetic aspects | Bacterial cell walls | Research | Cell culture | Membranes | Funding | Virulence | Tissue culture | Biology | Activation | Fungal infections | Macrophages | Cell surface | Genetic screening | Defects | Proteins | Fungi | Glucan | Polysaccharides | Receptors | Cell activation | Microorganisms | Toll-like receptors | Bone marrow | Catalysis | Adaptor proteins | Pathogens | Departments | Phenotypes | Cell walls | Chitin | Liquid chromatography | Pattern recognition | High-performance liquid chromatography | Mutants | Host-pathogen interactions | Medicine | Polymerase chain reaction | Glucans | TLR2 protein | MyD88 protein | Chitosan | Cell size | Intracellular | Avoidance | Trafficking
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 12/2016, Volume 311, Issue 6, pp. H1540 - H1559
The forkhead box O3a (FOXO3a) transcription factor has been shown to regulate glucose metabolism, muscle atrophy, and cell death in postmitotic cells. Its role... 
F0X03a | Heart failure | BN1P3 | Apoptosis | Calcium regulation | heart failure | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | calcium regulation | PROTEIN | INDUCIBLE FACTOR 1-ALPHA | BNIP3 | HEART-FAILURE | NEURON DEATH | apoptosis | AUTOPHAGY | HYPOXIA | HYPERTROPHY | PERIPHERAL VASCULAR DISEASE | FOXO3a | TRANSCRIPTION FACTOR | EXPRESSION | Mitochondria, Heart - ultrastructure | Dynamins - metabolism | Phosphorylation | Mitochondria, Heart - metabolism | Calcium - metabolism | Ventricular Function, Left - genetics | Heart Failure - physiopathology | Stress, Physiological | Sympathomimetics - pharmacology | Endoplasmic Reticulum - metabolism | Male | Mitochondria, Heart - drug effects | Electron Transport Complex IV - metabolism | Forkhead Box Protein O3 - genetics | Ventricular Remodeling | Phenylephrine - pharmacology | Mitochondrial Proteins - metabolism | Membrane Potential, Mitochondrial | Citrate (si)-Synthase - metabolism | Membrane Proteins - metabolism | Real-Time Polymerase Chain Reaction | Disease Models, Animal | Calcium-Binding Proteins - metabolism | Microscopy, Electron, Transmission | Echocardiography | Cell Survival | Rats | Heart Failure - metabolism | Rats, Sprague-Dawley | Blotting, Western | Forkhead Box Protein O3 - metabolism | Stroke Volume | Animals | Myocytes, Cardiac - drug effects | Fluorescent Antibody Technique | Myocytes, Cardiac - metabolism | In Vitro Techniques | Myocytes, Cardiac - ultrastructure | Transcription factors | Genetic aspects | Gene expression | Health aspects | Heart diseases | Signaling and Stress Response
Journal Article
Human Gene Therapy, ISSN 1043-0342, 08/2018, Volume 29, Issue 8, pp. 927 - 937
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 05/2016, Volume 67, Issue 17, pp. 2032 - 2046
Journal Article
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 10/2017, Volume 70, Issue 14, pp. 1744 - 1756
Increased protein phosphatase-1 in heart failure (HF) induces molecular changes deleterious to the cardiac cell. Inhibiting protein phosphatase-1 through the... 
chimeric adenoassociated virus | inhibitor-1 | mitral regurgitation | contractility | intracoronary gene therapy | CARDIAC-FUNCTION | CARDIAC & CARDIOVASCULAR SYSTEMS | DILATED CARDIOMYOPATHY | DELIVERY | REGURGITATION | DISEASE | DYSFUNCTION | EXPRESSION | Intracellular Signaling Peptides and Proteins - pharmacology | Enzyme Inhibitors - pharmacology | Heart Failure - physiopathology | Treatment Outcome | Heart Failure - metabolism | Drug Monitoring - methods | Drug Delivery Systems | Heart Failure - drug therapy | Dose-Response Relationship, Drug | Protein Phosphatase 1 - antagonists & inhibitors | Protein Phosphatase 1 - metabolism | Animals | Genetic Vectors - pharmacology | Swine | Heart Failure - etiology | Disease Models, Animal | Genetic Therapy - methods | Heart failure | Phosphatases | Gene therapy | Analysis | Health aspects | Genes | Heart | Phosphorylation | Diastolic pressure | Calcium | Phosphoprotein phosphatase | Cardiomyopathy | Cardiovascular disease | Myocytes | Kinases | Phosphatase | Contraction | Proteins | Ischemia | Regurgitation | Blood pressure | Safety | Heart diseases | Immune system | Medical research | Atrium | Immunoglobulins | Immune response | Gene transfer | Muscle contraction | Hogs | Inhibitors | Protein phosphatase | Ventricle | Laboratory animals | chimeric adeno-associated virus
Journal Article