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Journal of Rheumatology, ISSN 0315-162X, 03/2018, Volume 45, Issue 3, pp. 439 - 440
Journal Article
JOURNAL OF INFLAMMATION RESEARCH, ISSN 1178-7031, 2019, Volume 12, pp. 259 - 265
Journal Article
Nature, ISSN 0028-0836, 2014, Volume 506, Issue 7488, pp. 376 - +
A major challenge in human genetics is to devise a systematic strategy to integrate disease-associated variants with diverse genomic and biological data sets... 
RECEPTOR INHIBITION | VARIANTS | MULTIDISCIPLINARY SCIENCES | DISEASE | SUSCEPTIBILITY | RISK | LOCI | IDENTIFICATION | TRAITS | GENOME-WIDE ASSOCIATION | RESOURCE | European Continental Ancestry Group - genetics | Genetic Predisposition to Disease - genetics | Genome-Wide Association Study | Humans | Asian Continental Ancestry Group - genetics | Computational Biology | Male | Drug Repositioning | Molecular Targeted Therapy | Drug Discovery | Case-Control Studies | Arthritis, Rheumatoid - pathology | Mice, Knockout | Arthritis, Rheumatoid - metabolism | Arthritis, Rheumatoid - genetics | Animals | Arthritis, Rheumatoid - drug therapy | Alleles | Polymorphism, Single Nucleotide - genetics | Female | Hematologic Neoplasms - genetics | Mice | Hematologic Neoplasms - metabolism | Quantitative trait loci | Genome-wide association studies | Rheumatoid arthritis | Genetic research | Genetic aspects | Research | Human genetics | Disease | Hematology | Cytokines | Genes | Genomics | Genomes | Meta-analysis | Proteins | Confidence intervals | Hypotheses | Quality control | Epigenetics | Population | Genetics | Health risk assessment | Asians | Cancer | Index Medicus | Rheumatology and Autoimmunity | Clinical Medicine | Medical and Health Sciences | Medicin och hälsovetenskap | Reumatologi och inflammation | Virtual screening | Klinisk medicin
Journal Article
Journal Article
Nature Genetics, ISSN 1061-4036, 05/2012, Volume 44, Issue 5, pp. 511 - 516
Journal Article
Journal Article
Rinsho byori. The Japanese journal of clinical pathology, ISSN 0047-1860, 04/2015, Volume 63, Issue 4, pp. 485 - 490
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2011, Volume 108, Issue 36, pp. 14914 - 14919
Journal Article
Annals of the Rheumatic Diseases, ISSN 0003-4967, 11/2019, Volume 78, Issue 11, pp. 1480 - 1487
ObjectsAlthough the association of cigarette smoking (CS) with susceptibility to rheumatoid arthritis (RA) has been established, the impact of CS on... 
CRITERIA | CYCLIC CITRULLINATED PEPTIDE | HLA-DRB1 ALLELES | GENETICS | SUSCEPTIBILITY | CLASSIFICATION | RHEUMATOLOGY | JAPANESE PATIENTS | ARTHRITIS | ACPA | EROSIONS
Journal Article
Rinsho byori. The Japanese journal of clinical pathology, ISSN 0047-1860, 04/2015, Volume 63, Issue 4, p. 485
We have conducted genome-wide association studies (GWAS) for rheumatoid arthritis (RA). We previously found that myelin basic protein (MBP) is associated with... 
Arthritis, Rheumatoid - genetics | Genetic Predisposition to Disease - genetics | Genome-Wide Association Study | Meta-Analysis as Topic | Animals | Humans | T-Lymphocytes, Regulatory | Mice | Drug Discovery
Journal Article
Annals of the Rheumatic Diseases, ISSN 0003-4967, 02/2019, Volume 78, Issue 2, pp. 284 - 286
[...]we found that HLA-DRB1*04:05 was significantly associated with SHS in condition with RF, disease duration, cohort information and time-averaged DAS28... 
RHEUMATOLOGY | Rheumatoid arthritis | Rheumatology | Drb1 protein | Rheumatism | Remission | Histocompatibility antigen HLA | Health risk assessment
Journal Article
Immunity, ISSN 1074-7613, 07/2017, Volume 47, Issue 1, pp. 66 - 79.e5
Hypoxia augments inflammatory responses and osteoclastogenesis by incompletely understood mechanisms. We identified COMMD1 as a cell-intrinsic negative... 
hypoxia | arthritis | inflammation | glycolysis | E2F | macrophage | metabolism | COMMD1 | CKB | osteoclast | RHEUMATOID-ARTHRITIS | ACTIVATION | MITOCHONDRIAL BIOGENESIS | RNA-SEQ | NEGATIVE REGULATION | JOINT DESTRUCTION | MECHANISMS | DIFFERENTIATION | IMMUNOLOGY | CREATINE-KINASE | BONE-RESORPTION | RNA, Small Interfering - genetics | Signal Transduction | Humans | Mice, Inbred C57BL | Middle Aged | Cells, Cultured | Male | NF-kappa B - metabolism | E2F1 Transcription Factor - metabolism | Hypoxia - metabolism | Mice, Knockout | Animals | Adaptor Proteins, Signal Transducing - genetics | Female | Mice | Adaptor Proteins, Signal Transducing - metabolism | Arthritis, Rheumatoid - immunology | Macrophages - immunology | Osteogenesis - genetics | Disease Models, Animal | Glucose metabolism | Yuan (China) | Medical colleges | Rheumatoid factor | Analysis | Physiological aspects | Arthritis | Inflammation | Macrophages | Population genetics | Regulators | Phosphorylation | Transcription factors | Destruction | Genes | Biosynthesis | Kinases | Osteolysis | Proteins | E2F protein | Signal transduction | Clonal deletion | Metabolites | Rodents | Deletion | Tumor necrosis factor-TNF | Biocompatibility | TRANCE protein | Copper | Erosion | Cytokines | Metabolism | Gene expression | E2F1 protein | Quantitative trait loci | Osteoclastogenesis | Rheumatoid arthritis | Glycolysis | Hypoxia | Bone | Cancer
Journal Article
PLoS genetics, ISSN 1553-7390, 2013, Volume 9, Issue 3, p. e1003394
Journal Article