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NEUROSCIENCE RESEARCH, ISSN 0168-0102, 2008, Volume 61, pp. S128 - S128
Conference Proceeding
PLoS ONE, ISSN 1932-6203, 11/2014, Volume 9, Issue 11, pp. e111888 - e111888
Journal Article
Science, ISSN 0036-8075, 5/2004, Volume 304, Issue 5674, pp. 1147 - 1150
Journal Article
Journal Article
Journal Article
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 8, pp. e23238 - e23238
Aim: We previously found that chronic tuberous sclerosis protein 2 (TSC2) deletion induces activation of mammalian target of rapamycin Complex 1 (mTORC1) and... 
SKELETAL-MUSCLE | PKC-LAMBDA | BIOGENESIS | TUBEROUS SCLEROSIS GENE | PROTEIN-KINASE | RENAL CARCINOGENESIS | BIOLOGY | RESISTANCE | PANCREATIC BETA-CELLS | MICE | NATURAL-HISTORY | Organ Specificity - drug effects | Cell Line | Tumor Suppressor Proteins - metabolism | Multiprotein Complexes | Glucose - pharmacology | Mitochondria - metabolism | Mitochondria - drug effects | Sirolimus - pharmacology | Gene Knockdown Techniques | Mechanistic Target of Rapamycin Complex 1 | Insulin - metabolism | Insulin-Secreting Cells - metabolism | Animals | Proteins - metabolism | Tuberous Sclerosis Complex 2 Protein | Insulin-Secreting Cells - drug effects | Tumor Suppressor Proteins - deficiency | Gene Deletion | Mice | TOR Serine-Threonine Kinases | Insulin Secretion | Type 2 diabetes | Pancreatic beta cells | RNA | Genes | Tuberous sclerosis | Biosynthesis | Mitochondrial DNA | Insulin | Statistics | TOR protein | Tuberous sclerosis 2 protein | Health sciences | Hyperinsulinemia | Neurosciences | Phosphorylation | Insulinoma | Homeostasis | Biology | Activation | Glucose | Kinases | Sclerosis | Proteins | Mitochondria | Clonal deletion | Rodents | Transcription activation | Deletion | Pancreas | Deoxyribonucleic acid--DNA | University graduates | Enlargement | Medical research | Phenotypes | Secretion | Rapamycin | Gene expression | Ablation | Beta cells | Medicine | TSC2 protein | Musculoskeletal system | Insulin resistance | Diabetes | ATP | Endocrinology | Hypertrophy | Cancer | Apoptosis | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 10/2011, Volume 108, Issue 40, pp. 16825 - 16830
Once their safety is confirmed, human-induced pluripotent stem cells (hiPSCs), which do not entail ethical concerns, may become a preferred cell source for... 
Spinal cord | Neuroscience | Neurons | Induced pluripotent stem cells | Astrocytes | Motor ability | Neural stem cells | Transplantation | Physical trauma | Tissue grafting | Synaptic connection | Stem-cell-based medicine | Cell transplantation | Neurotrauma | DIRECTED DIFFERENTIATION | neurotrauma | ADULT RATS | MULTIDISCIPLINARY SCIENCES | HUMAN IPS CELLS | NEUROLOGICAL DISORDERS | synaptic connection | cell transplantation | HUMAN ASTROCYTES | stem-cell-based medicine | MESSENGER-RNA | NEURAL STEM/PROGENITOR CELLS | ENDOGENOUS REPAIR | GROWTH-FACTOR | LOCOMOTOR FUNCTION | Nerve Regeneration - genetics | Transduction, Genetic | Synapses - physiology | Humans | Induced Pluripotent Stem Cells - transplantation | Nerve Regeneration - physiology | Spinal Cord Injuries - surgery | Electrophysiology | Gene Expression Profiling | Mice, SCID | Reverse Transcriptase Polymerase Chain Reaction | Lentivirus | Regenerative Medicine - methods | Cell Movement - physiology | Rotarod Performance Test | Animals | Analysis of Variance | Female | Mice, Inbred NOD | Mice | Cell Differentiation - physiology | Physiological aspects | Care and treatment | Spinal cord injuries | Patient outcomes | Stem cells | Rodents | Tumors | Synaptogenesis | Myelin | Diabetes mellitus | Oct-4 protein | Immunodeficiency | c-Myc protein | Recovery of function | Neurotrophic factors | Spinal cord injury | Angiogenesis | Ethics | Regeneration | KLF4 protein | Oligodendrocytes | Fibroblasts | neurospheres | Index Medicus | Biological Sciences | stem-cell–based medicine
Journal Article