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Historica - Sborník prací historických, ISSN 1803-9561, 2017, Issue 52, pp. 325 - 326
Journal Article
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, ISSN 0027-8424, 07/2019, Volume 116, Issue 27, pp. 13414 - 13423
Journal Article
Nature, ISSN 0028-0836, 10/2011, Volume 478, Issue 7367, pp. 114 - 118
Left ventricular mass (LVM) is a highly heritable trait(1) and an independent risk factor for all-cause mortality(2). So far, genome-wide association studies... 
PATHOGENESIS | OXIDATIVE STRESS | REPLICATION | BIOGENESIS | MULTIDISCIPLINARY SCIENCES | CARDIOMYOPATHY | DISEASE | DEATH | MECHANISMS | LEFT-VENTRICULAR MASS | EXPRESSION | Receptors, Estrogen - metabolism | Hypertrophy, Left Ventricular - enzymology | Reactive Oxygen Species - metabolism | Chromosomes, Mammalian - genetics | Cardiomegaly - pathology | Male | Rats, Inbred Strains | Body Weight - genetics | Hypertrophy, Left Ventricular - genetics | Cell Respiration | Mitochondria - genetics | Hypertrophy, Left Ventricular - pathology | Organ Size - genetics | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Endodeoxyribonucleases - metabolism | Female | Endodeoxyribonucleases - deficiency | Gene Expression Regulation | Cardiomegaly - physiopathology | Rats | Lipid Metabolism | Mitochondria - metabolism | Cardiomegaly - enzymology | Mitochondria - pathology | Transcription Factors - metabolism | Genes, Mitochondrial - genetics | Endodeoxyribonucleases - genetics | Animals | Hypertrophy, Left Ventricular - physiopathology | Cardiomegaly - genetics | RNA-Binding Proteins - metabolism | Apoptosis | Crosses, Genetic | Quantitative Trait Loci - genetics | Nucleases | Mitochondria | Heart enlargement | Physiological aspects | Genetic aspects | Research | Diagnosis | Risk factors | Studies | Rodents | Biomarkers | Genetics | Cardiomyocytes | Genomes | Blood pressure | Kinases | Index Medicus
Journal Article
Cardiovascular Diabetology, ISSN 1475-2840, 05/2018, Volume 17, Issue 1, pp. 68 - 68
Journal Article
CESKA LITERATURA, ISSN 0009-0468, 1993, Volume 41, Issue 5, pp. 584 - 585
Journal Article
Proceedings of the National Academy of Sciences of the United States, ISSN 0027-8424, 07/2019, Volume 116, Issue 27, p. 13414
The molecular mechanisms regulating sympathetic innervation of the heart during embryogenesis and its importance for cardiac development and function remain to... 
Journal Article
Molecular and cellular biochemistry, ISSN 0300-8177, 07/2019, pp. 1 - 8
Adaptation to chronic hypoxia renders the heart more tolerant to ischemia/reperfusion injury. To evaluate changes in gene expression after adaptation to... 
Algorithms | Reperfusion | Ischemia | Genes | Myocardium | Hypoxia | Glyceraldehyde-3-phosphate dehydrogenase | Stability analysis | Gene expression | Adaptation
Journal Article
Journal Article
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 11/2017, Volume 112, pp. 157 - 157
Journal Article
Journal Article
BMC Endocrine Disorders, ISSN 1472-6823, 02/2014, Volume 14, Issue 1
Diabetic cardiomyopathy is associated with a number of functional and structural pathological changes such as left ventricular dysfunction, cardiac remodeling,... 
Type 2 diabetes | Physiological aspects | Endothelial growth factors | Analysis | Risk factors | Heart | Cardiomyopathy | Anatomy & physiology | Gene expression | Experiments | Proteins | Studies | Angiogenesis | Pathology | Hyperglycemia | Rodents | Adenoviruses | Hypoxia | Diabetes | Laboratory animals | Vascular endothelial growth factor | Apoptosis
Journal Article
Experimental and Clinical Cardiology, ISSN 1205-6626, 12/2010, Volume 15, Issue 4, p. 61
Journal Article
ISSN 1463-9076, 12/2018, Volume 2, Issue 48, pp. 3247 - 3256
The kinetics and mechanism of ferrate( iv ), ( v ) and ( vi ) transformations in water and in polar organic solvents (namely ethanol and tetrahydrofuran) have... 
Journal Article
Physiological Genomics, ISSN 1094-8341, 07/2018, Volume 50, Issue 7, pp. 532 - 541
Recently we have shown that adaptation to continuous normobaric hypoxia (CNH) decreases myocardial ischemia/reperfusion injury in spontaneously hypertensive... 
Hypoxia | SHR strains | Cardioprotection | Akt pathway | Mitochondrial genome | hypoxia | DEPENDENT ANION CHANNEL | PHYSIOLOGY | PROTEIN-KINASE-B | REPERFUSION INJURY | CARDIAC METABOLISM | mitochondrial genome | CELL BIOLOGY | HEXOKINASE-II | HIGH-ALTITUDE HYPOXIA | GLUCOSE-TRANSPORTER | GLUT4 TRANSLOCATION | GENETICS & HEREDITY | cardioprotection | ENDOPLASMIC-RETICULUM | EXPRESSION | Glucose Transport Proteins, Facilitative - metabolism | Rats, Inbred SHR | Mitochondria, Heart - metabolism | Species Specificity | Genome, Mitochondrial - genetics | Male | Signal Transduction - genetics | Hypoxia-Inducible Factor 1 - metabolism | Proto-Oncogene Proteins c-akt - genetics | Hypoxia-Inducible Factor 1 - genetics | Rats, Inbred BN | Animals | Adaptation, Physiological - genetics | Myocardium - metabolism | Glucose Transport Proteins, Facilitative - genetics | Hypertension - genetics | Hexokinase - genetics | Hexokinase - metabolism | Mitochondria, Heart - genetics | Proto-Oncogene Proteins c-akt - metabolism | Heart | Glucose kinase | Myocardial ischemia | AKT protein | Genomes | mRNA | Kinases | Western blotting | Proteins | Mitochondria | Reperfusion | Ischemia | Sarcoplasmic reticulum | Rodents | Protein transport | Localization | Adaptation | Hypertension | Phenotypes | AKT2 protein | Hexokinase | Polymerase chain reaction | Isoforms | Myocardium | Ventricle | Immunofluorescence
Journal Article