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Nature, ISSN 0028-0836, 12/2010, Volume 468, Issue 7324, pp. 696 - 700
Parkinson's disease is a pervasive, ageing-related neurodegenerative disease the cardinal motor symptoms of which reflect the loss of a small group of neurons,... 
PATHOGENESIS | CELLS | SUBSTANTIA-NIGRA NEURONS | MULTIDISCIPLINARY SCIENCES | DYSFUNCTION | MUTATIONS | MITOCHONDRIAL-DNA DELETIONS | CALCIUM-CHANNEL BLOCKERS | PARKINSONS-DISEASE | Oncogene Proteins - genetics | Oxidative Stress | Calcium - metabolism | Iridoid Glycosides - pharmacology | Male | Neurons - cytology | Cyclophilins - metabolism | Calcium Channels, L-Type - pharmacology | Brain - metabolism | Iridoids | Mitochondrial Proteins - metabolism | Gene Deletion | Superoxides - metabolism | Neurons - metabolism | Parkinson Disease - metabolism | Calcium Signaling | Dopamine - metabolism | Brain - cytology | Parkinson Disease - pathology | Purines - pharmacology | Mitochondrial Proteins - antagonists & inhibitors | Oncogene Proteins - metabolism | Mice, Transgenic | Mitochondria - metabolism | Calcium Channel Blockers - pharmacology | Ion Channels - antagonists & inhibitors | Peroxiredoxins | Protein Deglycase DJ-1 | Parkinson Disease - prevention & control | Animals | Biological Clocks - physiology | Ion Channels - metabolism | Dihydropyridines - pharmacology | Oncogene Proteins - deficiency | Mice | Calcium Channels, L-Type - metabolism | Uncoupling Protein 1 | Development and progression | Calcium channels | Parkinson's disease | Dopaminergic mechanisms | Health aspects | Green fluorescent protein | Proteins | Neurotransmitters | Neurons | Parkinsons disease
Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 06/2018, Volume 128, Issue 6, pp. 2266 - 2280
Journal Article
Nature neuroscience, ISSN 1097-6256, 01/2020, Volume 23, Issue 1, pp. 15 - 20
Monoamine oxidase (MAO) metabolizes cytosolic dopamine (DA), thereby limiting auto-oxidation, but is also thought to generate cytosolic hydrogen peroxide (H O... 
Mitochondria | Dopamine | Monoamine oxidase | Physiological aspects | Metabolism | Observations | Electron transport | Electron transport chain | Hydrogen peroxide | Oxidase | Oxidation | Anchoring | Amine oxidase (flavin-containing)
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 12/2013, Volume 49, Issue 6, pp. 885 - 891
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 2010, Volume 49, Issue 12, pp. 1925 - 1936
Low levels of reactive oxygen species (ROS) can function as redox-active signaling messengers, whereas high levels of ROS induce cellular damage. Menadione... 
Reactive oxygen species | Mitochondria | RoGFP | Free radicals | Redox cycling agents | Apoptosis | OXIDATIVE STRESS | BIOCHEMISTRY & MOLECULAR BIOLOGY | STRESS-INDUCED APOPTOSIS | INJURY | FLUORESCENT PROTEIN INDICATORS | DEFICIENT MICE | CYTOCHROME-C | MITOCHONDRIAL PERMEABILITY TRANSITION | CYCLOPHILIN-D | REDOX GENE-THERAPY | ENDOCRINOLOGY & METABOLISM | ISCHEMIA-REPERFUSION | Fibroblasts - enzymology | Oxidants - pharmacology | Reactive Oxygen Species - metabolism | Oxidative Stress | Apoptosis - drug effects | Cyclophilins - metabolism | Myocytes, Cardiac - enzymology | Glutathione - pharmacology | Cell Death - drug effects | Fibroblasts - metabolism | Recombinant Proteins - metabolism | Green Fluorescent Proteins - metabolism | Signal Transduction | Cytochromes c - metabolism | Mice, Inbred C57BL | Cells, Cultured | Mitochondria - metabolism | Antioxidants - pharmacology | Chick Embryo | Mice, Knockout | Vitamin K 3 - pharmacology | Poly(ADP-ribose) Polymerases - metabolism | Animals | Myocytes, Cardiac - drug effects | Fibroblasts - drug effects | Acetylcysteine - pharmacology | Myocytes, Cardiac - metabolism | Mice | Enzyme Activation | Poly (ADP-Ribose) Polymerase-1 | Mitochondria - physiology | bcl-X Protein - metabolism | Staurosporine - pharmacology | Cytochrome c | Analysis | Genetically modified organisms | Biochemistry | Mitochondrial DNA | Superoxide | Permeability | apoptosis | redox cycling agents | mitochondria
Journal Article
Nature Medicine, ISSN 1078-8956, 07/2019, Volume 25, Issue 7, pp. 1131 - 1142
Huntington’s disease (HD) is a dominantly inherited neurodegenerative disorder caused by a CAG trinucleotide expansion in the huntingtin gene (HTT), which... 
Huntingtons disease | Brain | Transcription factors | Animal models | Polyglutamine | Huntingtin | Neurodegenerative diseases | Neurons | Medical treatment | Trinucleotide repeats | Gene expression | Huntington's disease | Zinc | Proteins | Gene silencing | Molecular modelling | Alleles | Fibroblasts | Zinc finger proteins | Trinucleotide repeat diseases
Journal Article
BBA - Molecular Cell Research, ISSN 0167-4889, 2011, Volume 1813, Issue 7, pp. 1382 - 1394
Journal Article