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Journal Article
Journal of Cutaneous Pathology, ISSN 0303-6987, 12/2017, Volume 44, Issue 12, pp. 1075 - 1079
Journal Article
Journal of Dermatological Science, ISSN 0923-1811, 05/2018, Volume 90, Issue 2, pp. 123 - 134
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 10/2016, Volume 113, Issue 42, pp. 11919 - 11924
Immune checkpoint therapies, such as ipilimumab, induce dramatic antitumor responses in a subset of patients with advanced malignancies, but they may also... 
Toxicities | T cells | Prostate cancer | CD8 | Ipilimumab | CTLA-4 BLOCKADE | PD-1 BLOCKADE | MULTIDISCIPLINARY SCIENCES | toxicities | DOCETAXEL | CHEMOTHERAPY | LUNG-CANCER | prostate cancer | ADVANCED MELANOMA | ipilimumab | PROSTATE-CANCER | PHASE-3 TRIAL | DOUBLE-BLIND | NIVOLUMAB | T-Lymphocyte Subsets - immunology | Humans | Middle Aged | Antineoplastic Combined Chemotherapy Protocols - adverse effects | Male | Ipilimumab - therapeutic use | CD8-Positive T-Lymphocytes - metabolism | Prostatic Neoplasms - drug therapy | Severity of Illness Index | Prostatic Neoplasms - pathology | Disease Susceptibility | Drug-Related Side Effects and Adverse Reactions - etiology | Treatment Outcome | Ipilimumab - adverse effects | Antineoplastic Agents, Immunological - adverse effects | Antineoplastic Agents, Immunological - therapeutic use | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | T-Lymphocyte Subsets - metabolism | Biomarkers | Lymphocyte Count | Prostatic Neoplasms - complications | CD8-Positive T-Lymphocytes - immunology | Clonal Evolution - immunology | Drug-Related Side Effects and Adverse Reactions - diagnosis | Clinical Trials, Phase II as Topic | Drugs | Complications and side effects | Cell research | CD8 lymphocytes | Physiological aspects | Adverse and side effects | Dosage and administration | Research | Health aspects | Biological Sciences
Journal Article
Abdominal Radiology, ISSN 2366-004X, 09/2019, pp. 1 - 12
Adrenal adenoma is the most common adrenal lesion. Due to its wide prevalence, adrenal adenomas may demonstrate various imaging features. Thus, it is important... 
Diagnostic systems | Adenoma | Lesions | Imaging | Cortex | Tumors
Journal Article
BIOCHEMICAL PHARMACOLOGY, ISSN 0006-2952, 11/2015, Volume 98, Issue 1, pp. 144 - 156
Various coenzyme Q(CoQ) analogs have been reported as anti-inflammatory and antioxidant substances. However, coenzyme Q(0) (CoQ(0),... 
RNA INTERFERENCE | VITAMIN-E | Coenzyme Q | Inflammation | Endothelial cells | ADHESION MOLECULE EXPRESSION | IN-VITRO | NF-kappa B | Nrf2 | DISEASE | Heme oxigenase-1 | GENE-EXPRESSION | PHARMACOLOGY & PHARMACY | GROWTH-FACTOR | HEME OXYGENASE-1 | NF-KAPPA-B
Journal Article
Journal Article
JOURNAL OF CUTANEOUS PATHOLOGY, ISSN 0303-6987, 12/2017, Volume 44, Issue 12, pp. 1080 - 1086
Immunotherapies targeting cytotoxic T-lymphocyte-associated antigen 4 (CTLA-4) and the programmed cell death 1 (PD-1) receptor and its ligand (PD-L1) have... 
CHRONIC MYELOGENOUS LEUKEMIA | DERMATOLOGICAL TOXICITIES | BRAF INHIBITORS | MONOCLONAL-ANTIBODY | PATHOLOGY | immune checkpoint antibody | anti-PD-1 | DERMATOLOGY | anti-PD-L1 | METASTATIC MELANOMA | dermatologic toxicity | anti-CTLA-4 | OF-THE-LITERATURE | NEUTROPHILIC PANNICULITIS | CELL-CARCINOMA | CLINICOPATHOLOGICAL FEATURES | PHASE-2 TRIAL
Journal Article
Archives of Toxicology, ISSN 0340-5761, 10/2017, Volume 91, Issue 10, pp. 3341 - 3364
Flavokawain B (FKB), a naturally occurring chalcone in kava extracts, has been reported to possess anticancer activity. However, the effect of FKB on gastric... 
Biomedicine, general | Biomedicine | Environmental Health | Occupational Medicine/Industrial Medicine | ROS | AGS cells | ATG4B | Pharmacology/Toxicology | Flavokawain B | Autophagy | APOPTOSIS | DOWN-REGULATION | CYCLE ARREST | P38 MAPK | RATS | ROS GENERATION | KAVA EXTRACT | BREAST-CANCER | COLON-CANCER CELLS | IN-VIVO | TOXICOLOGY | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Humans | bcl-2-Associated X Protein - metabolism | Stomach Neoplasms - metabolism | Stomach Neoplasms - pathology | Autophagy - physiology | Stomach Neoplasms - drug therapy | Autophagy - drug effects | Xenograft Model Antitumor Assays | Proto-Oncogene Proteins c-bcl-2 - metabolism | Animals | Cysteine Endopeptidases - metabolism | Signal Transduction - drug effects | Mice, Nude | Cell Line, Tumor | Female | Flavonoids - pharmacology | Mice, Inbred BALB C | Cell Death - drug effects | Antineoplastic Agents, Phytogenic - pharmacology | Beclin-1 - metabolism | Autophagy-Related Proteins - metabolism | bcl-2-Associated X Protein - genetics | Prevention | Growth | Cell death | Biochemistry | Stomach cancer | Cells | Cancer | TOR protein | Bax protein | Bcl-2 protein | Acetylcysteine | Cytotoxicity | AKT protein | Activation | Caspase-3 | Anticancer properties | Proteins | Signal transduction | Cascades | Rodents | Biocompatibility | Inhibition | Pretreatment | Gastric cancer | Mortality | Poly(ADP-ribose) polymerase | JNK protein | Organelles | ErbB-2 protein | 1-Phosphatidylinositol 3-kinase | Molecular modelling | Hepatocytes | Lymphocytes B | Antitumor activity | Mice | In vivo methods and tests | Phagocytosis | Tumors | Apoptosis
Journal Article
BIOCHIMICA ET BIOPHYSICA ACTA-GENE REGULATORY MECHANISMS, ISSN 1874-9399, 02/2016, Volume 1859, Issue 2, pp. 246 - 261
Coenzyme Q (CoQ) analogs with variable number of isoprenoid units have been demonstrated as anti-inflammatory and antioxidant/pro-oxidant molecules. In this... 
VITAMIN-E | PROTEIN | OXIDATIVE DAMAGE | ANTIOXIDANT-RESPONSE ELEMENT | BIOCHEMISTRY & MOLECULAR BIOLOGY | KINASE | Coenzyme Q | Inflammation | Macrophages | BIOPHYSICS | P38 | Nrf2 | DISEASE | Antioxidant response element | NF-KAPPA-B | TRANSCRIPTION FACTOR | EXPRESSION | NF kappa B
Journal Article
Environmental Toxicology, ISSN 1520-4081, 08/2017, Volume 32, Issue 8, pp. 2070 - 2084
Cigarette smoke exposure activates several cellular mechanisms predisposing to atherosclerosis, including oxidative stress, dyslipidemia, and vascular... 
atherosclerosis | smooth muscle cells | cigarette smoke | inflammation | Antrodia camphorata | ROS | OXIDATIVE STRESS | ACTIVATION | ANGIOGENESIS | TRANSCRIPTION | WATER RESOURCES | FACTOR-KAPPA-B | INDUCTION | ENVIRONMENTAL SCIENCES | INHIBITION | TOXICOLOGY | NEOVASCULARIZATION | TUMOR-GROWTH | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Humans | NF-kappa B - metabolism | Lung - cytology | Transcription Factor AP-1 - metabolism | Inflammation - metabolism | Tobacco Products | Female | Muscle, Smooth, Vascular - drug effects | DNA Damage - drug effects | Cell Line | Atherosclerosis - pathology | Anti-Inflammatory Agents - pharmacology | Smoke - adverse effects | Rats | Antioxidants - pharmacology | Atherosclerosis - metabolism | Antrodia - chemistry | Muscle, Smooth, Vascular - pathology | Animals | Apolipoproteins E - genetics | Fibroblasts - drug effects | Culture Media - pharmacology | Tobacco - chemistry | Fibroblasts - cytology | Mice | Oxidative Stress - drug effects | Atherosclerosis - prevention & control | Platelet-derived growth factor | DNA damage | Smooth muscle | Inflammation | Triglycerides | Apolipoproteins | Prevention | Antioxidants | DNA | Atherosclerosis | Vascular endothelial growth factor | Smoking | Apoptosis | Cell culture | Oxidative stress | Transcription factors | Bax protein | Bcl-2 protein | Dyslipidemia | Stimulation | Smoke | Damage prevention | Degradation | Catalase | Apolipoprotein E | Attenuation | Inhibition | c-Fos protein | Deoxyribonucleic acid--DNA | NF-κB protein | Tobacco smoke | EGR-1 protein | Mortality | Oral administration | c-Jun protein | Cholesterol | Lungs | Cell death | Arteriosclerosis | Aberration | Cigarette smoke | Index Medicus
Journal Article