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Nature Medicine, ISSN 1078-8956, 2015, Volume 21, Issue 3, pp. 231 - 238
Journal Article
PLoS Biology, ISSN 1544-9173, 07/2016, Volume 14, Issue 7, p. e1002507
Journal Article
Cancer Research, ISSN 0008-5472, 08/2018, Volume 78, Issue 15, pp. 4215 - 4228
Syntaphilin (SNPH) inhibits the movement of mitochondria in tumor cells, preventing their accumulation at the cortical cytoskeleton and limiting the... 
CANCER-CELLS | E3 LIGASE | TRANSPORT | CHIP | PROTEIN | ONCOLOGY | TRACKING | TRAFFICKING | DEGRADATION | MICROTUBULE DYNAMICS | IDENTIFICATION | Ubiquitin | Regulators | Motility | Tumor cells | Trafficking | Cortex | Metastasis | Chemotaxis | Fission | Metastases | Proteins | Mitochondria | Ubiquitination | Tubulin | Bioenergetics | Dynamics | Proteomics | Dynamin | Cytoskeleton | Inhibition | Ubiquitin-protein ligase | Cancer | Tumors | tubulin | metastasis | syntaphilin | ubiquitin
Journal Article
Nature Communications, ISSN 2041-1723, 2013, Volume 4, Issue 1, pp. 2139 - 2139
Journal Article
Journal Article
Nature Communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 631 - 11
Journal Article
Journal Article
Nature Communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 4116 - 9
Inactivation of the subunits of SWI/SNF complex such as ARID1A is synthetically lethal with inhibition of EZH2 activity. However, mechanisms of de novo... 
METHYLATION | BRG1 | MULTIDISCIPLINARY SCIENCES | COMPLEXES | SYNTHETIC LETHALITY | LYMPHOMA | MUTANT CANCERS | MUTATIONS | BRM | CARCINOMA | CANCERS DEPEND | Enhancer of Zeste Homolog 2 Protein - antagonists & inhibitors | Humans | Gene Expression Regulation, Neoplastic | Indoles - administration & dosage | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Ovarian Neoplasms - genetics | Pyridones - administration & dosage | Female | Indoles - pharmacology | Ovarian Neoplasms - metabolism | Tumor Cells, Cultured | Nuclear Proteins - genetics | DNA Helicases - genetics | Ovarian Neoplasms - drug therapy | Aniline Compounds - administration & dosage | Enhancer of Zeste Homolog 2 Protein - genetics | Enhancer of Zeste Homolog 2 Protein - metabolism | Chromosomal Proteins, Non-Histone - metabolism | Nuclear Proteins - metabolism | Transcription Factors - genetics | Mice, SCID | Xenograft Model Antitumor Assays - methods | Chromosomal Proteins, Non-Histone - genetics | Mice, Knockout | Transcription Factors - metabolism | DNA Helicases - metabolism | Drug Resistance, Neoplasm - genetics | Animals | Cell Line, Tumor | Mice, Inbred NOD | Mutation | Pyridones - pharmacology | Sulfonamides - administration & dosage | Drug Resistance, Neoplasm - drug effects | Animal models | Inhibitors | Deactivation | SWI/SNF complex | Catalysis | Inhibition | Inactivation | Apoptosis | Catalytic subunits
Journal Article
PLoS Pathogens, ISSN 1553-7366, 07/2017, Volume 13, Issue 7, p. e1006517
The chemical probe C60 efficiently triggers Epstein-Barr Virus (EBV) reactivation from latency through an unknown mechanism. Here, we identify the Cullin... 
PROTEIN | TRANSCRIPTION | MICROBIOLOGY | CULLIN-RING LIGASES | LYTIC-SWITCH | UBIQUITIN LIGASE COMPLEX | CAND1 | THERAPY | BZLF1 | VIROLOGY | EBV | LATENCY | PARASITOLOGY | Antiviral Agents - pharmacology | Gene Expression Regulation, Viral - drug effects | Phosphorylation | Herpesvirus 4, Human - genetics | Epstein-Barr Virus Infections - virology | Herpesvirus 4, Human - physiology | Humans | Herpesvirus 4, Human - drug effects | Tumor Suppressor Protein p53 - metabolism | Ubiquitin - metabolism | Fullerenes - pharmacology | Ubiquitin - genetics | Transcription Factors - genetics | Tumor Suppressor Protein p53 - genetics | Cullin Proteins - genetics | Transcription Factors - metabolism | Virus Activation - drug effects | Protein Binding - drug effects | Cullin Proteins - metabolism | Epstein-Barr Virus Infections - genetics | Epstein-Barr Virus Infections - metabolism | Ubiquitin | Epstein-Barr virus | Genetic aspects | Buckminsterfullerene | Research | Tumor proteins | Visualization | Profiling | Funding | Stabilization | p53 Protein | Viruses | Infections | Activation | Kinases | Cullin | Proteins | Perturbation methods | Transcription activation | Libraries | Supervision | Deoxyribonucleic acid--DNA | Enzymes | Gene expression | Substrates | Latency | Medicine | Fullerenes | Lymphomas | Tumors | Deoxyribonucleic acid | DNA
Journal Article