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1. Full Text New genetic loci link adipose and insulin biology to body fat distribution
by Shungin, Dmitry and Winkler, Thomas W and Croteau-Chonka, Damien C and Ferreira, Teresa and Locke, Adam E and Mägi, Reedik and Strawbridge, Rona J and Pers, Tune H and Fischer, Krista and Justice, Anne E and Workalemahu, Tsegaselassie and Wu, Joseph M. W and Buchkovich, Martin L and Heard-Costa, Nancy L and Roman, Tamara S and ng, Alexander W and Song, Ci and Gustafsson, Stefan and Day, Felix R and Esko, Tonu and Fall, Tove and Kutalik, Zoltán and Luan, Jian'an and Randall, Joshua C and Scherag, Ané and Vedantam, Sailaja and Wood, Anew R and Chen, Jin and Fehrmann, Rudolf and Karjalainen, Juha and Kahali, Bratati and Liu, Ching-Ti and Schmidt, Ellen M and Absher, Devin and Amin, Najaf and Anderson, Denise and Beekman, Marian and Bragg-Gresham, Jennifer L and Buyske, Steven and Demirkan, Ayse and Ehret, Georg B and Feitosa, Mary F and Goel, Anuj and Jackson, Anne U and Johnson, Toby and Kleber, Marcus E and Kristiansson, Kati and Mangino, Massimo and Mateo Leach, Irene and Medina-Gomez, Carolina and Palmer, Cameron D and Pasko, Dorota and Pechlivanis, Sonali and Peters, Marjolein J and Prokopenko, Inga and Stančáková, Alena and Ju Sung, Yun and Tanaka, Toshiko and Teumer, Alexander and van Vliet-Ostaptchouk, Jana V and Yengo, Loïc and Zhang, Weihua and Albrecht, Eva and Ärnlöv, Johan and Arscott, Gillian M and Bandinelli, Stefania and Barrett, Amy and Bellis, Claire and Bennett, Amanda J and Berne, Christian and Blüher, Matthias and Böhringer, Stefan and Bonnet, Fabrice and Böttcher, Yvonne and Bruinenberg, Marcel and Carba, Delia B and Caspersen, Ida H and Clarke, Robert and Daw, E. Warwick and Deelen, Joris and Deelman, Ewa and Delgado, Graciela and Doney, Alex S. F and Eklund, Niina and Erdos, Michael R and Estrada, Karol and Eury, Elodie and Frieich, Nele and Garcia, Melissa E and Gieaitis, Vilmantas and Gigante, Bruna and Go, Alan S and Golay, Alain and Grallert, Harald and Grammer, Tanja B and Gräßler, Jürgen and Grewal, Jagvir and Groves, Christopher J and Haller, Toomas and Hallmans, Goran and ... and PAGE Consortium and LifeLines Cohort Study and CARDIOGRAMplusC4D Consortium and MuTHER Consortium and ICBP and ADIPOGEN Consortium and CKDGen Consortium and GEFOS Consortium and Int Endogene Consortium and GENIE Consortium and MAGIC Investigators and ReproGen Consortium and GLGC and ADIPOGen Consortium and International Endogene Consortium and The PAGE Consortium and The International Endogene Consortium and The MAGIC Investigators and The ReproGen Consortium and The CKDGen Consortium and The ADIPOGen Consortium and The ICBP and The CARDIOGRAMplusC4D Consortium and The GENIE Consortium and The GLGC and The GEFOS Consortium and The MuTHER Consortium and The LifeLines Cohort Study and Högskolan Dalarna and Akademin Utbildning, hälsa och samhälle and Medicinsk vetenskap
Nature, ISSN 0028-0836, 2015, Volume 518, Issue 7538, pp. 187 - U378
Body fat distribution is a heritable trait and a well-established predictor of adverse metabolic outcomes, independent of overall adiposity. To increase our... 
HUMAN HEIGHT | ABDOMINAL ADIPOSITY | MULTIDISCIPLINARY SCIENCES | COMMON SNPS | SUSCEPTIBILITY LOCI | GLYCEMIC TRAITS | FALSE DISCOVERY | MESENCHYMAL STEM-CELLS | GENOME-WIDE ASSOCIATION | ADIPOGENIC DIFFERENTIATION | SEXUAL-DIMORPHISM | Body Mass Index | Genome-Wide Association Study | Age Factors | Neovascularization, Physiologic - genetics | Epigenesis, Genetic | Humans | Male | Continental Population Groups - genetics | Sex Characteristics | Obesity - genetics | Europe - ethnology | Genome, Human - genetics | Adipose Tissue - metabolism | Insulin - metabolism | Models, Biological | Adipocytes - metabolism | Insulin Resistance - genetics | Polymorphism, Single Nucleotide - genetics | Female | Body Fat Distribution | Transcription, Genetic - genetics | Adipogenesis - genetics | Waist-Hip Ratio | Quantitative Trait Loci - genetics | Adipose tissues | Quantitative trait loci | Genetic research | Genetic aspects | Research | Metabolism | Health aspects | Studies | Body mass index | Genealogy | Body fat | Insulin resistance | Genetics | Genomes | Abdomen | Meta-analysis | Index Medicus | Life Sciences | Adipocytes/metabolism Adipogenesis/genetics Adipose Tissue/metabolism Age Factors Body Fat Distribution Body Mass Index Continental Population Groups/genetics Epigenesis, Genetic Europe/ethnology Female Genome, Human/genetics Genome-Wide Association Study Humans Insulin/metabolism Insulin Resistance/genetics Male Models, Biological Neovascularization, Physiologic/genetics Obesity/genetics Polymorphism, Single Nucleotide/genetics Quantitative Trait Loci/genetics Sex Characteristics Transcription, Genetic/genetics Waist-Hip Ratio | Clinical Medicine | Hälsa och välfärd | Medical and Health Sciences | Klinisk medicin | Medicin och hälsovetenskap | Health and Welfare
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2. Full Text Uric acid is a risk factor for myocardial infarction and stroke: The Rotterdam Study
by Bos, Michiel J and Koudstaal, Peter J and Hofman, Albert and Witteman, Jacqueline C.M and Breteler, Monique M.B
Stroke, ISSN 0039-2499, 06/2006, Volume 37, Issue 6, pp. 1503 - 1507
Background and Purpose - The role of uric acid as a risk factor for myocardial infarction is controversial, and little is known about its role as a risk factor... 
Myocardial infarction | Cerebrovascular disorders | Stroke | Epidemiology | Risk factors | MORTALITY | risk factors | myocardial infarction | FOLLOW-UP | stroke | BLOOD-PRESSURE | CLINICAL NEUROLOGY | epidemiology | ALL-CAUSE | HYPERURICEMIA | MIDDLE-AGED MEN | CARDIOVASCULAR-DISEASE | PERIPHERAL VASCULAR DISEASE | COHORT | cerebrovascular disorders | CORONARY-HEART-DISEASE | HYPERTENSION | Brain Ischemia - complications | Cardiovascular Diseases - etiology | Prospective Studies | Humans | Middle Aged | Risk Factors | Proportional Hazards Models | Male | Case-Control Studies | Stroke - etiology | Hypertension - complications | Female | Aged | Uric Acid - blood | Cerebral Hemorrhage - complications | Myocardial Infarction - etiology | Cohort Studies | Index Medicus
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3. Full Text Discovery and refinement of loci associated with lipid levels
by Willer, Cristen J and Schmidt, Ellen M and Sengupta, Sebanti and Peloso, Gina M and Gustafsson, Stefan and Kanoni, Stavroula and Ganna, Anea and Chen, Jin and Buchkovich, Martin L and Mora, Samia and Beckmann, Jacques S and Bragg-Gresham, Jennifer L and Chang, Hsing-Yi and Demirkan, Ayşe and den Hertog, Heleen M and Do, Ron and Donnelly, Louise A and Ehret, Georg B and Esko, Tõnu and Feitosa, Mary F and Ferreira, Teresa and Fischer, Krista and Fontanillas, Pierre and Fraser, Ross M and Freitag, Daniel F and Gurdasani, Deepti and Heikkilä, Kauko and Hyppönen, Elina and Isaacs, Aaron and Jackson, Anne U and Johansson, Asa and Johnson, Toby and Kaakinen, Marika and Kettunen, Johannes and Kleber, Marcus E and Li, Xiaohui and Luan, Jian'an and Lyytikäinen, Leo-Pekka and Magnusson, Patrik K. E and Mangino, Massimo and Mihailov, Evelin and Montasser, May E and Müller-Nurasyid, Martina and Nolte, Ilja M and O'Connell, Jeffrey R and Palmer, Cameron D and Perola, Markus and Petersen, Ann-Kristin and Sanna, Serena and Saxena, Richa and Service, Susan K and Shah, Sonia and Shungin, Dmitry and Sidore, Carlo and Song, Ci and Strawbridge, Rona J and Surakka, Ida and Tanaka, Toshiko and Teslovich, Tanya M and Thorleifsson, Gudmar and van den Herik, Evita G and Voight, Benjamin F and Volcik, Kelly A and Waite, Lindsay L and Wong, Anew and Wu, Ying and Zhang, Weihua and Absher, Devin and Asiki, Gershim and Barroso, Inês and Been, Latonya F and Bolton, Jennifer L and Bonnycastle, Lori L and Brambilla, Paolo and Burnett, Mary S and Cesana, Giancarlo and Dimitriou, Maria and Doney, Alex S. F and Döring, Angela and Elliott, Paul and Epstein, Stephen E and Eyjolfsson, Gudmundur Ingi and Gigante, Bruna and Goodarzi, Mark O and Grallert, Harald and Gravito, Martha L and Groves, Christopher J and Hallmans, Göran and Hartikainen, Anna-Liisa and Hayward, Caroline and Hernandez, Dena and Hicks, Anew A and Holm, Hilma and Hung, Yi-Jen and Illig, Thomas and Jones, Michelle R and Kaleebu, Pontiano and Kastelein, John J. P and Khaw, Kay-Tee and Kim, Eric and ... and Global Lipids Genetics Consortium and Medicinska fakulteten and Institutionen för folkhälsa och klinisk medicin and Institutionen för odontologi and Näringsforskning and Umeå universitet and Medicin
Nature genetics, ISSN 1061-4036, 2013, Volume 45, Issue 11, pp. 1274 - 1283
Levels of low-density lipoprotein (LDL) cholesterol, high-density lipoprotein (HDL) cholesterol, triglycerides and total cholesterol are heritable, modifiable... 
DENSITY-LIPOPROTEIN CHOLESTEROL | HEART-DISEASE | HOMEOSTASIS | METAANALYSIS | PLASMA | GENE | GENETICS & HEREDITY | SUSCEPTIBILITY LOCI | RISK | IDENTIFIES 13 | TRAITS | European Continental Ancestry Group - genetics | Genetic Predisposition to Disease | Genome-Wide Association Study | Lipids - genetics | Humans | Asian Continental Ancestry Group - genetics | Genotype | Cholesterol, HDL - genetics | Coronary Artery Disease - blood | Cholesterol, LDL - genetics | Lipids - blood | Coronary Artery Disease - genetics | Triglycerides - blood | Cholesterol, HDL - blood | Cholesterol, LDL - blood | African Continental Ancestry Group - genetics | Triglycerides - genetics | Quantitative trait loci | Proteolipids | Lipoproteins | Genetic aspects | Blood lipoproteins | Biological control systems | Observations | Identification and classification | Studies | Lipids | Genes | Cholesterol | Index Medicus | Basic Medicine | Medical Genetics | Medicinsk genetik | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper
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4. Full Text Common variants associated with plasma triglycerides and risk for coronary artery disease
by Do, Ron and Willer, Cristen J and Schmidt, Ellen M and Sengupta, Sebanti and Gao, Chi and Peloso, Gina M and Gustafsson, Stefan and Kanoni, Stavroula and Ganna, Anea and Chen, Jin and Buchkovich, Martin L and Mora, Samia and Beckmann, Jacques S and Bragg-Gresham, Jennifer L and Chang, Hsing-Yi and Demirkan, Ayşe and den Hertog, Heleen M and Donnelly, Louise A and Ehret, Georg B and Esko, Tõnu and Feitosa, Mary F and Ferreira, Teresa and Fischer, Krista and Fontanillas, Pierre and Fraser, Ross M and Freitag, Daniel F and Gurdasani, Deepti and Heikkilä, Kauko and Hyppönen, Elina and Isaacs, Aaron and Jackson, Anne U and Johansson, Asa and Johnson, Toby and Kaakinen, Marika and Kettunen, Johannes and Kleber, Marcus E and Li, Xiaohui and Luan, Jian'an and Lyytikäinen, Leo-Pekka and Magnusson, Patrik K. E and Mangino, Massimo and Mihailov, Evelin and Montasser, May E and Müller-Nurasyid, Martina and Nolte, Ilja M and O'Connell, Jeffrey R and Palmer, Cameron D and Perola, Markus and Petersen, Ann-Kristin and Sanna, Serena and Saxena, Richa and Service, Susan K and Shah, Sonia and Shungin, Dmitry and Sidore, Carlo and Song, Ci and Strawbridge, Rona J and Surakka, Ida and Tanaka, Toshiko and Teslovich, Tanya M and Thorleifsson, Gudmar and van den Herik, Evita G and Voight, Benjamin F and Volcik, Kelly A and Waite, Lindsay L and Wong, Anew and Wu, Ying and Zhang, Weihua and Absher, Devin and Asiki, Gershim and Barroso, Inês and Been, Latonya F and Bolton, Jennifer L and Bonnycastle, Lori L and Brambilla, Paolo and Burnett, Mary S and Cesana, Giancarlo and Dimitriou, Maria and Doney, Alex S. F and Döring, Angela and Elliott, Paul and Epstein, Stephen E and Eyjolfsson, Gudmundur Ingi and Gigante, Bruna and Goodarzi, Mark O and Grallert, Harald and Gravito, Martha L and Groves, Christopher J and Hallmans, Göran and Hartikainen, Anna-Liisa and Hayward, Caroline and Hernandez, Dena and Hicks, Anew A and Holm, Hilma and Hung, Yi-Jen and Illig, Thomas and Jones, Michelle R and Kaleebu, Pontiano and Kastelein, John J. P and Khaw, Kay-Tee and ... and Medicinska fakulteten and Institutionen för folkhälsa och klinisk medicin and Institutionen för odontologi and Näringsforskning and Umeå universitet and Medicin
Nature genetics, ISSN 1061-4036, 2013, Volume 45, Issue 11, pp. 1345 - 1352
Triglycerides are transported in plasma by specific triglyceride-rich lipoproteins; in epidemiological studies, increased triglyceride levels correlate with... 
CONTRIBUTE | THERAPY | CHOLESTEROL LEVELS | GENETICS & HEREDITY | CARDIOVASCULAR-DISEASE | ISCHEMIC-HEART-DISEASE | LOCI | MENDELIAN RANDOMIZATION | TYPE-2 DIABETES-MELLITUS | GENETIC EPIDEMIOLOGY | HIGH-DENSITY-LIPOPROTEIN | Humans | Risk Factors | Cholesterol, HDL - genetics | Coronary Artery Disease - blood | Triglycerides - metabolism | Cholesterol, LDL - genetics | Biological Transport | Coronary Artery Disease - genetics | Triglycerides - blood | Cholesterol, HDL - blood | Cholesterol, LDL - blood | Coronary Artery Disease - epidemiology | Polymorphism, Single Nucleotide | Triglycerides - genetics | Genetic susceptibility | Genetic variation | Triglycerides | Genetic aspects | Properties | Coronary heart disease | Identification and classification | Testing | Plasma | Cardiovascular disease | Lipids | Genomes | Estimates | Cholesterol | Index Medicus | Basic Medicine | Medical Genetics | Medicinsk genetik | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper
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5. Full Text Atrial Fibrillation and Cognitive Function
by Diener, Hans-Christoph and Hart, Robert G and Koudstaal, Peter J and Lane, Deirdre A and Lip, Gregory Y.H
Journal of the American College of Cardiology, ISSN 0735-1097, 02/2019, Volume 73, Issue 5, pp. 612 - 619
Numerous vascular risk factors and vascular diseases contribute to cognitive impairment and dementia. Many studies and registries show an association of atrial... 
dementia | atrial fibrillation | cognitive impairment | anticoagulation
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6. Full Text Vitamin D Status and Risk of Stroke: The Rotterdam Study
by Berghout, Brian P and Fani, Lana and Heshmatollah, Alis and Koudstaal, Peter J and Ikram, M Arfan and Zillikens, M Carola and Ikram, M Kamran
Stroke, ISSN 0039-2499, 09/2019, Volume 50, Issue 9, pp. 2293 - 2298
BACKGROUND AND PURPOSE—Recent findings suggest that vitamin D, a neuroprotective prohormone, is involved in the pathogenesis of cardiovascular disease.... 
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7. Full Text Rare variant in scavenger receptor BI raises HDL cholesterol and increases risk of coronary heart disease
by Zanoni, Paolo and Khetarpal, Sumeet A and Larach, Daniel B and Hancock-Cerutti, William F and Millar, John S and Cuchel, Marina and DerOhannessian, Stephanie and Kontush, Anatol and Surenan, Praveen and Saleheen, Danish and Trompet, Stella and Jukema, J. Wouter and de Craen, Anton and Deloukas, Panos and Sattar, Naveed and Ford, Ian and Packard, Chris and Majumder, Abdullah al Shafi and Alam, Dewan S and Di Angelantonio, Emanuele and Abecasis, Goncalo and Chowdhury, Rajiv and Erdmann, Jeanette and Nordestgaard, Børge G and Nielsen, Sune F and Tybjærg-Hansen, Anne and Schmidt, Ruth Frikke and Kuulasmaa, Kari and Liu, Dajiang J and Perola, Markus and Blankenberg, Stefan and Salomaa, Veikko and Männistö, Satu and Amouyel, Philippe and Arveiler, Dominique and Ferrieres, Jean and Müller-Nurasyid, Martina and Ferrario, Marco and Kee, Frank and Willer, Cristen J and Samani, Nilesh and Schunkert, Heribert and Butterworth, Adam S and Howson, Joanna M. M and Peloso, Gina M and Stitziel, Nathan O and Danesh, John and Kathiresan, Sekar and Rader, Daniel J and Watson, Sarah and Schmidt, Ellen M and Sengupta, Sebanti and Gustafsson, Stefan and Kanoni, Stavroula and Ganna, Anea and Chen, Jin and Buchkovich, Martin L and Mora, Samia and Beckmann, Jacques S and Bragg-Gresham, Jennifer L and Chang, Hsing-Yi and Demirkan, Ayşe and den Hertog, Heleen M and Do, Ron and Donnelly, Louise A and Ehret, Georg B and Esko, Tõnu and Feitosa, Mary F and Ferreira, Teresa and Fischer, Krista and Fontanillas, Pierre and Fraser, Ross M and Freitag, Daniel F and Gurdasani, Deepti and Heikkilä, Kauko and Hyppönen, Elina and Isaacs, Aaron and Jackson, Anne U and Johansson, Åsa and Johnson, Toby and Kaakinen, Marika and Kettunen, Johannes and Kleber, Marcus E and Li, Xiaohui and Luan, Jian'an and Lyytikäinen, Leo-Pekka and Magnusson, Patrik K. E and Mangino, Massimo and Mihailov, Evelin and Montasser, May E and Nolte, Ilja M and O'Connell, Jeffrey R and Palmer, Cameron D and Petersen, Ann-Kristin and Sanna, Serena and Saxena, Richa and Service, Susan K and Shah, Sonia and Shungin, Dmitry and Sidore, Carlo and ... and Consortium, CE and Global Lipids Genetics Consortium and CARDIoGRAM Exome Consortium and CHD Exome+ Consortium
Science, ISSN 0036-8075, 2016, Volume 351, Issue 6278, pp. 1166 - 1171
Scavenger receptor BI (SR-BI) is the major receptor for high-density lipoprotein (HDL) cholesterol (HDL-C). In humans, high amounts of HDL-C in plasma are... 
ESTER | SELECTIVE LIPID UPTAKE | METABOLISM | MULTIDISCIPLINARY SCIENCES | I SR-BI | TARGETED MUTATION | ATHEROSCLEROSIS | DEFICIENT MICE | EXPRESSION | HIGH-DENSITY-LIPOPROTEIN | TRANSGENIC MICE | Coronary Disease - blood | Humans | Middle Aged | Male | Risk | Scavenger Receptors, Class B - genetics | Genetic Variation | Proline - genetics | Homozygote | Animals | Coronary Disease - genetics | DNA Mutational Analysis | Cholesterol, HDL - blood | Female | Heterozygote | Aged | Mice | Protein Processing, Post-Translational | Scavenger Receptors, Class B - metabolism | Leucine - genetics | Amino Acid Substitution | Physiological aspects | Blood cholesterol | Coronary heart disease | Risk factors | Cholesterol, HDL | Health aspects | Genotype & phenotype | Cardiovascular disease | Lipoproteins | Cholesterol | Index Medicus
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