Journal of Comparative Neurology, ISSN 0021-9967, 04/2017, Volume 525, Issue 5, pp. 1094 - 1108
The mesolimbic dopamine (DA) circuitry determines which behaviors are positively reinforcing and therefore should be encoded in the memory to become a part of...
RRID: AB_11180326 | ventral tegmental area | RRID: AB_11180858 | RRID:AB_1524535 | nucleus accumbens | oxytocin receptors | dopamine | glutamate | FIELD | LATERAL HABENULA | RELEASE | NEUROSCIENCES | ADDICTION | ZOOLOGY | SYNAPSES | REGIONS | AMPHETAMINE | MIDBRAIN | Immunohistochemistry | Mice, Inbred C57BL | Nucleus Accumbens - metabolism | Male | Mice, Transgenic | Animals | In Situ Hybridization | Image Processing, Computer-Assisted | Ventral Tegmental Area - metabolism | Neural Pathways - metabolism | Glutamic Acid - metabolism | Mice | Neurons - metabolism | Receptors, Oxytocin - biosynthesis | Dopamine - metabolism | Tyrosine | Phenols | Glutamate | Gene expression | Neurons
RRID: AB_11180326 | ventral tegmental area | RRID: AB_11180858 | RRID:AB_1524535 | nucleus accumbens | oxytocin receptors | dopamine | glutamate | FIELD | LATERAL HABENULA | RELEASE | NEUROSCIENCES | ADDICTION | ZOOLOGY | SYNAPSES | REGIONS | AMPHETAMINE | MIDBRAIN | Immunohistochemistry | Mice, Inbred C57BL | Nucleus Accumbens - metabolism | Male | Mice, Transgenic | Animals | In Situ Hybridization | Image Processing, Computer-Assisted | Ventral Tegmental Area - metabolism | Neural Pathways - metabolism | Glutamic Acid - metabolism | Mice | Neurons - metabolism | Receptors, Oxytocin - biosynthesis | Dopamine - metabolism | Tyrosine | Phenols | Glutamate | Gene expression | Neurons
Journal Article
The Milbank Quarterly, ISSN 0887-378X, 12/2018, Volume 96, Issue 4, pp. 635 - 671
Policy Points A consensus regarding the need to orient health systems to address inequities is emerging, with much of this discussion targeting population...
social determinants of health | models (theoretical) | cohort studies | health equity | primary health care | primary care | quality of care | PSYCHOMETRIC PROPERTIES | SELF-EFFICACY | CHRONIC PAIN | SOCIAL DETERMINANTS | MENTAL-HEALTH | QOL 8-ITEM INDEX | PATIENT-CENTERED CARE | CULTURAL COMPETENCE | HEALTH CARE SCIENCES & SERVICES | BEHAVIOR-CHANGE | HEALTH POLICY & SERVICES | INTIMATE PARTNER VIOLENCE | Medical research | Primary health care | Analysis | Post-traumatic stress disorder | Medicine, Experimental | Medical care | Quality management | Chronic posttraumatic stress disorder | Health care | Intervention | Ageism | Identification methods | Health problems | Equity | Posttraumatic stress disorder | Mental depression | Clinical outcomes | Conditions | Disability | Pain | Sexism | Confidence | Investments | Mental health services | Patient satisfaction | Care management | Path analysis | Economic stress | Clinics | Violence | Aggression | Health professional-Patient communication | Data processing | Patients | Trauma | Health care services policy | Trust | Empirical analysis | Primary care | Chronic pain | Quality of life | Strain | Health status | Discrimination | Structured interviews | Comfort | Symptoms | Quality of care | Health disparities
social determinants of health | models (theoretical) | cohort studies | health equity | primary health care | primary care | quality of care | PSYCHOMETRIC PROPERTIES | SELF-EFFICACY | CHRONIC PAIN | SOCIAL DETERMINANTS | MENTAL-HEALTH | QOL 8-ITEM INDEX | PATIENT-CENTERED CARE | CULTURAL COMPETENCE | HEALTH CARE SCIENCES & SERVICES | BEHAVIOR-CHANGE | HEALTH POLICY & SERVICES | INTIMATE PARTNER VIOLENCE | Medical research | Primary health care | Analysis | Post-traumatic stress disorder | Medicine, Experimental | Medical care | Quality management | Chronic posttraumatic stress disorder | Health care | Intervention | Ageism | Identification methods | Health problems | Equity | Posttraumatic stress disorder | Mental depression | Clinical outcomes | Conditions | Disability | Pain | Sexism | Confidence | Investments | Mental health services | Patient satisfaction | Care management | Path analysis | Economic stress | Clinics | Violence | Aggression | Health professional-Patient communication | Data processing | Patients | Trauma | Health care services policy | Trust | Empirical analysis | Primary care | Chronic pain | Quality of life | Strain | Health status | Discrimination | Structured interviews | Comfort | Symptoms | Quality of care | Health disparities
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2017, Volume 12, Issue 7, p. e0180738
Activation of the angiotensin II type 2 receptor (AT2R) by administration of Compound 21 (C21), a selective AT2R agonist, induces neuroprotection in models of...
Receptor, Angiotensin, Type 2 - agonists | Animals | Neuroprotective Agents - pharmacology | Rats | Female | Male | Mice | Rats, Sprague-Dawley | Stroke - physiopathology | Stroke (Disease) | Care and treatment | Receptors | Angiotensin II receptor blockers | Angiotensin | Physiological aspects | Dosage and administration | Research | Health aspects | Risk factors | Occlusion | Neuroimaging | Neuroprotection | Brain | Neurosciences | Animal models | Genomics | Medical services | Activation | Ischemia | Cerebral blood flow | Rodents | Physiology | Localization | Angiotensin II | Activation analysis | Stemming | Medical research | Stroke | Neurons | Astrocytes | Transgenic mice | Microglia | Medicine | Brain research | Magnetic resonance imaging | Resonance | Receptor mechanisms | Laboratory animals | Veins & arteries
Receptor, Angiotensin, Type 2 - agonists | Animals | Neuroprotective Agents - pharmacology | Rats | Female | Male | Mice | Rats, Sprague-Dawley | Stroke - physiopathology | Stroke (Disease) | Care and treatment | Receptors | Angiotensin II receptor blockers | Angiotensin | Physiological aspects | Dosage and administration | Research | Health aspects | Risk factors | Occlusion | Neuroimaging | Neuroprotection | Brain | Neurosciences | Animal models | Genomics | Medical services | Activation | Ischemia | Cerebral blood flow | Rodents | Physiology | Localization | Angiotensin II | Activation analysis | Stemming | Medical research | Stroke | Neurons | Astrocytes | Transgenic mice | Microglia | Medicine | Brain research | Magnetic resonance imaging | Resonance | Receptor mechanisms | Laboratory animals | Veins & arteries
Journal Article
American journal of physiology. Heart and circulatory physiology, ISSN 0363-6135, 02/2016, Volume 310, Issue 3, pp. H404 - H415
ANG II is thought to increase sympathetic outflow by increasing oxidative stress and promoting local inflammation in the paraventricular nucleus (PVN) of the...
angiotensin II type 1 receptor | microglia | angiotensin II | Toll-like receptor | paraventricular nucleus | Microglia - metabolism | Reactive Oxygen Species - metabolism | Rats, Wistar | Minocycline - pharmacology | Oxidative Stress - immunology | Male | Angiotensin II Type 1 Receptor Blockers - pharmacology | Receptor, Angiotensin, Type 1 - genetics | Microglia - immunology | Reactive Oxygen Species - immunology | Paraventricular Hypothalamic Nucleus - immunology | Angiotensin II - metabolism | Microglia - drug effects | Renin-Angiotensin System - immunology | Losartan - pharmacology | Oxidative Stress - genetics | Rats | Renin-Angiotensin System - genetics | Inflammation | Toll-Like Receptor 4 - immunology | Toll-Like Receptor 4 - metabolism | Mice, Inbred C3H | Mice, Knockout | Angiotensin II - immunology | Immunity, Innate - immunology | Paraventricular Hypothalamic Nucleus - drug effects | Animals | Receptor, Angiotensin, Type 1 - metabolism | Paraventricular Hypothalamic Nucleus - metabolism | Anti-Bacterial Agents - pharmacology | Mice | Cardiovascular Neurohormonal Regulation
angiotensin II type 1 receptor | microglia | angiotensin II | Toll-like receptor | paraventricular nucleus | Microglia - metabolism | Reactive Oxygen Species - metabolism | Rats, Wistar | Minocycline - pharmacology | Oxidative Stress - immunology | Male | Angiotensin II Type 1 Receptor Blockers - pharmacology | Receptor, Angiotensin, Type 1 - genetics | Microglia - immunology | Reactive Oxygen Species - immunology | Paraventricular Hypothalamic Nucleus - immunology | Angiotensin II - metabolism | Microglia - drug effects | Renin-Angiotensin System - immunology | Losartan - pharmacology | Oxidative Stress - genetics | Rats | Renin-Angiotensin System - genetics | Inflammation | Toll-Like Receptor 4 - immunology | Toll-Like Receptor 4 - metabolism | Mice, Inbred C3H | Mice, Knockout | Angiotensin II - immunology | Immunity, Innate - immunology | Paraventricular Hypothalamic Nucleus - drug effects | Animals | Receptor, Angiotensin, Type 1 - metabolism | Paraventricular Hypothalamic Nucleus - metabolism | Anti-Bacterial Agents - pharmacology | Mice | Cardiovascular Neurohormonal Regulation
Journal Article
Medienimpulse, ISSN 2307-3187, 12/2015, Issue 4/2015
This text revisits the long-term project Hidden Curriculum (HC), initiated by Annette Krauss, that addresses unquestioned routines, hierarchies of knowledge...
body | hidden curriculum | discourse | situated knowledge
body | hidden curriculum | discourse | situated knowledge
Journal Article
Medienimpulse, ISSN 2307-3187, 09/2015, Issue 3/2015
This text revisits the long-term project Hidden Curriculum, initiated by Annette Krauss. The project addresses unquestioned routines, hierarchies of knowledge...
freire | rancière | hidden curriculum | geheimer lehrplan
freire | rancière | hidden curriculum | geheimer lehrplan
Journal Article
Physiology & Behavior, ISSN 0031-9384, 2010, Volume 100, Issue 5, pp. 525 - 534
Abstract The renin angiotensin system (RAS; most well-known for its critical roles in the regulation of cardiovascular function and hydromineral balance) has...
Psychiatry | Obesity | Adipose | Renin | Diabetes | Food intake | Angiotensin | HEPATIC TRIGLYCERIDE PRODUCTION | II TYPE-1 RECEPTOR | NECROSIS-FACTOR-ALPHA | INCREASED ENERGY-EXPENDITURE | BROWN ADIPOSE-TISSUE | DIET-INDUCED OBESITY | RANDOMIZED CLINICAL-TRIALS | BETA-CELL FUNCTION | CONVERTING ENZYME-INHIBITION | BEHAVIORAL SCIENCES | PSYCHOLOGY, BIOLOGICAL | BRUSH-BORDER MEMBRANE
Psychiatry | Obesity | Adipose | Renin | Diabetes | Food intake | Angiotensin | HEPATIC TRIGLYCERIDE PRODUCTION | II TYPE-1 RECEPTOR | NECROSIS-FACTOR-ALPHA | INCREASED ENERGY-EXPENDITURE | BROWN ADIPOSE-TISSUE | DIET-INDUCED OBESITY | RANDOMIZED CLINICAL-TRIALS | BETA-CELL FUNCTION | CONVERTING ENZYME-INHIBITION | BEHAVIORAL SCIENCES | PSYCHOLOGY, BIOLOGICAL | BRUSH-BORDER MEMBRANE
Journal Article
Brain Structure and Function, ISSN 1863-2653, 3/2016, Volume 221, Issue 2, pp. 891 - 912
Angiotensin-II acts at its type-1 receptor (AT1R) in the brain to regulate body fluid homeostasis, sympathetic outflow and blood pressure. However, the role of...
Hypertension | Neurology | Brain | Neurosciences | Biomedicine | Renin | Blood pressure | Cell Biology | AT2 | SYMPATHETIC AFFERENT REFLEX | NUCLEUS-TRACTUS-SOLITARIUS | SPONTANEOUSLY HYPERTENSIVE-RATS | MESSENGER-RNA EXPRESSION | ANATOMY & MORPHOLOGY | NEUROSCIENCES | HYPOTHALAMIC PARAVENTRICULAR NUCLEUS | BLOOD-PRESSURE | DORSAL MOTOR NUCLEUS | ROSTRAL VENTROLATERAL MEDULLA | MEDIAL PREFRONTAL CORTEX | LATERAL SEPTAL AREA | Immunohistochemistry | Tyrosine 3-Monooxygenase - metabolism | Central Nervous System - metabolism | Vesicular Glutamate Transport Protein 2 - metabolism | Male | Mice, Transgenic | Preoptic Area - metabolism | RNA, Messenger - metabolism | Brain - metabolism | Animals | Hypothalamus - metabolism | Receptor, Angiotensin, Type 2 - metabolism | Paraventricular Hypothalamic Nucleus - metabolism | Models, Animal | Mice | GABAergic Neurons - metabolism | RNA | Analysis | Journalists | Angiotensin | Physiological aspects | GABA | Genetic engineering | Glutamate | Proteins | Nervous system | Rodents | Neurobiology | blood pressure | brain | hypertension | renin
Hypertension | Neurology | Brain | Neurosciences | Biomedicine | Renin | Blood pressure | Cell Biology | AT2 | SYMPATHETIC AFFERENT REFLEX | NUCLEUS-TRACTUS-SOLITARIUS | SPONTANEOUSLY HYPERTENSIVE-RATS | MESSENGER-RNA EXPRESSION | ANATOMY & MORPHOLOGY | NEUROSCIENCES | HYPOTHALAMIC PARAVENTRICULAR NUCLEUS | BLOOD-PRESSURE | DORSAL MOTOR NUCLEUS | ROSTRAL VENTROLATERAL MEDULLA | MEDIAL PREFRONTAL CORTEX | LATERAL SEPTAL AREA | Immunohistochemistry | Tyrosine 3-Monooxygenase - metabolism | Central Nervous System - metabolism | Vesicular Glutamate Transport Protein 2 - metabolism | Male | Mice, Transgenic | Preoptic Area - metabolism | RNA, Messenger - metabolism | Brain - metabolism | Animals | Hypothalamus - metabolism | Receptor, Angiotensin, Type 2 - metabolism | Paraventricular Hypothalamic Nucleus - metabolism | Models, Animal | Mice | GABAergic Neurons - metabolism | RNA | Analysis | Journalists | Angiotensin | Physiological aspects | GABA | Genetic engineering | Glutamate | Proteins | Nervous system | Rodents | Neurobiology | blood pressure | brain | hypertension | renin
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2010, Volume 107, Issue 47, pp. 20529 - 20534
Individuals often eat calorically dense, highly palatable "comfort" foods during stress for stress relief. This article demonstrates that palatable food intake...
Psychological stress | Amygdala | Corticosterone | Comfort foods | Rats | Pleasure | Dehydration | Lesions | Food consumption | Behavioral neuroscience | Synaptophysin | Anxiety-related behavior | CAMP response element-binding protein | Calcium/calmodulin-dependent protein kinase | synaptophysin | HORMONAL RESPONSES | RAT BASOLATERAL AMYGDALA | PROTEIN-KINASE | MULTIDISCIPLINARY SCIENCES | COMFORT FOOD | OBESITY | cAMP response element-binding protein | LATERAL AMYGDALA | calcium/calmodulin-dependent protein kinase | anxiety-related behavior | LONG-TERM POTENTIATION | corticosterone | INTERNEURONS | PLASTICITY | CORTISOL | Cardiovascular Physiological Phenomena | Amygdala - physiopathology | Amygdala - drug effects | Pleasure - physiology | Stress, Psychological - drug therapy | Amygdala - metabolism | Sucrose - pharmacology | Behavior, Animal - physiology | Male | Telemetry | Amygdala - pathology | Animals | Analysis of Variance | Microarray Analysis | Hormones - blood | Stress, Psychological - physiopathology | Care and treatment | Stress management | Physiological aspects | Anxiety | Dosage and administration | Diagnosis | Binding proteins | Properties | Protein kinases | Methods | Cardiovascular system | Brain | Obesity | Sucrose | mRNA | Gene expression | Stress | Plasticity (functional) | Saccharin | Motivation | Food intake | Reinforcement | Beverages | Food | Biological Sciences | calcium | calmodulin-dependent protein kinase
Psychological stress | Amygdala | Corticosterone | Comfort foods | Rats | Pleasure | Dehydration | Lesions | Food consumption | Behavioral neuroscience | Synaptophysin | Anxiety-related behavior | CAMP response element-binding protein | Calcium/calmodulin-dependent protein kinase | synaptophysin | HORMONAL RESPONSES | RAT BASOLATERAL AMYGDALA | PROTEIN-KINASE | MULTIDISCIPLINARY SCIENCES | COMFORT FOOD | OBESITY | cAMP response element-binding protein | LATERAL AMYGDALA | calcium/calmodulin-dependent protein kinase | anxiety-related behavior | LONG-TERM POTENTIATION | corticosterone | INTERNEURONS | PLASTICITY | CORTISOL | Cardiovascular Physiological Phenomena | Amygdala - physiopathology | Amygdala - drug effects | Pleasure - physiology | Stress, Psychological - drug therapy | Amygdala - metabolism | Sucrose - pharmacology | Behavior, Animal - physiology | Male | Telemetry | Amygdala - pathology | Animals | Analysis of Variance | Microarray Analysis | Hormones - blood | Stress, Psychological - physiopathology | Care and treatment | Stress management | Physiological aspects | Anxiety | Dosage and administration | Diagnosis | Binding proteins | Properties | Protein kinases | Methods | Cardiovascular system | Brain | Obesity | Sucrose | mRNA | Gene expression | Stress | Plasticity (functional) | Saccharin | Motivation | Food intake | Reinforcement | Beverages | Food | Biological Sciences | calcium | calmodulin-dependent protein kinase
Journal Article
Journal of Virology, ISSN 0022-538X, 2015, Volume 89, Issue 13, pp. 6761 - 6772
Gene-engineered CD34(+) hematopoietic stem and progenitor cells (HSPCs) can be used to generate an HIV-1-resistant immune system. However, a certain threshold...
VIROLOGY | CD4 T-CELLS | IN-VIVO | LENTIVIRAL VECTOR | MOUSE MODEL | GENE-THERAPY | ANTIRETROVIRAL THERAPY | ZINC-FINGER NUCLEASES | EXPRESSION | LYMPHOCYTES | STRATEGIES | Disease Resistance | HIV Infections - virology | Humans | Transplantation | Mice, SCID | Viral Load | Gene Knockdown Techniques | HIV Infections - immunology | Hematopoietic Stem Cells - immunology | Receptors, CCR5 - metabolism | Receptors, HIV - antagonists & inhibitors | Animals | HIV-1 - physiology | Virus Attachment | Lentivirus - genetics | Genetic Vectors | Hematopoietic Stem Cells - virology | Genetic Therapy - methods
VIROLOGY | CD4 T-CELLS | IN-VIVO | LENTIVIRAL VECTOR | MOUSE MODEL | GENE-THERAPY | ANTIRETROVIRAL THERAPY | ZINC-FINGER NUCLEASES | EXPRESSION | LYMPHOCYTES | STRATEGIES | Disease Resistance | HIV Infections - virology | Humans | Transplantation | Mice, SCID | Viral Load | Gene Knockdown Techniques | HIV Infections - immunology | Hematopoietic Stem Cells - immunology | Receptors, CCR5 - metabolism | Receptors, HIV - antagonists & inhibitors | Animals | HIV-1 - physiology | Virus Attachment | Lentivirus - genetics | Genetic Vectors | Hematopoietic Stem Cells - virology | Genetic Therapy - methods
Journal Article
Pharmacology and Therapeutics, ISSN 0163-7258, 06/2013, Volume 138, Issue 3, pp. 428 - 440
Hypertension is an epidemic health concern and a major risk factor for the development of cardiovascular disease. Although there are available treatment...
Inflammation | Blood pressure | Renin | Sympathetic nervous system | Angiotensin | Microglia | MIGRATION INHIBITORY FACTOR | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | NECROSIS-FACTOR-ALPHA | NUCLEUS-TRACTUS-SOLITARII | HYPOTHALAMIC PARAVENTRICULAR NUCLEUS | DECREASES BLOOD-PRESSURE | II-INDUCED HYPERTENSION | ROSTRAL VENTROLATERAL MEDULLA | RENIN-ANGIOTENSIN SYSTEM | PHARMACOLOGY & PHARMACY | CENTRAL-NERVOUS-SYSTEM | Animals | Immune System - physiopathology | Obesity - immunology | Humans | Obesity - physiopathology | Renin-Angiotensin System - physiology | Hypertension - immunology | Sympathetic Nervous System - physiopathology | Hypertension - physiopathology | Hypertension | Obesity | Development and progression | Health aspects | angiotensin | blood pressure | inflammation | microglia | renin | sympathetic nervous system
Inflammation | Blood pressure | Renin | Sympathetic nervous system | Angiotensin | Microglia | MIGRATION INHIBITORY FACTOR | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | NECROSIS-FACTOR-ALPHA | NUCLEUS-TRACTUS-SOLITARII | HYPOTHALAMIC PARAVENTRICULAR NUCLEUS | DECREASES BLOOD-PRESSURE | II-INDUCED HYPERTENSION | ROSTRAL VENTROLATERAL MEDULLA | RENIN-ANGIOTENSIN SYSTEM | PHARMACOLOGY & PHARMACY | CENTRAL-NERVOUS-SYSTEM | Animals | Immune System - physiopathology | Obesity - immunology | Humans | Obesity - physiopathology | Renin-Angiotensin System - physiology | Hypertension - immunology | Sympathetic Nervous System - physiopathology | Hypertension - physiopathology | Hypertension | Obesity | Development and progression | Health aspects | angiotensin | blood pressure | inflammation | microglia | renin | sympathetic nervous system
Journal Article