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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2004, Volume 101, Issue 47, pp. 16419 - 16424
Stimulation of cells with various peptide growth factors induces the production of phosphatidylinositol 3,4,5-trisphosphate ( PIP ) through activation of... 
Phagocytes | Molecules | Biological Sciences | Cell growth | Phosphatases | NIH 3T3 cells | HEK293 cells | Memory interference | Cell lines | HeLa cells | Oxidation | Peroxiredoxin | Peptide growth factor receptor | Hydrogen peroxide | peroxiredoxin | PHOSPHATIDYLINOSITOL 3-KINASE | MULTIDISCIPLINARY SCIENCES | hydrogen peroxide | CYSTEINE-SULFINIC ACID | NECROSIS-FACTOR-ALPHA | peptide growth factor receptor | SACCHAROMYCES-CEREVISIAE | SIGNAL-TRANSDUCTION | TYROSINE-PHOSPHATASE 1B | REDOX REGULATION | IN-VIVO | HYDROGEN-PEROXIDE | NIH 3T3 Cells | Tumor Suppressor Proteins - antagonists & inhibitors | Humans | Protein Tyrosine Phosphatases - metabolism | Protein Tyrosine Phosphatases - antagonists & inhibitors | Protein Tyrosine Phosphatases - genetics | Transfection | PTEN Phosphohydrolase | Tumor Suppressor Proteins - genetics | Phosphatidylinositol Phosphates - biosynthesis | Peroxidases - metabolism | NADH, NADPH Oxidoreductases - metabolism | Phosphoric Monoester Hydrolases - antagonists & inhibitors | Cell Line | Insulin - pharmacology | NADH, NADPH Oxidoreductases - genetics | Gene Expression | Phosphoric Monoester Hydrolases - genetics | Tumor Suppressor Proteins - metabolism | Oxidation-Reduction | Signal Transduction | Proto-Oncogene Proteins c-sis | Platelet-Derived Growth Factor - pharmacology | Recombinant Proteins - pharmacology | NADPH Oxidase 1 | Hydrogen Peroxide - metabolism | Peroxiredoxins | Animals | Models, Biological | Mice | Epidermal Growth Factor - pharmacology | HeLa Cells | Peroxidases - genetics | Phosphoric Monoester Hydrolases - metabolism | Peptides | Oxidation-reduction reaction | Research
Journal Article
Journal of immunology (Baltimore, Md. : 1950), ISSN 0022-1767, 1/2019, Volume 202, Issue 2, pp. 428 - 440
Engagement of the B cell receptor (BCR) with antigens triggers signaling pathways for commitment of B-lymphocyte responses that can be regulated, in part, by... 
BCR | Duox1 | CD40 | T cell independent (TI) | RGS16 | ROS | IL-4 | H2O2 | CD19+ B cells | BCAP | T cell dependent (TD)
Journal Article
European Journal of Immunology, ISSN 0014-2980, 12/2012, Volume 42, Issue 12, pp. 3202 - 3211
Absence of phagocyte NADPH oxidase (NOX2) activity causes chronic granulomatous disease (CGD), a primary immunodeficiency characterized by recurrent bacterial... 
STAT5 | Chronic granulomatous disease | Reactive oxygen species | NADPH oxidase | T-BET/GATA-3 RATIO | CHRONIC GRANULOMATOUS-DISEASE | ANGIOTENSIN-II | IMMUNOLOGY | ARTHRITIS | LINEAGE COMMITMENT | REACTIVE OXYGEN | RECEPTOR STIMULATION | TH2 DIFFERENTIATION | GENE-EXPRESSION | GATA3 Transcription Factor - genetics | Th1 Cells - pathology | Bacterial Infections - enzymology | Reactive Oxygen Species - metabolism | Membrane Glycoproteins - metabolism | GATA3 Transcription Factor - immunology | Granulomatous Disease, Chronic - genetics | Granulomatous Disease, Chronic - metabolism | NADPH Oxidases - metabolism | STAT5 Transcription Factor - immunology | NADPH Oxidases - immunology | GATA3 Transcription Factor - biosynthesis | Th2 Cells - immunology | Interferon-gamma - metabolism | Granulomatous Disease, Chronic - immunology | Th1 Cells - immunology | Th1 Cells - metabolism | Cell Differentiation - genetics | STAT5 Transcription Factor - genetics | STAT5 Transcription Factor - metabolism | Signal Transduction - immunology | Bacterial Infections - genetics | Phosphorylation - genetics | Bacterial Infections - immunology | Reactive Oxygen Species - immunology | NADPH Oxidases - genetics | Receptors, Antigen, T-Cell - immunology | Phosphorylation - immunology | Interferon-gamma - genetics | Interleukin-4 - genetics | Membrane Glycoproteins - immunology | Oxidation-Reduction | Receptors, Antigen, T-Cell - metabolism | Interleukin-4 - metabolism | Signal Transduction - genetics | NADPH Oxidase 2 | Th2 Cells - metabolism | Membrane Glycoproteins - genetics | Mice, Knockout | Cell Differentiation - immunology | Interleukin-4 - immunology | Animals | Interferon-gamma - immunology | Mice | Receptors, Antigen, T-Cell - genetics | Oxidases | Influenza viruses | Antioxidants | Bacterial infections | Analysis | T cells | Health aspects | chronic granulomatous disease | reactive oxygen species
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2017, Volume 7, Issue 1, pp. 6361 - 13
Journal Article
Cell Reports, ISSN 2211-1247, 01/2020, Volume 30, Issue 3, pp. 783 - 792.e5
The physiological effects of the many germline mutations of TP53, encoding the tumor suppressor protein p53, are poorly understood. Here we report generating a... 
mouse model | ADRB3 | TP53 mutation | fat | cancer | lipolysis | Li-Fraumeni syndrome | BREAST-CANCER | CAPACITY | METABOLISM | GENE | TUMOR SUPPRESSION | DNA-BINDING COOPERATIVITY | RECEPTOR | CELL-CYCLE ARREST | PROMOTES | GAIN | CELL BIOLOGY
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2015, Volume 10, Issue 11, p. e0141550
Interleukin (IL)-23 and IL-12 are closely related in structure, and these cytokines regulate both innate and adaptive immunity. However, the precise signaling... 
IMMUNE-RESPONSE | ACTIVATION | PROTEIN | MULTIDISCIPLINARY SCIENCES | TOLL-LIKE RECEPTORS | RESISTANCE | INNATE | INFECTION | CYTOKINE | MICE | EXPRESSION | Up-Regulation | Humans | Enzyme Inhibitors - pharmacology | Phosphatidylinositol 3-Kinases - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | MAP Kinase Signaling System | Toxoplasma | Toxoplasmosis - pathology | Toxoplasmosis - metabolism | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Cell Line, Tumor | Interleukin-23 - biosynthesis | p38 Mitogen-Activated Protein Kinases - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Interleukin-12 - biosynthesis | Care and treatment | Interleukins | Patient outcomes | Physiological aspects | Monoclonal antibodies | Development and progression | Genetic aspects | Research | Health aspects | Networks | Adaptive structures | Phosphorylation | Interleukin | Intracellular signalling | AKT protein | Interleukin 23 | Biology | Infections | Activation | Tachyzoites | Parasites | Kinases | Immunity | Proteins | Signal transduction | Transfection | Pathways | Cascades | Gastroenterology | Toll-like receptors | Inhibition | Pretreatment | Immunoglobulins | Cytokines | Dendritic cells | Neutrophils | Extracellular signal-regulated kinase | MAP kinase | Interleukin 12 | JNK protein | siRNA | Adaptive immunity | TLR4 protein | Gene expression | 1-Phosphatidylinositol 3-kinase | Medicine | Monocytes | Hospitals | Inhibitors | TLR2 protein | Wortmannin | Ligands | Intracellular
Journal Article
Journal Article
Cytokine, ISSN 1043-4666, 11/2017, Volume 99, p. 91
To access, purchase, authenticate, or subscribe to the full-text of this article, please visit this link: http://dx.doi.org/10.1016/j.cyto.2017.07.007... 
Rheumatoid factor | Medical colleges | Interleukins | Tumor necrosis factor | Analysis | Immunotherapy | Arthritis | Drug discovery | Research institutes | T cells
Journal Article
Journal Article
Journal of Molecular Medicine, ISSN 0946-2716, 3/2016, Volume 94, Issue 3, pp. 287 - 289
To access, purchase, authenticate, or subscribe to the full-text of this article, please visit this link: http://dx.doi.org/10.1007/s00109-016-1399-y 
Human Genetics | Internal Medicine | Molecular Medicine | Biomedicine
Journal Article