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PLoS ONE, ISSN 1932-6203, 08/2017, Volume 12, Issue 8, p. e0181465
Journal Article
International Journal of Molecular Sciences, ISSN 1661-6596, 02/2016, Volume 17, Issue 2, pp. 206 - 206
Polymorphisms in certain inflammatory-related genes have been identified as putative differential risk factors of neurodegenerative diseases with abnormal... 
Tauopathies | Alzheimer’s disease | Cytokines | Creutzfeldt-Jakob’s disease | Toll-like receptors | Complement | Inflammation | Chemokines | Parkinson’s disease | Microglia | Parkinson's disease | MICROGLIAL ACTIVATION | toll-like receptors | BIOCHEMISTRY & MOLECULAR BIOLOGY | Creutzfeldt-Jakob's disease | APOLIPOPROTEIN-E GENE | PROINFLAMMATORY CYTOKINES | chemokines | MILD COGNITIVE IMPAIRMENT | FRONTOTEMPORAL LOBAR DEGENERATION | CHEMISTRY, MULTIDISCIPLINARY | FACTOR-ALPHA | inflammation | A-BETA | cytokines | tauopathies | TNF-ALPHA | complement | microglia | Alzheimer's disease | ANTIINFLAMMATORY DRUGS | GENOME-WIDE ASSOCIATION | Alzheimer Disease - etiology | Neurodegenerative Diseases - etiology | Humans | Transcriptome | Neurodegenerative Diseases - diagnosis | Alzheimer Disease - diagnosis | Brain - metabolism | Tauopathies - etiology | Inflammation - complications | Inflammation - metabolism | Parkinson Disease - metabolism | Diagnosis, Differential | Genetic Predisposition to Disease | Genetic Association Studies | RNA, Messenger - genetics | Risk Factors | Gene Expression Regulation | Neurodegenerative Diseases - metabolism | Disease Progression | Protein Aggregation, Pathological | Creutzfeldt-Jakob Syndrome - etiology | Creutzfeldt-Jakob Syndrome - metabolism | Creutzfeldt-Jakob Syndrome - diagnosis | Tauopathies - metabolism | Tauopathies - diagnosis | Alzheimer Disease - metabolism | Biomarkers | Brain - pathology | Inflammation - genetics | Parkinson Disease - diagnosis | Creutzfeldt-Jakob Syndrome - genetics | Parkinson Disease - etiology | Genotype & phenotype | Neurodegeneration | Polymorphism | Immune system
Journal Article
Molecular Neurobiology, ISSN 0893-7648, 07/2017, Volume 54, Issue 5, pp. 3119 - 3130
Lafora progressive myoclonus epilepsy (Lafora disease, LD) is a fatal rare autosomal recessive neurodegenerative disorder characterized by the accumulation of... 
Polyglucosan | Inflammation | Cytokines | Chemokines | Lafora disease | Microglia | OXIDATIVE STRESS | COMPLEX | PHOSPHATASE | ENDOPLASMIC-RETICULUM STRESS | AUTOPHAGY | GLYCOGEN ACCUMULATION | NEUROSCIENCES | MYOCLONUS EPILEPSY | PROTEASOME | NEURONAL CELLS | TRANSGENIC MICE | Inflammation - pathology | Microglia - metabolism | Astrocytes - pathology | Glial Fibrillary Acidic Protein - metabolism | RNA, Messenger - metabolism | Aging - genetics | Inflammation Mediators - metabolism | Microglia - pathology | Microfilament Proteins - metabolism | Inclusion Bodies - metabolism | Cytokines - genetics | Lafora Disease - pathology | Disease Models, Animal | Biomarkers - metabolism | Calcium-Binding Proteins - metabolism | Telencephalon - metabolism | Cytokines - metabolism | Dual-Specificity Phosphatases - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | Gene Expression Regulation | Ubiquitin-Protein Ligases - metabolism | Hippocampus - pathology | Disease Progression | Dual-Specificity Phosphatases - deficiency | Mice, Knockout | Hippocampus - metabolism | Animals | Cyclooxygenase 2 - metabolism | Ubiquitin-Protein Ligases - deficiency | Astrocytes - metabolism | Ubiquitin | Nervous system diseases | Phosphatases | Ligases | Analysis | Animal models | Disease | Genes | Epilepsy | Carbon tetrachloride | Inflammatory response | Inflammatory diseases | Interleukin 6 | Glucan | Coding | Rodents | Inclusion bodies | Interleukin 1 | Ubiquitin-protein ligase | Neurodegenerative diseases | Myoclonus | Astrocytes | Glial fibrillary acidic protein | Gene expression | CCL4 protein | Anti-inflammatory agents | Hereditary diseases | Neurological diseases | Evolutionary biology | Aggregates | Tumor necrosis factor | CXCL10 protein | Models | Mice | Cyclooxygenase-2 | Mutation | Cytoplasm | Micròglia | Quimiocines | Inflamació | Ubiqüitina | Malalties neurodegeneratives | Epilèpsia | cytokines | chemokines | inflammation | microglia | polyglucosan
Journal Article
Brain Pathology, ISSN 1015-6305, 03/2013, Volume 23, Issue 2, pp. 144 - 153
Thorn‐shaped astrocytes (TsA) are mainly localized in the periventricular white matter of the temporal lobe in a subgroup of aged individuals usually in the... 
tau | thorn‐shaped astrocytes | Alzheimer | truncation | ubiquitin | thorn-shaped astrocytes | NEUROFIBRILLARY TANGLES | PHOSPHORYLATION | PROTEIN-KINASE | PICKS-DISEASE | GLIAL FIBRILLARY TANGLES | PATHOLOGY | TAU PATHOLOGY | NEUROSCIENCES | TAUOPATHIES | CLINICAL NEUROLOGY | CONFORMATIONAL-CHANGES | PROGRESSIVE SUPRANUCLEAR PALSY | CORTICOBASAL DEGENERATION | Neurons - pathology | Phosphorylation | Humans | Astrocytes - pathology | Caspase 3 - metabolism | tau Proteins - metabolism | Glycogen Synthase Kinase 3 beta | Male | Alzheimer Disease - pathology | Neurofibrillary Tangles - metabolism | Nerve Fibers, Myelinated - pathology | Cell Shape | Aged, 80 and over | Female | Neurons - metabolism | Temporal Lobe - pathology | Nerve Fibers, Myelinated - metabolism | Neurofibrillary Tangles - pathology | Mitogen-Activated Protein Kinase 8 - metabolism | Axons - metabolism | Glycogen Synthase Kinase 3 - metabolism | Temporal Lobe - metabolism | Axons - pathology | Alzheimer Disease - metabolism | Aged | Astrocytes - metabolism | Ubiquitin | Brain | Aspartate | Alzheimer's disease | Amyloid beta-protein | Proteins | Alzheimers disease | Index Medicus | Immunohistochemistry | Temporal lobe | Neurodegenerative diseases | Astrocytes | Neurons | Antibodies | MAP kinase | Substantia alba | Caspase-3 | Neurofibrillary tangles | Tau protein | ubiquitination | Microtubules | DNA fragmentation | Aspartic acid
Journal Article
Brain Pathology, ISSN 1015-6305, 09/2012, Volume 22, Issue 5, pp. 636 - 653
Double‐transgenic amyloid precursor protein/presenilin 1 (APP/PS1) mice express a chimeric mouse/human APP bearing the Swedish mutation (Mo/HuAPP695swe) and a... 
synapses | β‐amyloid | mitochondria | APP/PS1 transgenic mice | tau phosphorylation | α‐synuclein | Alzheimer's disease | oxidative stress | ubiquitin‐proteasome system | ubiquitin-proteasome system | α-synuclein | β-amyloid | APP | BETA-SECRETASE ACTIVITY | CYTOCHROME-OXIDASE | ALPHA-SYNUCLEIN | PATHOLOGY | DYSTROPHIC NEURITES | NEUROSCIENCES | CLINICAL NEUROLOGY | SENILE PLAQUES | TAU-HYPERPHOSPHORYLATION | ss-amyloid | IN-VIVO | PS1 transgenic mice | PRECURSOR PROTEIN | TRANSGENIC MOUSE MODEL | a-synuclein | PARKINSONS-DISEASE | Alzheimer Disease - complications | Age Factors | Humans | tau Proteins - metabolism | Aspartic Acid Endopeptidases - genetics | RNA, Messenger - metabolism | Microscopy, Immunoelectron | Recognition (Psychology) - physiology | Alzheimer Disease - pathology | Brain - metabolism | Cysteine Endopeptidases - metabolism | Cognition Disorders - etiology | Amyloid beta-Peptides - metabolism | Brain - ultrastructure | Ubiquitin Thiolesterase - metabolism | Superoxide Dismutase - metabolism | Disease Models, Animal | Mitogen-Activated Protein Kinases | Microscopy, Electron, Transmission | Plaque, Amyloid - pathology | Gene Expression Regulation - genetics | Mice, Inbred C57BL | Presenilin-1 - genetics | Mice, Transgenic | Signal Transduction - genetics | Avoidance Learning - physiology | Mutation - genetics | Disease Progression | Neuropsychological Tests | Proteasome Endopeptidase Complex - genetics | Amyloid beta-Protein Precursor - genetics | Animals | Aspartic Acid Endopeptidases - metabolism | Brain - pathology | Plaque, Amyloid - metabolism | Mice | Plaque, Amyloid - ultrastructure | Proteasome Endopeptidase Complex - metabolism | Superoxide Dismutase-1 | Alzheimer Disease - genetics | alpha-Synuclein - metabolism | Messenger RNA | Proteases | Amyloid beta-protein | Amyloidosis | Genetic aspects | Mitochondrial DNA | Amyloidogenesis | Ubiquitin | Brain | Animal models | Neurodegenerative diseases | beta -Site APP cleaving enzyme 1 | Memory | Proteinase | beta -Amyloid | mRNA | Amyloid precursor protein | Axons | Mitochondria | Presenilin 1 | Post-translation | Mutation | Age | Plaques
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