Articles

Search Articles

Advanced search
Help

Catalogue

Articles

Databases

X
Search Filters
Format Format
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
animals (58) 58
mice (53) 53
immunology (51) 51
index medicus (39) 39
mice, inbred c57bl (26) 26
lymphocytes (24) 24
cytokines (22) 22
mice, inbred balb c (22) 22
t cells (20) 20
humans (18) 18
cells, cultured (16) 16
expression (16) 16
gene expression (16) 16
immune system (16) 16
mice, knockout (16) 16
research (15) 15
th2 cells - immunology (15) 15
responses (14) 14
in-vivo (13) 13
lymphocyte activation (13) 13
th1 cells - immunology (13) 13
flow cytometry (12) 12
lymphocytic choriomeningitis virus (12) 12
mice, transgenic (12) 12
multidisciplinary sciences (12) 12
t cell receptors (12) 12
cytokine (11) 11
differentiation (11) 11
interleukin-4 - biosynthesis (11) 11
th2 cells - metabolism (11) 11
antigens (10) 10
immunologic memory (10) 10
infections (10) 10
proteins (10) 10
biochemistry & molecular biology (9) 9
lymphocytic choriomeningitis virus - immunology (9) 9
t-cells (9) 9
activation (8) 8
article (8) 8
cell differentiation (8) 8
dendritic cells (8) 8
gata3 transcription factor (8) 8
ifn-gamma (8) 8
infection (8) 8
interferon-gamma (8) 8
physiological aspects (8) 8
th1 cells - metabolism (8) 8
transcription factor (8) 8
viruses (8) 8
analysis (7) 7
cd8-positive t-lymphocytes - immunology (7) 7
dna-binding proteins - genetics (7) 7
health aspects (7) 7
immune response (7) 7
infectious diseases (7) 7
inflammation (7) 7
interferon-gamma - metabolism (7) 7
interleukin-10 - biosynthesis (7) 7
interleukins (7) 7
lymphocytes t (7) 7
medicine, research & experimental (7) 7
receptor (7) 7
rodents (7) 7
signal transduction (7) 7
t lymphocytes (7) 7
th2 cells (7) 7
th2 cells - cytology (7) 7
trans-activators - genetics (7) 7
transcription factors (7) 7
bone marrow (6) 6
cell biology (6) 6
cytokines - metabolism (6) 6
female (6) 6
gene (6) 6
gene expression regulation (6) 6
immunology and allergy (6) 6
t-lymphocytes, helper-inducer - immunology (6) 6
viral infections (6) 6
adoptive transfer (5) 5
antigen (5) 5
arenaviridae infections - immunology (5) 5
biological sciences (5) 5
cd4-positive t-lymphocytes - immunology (5) 5
cell differentiation - immunology (5) 5
cell separation (5) 5
cells (5) 5
cytokines - biosynthesis (5) 5
disease (5) 5
dna-binding proteins - metabolism (5) 5
effector function (5) 5
gata3 transcription factor - metabolism (5) 5
genes (5) 5
immune-responses (5) 5
interferon (5) 5
lymphocyte activation - immunology (5) 5
lymphocytic choriomeningitis - immunology (5) 5
lymphocytic choriomeningitis virus - physiology (5) 5
memory (5) 5
ovalbumin - immunology (5) 5
survival (5) 5
more...
Language Language
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


1. Full Text Eosinophils are required for the maintenance of plasma cells in the bone marrow
by Roch, Toralf and Löhning, Max and Steinhauser, Gudrun and Chu, Van Trung and Fröhlich, Anja and Fillatreau, Simon and Lee, James J and Scheel, Tobias and Berek, Claudia
Nature Immunology, ISSN 1529-2908, 02/2011, Volume 12, Issue 2, pp. 151 - 159
Plasma cells are of crucial importance for long-term immune protection. It is thought that long-lived plasma cells survive in specialized niches in the bone... 
SURVIVAL | IMMUNE-RESPONSE | MEMORY B-CELLS | IN-VIVO | CXCR4 | CHEMOKINE | IMMUNOLOGY | STROMAL CELLS | MATURATION | EXPRESSION | APRIL | Tumor Necrosis Factor Ligand Superfamily Member 13 - immunology | Eosinophils - metabolism | Bone Marrow - immunology | Cells, Cultured | Adoptive Transfer | Blood Component Removal | Cell Survival - immunology | Eosinophils - immunology | Tumor Necrosis Factor Ligand Superfamily Member 13 - metabolism | Antigens, Differentiation - biosynthesis | Animals | Apoptosis - immunology | Bone Marrow - pathology | Interleukin-6 - immunology | Immunologic Memory | Plasma Cells - metabolism | Eosinophils - pathology | Mice | Mice, Inbred BALB C | Interleukin-6 - metabolism | Plasma Cells - cytology | Plasma Cells - immunology | Physiological aspects | Bone marrow | Plasma cells | Research | Eosinophils
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
2. Full Text The alarmin IL-33 promotes regulatory T-cell function in the intestine
by Schiering, Chris and Schiering, Chris and Krausgruber, Thomas and Krausgruber, Thomas and Chomka, Agnieszka and Chomka, Agnieszka and Fröhlich, Anja and Fröhlich, Anja and Adelmann, Krista and Adelmann, Krista and Wohlfert, Elizabeth A and Wohlfert, Elizabeth A and Pott, Johanna and Pott, Johanna and Griseri, Thibault and Griseri, Thibault and Bollrath, Julia and Bollrath, Julia and Hegazy, Ahmed N and Hegazy, Ahmed N and Harrison, Oliver J and Harrison, Oliver J and Owens, Benjamin M. J and Owens, Benjamin M J and Löhning, Max and Löhning, Max and Belkaid, Yasmine and Belkaid, Yasmine and Fallon, Padraic G and Fallon, Padraic G and Powrie, Fiona and Powrie, Fiona
Nature, ISSN 0028-0836, 2014, Volume 513, Issue 7519, pp. 564 - 568
FOXP3+ regulatory T cells (T-reg cells) are abundant in the intestine, where they prevent dysregulated inflammatory responses to self and environmental... 
DEPENDENT COLITIS | RESPONSES | COMPLEX | INFLAMMATORY-BOWEL-DISEASE | MULTIDISCIPLINARY SCIENCES | TRANSCRIPTION FACTOR FOXP3 | ULCERATIVE-COLITIS | AMELIORATES EXPERIMENTAL COLITIS | CYTOKINE | MICE | SOLUBLE ST2 | Colon - cytology | Inflammation - pathology | Immunity, Mucosal | Colitis - pathology | Colon - immunology | Male | Interleukins - metabolism | Intestines - immunology | T-Lymphocytes, Regulatory - immunology | Receptors, Interleukin - metabolism | Signal Transduction - immunology | Inflammation - metabolism | Interleukins - immunology | T-Lymphocytes, Regulatory - cytology | Female | Colitis - immunology | Disease Models, Animal | Intestines - pathology | Colon - pathology | Interleukin-33 | Mice, Inbred C57BL | Thymus Gland - cytology | Inflammation - immunology | Interleukin-23 - immunology | Interleukins - antagonists & inhibitors | Animals | Mice | Transforming Growth Factor beta - metabolism | Intestines - cytology | Immune response | Interleukins | Gastrointestinal diseases | Physiological aspects | Genetic aspects | Research | T cells | Proteins | Signal transduction | Phosphorylation | Transcription factors | Rodents | T cell receptors | Colon | RNA polymerase | Gene expression | Binding sites
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
3. Full Text T-bet– and STAT4–dependent IL-33 receptor expression directly promotes antiviral Th1 cell responses
by Claudia Baumann and Weldy V. Bonilla and Anja Fröhlich and Caroline Helmstetter and Michael Peine and Ahmed N. Hegazy and Daniel D. Pinschewer and Max Löhning
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2015, Volume 112, Issue 13, pp. 4056 - 4061
During infection, the release of damage-associated molecular patterns, so-called “alarmins,” orchestrates the immune response. The alarmin IL-33 plays a role... 
ST2 | CD4 T cell | Virus infection | Th1 cell | IL-33 | IFN-GAMMA PRODUCTION | MULTIDISCIPLINARY SCIENCES | EFFECTOR FUNCTION | CHORIOMENINGITIS VIRUS-INFECTION | CYTOKINE | virus infection | SELECTIVE EXPRESSION | NK CELLS | IMMUNE-RESPONSES | MICE | DIFFERENTIATION | TRANSCRIPTION FACTOR | CD8-Positive T-Lymphocytes - cytology | Cell Separation | Interleukin-33 | Mice, Inbred C57BL | Gene Expression Regulation | STAT4 Transcription Factor - metabolism | Gene Expression Profiling | Interferon-gamma - metabolism | Th1 Cells - immunology | T-Box Domain Proteins - metabolism | L-Selectin - metabolism | Animals | Flow Cytometry | Lymphocytic choriomeningitis virus | T-Lymphocytes - cytology | Interleukins - immunology | Hyaluronan Receptors - metabolism | Cell Differentiation | Mice | Th1 Cells - cytology | CD8-Positive T-Lymphocytes - immunology | Arenaviridae Infections - immunology | Gene expression | Immune response | Research | Biological Sciences
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
4. Full Text IL-33 in T Cell Differentiation, Function, and Immune Homeostasis
by Peine, Michael and Marek, Roman M and Löhning, Max
Trends in Immunology, ISSN 1471-4906, 2016, Volume 37, Issue 5, pp. 321 - 333
Recent studies have highlighted a role for the alarmin interleukin (IL)-33 in CD4+ and CD8+ T cell activation and function, and have also revealed important... 
Allergy and Immunology | STAT4/STAT5 | alarmin IL-33 | T-bet/GATA-3/Foxp3 | ST2/T1/IL-1RL1 | Th1/Th2/Treg/CTL | CD4+ and CD8+ T cell subsets | Alarmin IL-33 | CD4 | and CD8 | T cell subsets | IL-1-LIKE CYTOKINE IL-33 | TH2 CELLS | ADAPTER MOLECULE MYD88 | IFN-GAMMA | MAST-CELLS | CHORIOMENINGITIS VIRUS-INFECTION | INTERFERON-GAMMA PRODUCTION | INNATE LYMPHOID-CELLS | INTERLEUKIN-1 RECEPTOR | IMMUNOLOGY | TRANSCRIPTION FACTOR GATA-3 | T-Lymphocyte Subsets - immunology | Gene Expression Regulation - immunology | Lymphocyte Activation | Humans | Homeostasis | Interleukin-33 - immunology | T-Lymphocytes, Regulatory - immunology | Feedback, Physiological | Animals | Cell Differentiation | Interleukin-1 Receptor-Like 1 Protein - metabolism | GATA3 Transcription Factor - metabolism | Interleukin-1 Receptor-Like 1 Protein - genetics | Interleukins | T cells | Cell differentiation | Osteoarthritis | Antigens | Signal transduction | Transcription factors | Cytokines | Lymphocytes | Cytotoxicity | T cell receptors | Gene expression
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
5. Full Text Type I Interferon Protects Antiviral CD8+ T Cells from NK Cell Cytotoxicity
by Xu, Haifeng C and Grusdat, Melanie and Pandyra, Aleksandra A and Polz, Robin and Huang, Jun and Sharma, Piyush and Deenen, René and Köhrer, Karl and Rahbar, Ramtin and Diefenbach, Andreas and Gibbert, Kathrin and Löhning, Max and Höcker, Lena and Waibler, Zoe and Häussinger, Dieter and Mak, Tak W and Ohashi, Pamela S and Lang, Philipp A and Lang, Karl S
Immunity, ISSN 1074-7613, 06/2014, Volume 40, Issue 6, pp. 949 - 960
Despite development of new antiviral drugs, viral infections are still a major health problem. The most potent antiviral defense mechanism is the innate... 
PLASMACYTOID DENDRITIC CELLS | EFFECTOR | NATURAL-KILLER-CELLS | VIRUS | THERAPY | IMMUNE-RESPONSES | CHRONIC HEPATITIS-C | INNATE | RECEPTOR | IMMUNOLOGY | TRANSGENIC MICE | Lymphocytic choriomeningitis virus - immunology | Lymphocytic Choriomeningitis - virology | Mice, Inbred C57BL | Cells, Cultured | Virus Replication - immunology | Basic-Leucine Zipper Transcription Factors - genetics | Immunity, Innate | Interferon Type I - immunology | Lymphocytic Choriomeningitis - immunology | Mice, Knockout | Signal Transduction - immunology | Animals | Receptor, Interferon alpha-beta - immunology | Killer Cells, Natural - immunology | Mice | Receptor, Interferon alpha-beta - genetics | CD8-Positive T-Lymphocytes - immunology | Cytotoxicity, Immunologic | Perforin - biosynthesis | Cytokines | Lymphocytes | Cytotoxicity | Infections | Gene expression | Experiments | Viral infections | Immune system | Chronic illnesses
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
6. Full Text Professional Memory CD4 + T Lymphocytes Preferentially Reside and Rest in the Bone Marrow
by Tokoyoda, Koji and Zehentmeier, Sandra and Hegazy, Ahmed N and Albrecht, Inka and Grün, Joachim R and Löhning, Max and Radbruch, Andreas
Immunity, ISSN 1074-7613, 2009, Volume 30, Issue 5, pp. 721 - 730
CD4 T lymphocytes are key to immunological memory. Here we show that in the memory phase of specific immune responses, most of the memory CD4 T lymphocytes had... 
CELLIMMUNO | SURVIVAL | CELL MEMORY | CD8(+) | LYMPHOID HOMEOSTASIS | CD8-T-CELL MEMORY | CD4-T-CELL HELP | ANTIBODY | IMMUNOLOGY | HOMEOSTATIC PROLIFERATION | EXPRESSION | ACUTE INFECTION | Gene Expression | Bone Marrow - immunology | Oligonucleotide Array Sequence Analysis | Stromal Cells - metabolism | CD4-Positive T-Lymphocytes - metabolism | Stromal Cells - immunology | CD4-Positive T-Lymphocytes - immunology | Animals | B-Lymphocytes - immunology | Interleukin-7 - immunology | Antigens, Ly - immunology | Down-Regulation - immunology | Integrin alpha2 - immunology | Immunologic Memory | Mice | Interleukin-7 - metabolism | Spleen | Competition | Antigens | Plasma | Microscopy | Lymphocytes | Homeostasis | T cell receptors | Gene expression | Immune system
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
7. Full Text Interferons Direct Th2 Cell Reprogramming to Generate a Stable GATA-3 +T-bet + Cell Subset with Combined Th2 and Th1 Cell Functions
by Hegazy, Ahmed N and Peine, Michael and Helmstetter, Caroline and Panse, Isabel and Fröhlich, Anja and Bergthaler, Andreas and Flatz, Lukas and Pinschewer, Daniel D and Radbruch, Andreas and Löhning, Max
Immunity, ISSN 1074-7613, 2010, Volume 32, Issue 1, pp. 116 - 128
Current T cell differentiation models invoke separate T helper 2 (Th2) and Th1 cell lineages governed by the lineage-specifying transcription factors GATA-3... 
CELLIMMUNO | LINEAGE COMMITMENT | CD4(+) T-CELLS | FATE DETERMINATION | IMMUNOLOGY | TRANSCRIPTION FACTOR GATA-3 | LYMPHOCYTIC CHORIOMENINGITIS VIRUS | T(H)1 CELLS | VIRAL-INFECTION | HELPER LYMPHOCYTES | STAT4 ACTIVATION | IFN-ALPHA | Lymphocytic choriomeningitis virus - immunology | T-Lymphocyte Subsets - immunology | T-Lymphocyte Subsets - cytology | GATA3 Transcription Factor - immunology | T-Box Domain Proteins - immunology | Th2 Cells - immunology | Adoptive Transfer | Interferon-gamma - metabolism | Th1 Cells - immunology | Th1 Cells - metabolism | Lymphocyte Activation - immunology | Flow Cytometry | GATA3 Transcription Factor - metabolism | Cytokines - immunology | Cell Separation | Mice, Inbred C57BL | Th2 Cells - cytology | Th2 Cells - metabolism | T-Box Domain Proteins - metabolism | Mice, Knockout | Cell Differentiation - immunology | Animals | T-Lymphocyte Subsets - metabolism | Interferon-gamma - immunology | Mice | Th1 Cells - cytology | Proteins | Transcription factors | Cytokines | Protein expression | T cell receptors | Infections | Viral infections | Immune system
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
8. Full Text The microRNA miR-182 is induced by IL-2 and promotes clonal expansion of activated helper T lymphocytes
by Haftmann, Claudia and Flossdorf, Michael and Baumgrass, Ria and Bosio, Andreas and Höfer, Thomas and Li, Na and Chen, Wei and Hatam, Farahnaz and Stittrich, Anna-Barbara and Panse, Isabel and Heinz, Gitta Anne and Grün, Joachim and Riedel, Rene and Fuhrmann, Franziska and Schulze, Felix-Michael and Radbruch, Andreas and Bissels, Ute and Thiel, Andreas and Hammoud, Ben and Hegazy, Ahmed Nabil and Chang, Hyun-Dong and Mollenkopf, Hans-Joachim and Löhning, Max and Rajewsky, Nikolaus and Mashreghi, Mir-Farzin and Fang, Zhuo and Loddenkemper, Christoph and Jahn, Angelina and Matz, Mareen and Dong, Jun and Sgouroudis, Evridiki and Kühl, Anja Andrea
Nature Immunology, ISSN 1529-2908, 11/2010, Volume 11, Issue 11, pp. 1057 - 1062
After being activated by antigen, helper T lymphocytes switch from a resting state to clonal expansion. This switch requires inactivation of the transcription... 
FORKHEAD TRANSCRIPTION FACTOR | CELLS | IN-VIVO | EFFECTOR FUNCTION | FACTOR FKHR-L1 | PROLIFERATION | DIFFERENTIATION | IMMUNOLOGY | EXPRESSION | INTERLEUKIN-7 RECEPTOR | FOXO1 | Cell Proliferation | Humans | Mice, Inbred C57BL | Cells, Cultured | MicroRNAs - immunology | Animals | Interleukin-2 - immunology | T-Lymphocytes, Helper-Inducer - immunology | T-Lymphocytes, Helper-Inducer - cytology | Arthritis - immunology | Mice | Mice, Inbred BALB C | Disease Models, Animal | Antigens | MicroRNA | Interleukins | Analysis | Physiological aspects | T cells | Health aspects
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
9. Full Text Viral replicative capacity is the primary determinant of lymphocytic choriomeningitis virus persistence and immunosuppression
by Andreas Bergthaler and Lukas Flatz and Ahmed N. Hegazy and Susan Johnson and Edit Horvath and Max Löhning and Daniel D. Pinschewer and Rolf M. Zinkernagel
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2010, Volume 107, Issue 50, pp. 21641 - 21646
The Clone 13 (Cl13) strain of lymphocytic choriomeningitis virus is widely studied as a model of chronic systemic viral infection. Here, we used reverse... 
T lymphocytes | Immunosuppression | Lymphocytic choriomeningitis virus | RNA | Dendritic cells | Exhaustion | Cell lines | Viruses | Infections | Genetic mutation | Viral polymerase | Viral tropism | Persistent viral infections | Plasmacytoid dendritic cell | Acquired immunodeficiency syndrome | CHRONIC INFECTION | RNA ANALOGS | MULTIDISCIPLINARY SCIENCES | EFFECTOR FUNCTION | TRANSCRIPTION | plasmacytoid dendritic cell | POLYMERASE | CD8(+) T-CELLS | acquired immunodeficiency syndrome | PLASMACYTOID DENDRITIC CELLS | persistent viral infections | viral tropism | viral polymerase | HEPATITIS-C VIRUS | IN-VIVO | MICE | Lymphocytic choriomeningitis virus - immunology | Genome, Viral | Viremia | Mice, Inbred C57BL | Viral Proteins - genetics | Viral Proteins - metabolism | Point Mutation | Animals | Virus Replication | Lymphocytic choriomeningitis virus - genetics | Lymphocytic choriomeningitis virus - physiology | Mice | CD8-Positive T-Lymphocytes - immunology | Viral meningitis | Physiological aspects | Research | Properties | Immunotherapy | Biological Sciences
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
10. Full Text IL-33 receptor-expressing regulatory t cells are highly activated, Th2 biased and suppress CD4 T Cell proliferation through IL-10 and TGFβ Release
by Siede, Julia and Fröhlich, Anja and Datsi, Angeliki and Hegazy, Ahmed N and Varga, Domonkos V and Holecska, Vivien and Saito, Hirohisa and Nakae, Susumu and Löhning, Max
PLoS ONE, ISSN 1932-6203, 08/2016, Volume 11, Issue 8, p. e0161507
Immunomodulatory Foxp3(+) regulatory T cells (Tregs) form a heterogeneous population consisting of subsets with different activation states, migratory... 
RESPONSES | VISCERAL ADIPOSE-TISSUE | LYMPHOID ORGANS | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | REG CELLS | IN-VIVO | MEDIATED SUPPRESSION | MICE | CYTOKINE | TRANSCRIPTION FACTOR GATA-3 | GATA3 Transcription Factor - genetics | Cell Proliferation | GATA3 Transcription Factor - immunology | Interleukin-13 - immunology | Interleukin-5 - genetics | Interleukin-1 Receptor-Like 1 Protein - deficiency | Interleukin-33 - immunology | T-Lymphocytes, Regulatory - immunology | Interleukin-13 - genetics | T-Lymphocytes, Regulatory - cytology | Interleukin-1 Receptor-Like 1 Protein - genetics | Transforming Growth Factor beta - immunology | Signal Transduction | Lymphocyte Activation | Interleukin-1 Receptor-Like 1 Protein - immunology | Mice, Inbred C57BL | Gene Expression Regulation | Immunophenotyping | Mice, Knockout | Interleukin-5 - immunology | Interleukin-33 - genetics | Animals | Transforming Growth Factor beta - genetics | Interleukin-10 - genetics | Mice | Interleukin-10 - immunology | Cell Lineage - immunology | Cell proliferation | Homing behavior | Transcription factors | Laboratories | Activation | Lymphocytes T | Arthritis | Homing | Genotype & phenotype | Receptors | Cell growth | Immunology | Interleukin 5 | Lymphocytes | Animal tissues | Rheumatism | Foxp3 protein | Growth factors | Antigens | Cytokines | Immunoregulation | Immunomodulation | Rheumatology | Inflammation | Interleukin 13 | Lymphoid tissue | Gene expression | Chemokine receptors | CD4 antigen | Medicine | Interleukin 10 | GATA-3 protein | Clinical medicine
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
11. Full Text Superoxide Dismutase 1 Protects Hepatocytes from Type I Interferon-Driven Oxidative Damage
by Bhattacharya, Anannya and Hegazy, Ahmed N and Deigendesch, Nikolaus and Kosack, Lindsay and Cupovic, Jovana and Kandasamy, Richard K and Hildebrandt, Andrea and Merkler, Doron and Kühl, Anja A and Vilagos, Bojan and Schliehe, Christopher and Panse, Isabel and Khamina, Kseniya and Baazim, Hatoon and Arnold, Isabelle and Flatz, Lukas and Xu, Haifeng C and Lang, Philipp A and Aderem, Alan and Takaoka, Akinori and Superti-Furga, Giulio and Colinge, Jacques and Ludewig, Burkhard and Löhning, Max and Bergthaler, Andreas
Immunity, ISSN 1074-7613, 11/2015, Volume 43, Issue 5, pp. 974 - 986
Tissue damage caused by viral hepatitis is a major cause of morbidity and mortality worldwide. Using a mouse model of viral hepatitis, we... 
PATHOGENESIS | VIRUS-INFECTION | MACROPHAGES | LIVER FIBROSIS | DISEASE | FUNCTIONAL-ROLE | ALPHA | IMMUNOLOGY | STRESS | T-CELLS | VIRAL-INFECTION | Hepatitis, Viral, Animal - immunology | Oxidation-Reduction | Antioxidants - metabolism | Mice, Inbred C57BL | Oxidative Stress - immunology | Inflammation - immunology | Hepatocytes - immunology | Interferon Type I - immunology | Immunity, Innate - immunology | Signal Transduction - immunology | Superoxide Dismutase - immunology | Liver - immunology | Animals | Killer Cells, Natural - immunology | Transcription, Genetic - immunology |