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Journal Article
Molecular Neurobiology, ISSN 0893-7648, 3/2016, Volume 53, Issue 2, pp. 1080 - 1091
Journal Article
Molecular Neurobiology, ISSN 0893-7648, 9/2018, Volume 55, Issue 9, pp. 7487 - 7499
Inhibition of microglial over-activation is an important strategy to counter balance neurodegenerative progression. We previously demonstrated that the... 
Neurology | Neurosciences | Biomedicine | BDNF | Cox-2 | Astrocytes | Neurobiology | Neuroinflammation | Cell Biology | Microglia | BV-2 MICROGLIA | Neurainflammation | PROTEIN-KINASE ACTIVATION | NEUROSCIENCES | NEURODEGENERATIVE DISEASES | RAT MODEL | NEURAL STEM-CELLS | ERYTHROPOIETIN RECEPTOR | SONIC HEDGEHOG PATHWAY | PARKINSONS-DISEASE | MEDIATED NEUROTOXICITY | INNATE IMMUNITY | Astrocytes - drug effects | Cell Line | Inflammation - pathology | Microglia - metabolism | Microglia - drug effects | Signal Transduction | Anti-Inflammatory Agents - pharmacology | Minocycline - pharmacology | Humans | Hedgehog Proteins - metabolism | Culture Media, Conditioned - pharmacology | Lipopolysaccharides | Animals | Neuroprotective Agents - pharmacology | Models, Biological | Cyclooxygenase 2 - metabolism | Inflammation Mediators - metabolism | Microglia - pathology | Brain-Derived Neurotrophic Factor - metabolism | Catechin - analogs & derivatives | Mice | Erythropoietin - metabolism | Catechin - pharmacology | Astrocytes - metabolism | Nervous system diseases | Analysis | Glycoproteins | Brain | Cell survival | AMP | Neurodegenerative diseases | Erythropoietin | Medical treatment | Epigallocatechin-3-gallate | Homeostasis | Inflammation | Kinases | Neurotrophic factors | Neuromodulation | Signal transduction | Epigallocatechin gallate | Brain-derived neurotrophic factor | Protein kinase | Adenosine kinase | Hedgehog protein | Microglial cells | Minocycline | Cyclooxygenase-2
Journal Article
International Journal of Developmental Neuroscience, ISSN 0736-5748, 12/2018, Volume 71, pp. 46 - 51
Activation of peroxisome proliferator-activated receptor alpha (PPARα) has been reported to modulate cell proliferation, migration, and differentiation in... 
Neuronal differentiation | Alzheimer’s disease | Mitogen-activated protein kinase pathway | PPAR-alpha | Alzheimer's disease | TRANSCRIPTION FACTORS | ALZHEIMERS-DISEASE | N-TERMINAL KINASE | DEVELOPMENTAL BIOLOGY | GLIAL-CELL PROLIFERATION | EMBRYONAL CARCINOMA | NEUROSCIENCES | RETINOIC ACID | PPAR | GROWTH | CYCLIN-DEPENDENT KINASE-5 | AGONIST FENOFIBRATE | PPAR alpha - pharmacology | Microtubule-Associated Proteins - metabolism | Humans | Teratocarcinoma - pathology | Cyclin-Dependent Kinase 5 - metabolism | Prostaglandins E, Synthetic - pharmacology | Nestin - metabolism | Cell Movement - drug effects | Analysis of Variance | Signal Transduction - drug effects | Time Factors | Cell Differentiation - drug effects | Cell Line, Tumor | Gene Expression Regulation, Neoplastic - drug effects | Neurons - drug effects | Tretinoin - pharmacology | Medical colleges | Nervous system diseases | Neurons | Physiological aspects | Cell differentiation | Embryonic stem cells | Mitogens | Protein kinases | Tretinoin | Cell proliferation | NeuroD protein | Nestin | Leukocyte migration | Astrocytes | Recovery of function | Extracellular signal-regulated kinase | MAP kinase | Activation | Kinases | Cyclin-dependent kinase 5 | Neurotrophin 2 | Neurological diseases | Polymerase chain reaction | Proteins | Cell growth | MAP2 protein | Protein kinase | Signal processing | Differentiation | Retinoic acid | Neurological disorders | Cell migration
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2012, Volume 7, Issue 11, p. e50138
Background: Desipramine is known principally as a tricyclic antidepressant drug used to promote recovery of depressed patients. It has also been used in a... 
BV-2 MICROGLIA | OXIDATIVE STRESS | SIGNALING PATHWAY | PROMISING TARGET | MULTIDISCIPLINARY SCIENCES | SUPPRESSES NEUROINFLAMMATORY RESPONSES | ENDOTHELIAL-CELLS | ANXIETY DISORDERS | MAJOR DEPRESSION | UP-REGULATION | PARKINSONS-DISEASE | Fluoxetine - pharmacology | Rhodamines - pharmacology | Heme Oxygenase-1 - metabolism | Signal Transduction | Cell Survival | Trypan Blue - pharmacology | Enzyme Inhibitors - pharmacology | Rats | Extracellular Signal-Regulated MAP Kinases - metabolism | Neurodegenerative Diseases - metabolism | Desipramine - pharmacology | Animals | Cell Nucleus - metabolism | MAP Kinase Kinase 4 - metabolism | Oxidopamine - pharmacology | Transfection | Dopaminergic Neurons - metabolism | NF-E2-Related Factor 2 - metabolism | Cell Death | Antidepressive Agents - pharmacology | Mice | Neurons - drug effects | RNA, Small Interfering - metabolism | Infection | Enzymes | RNA | Neurons | Cell death | Desipramine | Heme | Serotonin uptake inhibitors | Biochemistry | Investigations | Neuroprotection | Disease | Reverse transcription | Activation | Mental depression | Kinases | Proteins | Signal transduction | Pathways | Rodents | Life sciences | Pretreatment | Deoxyribonucleic acid--DNA | Dopamine receptors | Medical research | Dopamine | Serotonin | Fluoxetine | Mortality | 6-Hydroxydopamine | Extracellular signal-regulated kinase | Heme oxygenase (decyclizing) | JNK protein | Pharmacology | siRNA | Gene expression | Rotenone | Oxygenase | Signaling | Gene amplification | Brain research | Inhibitors | Electrophoretic mobility | Apoptosis | Deoxyribonucleic acid | DNA
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2014, Volume 9, Issue 3, p. e91167
Journal Article
Journal Article