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serum amyloid a protein - metabolism (7) 7
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1. Full Text Improved Protective Effect of Umbilical Cord Stem Cell Transplantation on Cisplatin-Induced Kidney Injury in Mice Pretreated with Antithymocyte Globulin
by Večerić-Haler, Željka and Erman, Andreja and Cerar, Anton and Motaln, Helena and Kološa, Katja and Turnšek, Tamara Lah and Sodin Šemrl, Snežna and Lakota, Katja and Mrak-Poljšak, Katjuša and Škrajnar, Špela and Kranjc, Simona and Arnol, Miha and Perše, Martina
Stem Cells International, ISSN 1687-966X, 2016, Volume 2016, pp. 3585362 - 12
Mesenchymal stem cells (MSCs) are recognised as a promising tool to improve renal recovery in experimental models of cisplatin-induced acute kidney injury.... 
MESENCHYMAL STROMAL CELLS | IN-VITRO | THERAPY | NECROPTOSIS | DISEASE | MECHANISMS | RENAL INJURY | EXPRESSION | T-CELLS | NEPHROTOXICITY | CELL & TISSUE ENGINEERING | Urea | Cell death | Analysis | Stem cells | Transplantation | B cells | Cisplatin | Immunology | Transplants & implants | Lymphocytes | Cloning | Morphology | Colleges & universities | Inflammation | Umbilical cord | Immune system
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2. Full Text Tenascin-C drives persistence of organ fibrosis
by Bhattacharyya, Swati and Wang, Wenxia and Morales-Nebreda, Luisa and Feng, Gang and Wu, Minghua and Zhou, Xiaodong and Lafyatis, Robert and Lee, Jungwha and Hinchcliff, Monique and Feghali-Bostwick, Carol and Lakota, Katja and Budinger, G. R. Scott and Raparia, Kirtee and Tamaki, Zenshiro and Varga, John
Nature Communications, ISSN 2041-1723, 06/2016, Volume 7, Issue 1, p. 11703
The factors responsible for maintaining persistent organ fibrosis in systemic sclerosis (SSc) are not known but emerging evidence implicates toll-like... 
ACTIVATION | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | MOUSE | GENE-EXPRESSION | LUNG INJURY | DEFICIENT MICE | SKIN | SYSTEMIC-SCLEROSIS | RECEPTOR 4 | PULMONARY-FIBROSIS | Up-Regulation | Tenascin - genetics | Skin - metabolism | Humans | Middle Aged | Scleroderma, Systemic - pathology | Male | Fibrosis - metabolism | Tenascin - metabolism | Case-Control Studies | Tenascin - pharmacology | Collagen - drug effects | Adult | Female | Cell Differentiation | Collagen - genetics | Skin - pathology | Disease Models, Animal | Fibroblasts - metabolism | Fibrosis - genetics | Lung - pathology | Signal Transduction | Scleroderma, Systemic - metabolism | Cells, Cultured | Gene Expression Regulation | Scleroderma, Systemic - genetics | Myofibroblasts - drug effects | Toll-Like Receptor 4 - metabolism | Animals | Lung - drug effects | Aged | Mice
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3. Full Text Short lymphocyte, but not granulocyte, telomere length in a subset of patients with systemic sclerosis
by Lakota, Katja and Hanumanthu, Vidya Sagar and Agrawal, Rishi and Carns, Mary and Armanios, Mary and Varga, John
Annals of the Rheumatic Diseases, ISSN 0003-4967, 08/2019, Volume 78, Issue 8, pp. 1142 - 1142
Correspondence to Dr John Varga, Northwestern Scleroderma Program, Feinberg School of Medicine, Northwestern University, Chicago, USA; j-varga@northwestern.edu... 
autoantibodies | systemic sclerosis | pulmonary fibrosis | INTERSTITIAL LUNG-DISEASE | PULMONARY | SCLERODERMA | RHEUMATOLOGY | Flow cytometry | Immunoglobulins | Autoantibodies | Raynaud disease | Lung diseases | Rheumatology | Arthritis | RNA polymerase | Hybridization | DNA-directed RNA polymerase | Studies | Telomeres | Pulmonary fibrosis | Granulocytes | Lymphocytes | Systemic sclerosis | Fibrosis | Mutation | Leukocytes (granulocytic) | Scleroderma | Telomerase | Age | Smoking
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4. Full Text Naturally occurring antibodies against serum amyloid A reduce IL-6 release from peripheral blood mononuclear cells
by Kuret, Tadeja and Lakota, Katja and Mali, Polonca and Čučnik, Saša and Praprotnik, Sonja and Tomšič, Matija and Sodin-Semrl, Snezna
PLoS ONE, ISSN 1932-6203, 04/2018, Volume 13, Issue 4, p. e0195346
Serum amyloid A (SAA) is a sensitive inflammatory marker rapidly increased in response to infection, injury or trauma during the acute phase. Resolution of the... 
C-REACTIVE PROTEIN | RHEUMATOID-ARTHRITIS | AUTOANTIBODIES | ACUTE-PHASE PROTEINS | MESSENGER-RNA EXPRESSION | INTRAVENOUS IMMUNOGLOBULIN | MULTIDISCIPLINARY SCIENCES | RECEPTOR ANTIBODY | DISEASE | NECROSIS-FACTOR-ALPHA | AA AMYLOIDOSIS | Viral antibodies | Amyloid beta-protein | Physiological aspects | Antibodies | Genetic aspects | Inflammation | Research | Interleukin-6 | Intravenous administration | Leukocytes (mononuclear) | Blood | Inflammatory diseases | Interleukin 6 | Proteins | Kawasaki disease | Peripheral blood mononuclear cells | Amyloid | Chronic obstructive pulmonary disease | Enzyme-linked immunosorbent assay | Antigens | Immunoglobulins | Autoantibodies | Cytokines | Blood & organ donations | Neutrophils | Rheumatology | Trauma | Blood donors | Rheumatoid arthritis | Avidity | Cancer
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5. Full Text Neutralizing effects of anti-infliximab antibodies on synergistically-stimulated human coronary artery endothelial cells
by Ogrič, Manca and Poljšak, Katjuša Mrak and Lakota, Katja and Žigon, Polona and Praprotnik, Sonja and Semrl, Snezna Sodin and Čučnik, Saša
Atherosclerosis, ISSN 0021-9150, 12/2019, Volume 291, pp. 1 - 8
Patients with rheumatic diseases have an increased risk of atherosclerosis with up-regulated serum amyloid A (SAA), tumor necrosis factor-α (TNF-α) and... 
Anti-Infliximab antibodies | Infliximab | Atherosclerosis | Rheumatic disease
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6. Full Text Serum amyloid a is a marker for pulmonary involvement in systemic sclerosis
by Lakota, Katja and Carns, Mary and Podlusky, Sofia and Mrak-Poljsak, Katjusa and Hinchcliff, Monique and Lee, Jungwha and Tomsic, Matija and Sodin-Semrl, Snezna and Varga, John
PLoS ONE, ISSN 1932-6203, 01/2015, Volume 10, Issue 1, p. e0110820
Inflammation in systemic sclerosis (SSc) is a prominent, but incompletely characterized feature in early stages of the disease. The goal of these studies was... 
INTERSTITIAL LUNG-DISEASE | C-REACTIVE PROTEIN | OVEREXPRESSION | RESPONSES | ACTIVATION | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | SKIN | INTERLEUKIN-6 | CORONARY-ARTERY | VALIDITY | Blood Sedimentation | Lung - pathology | Scleroderma, Systemic - blood | Serum Amyloid A Protein - metabolism | C-Reactive Protein | Humans | Self Report | Scleroderma, Systemic - pathology | Lung - physiopathology | Biomarkers - blood | Patient Outcome Assessment | Scleroderma, Systemic - epidemiology | Case-Control Studies | Lung - metabolism | Respiratory Function Tests | Interleukin-6 - metabolism | Fibroblasts - metabolism | Systemic scleroderma | Inflammation | Scleroderma (Disease) | Multiplexing | Arthritis | Interleukin 6 | Biological effects | Dyspnea | Fibroblasts | Tumor necrosis factor-TNF | Amyloid | Carbon monoxide | Scleroderma | Interleukin 8 | Recombinant | Hypertension | Cytokines | Incubation | Review boards | Pulmonary arteries | Mortality | Lung diseases | Rheumatology | RNA polymerase | Preventive medicine | Patients | Serum levels | Lungs | Systemic sclerosis | Coronary vessels | Proteomics | Biomarkers | Skin | Respiration | Health risk assessment | Chemokines | Veins & arteries
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7. Full Text An orally-active adiponectin receptor agonist mitigates cutaneous fibrosis, inflammation and microvascular pathology in a murine model of systemic sclerosis
by Yamashita, Takashi and Lakota, Katja and Taniguchi, Takashi and Yoshizaki, Ayumi and Sato, Shinichi and Hong, Wen and Zhou, Xingchun and Sodin-Semrl, Snezn and Fang, Feng and Asano, Yoshihide and Varga, John
Scientific Reports, ISSN 2045-2322, 12/2018, Volume 8, Issue 1, pp. 11843 - 14
The hallmarks of systemic sclerosis (SSc) are autoimmunity, microangiopathy and fibrosis. Skin fibrosis is accompanied by attrition of the dermal white adipose... 
INTERSTITIAL LUNG-DISEASE | CELLS | GROWTH-FACTOR-BETA | SCLERODERMA | ENDOTHELIAL-MESENCHYMAL TRANSITION | MULTIDISCIPLINARY SCIENCES | DERMAL FIBROBLASTS | SKIN SCLEROSIS | PULMONARY VASCULAR INVOLVEMENT | EXPRESSION | CLINICAL-SIGNIFICANCE | Phenotypes | Animal models | Adipose tissue | Immune response | Mesenchyme | Helper cells | Lymphocytes T | Adiponectin | Bleomycin | Systemic sclerosis | Fibrosis | Fibroblasts | Skin diseases | Microvasculature
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8. Full Text Clinically important neutralizing anti-drug antibodies detected with an in-house competitive ELISA
by Ogrič, Manca and Ogrič, Manca and Žigon, Polona and Žigon, Polona and Lakota, Katja and Lakota, Katja and Praprotnik, Sonja and Praprotnik, Sonja and Drobne, David and Drobne, David and Štabuc, Borut and Štabuc, Borut and Sodin-Semrl, Snezna and Sodin-Semrl, Snezna and Čučnik, Saša and Čučnik, Saša
Clinical Rheumatology, ISSN 0770-3198, 2/2019, Volume 38, Issue 2, pp. 361 - 370
Therapeutic drug monitoring of TNF-alpha inhibitors is crucial for evaluating patients with inflammatory diseases on a personalized level. It has been... 
Reporter gene assay | Anti-infliximab antibodies | Medicine & Public Health | Anti-adalimumab antibodies | Competitive ELISA | Rheumatology | RHEUMATOID-ARTHRITIS | ASSAY | ADALIMUMAB | INFLIXIMAB ANTIBODY | GENERATION | RHEUMATOLOGY | BIOPHARMACEUTICALS | HARMONIZATION | Viral antibodies | Analysis | Resveratrol | Antibodies | Medical records | Rankings | Enzyme-linked immunosorbent assay | Biopharmaceutics | Immunoglobulins | Reporter gene | Infliximab | Monoclonal antibodies | TNF inhibitors | Tumor necrosis factor-α | Patients | Inflammatory diseases
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9. Full Text Adiponectin is an endogenous anti-fibrotic mediator and therapeutic target
by Marangoni, Roberta G and Masui, Yuri and Fang, Feng and Korman, Benjamin and Lord, Gabriel and Lee, Junghwa and Lakota, Katja and Wei, Jun and Scherer, Philipp E and Otvos, Laszlo and Yamauchi, Toshimasa and Kubota, Naoto and Kadowaki, Takashi and Asano, Yoshihide and Sato, Shinichi and Tourtellotte, Warren G and Varga, John
Scientific Reports, ISSN 2045-2322, 12/2017, Volume 7, Issue 1, pp. 4397 - 12
Skin fibrosis in systemic sclerosis (SSc) is accompanied by attrition of dermal white adipose tissue (dWAT) and reduced levels of circulating adiponectin.... 
SCLERODERMA | PROFIBROTIC RESPONSES | MULTIDISCIPLINARY SCIENCES | ACTIVATED-RECEPTOR-GAMMA | GENE-EXPRESSION | DERMAL ADIPOCYTES | SKIN FIBROSIS | SYSTEMIC-SCLEROSIS | CIRCULATING ADIPONECTIN | ADIPOSE-TISSUE | BETA | Adiponectin - pharmacology | Fibrosis - drug therapy | Skin - metabolism | Humans | Scleroderma, Systemic - metabolism | Scleroderma, Systemic - pathology | Receptors, Adiponectin - metabolism | Mice, Transgenic | Fibrosis - metabolism | Mice, Knockout | Animals | Scleroderma, Systemic - etiology | Signal Transduction - drug effects | Biopsy | Fibroblasts - drug effects | Fluorescent Antibody Technique | Adiponectin - metabolism | Mice | Fibrosis - pathology | Oligopeptides - pharmacology | Skin - pathology | Disease Models, Animal | Fibroblasts - metabolism | Signal transduction | Adipose tissue | Medical innovations | Systemic sclerosis | Fibrosis | Transgenic mice | Fibroblasts | Skin | Adiponectin | Peritoneum
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10. Full Text Interleukin-1β Induces Intracellular Serum Amyloid A1 Expression in Human Coronary Artery Endothelial Cells and Promotes its Intercellular Exchange
by Kuret, Tadeja and Sodin-Šemrl, Snežna and Mrak-Poljšak, Katjuša and Čučnik, Saša and Lakota, Katja and Erman, Andreja
Inflammation, ISSN 0360-3997, 8/2019, Volume 42, Issue 4, pp. 1413 - 1425
Serum amyloid A (SAA) is an acute-phase protein with important, pathogenic role in the development of atherosclerosis. Since dysfunctional endothelium... 
atherosclerosis | Pathology | Biomedicine | Immunology | intercellular transport | serum amyloid A | Rheumatology | Internal Medicine | Pharmacology/Toxicology | IL-1β | human coronary artery endothelial cells | C-REACTIVE PROTEIN | ACTIVATION | IL-1 beta | INDUCTION | IMMUNOLOGY | IL-6 | CELL BIOLOGY | ACUTE-PHASE | MESSENGER-RNA | ISOTYPE | DISEASE | GENE-EXPRESSION | SAA1 | Flow cytometry | Atherogenesis | Intercellular signalling | Dexamethasone | Coronary artery | Nanotubes | Inflammation | Gene expression | Endothelial cells | Western blotting | Endothelium | Interleukin 6 | Acute phase substances | Arteriosclerosis | Coronary vessels | Atherosclerosis | Amyloid | Intracellular | Enzyme-linked immunosorbent assay
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11. Full Text Gene and miRNA expression in giant cell arteritis—a concise systematic review of significantly modified studies
by Kuret, Tadeja and Kuret, Tadeja and Burja, Blaž and Burja, Blaž and Feichtinger, Julia and Feichtinger, Julia and Thallinger, Gerhard G and Thallinger, Gerhard G and Frank-Bertoncelj, Mojca and Frank-Bertoncelj, Mojca and Lakota, Katja and Lakota, Katja and Žigon, Polona and Žigon, Polona and Sodin-Semrl, Snezna and Sodin-Semrl, Snezna and Čučnik, Saša and Čučnik, Saša and Tomšič, Matija and Tomšič, Matija and Hočevar, Alojzija and Hočevar, Alojzija
Clinical Rheumatology, ISSN 0770-3198, 2/2019, Volume 38, Issue 2, pp. 307 - 316
Giant cell arteritis (GCA) is a systemic vasculitis in individuals older than 50 years, characterized by headaches, visual disturbances, painful scalp, jaw... 
Vasculitis | Medicine & Public Health | Temporal artery biopsy | Rheumatology | miRNA | Giant cell arteritis | mRNA expression | WALL | INFLAMED ARTERIES | POLYMYALGIA-RHEUMATICA | TISSUE | MARKERS | RHEUMATOLOGY | BIOMARKERS | EPIGENETICS | TEMPORAL ARTERIES | INFLAMMATION | ASSOCIATION | Occlusion | Headache | Jaw | MiRNA | Stenosis | Systematic review | Arteries | Arteritis | Biopsy | Scalp | Epigenetics | Cardiovascular diseases | Systemic vasculitis
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12. Full Text Utility of serological biomarkers for giant cell arteritis in a large cohort of treatment-naïve patients
by Burja, Blaž and Burja, Blaž and Feichtinger, Julia and Feichtinger, Julia and Lakota, Katja and Lakota, Katja and Thallinger, Gerhard G and Thallinger, Gerhard G and Sodin-Semrl, Snezna and Sodin-Semrl, Snezna and Kuret, Tadeja and Kuret, Tadeja and Rotar, Žiga and Rotar, Žiga and Ješe, Rok and Ješe, Rok and Žigon, Polona and Žigon, Polona and Čučnik, Saša and Čučnik, Saša and Mali, Polonca and Mali, Polonca and Praprotnik, Sonja and Praprotnik, Sonja and Tomšič, Matija and Tomšič, Matija and Hočevar, Alojzija and Hočevar, Alojzija
Clinical Rheumatology, ISSN 0770-3198, 2/2019, Volume 38, Issue 2, pp. 317 - 329
Early diagnosis and treatment of giant cell arteritis (GCA) is crucial for preventing ischemic complications. Multiple serological markers have been... 
Relapse | Prognosis | Medicine & Public Health | Complications | Rheumatology | Biomarkers | Giant cell arteritis | Clustering | RISK | RHEUMATOLOGY | DISEASE-ACTIVITY | TOCILIZUMAB | INFLAMMATION | SERUM MARKERS | ASSOCIATION | Medical research | Care and treatment | Haptoglobin | Analysis | Medicine, Experimental | Protein C | Biological markers | Blood donors | Immunoglobulin G | Macrophage colony-stimulating factor | Principal components analysis | Interleukin 23 | Matrix metalloproteinase | Tumor necrosis factor-α | a-fetoprotein | Patients | Interleukin 18 | Arteritis | Gelatinase A | Nephelometry | Interleukin 6 | Acute phase substances | Ischemia | Interstitial collagenase | Enzyme-linked immunosorbent assay
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