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Nature Genetics, ISSN 1061-4036, 2014, Volume 46, Issue 5, pp. 503 - 509
Journal Article
Neurobiology of Disease, ISSN 0969-9961, 2005, Volume 20, Issue 2, pp. 401 - 411
Lewy bodies (LBs) are the characteristic inclusions of Parkinson's disease brain but the mechanism responsible for their formation is obscure. Lewy bodies... 
Immunohistochemistry | Synphilin-1 | Yeast two-hybrid analysis | Aggresomes | Proteasome inhibitor | Distribution | Immunoelectron microscopy | Parkin | Lewy bodies | MG-132 | proteasome | immunoelectron microscopy | DEMENTIA | SYNUCLEIN-INTERACTING PROTEIN | ALPHA-SYNUCLEIN | distribution | NEUROSCIENCES | NEURODEGENERATIVE DISEASES | INCLUSION-BODIES | inhibitor | MESSENGER-RNA | GENE | aggresomes | yeast two-hybrid analysis | DEGRADATION | synphilin-1 | immunohistochemistry | MUTATIONS | LEWY-BODY FORMATION | Lewy Bodies - ultrastructure | Substantia Nigra - physiopathology | Neurons - pathology | Substantia Nigra - pathology | Humans | Middle Aged | Cerebral Cortex - pathology | Male | Substantia Nigra - metabolism | Cerebral Cortex - metabolism | Cerebral Cortex - physiopathology | Brain - metabolism | Neurons - ultrastructure | Aged, 80 and over | Female | Neurons - metabolism | Parkinson Disease - metabolism | Lewy Bodies - metabolism | Lewy Bodies - pathology | Peptide Hydrolases - metabolism | Microscopy, Electron, Transmission | Parkinson Disease - pathology | Brain - physiopathology | Enzyme Inhibitors - pharmacology | Ubiquitin-Protein Ligases - metabolism | Parkinson Disease - physiopathology | Nerve Tissue Proteins - metabolism | Carrier Proteins - metabolism | Brain - pathology | Cell Line, Tumor | Aged | Proteasome Endopeptidase Complex - metabolism | Proteasome Inhibitors | Proteins | Brain | Neurosciences | Parkinson's disease | Neurons
Journal Article
European Journal of Immunology, ISSN 0014-2980, 01/1998, Volume 28, Issue 1, pp. 339 - 350
Antibodies binding to double‐stranded (ds) DNA are strongly associated with renal involvement in patients with systemic lupus erythematosus (SLE). We have... 
Lupus | Autoantibody | DNA sequence | Nephritis | Antigen binding | LONG-TERM | AUTOANTIBODIES | autoantibody | REACTIVITY | IDIOTYPE PR4 | IMMUNOLOGY | SYSTEMIC LUPUS | lupus | ERYTHEMATOSUS | REGION | nephritis | antigen binding | SEGMENTS | HYBRIDOMAS | EXPRESSION | Antibody Specificity | Immunoglobulin G - isolation & purification | Antibodies, Antinuclear - isolation & purification | Antibodies, Antinuclear - immunology | Humans | DNA, Complementary - genetics | Kidney - immunology | Molecular Sequence Data | Structure-Activity Relationship | Antigen-Antibody Reactions | Antibodies, Antinuclear - chemistry | Immunoglobulin G - immunology | Base Sequence | Female | Antibodies, Monoclonal - chemistry | Antibodies, Monoclonal - immunology | Proteinuria - etiology | Binding, Competitive | Lupus Nephritis - pathology | Amino Acid Sequence | DNA - immunology | Immunoglobulin G - chemistry | Autoimmune Diseases - immunology | Models, Molecular | Antibody Affinity | Lupus Nephritis - immunology | Antibodies, Monoclonal - isolation & purification | Mice, SCID | Microscopy, Electron | Sequence Homology, Nucleic Acid | Hybridomas - transplantation | Kidney - ultrastructure | Antibodies, Antinuclear - genetics | Genes, Immunoglobulin | Antibodies, Monoclonal - genetics | DNA, Single-Stranded - immunology | Sequence Alignment | Animals | Immunoglobulin G - genetics | Protein Conformation | Mice | Immunoglobulin Heavy Chains - genetics
Journal Article
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 07/2006, Volume 98, Issue 1, pp. 156 - 169
Following our identification of PTEN‐induced putative kinase 1 (PINK1) gene mutations in PARK6‐linked Parkinson's disease (PD), we have recently reported that... 
autosomal‐recessive juvenile parkinsonism | microtubule organizing centre | ubiquitin–proteasome system | Parkinson's disease | mitochondrial processing peptidase | Lewy body | PROTEIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | ALPHA-SYNUCLEIN | RECESSIVE PARKINSONISM | SUBSTANTIA-NIGRA | NEUROSCIENCES | ubiquitin-proteasome system | INCLUSION-BODIES | DISEASE | autosomal-recessive juvenile parkinsonism | MUTATIONS | MITOCHONDRIAL INNER MEMBRANE | LEWY BODIES | EARLY-ONSET PARKINSONISM | Protein Kinases - metabolism | Protein Kinases - genetics | Humans | Proteasome Endopeptidase Complex | Mitochondria - ultrastructure | Brain - metabolism | Stress, Physiological - chemically induced | Transfection - methods | Stress, Physiological - metabolism | Brain - ultrastructure | Cloning, Molecular - methods | Parkinson Disease - metabolism | Microscopy, Immunoelectron - methods | Cell Line | Green Fluorescent Proteins - metabolism | Blotting, Western - methods | Cricetinae | Parkinson Disease - pathology | Fluorescent Antibody Technique - methods | Enzyme Inhibitors - pharmacology | Mutant Proteins - genetics | Mitochondria - metabolism | Mutant Proteins - physiology | Parkinson Disease - genetics | Nerve Tissue Proteins - metabolism | Leupeptins - pharmacology | Animals | Cysteine Proteinase Inhibitors - pharmacology | Brain - pathology | Proteins | Enzymes | Brain | Mutation | Genes | Parkinsons disease
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 08/2007, Volume 293, Issue 2, pp. 928 - 938
Urocortin (UCN) protects hearts against ischemia and reperfusion injury whether given before ischemia or at reperfusion. Here we investigate the roles of PKC,... 
Peptide hormones | Oxygen radicals | Mitochondria | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | mitochondria | CARDIAC MYOCYTES | DELTA-PKC | oxygen radicals | peptide hormones | CYCLOSPORINE-A | K-ATP CHANNEL | GENE-EXPRESSION | EPSILON ACTIVATION | PERIPHERAL VASCULAR DISEASE | PKC-EPSILON | ISCHEMIA-REPERFUSION | ADENINE-NUCLEOTIDE TRANSLOCASE | MURINE HEART | Urocortins | Mitochondria, Heart - metabolism | Reactive Oxygen Species - metabolism | Mitochondrial Membrane Transport Proteins - antagonists & inhibitors | Calcium - metabolism | Mitochondria, Heart - pathology | Myocardial Contraction - drug effects | Male | Mitochondria, Heart - drug effects | Protein Carbonylation - drug effects | Necrosis | Cardiotonic Agents - therapeutic use | Myocardial Reperfusion Injury - pathology | Time Factors | Protein Kinase C - metabolism | Myocardium - metabolism | Corticotropin-Releasing Hormone - therapeutic use | Cell Death - drug effects | Animals, Newborn | Heart - physiopathology | Cell Line | Mitochondrial Membrane Transport Proteins - metabolism | Alkaloids - pharmacology | Ventricular Pressure - drug effects | Rats | Myocardium - pathology | Protein Kinase C - antagonists & inhibitors | Antioxidants - pharmacology | Cardiotonic Agents - pharmacology | Rats, Sprague-Dawley | Benzophenanthridines - pharmacology | Myocardial Reperfusion Injury - physiopathology | Corticotropin-Releasing Hormone - pharmacology | Myocardial Reperfusion Injury - metabolism | Antioxidants - therapeutic use | Animals | Myocytes, Cardiac - drug effects | Signal Transduction - drug effects | Heart - drug effects | Myocytes, Cardiac - metabolism | Protein Kinase Inhibitors - pharmacology | Oxidative Stress - drug effects | In Vitro Techniques | Myocardial Reperfusion Injury - prevention & control | reperfusion
Journal Article
Pediatric Neurology, ISSN 0887-8994, 2015, Volume 52, Issue 2, pp. 214 - 217.e7
Abstract Objective Migraine affects approximately 4%-11% of elementary school children; yet reaching a diagnosis in this age group can be challenging. The goal... 
Pediatrics | Neurology | questionnaire | migraine | children | specificity | diagnosis | sensitivity | validation | CRITERIA | POPULATION | CLINICAL NEUROLOGY | HEADACHE | ADOLESCENTS | PEDIATRICS | Prospective Studies | Outcome Assessment (Health Care) | Humans | Sensitivity and Specificity | Migraine Disorders - diagnosis | Child, Preschool | Female | Male | Surveys and Questionnaires | Child | Surveys | Elementary school students | Migraine
Journal Article
Journal Article
Basic Research in Cardiology, ISSN 0300-8428, 4/2001, Volume 96, Issue 3, pp. 227 - 236
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2008, Volume 3, Issue 6, pp. e2455 - e2455
Journal Article