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2011, Advances in surgical pathology series, ISBN 1608316173, xvi, 348
Advances in Surgical Pathology: Gastric Cancer provides a concise, updated review of the pathological characteristics of gastric cancer, with an emphasis on... 
Stomach Neoplasms | pathology | Stomach | Cancer
Book
Gastrointestinal Endoscopy, ISSN 0016-5107, 11/2018, Volume 88, Issue 5, pp. 816 - 817
Journal Article
Virchows Archiv, ISSN 0945-6317, 1/2018, Volume 472, Issue 1, pp. 1 - 2
To access, purchase, authenticate, or subscribe to the full-text of this article, please visit this link: http://dx.doi.org/10.1007/s00428-017-2284-y 
Medicine & Public Health | Pathology | PATHOLOGY | Gastrointestinal system | Gastrointestinal tract | Inflammation | Index Medicus
Journal Article
Human Pathology, ISSN 0046-8177, 2014, Volume 45, Issue 2, pp. 431 - 432
  Taken together, the articles of Ochiai et al and previously those of Tava et al [3] and Cassaro et al [4], plus our recent findings, lay out the importance... 
Pathology | INDEFINITE | PATHOLOGY | NEOPLASIA | LESIONS | Asian Continental Ancestry Group - ethnology | Precancerous Conditions - diagnosis | Stomach - pathology | Humans | Female | Gastric Mucosa - pathology | Male | Gastritis, Atrophic - diagnosis | Mucous membrane | Index Medicus
Journal Article
The American Journal of Surgical Pathology, ISSN 0147-5185, 01/2014, Volume 38, Issue 1, p. 145
Journal Article
Pathology, ISSN 0031-3025, 02/2016, Volume 48, pp. S125 - S125
Journal Article
American Journal of Clinical Pathology, ISSN 0002-9173, 04/2018, Volume 149, Issue 6, pp. 461 - 473
Abstract Objectives Gastric adenocarcinoma of the fundic gland type (GA-FG) is a newly described entity with a lack of awareness amongst general surgical... 
Journal Article
Journal of Clinical Oncology, ISSN 0732-183X, 12/2011, Volume 29, Issue 36, pp. 4803 - 4810
Journal Article
Journal Article
Hepatology, ISSN 0270-9139, 04/2014, Volume 59, Issue 4, pp. 1577 - 1590
Hepatocellular carcinoma (HCC) is the most rapidly increasing cause of cancer‐related mortality in the United States. Because of the lack of viable treatment... 
FUNCTIONAL POLYMORPHISM | HEPATIC STELLATE CELLS | CIRRHOSIS | DISEASE | GENE-EXPRESSION | TARGETS | MICE | MECHANISMS | GASTROENTEROLOGY & HEPATOLOGY | ERLOTINIB | HEPATOCARCINOGENESIS | Erlotinib Hydrochloride | Prognosis | Rats, Wistar | Carcinoma, Hepatocellular - prevention & control | Humans | Transcriptome | Hepatic Stellate Cells - metabolism | Receptor, Epidermal Growth Factor - drug effects | Hepatocytes - pathology | Male | Receptor, Epidermal Growth Factor - metabolism | Diethylnitrosamine - adverse effects | Liver Neoplasms - pathology | Phosphorylation - drug effects | Liver Cirrhosis - genetics | Hepatic Stellate Cells - pathology | Hepatocytes - drug effects | Disease Models, Animal | Hepatic Stellate Cells - drug effects | Liver Neoplasms - prevention & control | Liver Cirrhosis - etiology | Liver Cirrhosis - prevention & control | Carbon Tetrachloride - adverse effects | Cells, Cultured | Bile Ducts - physiopathology | Rats | Mice, Inbred Strains | Disease Progression | Animals | Quinazolines - therapeutic use | Carcinoma, Hepatocellular - pathology | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Cell Proliferation - drug effects | Mice | Ligation - adverse effects | Quinazolines - pharmacology | Liver cancer | Epidermal growth factor | Rodents | Mortality | Hepatology | Gene expression | Liver cirrhosis | Index Medicus
Journal Article
The American Journal of Surgical Pathology, ISSN 0147-5185, 05/2017, Volume 41, Issue 5, pp. 643 - 654
Programmed cell death protein 1 (PD-1) blocking agents are novel immunotherapeutics used for treatment of advanced-stage malignancies. They have shown promise... 
Anti-CTLA-4 antibody | Colitis | Anti-PD-1 antibody | Immunotherapy | SURGERY | AUTOIMMUNE ENTEROPATHY | colitis | GASTROINTESTINAL-TRACT | COLON BIOPSIES | PATHOLOGY | VERSUS-HOST-DISEASE | APOPTOTIC ENTEROPATHY | anti-PD-1 antibody | CELL LUNG-CANCER | UNTREATED MELANOMA | IMMUNE CHECKPOINT BLOCKADE | ADVANCED MELANOMA | anti-CTLA-4 antibody | immunotherapy | IPILIMUMAB | Predictive Value of Tests | Apoptosis - drug effects | Humans | Middle Aged | Colon - drug effects | Colitis - pathology | Colon - immunology | Male | Programmed Cell Death 1 Receptor - antagonists & inhibitors | Colectomy | Atrophy | Enterocolitis, Pseudomembranous - chemically induced | Colitis - therapy | Antineoplastic Agents - adverse effects | Colitis - chemically induced | Colonoscopy | Enterocolitis, Pseudomembranous - immunology | Inflammatory Bowel Diseases - pathology | Aged, 80 and over | Anti-Inflammatory Agents - therapeutic use | Adult | Colitis, Lymphocytic - pathology | Female | Retrospective Studies | Colitis - immunology | Severity of Illness Index | Colitis, Lymphocytic - chemically induced | Diagnosis, Differential | Colon - pathology | Antibodies - adverse effects | Remission Induction | Enterocolitis, Pseudomembranous - pathology | Immunotherapy - adverse effects | Biopsy | Colitis, Lymphocytic - immunology | Aged | Programmed Cell Death 1 Receptor - immunology | Index Medicus
Journal Article
Journal Article
Hepatology, ISSN 0270-9139, 04/2014, Volume 59, Issue 4, pp. 1435 - 1447
Sorafenib—a broad kinase inhibitor—is a standard therapy for advanced hepatocellular carcinoma (HCC) and has been shown to exert antifibrotic effects in liver... 
ADVANCED HEPATOCELLULAR-CARCINOMA | IN-VITRO | HEPATIC STELLATE CELLS | ANGIOGENESIS | MICROENVIRONMENT | PATHWAY | PHASE-II | CXCR4 | GROWTH-FACTOR | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | Niacinamide - analogs & derivatives | Liver - pathology | Male | Liver Cirrhosis - chemically induced | Liver - drug effects | Liver Cirrhosis - metabolism | Hepatocyte Growth Factor - genetics | Liver Neoplasms - pathology | Disease Models, Animal | Receptors, Platelet-Derived Growth Factor - antagonists & inhibitors | Liver - metabolism | Receptors, Chemokine - metabolism | Carbon Tetrachloride - adverse effects | Proto-Oncogene Proteins - genetics | Proto-Oncogene Proteins - deficiency | Mice, Inbred C3H | Mice, Knockout | Receptors, CXCR4 - metabolism | Hepatocyte Growth Factor - deficiency | Animals | Chemokine CXCL12 - metabolism | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - metabolism | Cell Line, Tumor | Liver Cirrhosis - pathology | Myeloid Cells - metabolism | Signal Transduction - physiology | Mice | Phenylurea Compounds - pharmacology | CD11b Antigen - metabolism | Myeloid Cells - pathology | Niacinamide - pharmacology | Receptors, Platelet-Derived Growth Factor - drug effects | Carcinoma, Hepatocellular - metabolism | Cell Movement | Hypoxia | Kinases | Liver cirrhosis | Hepatology | Index Medicus
Journal Article
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