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Nature genetics, ISSN 1061-4036, 03/2010, Volume 42, Issue 3, pp. 197 - 199
HUMANS | GENERAL-POPULATION | LEUCINE-RICH REPEAT | LOCUS | UMCG Approved | GENOME-WIDE ASSOCIATION | Life Sciences & Biomedicine | Genetics & Heredity | Science & Technology | Fundamental and applied biological sciences. Psychology | Biological and medical sciences | Genetics of eukaryotes. Biological and molecular evolution | RNA - physiology | Genome-Wide Association Study | Meta-Analysis as Topic | Age Factors | Gene Frequency | Humans | Polymorphism, Single Nucleotide - physiology | RNA - genetics | Chromosomes, Human, Pair 3 | Telomerase - genetics | Aging - genetics | Telomerase - physiology | Time Factors | Telomere - metabolism | Quantitative Trait Loci | Cohort Studies | Telomere - genetics | Physiological aspects | Telomeres | Genetic aspects | Structure | Genetic variation | Aging | Genetics | Gene therapy | Chromosomes | Apoptosis | Index Medicus
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Trends in cardiovascular medicine, ISSN 1050-1738, 08/2018, Volume 28, Issue 6, pp. 369 - 379
Myocardial infarction | Heart failure | Immune modulation/therapy | Inflammation | Cytokines | C-REACTIVE PROTEIN | RHEUMATOID-ARTHRITIS | INTERLEUKIN-1 RECEPTOR ANTAGONIST | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | PRESERVED EJECTION FRACTION | FACTOR-ALPHA | TUMOR-NECROSIS-FACTOR | PERCUTANEOUS CORONARY INTERVENTION | TNF-ALPHA | ST-SEGMENT ELEVATION | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Inflammation Mediators - immunology | Humans | Translational Medical Research | Heart Failure - physiopathology | Myocardial Infarction - immunology | Heart Failure - drug therapy | Cardiovascular Agents - therapeutic use | Animals | Anti-Inflammatory Agents - adverse effects | Myocardial Infarction - drug therapy | Signal Transduction - drug effects | Heart Failure - immunology | Anti-Inflammatory Agents - therapeutic use | Myocardial Infarction - physiopathology | Inflammation Mediators - antagonists & inhibitors | Cytokines - antagonists & inhibitors | Cardiovascular Agents - adverse effects | Cytokines - immunology | Heart | Rheumatoid factor | Medical research | Interleukins | Analysis | Medicine, Experimental | Arthritis | Heart attack | Animal models | Heart attacks | Clinical trials | Cardiovascular disease | Interleukin 6 | Ischemia | CXC chemokines | Interleukin 6 receptors | Rodents | Interleukin 1 | Tumor necrosis factor-TNF | Experimental research | Heart diseases | Interleukin 8 | Age | Immune system | Antigens | Translation | Interleukin 1 receptor antagonist | Mortality | Cardiomyocytes | Tumor necrosis factor-α | Gene expression | Studies | Monocytes | Monocyte chemoattractant protein | Rheumatoid arthritis | Coronary vessels | Cardiac function | TNF inhibitors | Monocyte chemoattractant protein 1 | Chemokines | Apoptosis | Veins & arteries | Index Medicus
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Circulation research, ISSN 0009-7330, 2018, Volume 122, Issue 6, pp. 846 - 854
Catecholamines | Genetics | Dopamine | Orthostatic | Sympathetic nervous system | Hypotension | Cardiac & Cardiovascular Systems | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Catecholamines - metabolism | Humans | Mice, Inbred C57BL | Secretory Vesicles - metabolism | Codon, Nonsense | Cytochrome b Group - genetics | Hypotension, Orthostatic - genetics | Syndrome | Ascorbic Acid - metabolism | Brain - metabolism | Animals | Norepinephrine - metabolism | Adrenal Glands - metabolism | Hypotension, Orthostatic - pathology | Pedigree | Adult | Female | Mice | Care and treatment | Gene mutations | Hypotension, Orthostatic | Causes of | Cytochromes | Genetic aspects | Research | Neurotransmitter receptors | Risk factors | Cytochrome | Epinephrine | Phenotypes | Ascorbic acid | Adrenal glands | Transcription | Nonsense mutation | Hydroxylase | Fetuses | Ribonucleic acid--RNA | DBH gene | Polymerase chain reaction | Secretory vesicles | Metabolites | Genetic analysis | Norepinephrine | Mutation | Gene mapping | hypotensin | catecholamines | genetics | adrenergic regulation | dopamine β-hydroxylase | sympathetic nervous system | CYB561 | dopamine | Orthostatic hypotension | low blood pressure
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Journal of the American College of Cardiology, ISSN 0735-1097, 04/2016, Volume 67, Issue 13, p. 2292
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Genetica, ISSN 0016-6707, 03/2012, Volume 140, Issue 1-3, pp. 53 - 63
Recombination | Reproductive mode | Genetic variability | Linkage | Microsatellites | SOCIAL INSECTS | PARASITIC WASP | HOST EXPLOITATION | Venturia canescens | GRAVENHORST HYMENOPTERA | SEX DETERMINATION | VIRUS-LIKE PARTICLES | RECOMBINATION RATES | LINKAGE ANALYSIS | HONEYBEE APIS-MELLIFERA | BOMBUS-TERRESTRIS | Life Sciences | Human Genetics | Life Sciences, general | Microbial Genetics and Genomics | Plant Genetics & Genomics | Animal Genetics and Genomics | Life Sciences & Biomedicine | Genetics & Heredity | Science & Technology | Haplotypes | Parthenogenesis - genetics | Genome, Insect - genetics | Reproduction - genetics | Wasps - classification | Male | Electron Transport Complex IV - genetics | Phylogeny | Wasps - genetics | Microsatellite Repeats - genetics | Genetic Variation | Animals | DNA, Mitochondrial - genetics | Genome Size - genetics | Female | Crosses, Genetic | Genetic research | Wasps | Index Medicus | Genetic markers | Genomes | Population dynamics | Foraging behavior | Sex
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Clinical research in cardiology, ISSN 1861-0684, 10/2015, Volume 104, Issue 10, pp. 812 - 821
CHRONIC HEART-FAILURE | ST-elevation myocardial infarction | OXIDATIVE STRESS | SEGMENT ELEVATION | RISK | IMPAIRMENT | Telomeres | Left ventricular ejection fraction | CARDIOVASCULAR-DISEASE | STEMI | SENESCENCE | Metformin | ASSOCIATION | PRIMARY PERCUTANEOUS INTERVENTION | Medicine & Public Health | Cardiology | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Myocardial Infarction - genetics | Myocardial Infarction - epidemiology | Metformin - therapeutic use | Humans | Middle Aged | Netherlands - epidemiology | Male | Leukocytes | Telomere Shortening - genetics | Incidence | Myocardial Infarction - therapy | Ventricular Dysfunction, Left - therapy | Ventricular Dysfunction, Left - genetics | Female | Genetic Predisposition to Disease - epidemiology | Hypoglycemic Agents - therapeutic use | Ventricular Dysfunction, Left - epidemiology | Acute Disease | Genetic Predisposition to Disease - genetics | Double-Blind Method | Risk Assessment | Comorbidity | Placebo Effect | Treatment Outcome | Combined Modality Therapy | Percutaneous Coronary Intervention - statistics & numerical data | Causality | Longitudinal Studies | Heart failure | Transluminal angioplasty | Heart | Glucose | Chromosomes | Heart attack | Dextrose | Natriuretic peptides | Index Medicus | Original Paper
Journal Article
Hypertension (Dallas, Tex. 1979), ISSN 0194-911X, 07/2020, Volume 76, Issue 1, pp. 195 - 205
INSULIN | blood pressure | epigenome | METAANALYSIS | Twin study | GLUCOSE | SUSCEPTIBILITY | DISEASE | DNA methylation | STRESS | hypertension | GENOME-WIDE ASSOCIATION | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Genes | Analysis | DNA | Genetic research | Blood pressure | Methylation | Gene expression | Index Medicus
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Twenty-eight genetic loci associated with ST-T-wave amplitudes of the electrocardiogram
Human molecular genetics, ISSN 0964-6906, 2016, Volume 25, Issue 10, pp. 2093 - 2103
Biochemistry & Molecular Biology | Genetics & Heredity | Life Sciences & Biomedicine | Science & Technology | Brugada Syndrome - genetics | Shab Potassium Channels - genetics | Genetic Predisposition to Disease | Genome-Wide Association Study | Basic Helix-Loop-Helix Transcription Factors - genetics | Brugada Syndrome - physiopathology | Humans | Repressor Proteins - genetics | Male | Arrhythmias, Cardiac - physiopathology | Shal Potassium Channels - genetics | Heart Conduction System - physiopathology | Adaptor Proteins, Signal Transducing - genetics | Death, Sudden, Cardiac - pathology | Electrocardiography | Polymorphism, Single Nucleotide - genetics | Female | NAV1.5 Voltage-Gated Sodium Channel - genetics | Cardiac Conduction System Disease | Arrhythmias, Cardiac - genetics | Index Medicus | Association Studies
Journal Article
Hypertension (Dallas, Tex. 1979), ISSN 0194-911X, 03/2013, Volume 61, Issue 3, pp. 602 - +
blood pressure | RENIN-ANGIOTENSIN | KALLIKREIN-KININ | HOMEOSTASIS | adrenomedullin | HEART-FAILURE | cardiovascular | IMMUNOLUMINOMETRIC ASSAY | BLOOD-PRESSURE | BIOMARKERS | GENERAL-POPULATION | kallikrein | genome-wide association study | REGIONAL PRO-ADRENOMEDULLIN | endothelin-1 | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Cardiology. Vascular system | Biological and medical sciences | Arterial hypertension. Arterial hypotension | Medical sciences | Clinical manifestations. Epidemiology. Investigative techniques. Etiology | Blood and lymphatic vessels | European Continental Ancestry Group - genetics | Genome-Wide Association Study | Protein Precursors - genetics | Protein Precursors - blood | Endothelin-1 - genetics | Humans | Middle Aged | Adrenomedullin - blood | Kallikreins - genetics | European Continental Ancestry Group - statistics & numerical data | Male | Kallikrein-Kinin System - genetics | Adrenomedullin - genetics | Endothelin-1 - blood | Epistasis, Genetic | Peptide Fragments - blood | Adult | Female | Aged | Polymorphism, Single Nucleotide | Peptide Fragments - genetics | Index Medicus
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Inhibition of Interleukin-6 Receptor in a Murine Model of Myocardial Ischemia-Reperfusion
PloS one, ISSN 1932-6203, 12/2016, Volume 11, Issue 12, pp. e0167195 - e0167195
RHEUMATOID-ARTHRITIS | HEART | INFARCTION | DISEASE | ANTIBODY | MICE | MECHANISMS | CARDIOMYOCYTES | IL-6 RECEPTOR | TOCILIZUMAB | Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Heart - physiopathology | Antibodies, Monoclonal - pharmacology | Mice, Inbred C57BL | Male | Myocardium - pathology | Receptors, Interleukin-6 - antagonists & inhibitors | Myocardial Infarction - metabolism | Fibrosis - metabolism | Hypertrophy - metabolism | Myocardial Reperfusion Injury - physiopathology | Myocardial Reperfusion Injury - metabolism | Animals | Time Factors | Interleukin-6 - blood | Myocardial Infarction - pathology | Myocardium - metabolism | Heart - drug effects | Hemodynamics - drug effects | Myocardial Infarction - prevention & control | Interleukin-6 - metabolism | Ventricular Remodeling - drug effects | Microscopy, Fluorescence | Disease Models, Animal | Receptors, Interleukin-6 - metabolism | Prevention | Physiological aspects | Myocardial ischemia | Genetic aspects | Research | Drug therapy | Reperfusion injury | Interleukin-6 | Myocardial infarction | Heart | Animal models | Nuclear magnetic resonance--NMR | Heart attacks | Immunoglobulin G | Interleukin | Cardiovascular disease | Interleukin 6 | Signal transduction | Reperfusion | Ischemia | Rodents | Surgery | Interleukin 6 receptors | Cardiology | Heart diseases | Stretching | Heart failure | Cytokines | Magnetic resonance | Coronary artery | Glycoproteins | Inflammation | Coronary artery disease | Ostomy | Injury prevention | Magnetic resonance imaging | Rheumatoid arthritis | Coronary vessels | Fibrosis | Monoclonal antibodies | Infarction | Mice | Ventricle | Hemodynamics | Cardiovascular diseases | Respiration | Hypertrophy | Apoptosis | Index Medicus | Remodeling | Nuclear magnetic resonance | NMR
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