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Nature Immunology, ISSN 1529-2908, 04/2003, Volume 4, Issue 4, pp. 321 - 329
Journal Article
Nature Immunology, ISSN 1529-2908, 05/2010, Volume 11, Issue 5, pp. 411 - 418
Sensors of pathogens, such as Toll-like receptors (TLRs), detect microbes to activate transcriptional programs that orchestrate adaptive responses to specific... 
UNFOLDED PROTEIN RESPONSE | PATHWAY | ER STRESS | HOST-DEFENSE | ENDOPLASMIC-RETICULUM STRESS | FRANCISELLA-TULARENSIS INFECTION | IMMUNOLOGY | LIVE VACCINE STRAIN | INFLAMMATORY RESPONSE | NEGATIVE REGULATOR | SYSTEMS BIOLOGY | RNA, Small Interfering - genetics | Endoribonucleases - genetics | Tularemia - metabolism | Toll-Like Receptor 2 - genetics | Membrane Glycoproteins - metabolism | Lipopeptides - pharmacology | Transcriptional Activation - drug effects | NADPH Oxidases - metabolism | Tularemia - genetics | Protein Splicing - genetics | Transcriptional Activation - immunology | X-Box Binding Protein 1 | Protein Splicing - drug effects | NADPH Oxidases - genetics | Stress, Physiological - drug effects | TNF Receptor-Associated Factor 6 - genetics | Transcription Factor CHOP - biosynthesis | Transcription Factors - immunology | Cytokines - genetics | Protein-Serine-Threonine Kinases - metabolism | DNA-Binding Proteins - immunology | Endoribonucleases - metabolism | Macrophages - pathology | Stress, Physiological - genetics | Myeloid Differentiation Factor 88 - genetics | Toll-Like Receptor 4 - genetics | Signal Transduction - genetics | Toll-Like Receptor 4 - immunology | Toll-Like Receptor 2 - metabolism | Toll-Like Receptor 4 - metabolism | Mice, Knockout | Macrophages - metabolism | Signal Transduction - drug effects | Tunicamycin - pharmacology | Lipopolysaccharides - pharmacology | Toll-Like Receptor 2 - immunology | Mice | Stress, Physiological - immunology | Endoribonucleases - immunology | Transcription Factor CHOP - genetics | NADPH Oxidases - immunology | DNA-Binding Proteins - metabolism | Signal Transduction - immunology | Macrophages - virology | Mice, Mutant Strains | Tularemia - immunology | Membrane Glycoproteins - immunology | Macrophages - immunology | Cytokines - immunology | Cell Line | Francisella tularensis - immunology | Protein Splicing - immunology | Protein-Serine-Threonine Kinases - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Immunity, Innate | NADPH Oxidase 2 | Regulatory Factor X Transcription Factors | Mice, Inbred C3H | Membrane Glycoproteins - genetics | Transcription Factors - metabolism | Animals | TNF Receptor-Associated Factor 6 - metabolism | Macrophages - drug effects | Protein-Serine-Threonine Kinases - immunology | Francisella tularensis - pathogenicity | Myeloid Differentiation Factor 88 - metabolism | Cytokines - biosynthesis | Cell receptors | Transcription factors | Immune response | Physiological aspects | Genetic aspects | Research | Health aspects | Francisella tularensis | Index Medicus
Journal Article
Science, ISSN 0036-8075, 6/2008, Volume 320, Issue 5882, pp. 1492 - 1496
Journal Article
Cell, ISSN 0092-8674, 09/2008, Volume 134, Issue 5, pp. 743 - 756
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 5/2011, Volume 108, Issue 21, pp. 8885 - 8890
Journal Article
Cell Metabolism, ISSN 1550-4131, 10/2012, Volume 16, Issue 4, pp. 487 - 499
Journal Article
Journal Article
Nature Immunology, ISSN 1529-2908, 07/2015, Volume 16, Issue 8, pp. 829 - 837
The transcription factor XBP1 has been linked to the development of highly secretory tissues such as plasma cells and Paneth cells, yet its function in... 
ALTERNATIVELY ACTIVATED MACROPHAGES | UNFOLDED PROTEIN RESPONSE | MESSENGER-RNA | ER-STRESS | BONE-MARROW | DISEASE | GENE-EXPRESSION | MICE | IMMUNOLOGY | PLASMA-CELL DIFFERENTIATION | MAJOR BASIC-PROTEIN | Endoribonucleases - genetics | Eosinophils - metabolism | Humans | Gene Expression Profiling | Endoplasmic Reticulum Stress - genetics | Eosinophils - immunology | Granulocyte Precursor Cells - ultrastructure | Endoplasmic Reticulum Stress - immunology | X-Box Binding Protein 1 | DNA-Binding Proteins - metabolism | Cell Differentiation - genetics | Signal Transduction - immunology | Flow Cytometry | Gene Expression - immunology | HEK293 Cells | Transcription Factors - immunology | Protein-Serine-Threonine Kinases - metabolism | Granulocyte Precursor Cells - immunology | Eosinophils - ultrastructure | Microscopy, Electron, Transmission | DNA-Binding Proteins - immunology | Endoribonucleases - metabolism | Granulocyte Precursor Cells - metabolism | Mice, Inbred C57BL | Cells, Cultured | Protein-Serine-Threonine Kinases - genetics | Mice, Transgenic | Signal Transduction - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Reverse Transcriptase Polymerase Chain Reaction | Regulatory Factor X Transcription Factors | Mice, Knockout | Transcription Factors - metabolism | Cell Differentiation - immunology | Microscopy, Confocal | Animals | Protein-Serine-Threonine Kinases - immunology | Mice, Inbred BALB C | Endoribonucleases - immunology | Transcription factors | Genetic aspects | Properties | Cell differentiation | Eosinophils | Immune system | Index Medicus
Journal Article
Endocrinology, ISSN 0013-7227, 2018, Volume 159, Issue 9, pp. 3177 - 3186
Journal Article
Science, ISSN 0036-8075, 10/2004, Volume 306, Issue 5695, pp. 457 - 461
Obesity contributes to the development of type 2 diabetes, but the underlying mechanisms are poorly understood. Using cell culture and mouse models, we show... 
Obesity | Phosphorylation | Receptors | Quantification | Liver | Lead | Insulin resistance | Type 2 diabetes mellitus | Insulin | Research Article | Type 1 diabetes mellitus | UNFOLDED PROTEIN RESPONSE | MESSENGER-RNA | ER STRESS | JNK | MULTIDISCIPLINARY SCIENCES | KINASE | IRE1 | RESISTANCE | TRANSCRIPTION FACTOR | XBP-1 | TRANSLATIONAL CONTROL | eIF-2 Kinase - metabolism | Endoplasmic Reticulum - metabolism | Homeostasis | Muscle, Skeletal - metabolism | Diabetes Mellitus, Type 2 - metabolism | Phosphoproteins - metabolism | X-Box Binding Protein 1 | DNA-Binding Proteins - metabolism | Adipose Tissue - metabolism | Insulin Receptor Substrate Proteins | Membrane Proteins - metabolism | Nuclear Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | Signal Transduction | Liver - metabolism | Cells, Cultured | Insulin Resistance | Rats | Nuclear Proteins - metabolism | DNA-Binding Proteins - genetics | Regulatory Factor X Transcription Factors | Obesity - metabolism | Insulin - metabolism | Animals | Mitogen-Activated Protein Kinase 8 | Tunicamycin - pharmacology | Glucose - metabolism | Mice, Obese | Receptor, Insulin - metabolism | Mice | Mice, Inbred BALB C | Transcription Factors | Mutation | Mitogen-Activated Protein Kinases - metabolism | Type 2 diabetes | Causes of | Stress (Physiology) | Research | Endoplasmic reticulum | Cell culture | Enzymes | Medical research | Diabetes | Index Medicus
Journal Article
Journal Article