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Cancer Research, ISSN 0008-5472, 07/2017, Volume 77, Issue 13 Supplement, pp. 3 - 3
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 07/2017, Volume 23, Issue 14, pp. 3711 - 3720
Purpose: To explore whether a cross-talk exists between PARP inhibition and PD-L1/PD-1 immune checkpoint axis, and determine whether blockade of PD-L1/PD-1... 
CELL LUNG-CANCER | BREAST-CANCER | OVEREXPRESSION | ACTIVATION | THERAPY | PEMBROLIZUMAB | ONCOLOGY | OVARIAN-CANCER | PROVIDES | COMBINATION | CHEMOTHERAPY | Tumor Microenvironment - drug effects | Poly (ADP-Ribose) Polymerase-1 - immunology | Breast Neoplasms - immunology | Humans | Poly(ADP-ribose) Polymerase Inhibitors - immunology | Lymphocytes, Tumor-Infiltrating - drug effects | B7-H1 Antigen - genetics | B7-H1 Antigen - immunology | Breast Neoplasms - drug therapy | Poly (ADP-Ribose) Polymerase-1 - antagonists & inhibitors | Poly(ADP-ribose) Polymerase Inhibitors - administration & dosage | Tumor Microenvironment - immunology | Xenograft Model Antitumor Assays | Immunosuppression | Animals | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Cell Line, Tumor | Female | Mice | Gene Expression Regulation, Neoplastic - drug effects | Programmed Cell Death 1 Receptor - immunology | Programmed Cell Death 1 Receptor - genetics | Lymphocytes, Tumor-Infiltrating - immunology | Cell culture | Flow cytometry | Biotechnology | Animal models | PD-1 protein | Immunoblotting | Lymphocytes T | Immunity | Anticancer properties | Lymphocytes | Xenografts | Tumor-infiltrating lymphocytes | Tumor cells | Poly(ADP-ribose) polymerase | Breast cancer | Tumor cell lines | Inhibitors | Immune checkpoint | Experimental design | PD-L1 protein | Cell lines | Breast | In vivo methods and tests | Tumors | Cancer | Index Medicus
Journal Article
Nature Communications, ISSN 2041-1723, 08/2016, Volume 7, Issue 1, pp. 12632 - 12632
Extracellular interaction between programmed death ligand-1 (PD-L1) and programmed cell death protein-1 (PD-1) leads to tumour-associated immune escape. Here... 
EPITHELIAL-MESENCHYMAL TRANSITION | B7 FAMILY-MEMBER | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | IN-VIVO | N-LINKED GLYCANS | DEGRADATION | PROMOTES TUMORIGENESIS | PD-1 | CANCER | EXPRESSION | Phosphorylation | Breast Neoplasms - immunology | Humans | Antineoplastic Agents - therapeutic use | Lymphocyte Activation - immunology | Ubiquitination | beta-Transducin Repeat-Containing Proteins - metabolism | T-Lymphocytes - metabolism | T-Lymphocytes - drug effects | Female | Antineoplastic Agents - pharmacology | Gefitinib | Breast - pathology | Tumor Escape - immunology | Programmed Cell Death 1 Receptor - metabolism | Epidermal Growth Factor - metabolism | Glycosylation | B7-H1 Antigen - immunology | Breast Neoplasms - drug therapy | Glycogen Synthase Kinase 3 beta - metabolism | Immunologic Surveillance - immunology | Xenograft Model Antitumor Assays | B7-H1 Antigen - metabolism | Animals | Breast Neoplasms - pathology | Quinazolines - therapeutic use | Protein Stability - drug effects | Cell Line, Tumor | T-Lymphocytes - immunology | Mice | Mice, Inbred BALB C | Quinazolines - pharmacology | Animal models | PD-1 protein | Lymphocytes T | Kinases | Inactivation | Immunity | Proteins | Signal transduction | Epidermal growth factor | Growth factors | Deactivation | Glycogen | Mortality | Glycogen synthase kinase 3 | Breast cancer | Immunosuppression | Cell death | PD-L1 protein | Ligands | Cancer | Tumors | Apoptosis | Index Medicus
Journal Article
Cancer Cell, ISSN 1535-6108, 02/2018, Volume 33, Issue 2, pp. 187 - 201.e10
Journal Article
Molecular Cell, ISSN 1097-2765, 08/2018, Volume 71, Issue 4, pp. 606 - 620.e7
Metformin has been reported to possess antitumor activity and maintain high cytotoxic T lymphocyte (CTL) immune surveillance. However, the functions and... 
ERAD | ER accumulation | cancer immunotherapy | PD-L1 | metformin | glycosylation | immune checkpoint blockade | BREAST-CANCER | CELLS | ACTIVATED PROTEIN-KINASE | ADVANCED MELANOMA | BIOCHEMISTRY & MOLECULAR BIOLOGY | AMPK | MECHANISMS | QUALITY-CONTROL | GLYCOSYLATION | EXPRESSION | CHECKPOINT BLOCKADE | CELL BIOLOGY | Endoplasmic Reticulum-Associated Degradation | Mammary Glands, Human - immunology | Phosphorylation | Epithelial Cells - drug effects | Humans | Melanoma, Experimental - drug therapy | Gene Expression Regulation, Neoplastic | Endoplasmic Reticulum - metabolism | T-Lymphocytes, Cytotoxic - drug effects | Melanoma, Experimental - immunology | AMP-Activated Protein Kinases - immunology | Endoplasmic Reticulum - drug effects | Female | Antineoplastic Agents - pharmacology | Epithelial Cells - cytology | Mammary Glands, Human - drug effects | T-Lymphocytes, Cytotoxic - immunology | Mammary Glands, Human - cytology | Endoplasmic Reticulum - genetics | Metformin - pharmacology | Melanoma, Experimental - pathology | CTLA-4 Antigen - genetics | Glycosylation | CTLA-4 Antigen - immunology | B7-H1 Antigen - genetics | B7-H1 Antigen - immunology | Serine - metabolism | Hypoglycemic Agents - pharmacology | Animals | Melanoma, Experimental - genetics | Epithelial Cells - immunology | Cell Line, Tumor | Mice, Inbred NOD | Mice | T-Lymphocytes, Cytotoxic - cytology | AMP-Activated Protein Kinases - genetics | Metformin | T cells | Proteolysis | Protein kinases | Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 12/2010, Volume 285, Issue 49, pp. 38720 - 38729
Accumulating evidence indicates that endocytosis plays an essential role in the nuclear transport of the ErbB family members, such as epidermal growth factor... 
BREAST-CANCER | GROWTH-FACTOR RECEPTOR | IMPORTIN | PATHWAY | PROTEIN TRANSLOCATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | CELL-SURFACE RECEPTORS | BIOLOGY | ENDOPLASMIC-RETICULUM | PROGNOSTIC VALUE | EXPRESSION | Intracellular Trafficking | Nuclear Transport | Breast Cancer | Oncogene | Cell Biology | Receptor Tyrosine Kinase
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 12/2015, Volume 125, Issue 12, pp. 4529 - 4543
Journal Article
Future oncology (London, England), 03/2018, Volume 14, Issue 6, p. 515
Sonidegib, a hedgehog pathway inhibitor, was approved by the US FDA for the treatment of locally advanced basal cell carcinoma which cannot be readily treated... 
Journal Article