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1. Xiang cun Jidu tu yu ru jia lun li
: Yu xi Li Cun jiao hui ge an yan jiu = Rural Christians and Confucian ethics : a case on church of Li village
by Li, Huawei
2013, Di 1 ban., Zong jiao xue li lun yan jiu cong shu, ISBN 7509740312, 5, 6, 5, 318
Religion | China Religion | Confucian ethics | Rural churches | Christianity | China
Book
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2. Full Text The functions and regulation of the PTEN tumour suppressor: new modes and prospects
by Lee, Yu-Ru and Chen, Ming and Pandolfi, Pier Paolo
Nature reviews. Molecular cell biology, ISSN 1471-0072, 09/2018, Volume 19, Issue 9, pp. 547 - 562
Life Sciences & Biomedicine | Science & Technology | Cell Biology | Neoplasms - metabolism | Tumor Suppressor Proteins - metabolism | Animals | Humans | PTEN Phosphohydrolase - metabolism | Neoplasms - pathology | Disease Progression | Cell survival | Restoration | Isoforms | Dimers | Phosphatase | Metabolism | PTEN protein | Cell migration | Biological activity | Tumors | Cancer
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3. Full Text Matrix stiffness regulation of integrin‐mediated mechanotransduction during osteogenic differentiation of human mesenchymal stem cells
by Shih, Yu‐Ru V and Tseng, Kuo‐Fung and Lai, Hsiu‐Yu and Lin, Chi‐Hung and Lee, Oscar K
Journal of bone and mineral research, ISSN 0884-0431, 04/2011, Volume 26, Issue 4, pp. 730 - 738
mesenchymal stem cell | matrix stiffness | integrin | mechanotransduction | osteogenic differentiation | Life Sciences & Biomedicine | Endocrinology & Metabolism | Science & Technology | Fundamental and applied biological sciences. Psychology | Biological and medical sciences | Vertebrates: osteoarticular system, musculoskeletal system | Skeleton and joints | RNA, Small Interfering - genetics | Osteogenesis - physiology | Gene Expression - drug effects | Gene Expression - genetics | Humans | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Collagen Type I - chemistry | Osteocalcin - genetics | Acrylic Resins - chemical synthesis | Integrin alpha2 - metabolism | Mechanotransduction, Cellular - physiology | rho-Associated Kinases - antagonists & inhibitors | Elasticity - physiology | Mesenchymal Stromal Cells - cytology | Collagen Type I - genetics | rho-Associated Kinases - metabolism | Extracellular Matrix - physiology | Myosin-Light-Chain Phosphatase - metabolism | Osteoblasts - cytology | Phosphorylation - drug effects | Cell Differentiation - physiology | Focal Adhesion Kinase 1 - metabolism | Mesenchymal Stromal Cells - drug effects | Calcification, Physiologic - drug effects | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Osteoblasts - drug effects | Calcification, Physiologic - physiology | Mesenchymal Stromal Cells - metabolism | Acrylic Resins - chemistry | beta Catenin - metabolism | Biomechanical Phenomena | Integrin alpha2 - genetics | Mitogen-Activated Protein Kinase 3 - metabolism | Cell Differentiation - drug effects | Protein Kinase Inhibitors - pharmacology | Osteoblasts - metabolism | Focal Adhesion Kinase 1 - antagonists & inhibitors | Core Binding Factor Alpha 1 Subunit - genetics | Mitogen-Activated Protein Kinase 1 - metabolism | Index Medicus
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4. Full Text Hypoxia promotes proliferation and osteogenic differentiation potentials of human mesenchymal stem cells
by Hung, Shun‐Pei and Ho, Jennifer H and Shih, Yu‐Ru V and Lo, Ting and Lee, Oscar K
Journal of orthopaedic research, ISSN 0736-0266, 02/2012, Volume 30, Issue 2, pp. 260 - 266
migration | hypoxia | differentiation | proliferation | mesenchymal stem cells | Life Sciences & Biomedicine | Orthopedics | Science & Technology | Cell Hypoxia | Mesenchymal Stromal Cells - cytology | Cell Proliferation | Humans | Gene Expression Regulation | Cell Differentiation | Chondrogenesis | Mesenchymal Stromal Cells - physiology | Osteogenesis | Adipogenesis | Cell Movement | Index Medicus
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5. Full Text Stem Cell Therapy for Liver Disease: Parameters Governing the Success of Using Bone Marrow Mesenchymal Stem Cells
by Kuo, Tom K and Hung, Shun–Pei and Chuang, Chiao–Hui and Chen, Chien–Tsun and Shih, Yu–Ru V and Fang, Szu–Ching Y and Yang, Vincent W and Lee, Oscar K
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 2008, Volume 134, Issue 7, pp. 2111 - 2121.e3
Gastroenterology and Hepatology | Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Liver - pathology | Cell Proliferation | Reactive Oxygen Species - metabolism | Oxidative Stress | Antioxidants - metabolism | Carbon Tetrachloride | Humans | Hepatocytes - pathology | Hepatocytes - metabolism | Liver - physiopathology | Liver Failure, Acute - physiopathology | Time Factors | Bone Marrow Transplantation | Cell Differentiation | Liver - surgery | Hepatocytes - transplantation | Disease Models, Animal | Liver Regeneration | Cell Survival | Liver - metabolism | Cells, Cultured | Mesenchymal Stromal Cells - metabolism | Liver Failure, Acute - chemically induced | Liver Failure, Acute - metabolism | Mice, SCID | Animals | Liver Failure, Acute - surgery | Mice, Inbred NOD | Mice | Mesenchymal Stem Cell Transplantation | Stem cell research | Transplantation | Liver diseases | Analysis | Stem cells | Index Medicus | Abridged Index Medicus
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6. Full Text An aberrant SREBP-dependent lipogenic program promotes metastatic prostate cancer
by Chen, Ming and Zhang, Jiangwen and Sampieri, Katia and Clohessy, John G and Mendez, Lourdes and Gonzalez-Billalabeitia, Enrique and Liu, Xue-Song and Lee, Yu-Ru and Fung, Jacqueline and Katon, Jesse M and Menon, Archita Venugopal and Webster, Kaitlyn A and Ng, Christopher and Palumbieri, Maria Dilia and Diolombi, Moussa S and Breitkopf, Susanne B and Teruya-Feldstein, Julie and Signoretti, Sabina and Bronson, Roderick T and Asara, John M and Castillo-Martin, Mireia and Cordon-Cardo, Carlos and Pandolfi, Pier Paolo
Nature genetics, ISSN 1061-4036, 02/2018, Volume 50, Issue 2, pp. 206 - 218
Life Sciences & Biomedicine | Genetics & Heredity | Science & Technology | PTEN Phosphohydrolase - genetics | Prostatic Neoplasms - metabolism | Prostatic Neoplasms - pathology | Cell Proliferation - genetics | PC-3 Cells | Humans | Mice, Inbred C57BL | Male | Sterol Regulatory Element Binding Proteins - genetics | Cell Transformation, Neoplastic - metabolism | Mice, Knockout | Metabolic Networks and Pathways - genetics | Neoplasm Metastasis | Animals | Prostatic Neoplasms - genetics | Lipogenesis - genetics | Cell Transformation, Neoplastic - genetics | Cell Line, Tumor | Mice | Sterol Regulatory Element Binding Proteins - physiology | Lipids | Genetic aspects | Research | Diagnosis | Gene mutations | Prostate cancer | Disease | Activation | Metastasis | Kinases | Inactivation | High fat diet | Metastases | Proteins | Rodents | Cooperation | Sterol regulatory element-binding protein | Deactivation | Mortality | MAP kinase | Metabolism | Gene expression | Fatty acids | Diet | Mutation | Prostate | PTEN protein | Cancer | Tumors | Index Medicus
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7. Full Text Adjunctive traditional Chinese medicine therapy improves survival in patients with advanced breast cancer: A population‐based study
by Lee, Yuan‐Wen and Chen, Ta‐Liang and Shih, Yu‐Ru Vernon and Tsai, Chu‐Lin and Chang, Chuen‐Chau and Liang, Hung‐Hua and Tseng, Sung‐Hui and Chien, Shu‐Chen and Wang, Ching‐Chiung
Cancer, ISSN 0008-543X, 05/2014, Volume 120, Issue 9, pp. 1338 - 1344
National Health Insurance Research Database | breast cancer | traditional Chinese medicine | taxane | complementary and alternative medicine | Life Sciences & Biomedicine | Oncology | Science & Technology | Gynecology. Andrology. Obstetrics | Mammary gland diseases | Biological and medical sciences | Multiple tumors. Solid tumors. Tumors in childhood (general aspects) | Medical sciences | Tumors | Humans | Middle Aged | Proportional Hazards Models | Medicine, Chinese Traditional - methods | Breast Neoplasms - drug therapy | Taxoids - therapeutic use | Taiwan - epidemiology | Survival Analysis | Breast Neoplasms - mortality | Female | Retrospective Studies | Chemotherapy, Adjuvant | Drugs, Chinese Herbal - therapeutic use | Phytotherapy - statistics & numerical data | Cohort Studies | Care and treatment | Medicine, Chinese | Breast cancer | Research | Health aspects | Patient outcomes | Index Medicus | Abridged Index Medicus
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8. PTEN Mouse Models of Cancer Initiation and and Progression
by Lee, Yu-Ru and Pandolfi, Pier Paolo
Cold Spring Harbor perspectives in medicine, ISSN 2157-1422, 02/2020, Volume 10, Issue 2
Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine
Journal Article
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9. Full Text Reactivation of PTEN tumor suppressor for cancer treatment through inhibition of a MYC-WWP1 inhibitory pathway
by Lee, Yu-Ru and Chen, Ming and Lee, Jonathan D and Zhang, Jinfang and Lin, Shu-Yu and Fu, Tian-Min and Chen, Hao and Ishikawa, Tomoki and Chiang, Shang-Yin and Katon, Jesse and Zhang, Yang and Shulga, Yulia V and Bester, Assaf C and Fung, Jacqueline and Monteleone, Emanuele and Wan, Lixin and Shen, Chen and Hsu, Chih-Hung and Papa, Antonella and Clohessy, John G and Teruya-Feldstein, Julie and Jain, Suresh and Wu, Hao and Matesic, Lydia and Chen, Ruey-Hwa and Wei, Wenyi and Pandolfi, Pier Paolo
Science (American Association for the Advancement of Science), ISSN 0036-8075, 05/2019, Volume 364, Issue 6441, p. 651
Neoplasms - metabolism | PTEN Phosphohydrolase - genetics | Tumor Suppressor Proteins - metabolism | Humans | Protein Multimerization | PTEN Phosphohydrolase - metabolism | Ubiquitin-Protein Ligases - antagonists & inhibitors | Male | Neoplasms - drug therapy | Carcinogenesis - drug effects | Ubiquitination - drug effects | Tumor Suppressor Proteins - genetics | HEK293 Cells | Proto-Oncogene Proteins c-myc - antagonists & inhibitors | Indoles - pharmacology | Indoles - therapeutic use | Proto-Oncogene Proteins c-myc - genetics | Ubiquitin-Protein Ligases - genetics | Anticarcinogenic Agents - pharmacology | Anticarcinogenic Agents - therapeutic use | Prevention | Care and treatment | Disease susceptibility | Cancer | Tumors | Analysis | Tumor suppressor genes | Genetic aspects | Ubiquitin-proteasome system | Multiple hamartoma syndrome | Regulators | Transformation | Vegetables | AKT protein | Inactivation | Phosphatase | Cancer therapies | Recruitment | Proteins | Signal transduction | Fibroblasts | Tumorigenesis | Inhibition | Dimerization | Computer simulation | Mass spectroscopy | Ablation | 1-Phosphatidylinositol 3-kinase | Molecular modelling | Inhibitors | Mice | Mutation | Mass spectrometry | Ubiquitin | Animal models | Senescence | Immunoprecipitation | Transcription | Indole-3-carbinol | Activation | Myc protein | Kinases | Evolutionary conservation | Localization | Ubiquitin-protein ligase | Deactivation | Pharmacology | Suppressors | Signaling | Depletion | Strategy | Dimers | Neoplasia | Prostate cancer | Prostate | PTEN protein | Index Medicus
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