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Nature, ISSN 0028-0836, 05/2016, Volume 534, Issue 7605, pp. 55 - 62
Somatic mutations have been extensively characterized in breast cancer, but the effects of these genetic alterations on the proteomic landscape remain poorly... 
PATHWAYS | HETEROGENEITY | PIK3CA MUTATIONS | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | GENES | BIOLOGY | RECEPTOR | EXPRESSION | SIGNATURE | REVEALS | Protein Kinases - metabolism | Focal Adhesion Kinase 1 - genetics | Receptor, Epidermal Growth Factor - genetics | Protein Kinases - genetics | Cyclin-Dependent Kinases - metabolism | Receptor, ErbB-2 - genetics | Receptors, G-Protein-Coupled - metabolism | Genomics | Humans | Gene Expression Regulation, Neoplastic | Receptor, ErbB-2 - metabolism | Phosphoproteins - metabolism | Receptor-Interacting Protein Serine-Threonine Kinase 2 - genetics | Tumor Suppressor Protein p53 - genetics | Breast Neoplasms - metabolism | Receptor-Interacting Protein Serine-Threonine Kinase 2 - metabolism | Breast Neoplasms - enzymology | Receptor, Epidermal Growth Factor - metabolism | Phosphoproteins - analysis | Mass Spectrometry | src-Family Kinases - metabolism | Female | Cyclin-Dependent Kinases - genetics | Focal Adhesion Kinase 1 - metabolism | Chromosomes, Human, Pair 5 - genetics | Breast Neoplasms - classification | Chromosome Deletion | p21-Activated Kinases - genetics | Signal Transduction | Molecular Sequence Annotation | Calcium-Binding Proteins - deficiency | Phosphoproteins - genetics | Mutation - genetics | S-Phase Kinase-Associated Proteins - metabolism | p21-Activated Kinases - metabolism | Phosphatidylinositol 3-Kinases - genetics | Breast Neoplasms - genetics | Class I Phosphatidylinositol 3-Kinases | Proteomics | S-Phase Kinase-Associated Proteins - genetics | Receptors, G-Protein-Coupled - genetics | src-Family Kinases - genetics | Calcium-Binding Proteins - genetics | Breast cancer | Genetic aspects | Research | Oncology, Experimental | Cancer | Physiological aspects | Methods | Mutation (Biology) | Proteins | Gene amplification | Peptides | Protein expression | Genomes | Mutation | Kinases | Deoxyribonucleic acid--DNA | Tumors
Journal Article
Molecular & Cellular Oncology, ISSN 2372-3556, 11/2018, Volume 5, Issue 6, p. e1526005
Estrogen receptor alpha gene (ESR1) fusion transcripts have been identified in breast cancer but their role in breast cancer is not completely understood.... 
endocrine therapy resistance | ESR1 fusions | breast cancer | metastasis | EMT
Journal Article
Journal of High Energy Physics, ISSN 1029-8479, 04/2017, Volume 8, Issue 26
JHEP08 (2017) 026 A measurement of the splitting scales occuring in the $k_\mathrm{t}$ jet-clustering algorithm is presented for final states containing a $Z$... 
Physics - High Energy Physics - Experiment | Physics | High Energy Physics - Experiment
Journal Article
MOLECULAR CANCER RESEARCH, ISSN 1541-7786, 05/2019, Volume 17, Issue 5, pp. 1024 - 1035
Aberrant activation of the Hedgehog signaling pathway has been linked to the formation of numerous cancer types, including the myogenic soft tissue sarcoma,... 
EWINGS-SARCOMA | ALVEOLAR RHABDOMYOSARCOMAS | ORIGIN | GENE | ONCOLOGY | MICE | TRANSCRIPTIONAL TARGET | CELL | PRECURSORS | CD99 | EWS/FLI | CELL BIOLOGY
Journal Article
Journal Article
Cancer Research, ISSN 0008-5472, 07/2018, Volume 78, Issue 13 Supplement, pp. 1814 - 1814
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 12/2017, Volume 196, Issue 11, pp. 1443 - 1455
Rationale: Vascular endothelial growth factor down-regulates microRNA-1 (miR-1) in the lung endothelium, and endothelial cells play a critical role in tumor... 
MicroRNA-1 | Angiogenesis | Vascular endothelial growth factor blockers | Tumor microenvironment | Lung cancer | vascular endothelial growth factor blockers | SQUAMOUS-CELL-CARCINOMA | ACTIVATION | TYROSINE PHOSPHORYLATION | MIR-1 | microRNA-1 | angiogenesis | DOWN-REGULATION | RECEPTOR | THROMBOPOIETIN | CANCER | lung cancer | RESPIRATORY SYSTEM | C-MPL | tumor microenvironment | EXPRESSION | CRITICAL CARE MEDICINE | Carcinoma, Non-Small-Cell Lung - pathology | Lung - pathology | Carcinoma, Non-Small-Cell Lung - blood supply | Endothelial Cells - metabolism | Lung Neoplasms - metabolism | Carcinoma, Non-Small-Cell Lung - metabolism | Lung Neoplasms - pathology | MicroRNAs - metabolism | Vascular Endothelial Growth Factor A - metabolism | Mice, Knockout | Neovascularization, Pathologic - pathology | Animals | Polymerase Chain Reaction | Survival Analysis | Lung - metabolism | Mice | Neovascularization, Pathologic - metabolism | Lung - blood supply | Disease Models, Animal | Lung Neoplasms - blood supply | Cell culture | Phosphorylation | Statistical analysis | Cytokines | Gene expression | Kinases | Patients | Cell adhesion & migration | Endothelium | Polymerase chain reaction | Proteins | Transgenic animals | MicroRNAs | Rodents | Vascular endothelial growth factor | Tumors | Index Medicus | Abridged Index Medicus | Original
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 11/2008, Volume 205, Issue 12, pp. 2711 - 2716
Pulmonary alveolar proteinosis (PAP) is a rare lung disorder in which surfactant-derived lipoproteins accumulate excessively within pulmonary alveoli, causing... 
RECEPTOR-DEFICIENT MICE | AUTOANTIBODIES | MEDICINE, RESEARCH & EXPERIMENTAL | COLONY-STIMULATING FACTOR | DISEASE | MUTATION | LUNG PATHOLOGY | IMMUNOLOGY | EXPRESSION | COMMON BETA-CHAIN | RESPIRATORY-FAILURE | SUBUNIT | Brief Definitive Reports
Journal Article
Journal Article
by Sullivan, Patrick and Yuan, Maoli and Coleman, John and Destefano, Joanne and Zhang, Xinsheng and Yates, Nicole and Barouch, Dan and Ackerman, Margaret and Decamp, Allan and Alter, Galit and Tomaras, Georgia and Tomaras, Georgia and Montefiori, David and Montefiori, David and Rasmussen, Beth and Morrow, Gavin and Koff, Wayne and Kashuba, Angela and Kashuba, Angela and Mascola, John and Picker, Louis and Kent, Stephen and Selhorst, Philippe and Masson, Lindi and Ngandu, Nobubelo and Rademeyer, Cecilia and Karim, Quarraisha Abdool and Kijak, Gustavo and Barouch, Dan and Rasheed, Ata Ur and Havenar-Daughton, Colin and Pham, Matthew and Kovalenkov, Yevgeniy and Gumber, Sanjeev and Johnson, Matthew and Wrammert, Jens and Villinger, Francois and Haynes, Barton and Haynes, Barton and Fox, Christopher and Reed, Steve and Vasilakos, John and Tomai, Mark and Crotty, Shane and Ahmed, Rafi and Pulendran, Bali and Fukazawa, Yoshinori and Lifson, Jeffrey and Lifson, Jeffrey and Moore, Penny and Chatora, Kumbirai and Skorochod, Elisabeth and Madidi, Ngonidzashe and Gomez, Anabel and Ncube, Getrude and Sundaram, Maaya and Kretschmer, Steve and Pauthner, Matthias and Briney, Bryan and Lee, Jeong Hyun and Saye-Francisco, Karen and Hsueh, Jessica and Ramos, Alejandra and Le, Khoa and Jones, Meaghan and Jardine, Joseph and Bastidas, Raiza and Sarkar, Anita and Liang, Chi-Hui and Wilson, Ian and Ward, Andrew and Zhu, Jiang and Poignard, Pascal and Burton, Dennis and Krebs, Shelly and Law, Wiliam and Gift, Syna and Chenine, Agnes and Rolland, Morgane and Moody, M. Anthony and Jarosinski, Marissa and Georgiev, Ivelin and Chuang, Gwo-Yu and Chuang, Gwo-Yu and Asokan, Mangaiarkarasi and Bailer, Robert T and Cale, Evan M and Louder, Mark and Kwong, Peter D and Polonis, Victoria and Tovanabutra, Sodsai and Robb, Merlin and Mascola, John R and Wibmer, Constantinos Kurt and Wibmer, Constantinos Kurt and Madzorera, Vimbai S and Doria-Rose, Nicole A and Morris, Lynn and Moore, Penny L and Anthony, Colin and ...
AIDS Research and Human Retroviruses, ISSN 0889-2229, 10/2016, Volume 32, Issue S1, pp. 1 - 409
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Journal Article