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PLoS One, ISSN 1932-6203, 2013, Volume 8, Issue 9, pp. e75091 - e75091
Objective:Neuronal damage is the morphological substrate of persisting neurological disability. Neurofilaments (Nf) are cytoskeletal proteins of neurons and... 
HEAVY-CHAIN | MULTIPLE-SCLEROSIS | BRAIN-INJURY | AXONAL DAMAGE MARKERS | SUBUNIT PNF-H | ALZHEIMERS-DISEASE | MULTIDISCIPLINARY SCIENCES | GUILLAIN-BARRE-SYNDROME | NF-H | AMYOTROPHIC-LATERAL-SCLEROSIS | CEREBROSPINAL-FLUID NEUROFILAMENT | Guillain-Barre Syndrome - cerebrospinal fluid | Immunoassay | Electrochemical Techniques | Humans | Middle Aged | Amyotrophic Lateral Sclerosis - diagnosis | Male | Biomarkers - blood | Luminescent Measurements | Alzheimer Disease - cerebrospinal fluid | Amyotrophic Lateral Sclerosis - blood | Case-Control Studies | Alzheimer Disease - diagnosis | Neurofilament Proteins - blood | Guillain-Barre Syndrome - diagnosis | Amyotrophic Lateral Sclerosis - cerebrospinal fluid | Aged, 80 and over | Guillain-Barre Syndrome - blood | Adult | Female | Aged | Alzheimer Disease - blood | Development and progression | Amyotrophic lateral sclerosis | Nervous system diseases | Cytoskeletal proteins | Guillain-Barre syndrome | Alzheimer's disease | Drugs | Neuroprotection | Neurosciences | Nuclear magnetic resonance--NMR | Disease | Laboratories | Central nervous system | Clinical trials | Chains | Systematic review | Cerebrospinal fluid | Blood | Sclerosis | Proteins | Neurodegeneration | Light | Neurofilaments | Longitudinal studies | Bioindicators | Light levels | Medical research | Immunoglobulins | Neurodegenerative diseases | Electrochemiluminescence | Dentistry | Patients | Substrates | Blood levels | Neurological diseases | Medicine | Serum levels | Neurology | Brain research | Hospitals | Correlation analysis | Neurological complications | Structural damage | Biomarkers | Cytoskeleton | Index Medicus | Nuclear magnetic resonance | NMR
Journal Article
Nature Communications, ISSN 2041-1723, 12/2017, Volume 8, Issue 1, pp. 2081 - 14
Journal Article
Journal Article
Journal of Neuroimmunology, ISSN 0165-5728, 2010, Volume 220, Issue 1, pp. 114 - 119
Abstract Background The loss of neurological function is closely related to axonal damage. Neurofilament subunits are concentrated in neurons and axons and... 
Neurology | Allergy and Immunology | Neurofilament heavy chain (NfH) | Amyotrophic lateral sclerosis | Guillain–Barré syndrome | Multiple sclerosis | Mild cognitive impairment/Alzheimer's disease | Electrochemiluminescence (ECL) | Guillain-Barré syndrome | SLOW AXONAL-TRANSPORT | Guillain Barre syndrome | ANEURYSMAL SUBARACHNOID HEMORRHAGE | GUILLAIN-BARRE-SYNDROME | MARKERS | IMMUNOLOGY | CEREBROSPINAL-FLUID | NEUROSCIENCES | DEGENERATION | MULTIPLE-SCLEROSIS | NF-H | CSF | INTERMEDIATE-FILAMENTS | Guillain-Barre Syndrome - cerebrospinal fluid | Predictive Value of Tests | Multiple Sclerosis - diagnosis | Amyotrophic Lateral Sclerosis - physiopathology | Humans | Middle Aged | Nerve Degeneration - physiopathology | Amyotrophic Lateral Sclerosis - diagnosis | Neurodegenerative Diseases - diagnosis | Male | Nerve Degeneration - cerebrospinal fluid | Alzheimer Disease - diagnosis | Neurofilament Proteins - cerebrospinal fluid | Multiple Sclerosis - physiopathology | Amyotrophic Lateral Sclerosis - cerebrospinal fluid | Neurofilament Proteins - analysis | Sensitivity and Specificity | Immunoassay - methods | Adult | Female | Subarachnoid Hemorrhage - diagnosis | Nerve Degeneration - diagnosis | Subarachnoid Hemorrhage - cerebrospinal fluid | Alzheimer Disease - physiopathology | Reproducibility of Results | Multiple Sclerosis - cerebrospinal fluid | Biomarkers - analysis | Guillain-Barre Syndrome - physiopathology | Alzheimer Disease - cerebrospinal fluid | Neurodegenerative Diseases - cerebrospinal fluid | Luminescent Measurements - methods | Subarachnoid Hemorrhage - physiopathology | Neurodegenerative Diseases - physiopathology | Guillain-Barre Syndrome - diagnosis | Electrochemical Techniques - methods | Aged | Biomarkers - cerebrospinal fluid | Index Medicus
Journal Article
Nature Communications, ISSN 2041-1723, 2017, Volume 8
. Protein misfolding and aggregation is increasingly being recognized as a cause of disease. In Alzheimers disease the amyloid-beta peptide (A beta) misfolds... 
Biological Sciences | Naturvetenskap | Biokemi och molekylärbiologi | Biologiska vetenskaper | Biochemistry and Molecular Biology | Natural Sciences
Journal Article
Diabetologia, ISSN 0012-186X, 8/2018, Volume 61, Issue 8, pp. 1748 - 1757
Multiplex proteomics could improve understanding and risk prediction of major adverse cardiovascular events (MACE) in type 2 diabetes. This study assessed 80... 
Type 2 diabetes | Medicine & Public Health | Human Physiology | Metabolic Diseases | Major adverse cardiovascular event | Proteomics | Biomarkers | Internal Medicine | Risk | KIDNEY-DISEASE | RISK-FACTORS | MYOCARDIAL-INFARCTION | ATHEROSCLEROSIS | PROTEIN BIOMARKERS | HEART-DISEASE | PLASMA-LEVELS | INTERLEUKIN-27 | ENDOCRINOLOGY & METABOLISM | ASSOCIATION | CORONARY-ARTERY-DISEASE | Biomarkers - metabolism | Humans | Middle Aged | Risk Factors | Proportional Hazards Models | Male | Inflammation | Atherosclerosis - metabolism | Diabetes Mellitus, Type 2 - diagnosis | Cardiovascular Diseases - complications | Sweden | Adult | Female | Aged | Proteomics - methods | Diabetes Mellitus, Type 2 - complications | Cardiovascular Diseases - diagnosis | Proteins | Medical research | Analysis | Medicine, Experimental | Epidemiology | Tumor necrosis factor receptors | Fibroblast growth factor | Diabetes mellitus | Matrix metalloproteinase | Interleukin 27 | Risk factors | Learning algorithms | Tumor necrosis factor | Metalloproteinase | TRAIL protein | Diabetes | Cardiovascular diseases | Diabetes mellitus (non-insulin dependent) | Apoptosis | Index Medicus | Clinical Medicine | Medical and Health Sciences | Endokrinologi och diabetes | Medicin och hälsovetenskap | Klinisk medicin | Biomarkers; Major adverse cardiovascular event; Proteomics; Risk; Type 2 diabetes | Endocrinology and Diabetes
Journal Article
Alzheimer's & Dementia: The Journal of the Alzheimer's Association, ISSN 1552-5260, 07/2018, Volume 14, Issue 7, pp. P727 - P727
Journal Article
BBA - Proteins and Proteomics, ISSN 1570-9639, 08/2015, Volume 1854, Issue 8, p. 835
The BRICHOS domain is associated with human amyloid disease, and it efficiently prevents amyloid fibril formation of the amyloid [beta]-peptide (A[beta]) in... 
Mass spectrometry | Crosslinked polymers | Detergents, Synthetic | Proteolysis | Amyloid beta-protein | Protein C | Alzheimer's disease
Journal Article
BIOCHIMICA ET BIOPHYSICA ACTA-PROTEINS AND PROTEOMICS, ISSN 1570-9639, 08/2015, Volume 1854, Issue 8, pp. 835 - 843
The BRICHOS domain is associated with human amyloid disease, and it efficiently prevents amyloid fibril formation of the amyloid beta-peptide (A beta) in vitro... 
DOMAIN | SURFACTANT PROTEIN-C | DEMENTIA | MECHANISM | ALZHEIMERS-DISEASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | CHAPERONE | Molecular chaperone | BIOPHYSICS | BIOSYNTHESIS | AMYLOID-BETA-PEPTIDE | Alzheimer's disease | Protein structure | TRANSMEMBRANE SEGMENT | TRANSFORMS | Biokemi och molekylärbiologi | Biochemistry and Molecular Biology
Journal Article
Journal of Neurology, ISSN 0340-5354, 2/2007, Volume 254, Issue 2, pp. 160 - 168
Journal Article
by Villeneuve, Sylvia and Vogel, Jacob W and Gonneaud, Julie and Pichet Binette, Alexa and Rosa-Neto, Pedro and Gauthier, Serge and Bateman, Randall J and Fagan, Anne M and Morris, John C and Benzinger, Tammie L.S and Johnson, Sterling C and Breitner, John C.S and Poirier, Judes and Barkun, Alan and Evans, Alan and Salaciak, Alyssa and Tam, Angela and Labonté, Anne and Faubert, Anne-Marie and Mathieu, Axel and Courcot, Blandine and Hyman, Bradley T and Madjar, Cécile and Carrier, Charles Edouard and Dansereau, Christian and Kazazian, Christina and Tardif, Christine and Lepage, Claude and Cuello, Claudio and Debacker, Clément and Jack, Clifford R and Picard, Cynthia and Maillet, David and Fontaine, David and Knopman, David S and Michaud, Diane and Couture, Doris and Dea, Doris and Teigner, Eduard and Anthal, Elena and Yu, Elsa and Vachon-Presseau, Etienne and Saint-Fort, Eunice Farah and Ferdinand, Fabiola and Benbouhoud, Fatiha and Pogossova, Galina and Maultaup, Gerhard and Mayrand, Ginette and Duclair, Guerda and Ayranci, Gülebru and Gagné, Guylaine and Newbold-Fox, Holly and Leppert, Illana and Vallée, Isabelle and Near, Jamie and Brandt, Jason and Maltais, Jean-Robert and Leoutsakos, Jeannie-Marie and Tremblay-Mercier, Jennifer and Pruessner, Jens and Frenette, Joanne and Frappier, Josée and Kat, Justin and Miron, Justin and Wan, Karen and Cheewakriengkrai, Laksanun and Mahar, Laura and Carmo, Leopoldina and Münter, Lisa-Marie and Collins, Louis and Théroux, Louise and Dadar, Mahsa and Chakravarty, Mallar and Dufour, Marianne and Lafaille-Magnan, Marie-Elyse and Dauar-Tedeschi, Marina and Sager, Mark A and Eisenberg, Mark and Appleby, Melissa and Savard, Mélissa and Tuwaig, Miranda and Petkova, Mirela and Rajah, Natasha and Aisen, Paul and Toussaint, Paule-Joanne and Kostopoulos, Penelope and Bellec, Pierre and Etienne, Pierre and Tariot, Pierre N and Orban, Pierre and Rioux, Pierre and Meyer, Pierre-François and El-Khoury, Rana and Sperling, Reisa A and Desautels, René and Gordon, Renee and Giles, Renuka and Hoge, Rick and Thomas, Ronald G and Das, Samir and ... and Presymptomatic Evaluation Novel Ex and Presymptomatic Evaluation of Novel or Experimental Treatments for Alzheimer Disease (PREVENT-AD) Research Group and for the Presymptomatic Evaluation of Novel or Experimental Treatments for Alzheimer Disease (PREVENT-AD) Research Group
JAMA Neurology, ISSN 2168-6149, 05/2018, Volume 75, Issue 5, pp. 608 - 619
IMPORTANCE Alzheimer disease (AD) develops during several decades. Presymptomatic individuals might be the best candidates for clinical trials, but their... 
COGNITIVE DECLINE | ADULT CHILDREN | DEMENTIA | OLDER-ADULTS | REGIONS | A-BETA | RISK | TAU | POLYMORPHISM | CLINICAL NEUROLOGY | Online First | Original Investigation | Research
Journal Article