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Methods in Enzymology, ISSN 0076-6879, 2010, Volume 484, Issue C, pp. 591 - 612
TRP channels participate in many cellular processes including cell death. These channels mediate these effects mainly by changing the cellular concentration of... 
Patch-Clamp Techniques | Biological Assay - methods | Animals | Humans | TRPC Cation Channels - metabolism | Index Medicus
Journal Article
Neuron, ISSN 0896-6273, 05/2019, Volume 102, Issue 4, pp. 801 - 812.e5
Journal Article
The Journal of Physiology, ISSN 0022-3751, 02/2017, Volume 595, Issue 3, pp. 713 - 738
Neuroinflammation associated with CNS insults leads to neuronal hyperexcitability, which may culminate in epileptiform discharges. Application of the endotoxin... 
intrinsic excitability | M‐current | CA1 | hippocampus | neuroinflammation | M-current | Inflammation | Neurons | Mitogens | Analysis | Potassium | Rodents | Research Paper | Neurological Conditions, Disorders and Treatments | Neuroscience ‐ Cellular | Molecular | Cellular and Molecular Neuroscience
Journal Article
Journal Article
Journal of Physiology (London), ISSN 0022-3751, 02/2017, Volume 595, Issue 3, pp. 713 - 738
Key points * Neuroinflammation associated with CNS insults leads to neuronal hyperexcitability, which may culminate in epileptiform discharges. * Application... 
Journal Article
The Journal of Physiology, ISSN 0022-3751, 02/2017, Volume 595, Issue 3, pp. 713 - 738
Journal Article
Journal of Neurophysiology, ISSN 0022-3077, 01/2015, Volume 113, Issue 2, pp. 601 - 619
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2012, Volume 7, Issue 9, pp. e44023 - e44023
Increased expression of the transient receptor potential vanilloid 1 (TRPV1) channels, following nerve injury, may facilitate the entry of QX-314 into... 
SCIATIC-NERVE | NOCICEPTIVE NEURONS | POTASSIUM CHANNELS | MULTIDISCIPLINARY SCIENCES | DIFFERENTIAL CHANGES | NUCLEUS CAUDALIS | GENE-EXPRESSION | SCHWANN-CELLS | DORSAL-ROOT GANGLION | INFERIOR ALVEOLAR NERVE | SENSORY NEURONS | Lidocaine - pharmacology | Capsaicin - pharmacology | Pain Management | Neuralgia - metabolism | Myelin Sheath - drug effects | Nociceptors - physiology | Male | Mandibular Nerve - pathology | Trigeminal Ganglion - drug effects | Anesthetics, Local - pharmacology | Trigeminal Nerve Injuries - metabolism | TRPV Cation Channels - metabolism | Pain Threshold - drug effects | Neuralgia - physiopathology | Mandibular Nerve - drug effects | Gene Expression | Myelin Sheath - physiology | TRPV Cation Channels - agonists | Trigeminal Nerve Injuries - drug therapy | Hyperalgesia - prevention & control | Rats | Trigeminal Ganglion - metabolism | Rats, Sprague-Dawley | Neuralgia - drug therapy | TRPV Cation Channels - genetics | Lidocaine - analogs & derivatives | Animals | Nerve Regeneration - drug effects | Trigeminal Ganglion - physiopathology | Nociceptors - metabolism | Trigeminal Nerve Injuries - physiopathology | Nociceptors - drug effects | Care and treatment | Pain | Neurosciences | Change detection | Capsaicin | Neurobiology | Science | Capsaicin receptors | Diabetic neuropathy | Neuropathy | Channels | Transient receptor potential proteins | Receptors | Rodents | Pain management | Physiology | Alveoli | Injuries | University graduates | Medical research | Pain perception | Neurons | Anatomy & physiology | Dentistry | Gene expression | Skin | Mechanical stimuli | Trigeminal ganglion | Index Medicus | Nerves | transient receptor potential proteins
Journal Article
Journal Article
Cell Calcium, ISSN 0143-4160, 2008, Volume 45, Issue 3, pp. 300 - 309
Journal Article
Frontiers in Molecular Neuroscience, ISSN 1662-5099, 06/2017, Volume 10, pp. 181 - 181
Journal Article
Pain, ISSN 0304-3959, 06/2019, Volume 160, Issue 6, pp. 1281 - 1296
Endogenous inflammatory mediators contribute to the pathogenesis of pain by acting on nociceptors, specialized sensory neurons that detect noxious stimuli.... 
NERVOUS-SYSTEM | SIGNALING PATHWAYS | SPINAL-CORD | Kv7 channels | RAT SENSORY NEURONS | NEUROSCIENCES | CLINICAL NEUROLOGY | M-current | KV7 POTASSIUM CHANNELS | RECEPTOR MESSENGER-RNAS | SKIN EPITHELIAL-CELLS | BETA-RECEPTORS | PDGF | ANESTHESIOLOGY | FORMALIN TEST | Inflammatory pain | K+ CHANNELS | Nociceptor excitability | Research Paper
Journal Article
BBA - Molecular Cell Research, ISSN 0167-4889, 12/2016, Volume 1863, Issue 12, pp. 2868 - 2880
The nociceptive noxious heat-activated receptor - TRPV1, conducts calcium and sodium, thus producing a depolarizing receptor potential, leading to activation... 
Nociception | Pain | TRPV1 | Capsaicin | NCLX | Mitochondrial calcium signaling | ACTIVATION | ESSENTIAL COMPONENT | NA+/CA2+ EXCHANGE | ROOT GANGLION NEURONS | MEMBRANE | BIOCHEMISTRY & MOLECULAR BIOLOGY | RECEPTOR | CELL-DEATH | CELL BIOLOGY | INDUCED CA2+ RELEASE | PRIMARY SENSORY NEURONS | RNA, Small Interfering - genetics | Calcium Channels - metabolism | Calcium - metabolism | Capsaicin - pharmacology | Humans | Male | Molecular Imaging | Ganglia, Spinal - cytology | Sodium-Calcium Exchanger - genetics | Sodium - metabolism | Membrane Potential, Mitochondrial - drug effects | TRPV Cation Channels - metabolism | Sodium-Calcium Exchanger - metabolism | TRPV Cation Channels - antagonists & inhibitors | Egtazic Acid - analogs & derivatives | HEK293 Cells | Membrane Potential, Mitochondrial - physiology | Sodium-Calcium Exchanger - antagonists & inhibitors | Single-Cell Analysis | Calcium Channels - genetics | Action Potentials - drug effects | Gene Expression Regulation | Rats | Nociceptors - cytology | Rats, Sprague-Dawley | TRPV Cation Channels - genetics | Action Potentials - physiology | Animals | Egtazic Acid - pharmacology | Nociceptors - metabolism | Primary Cell Culture | Ganglia, Spinal - drug effects | Nociceptors - drug effects | Ganglia, Spinal - metabolism | RNA, Small Interfering - metabolism | Cell death | Machinery | Magneto-electric machines
Journal Article