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1986, Di 1 ban., 4, 4, 2, 300
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西部:新文学(上), ISSN 1671-3311, 2011, Issue 9X, pp. 91 - 94
<正>我和樱妹是同年同月同日生,在老家称老庚,两家相隔不到百米。因我一个堂叔娶了她的姑姑,我叫她爸妈为舅舅、舅娘,两家关系甚好。儿时我们经常在一起摆家家做游戏。一次我们摆家家扮新郎新娘结婚闹洞房时,我们学大人 
我不知道 | 新郎新娘 | 我一 | 辗转难眠 | 杀猪 | 给你 | 对我说 | 爆响 | 一个女孩子 | 天都
Journal Article
by 李海
职教通讯, ISSN 1674-7747, 2017, Issue 34, pp. 23 - 29
Journal Article
艺术市场, ISSN 1671-7910, 2013, Issue 5, pp. 108 - 111
Journal Article
STEM CELLS, ISSN 1066-5099, 03/2019, Volume 37, Issue 3, pp. 430 - 440
Journal Article
Journal Article
STEM CELLS, ISSN 1066-5099, 09/2013, Volume 31, Issue 9, pp. 1775 - 1784
Journal Article
BMC public health, ISSN 1471-2458, 01/2020, Volume 20, Issue 1, pp. 133 - 10
Metabolic syndrome (MS) has abruptly increased in China in the past two decades, gradually representing an important public health threat over the years. Here,... 
Metabolic syndrome | Associated factors | Prevalence
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2014, Volume 9, Issue 7, pp. e98973 - e98973
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2011, Volume 6, Issue 9, p. e24724
Ca2+/calmodulin-dependent protein kinase II delta B (CaMKII delta B) is one of the predominant isoforms of CaMKII in the heart. The precise role of CaMKII... 
PRESSURE-OVERLOAD | MYOCYTES | SARCOPLASMIC-RETICULUM CA2+-ATPASE | TRANSGENIC MOUSE | CARDIAC-HYPERTROPHY | BIOLOGY | CARDIOMYOCYTES | CA2+/CALMODULIN-DEPENDENT PROTEIN-KINASE | NA+-CA2+ EXCHANGER GENE | UP-REGULATION | FAILURE | Immunohistochemistry | Body Weight | Calcium - metabolism | Heart - physiology | Male | Organ Size - physiology | Heart Failure - metabolism | Sodium-Calcium Exchanger - genetics | Lung - physiology | Sarcoplasmic Reticulum Calcium-Transporting ATPases | Aortic Diseases - metabolism | Blotting, Western | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - genetics | Animals | Sodium-Calcium Exchanger - metabolism | Aortic Diseases - complications | Lung - metabolism | Mice | Heart Failure - etiology | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - metabolism | Heart | Histone deacetylase | Neurosciences | Phosphorylation | Transcription | Body weight | Neurobiology | Homeostasis | Immunoblotting | Kinases | Ca2+-transporting ATPase | Proteins | Antagonism | Na+/Ca2+-exchanging ATPase | Toxicology | Transfection | Myocyte-specific enhancer-binding factor 2 | Transgenic animals | Tibia | Rodents | Calcium-binding protein | Aorta | Heart diseases | Pharmaceutical sciences | Calmodulin | Calcium homeostasis | Heart failure | Calcium (intracellular) | Cardiomyocytes | Pharmacology | Mammals | Gene expression | Signaling | Lungs | Isoforms | Ventricle | Diabetes | Aberration | Laboratory animals | NCX1 protein | Endoplasmic reticulum | Adenosine triphosphatase | Ca2+/calmodulin-dependent protein kinase II | Apoptosis
Journal Article
Nature Neuroscience, ISSN 1097-6256, 02/2012, Volume 15, Issue 2, pp. 267 - 273
Dysfunction of fast-spiking, parvalbumin-positive (FS-PV) interneurons is implicated in the pathogenesis of epilepsy. ErbB4, a key Neuregulin 1 (NRG1)... 
PARVALBUMIN-POSITIVE INTERNEURONS | NEURAL DEVELOPMENT | PRIMARY VISUAL-CORTEX | NMDA RECEPTORS | POTASSIUM CHANNELS | ERBB4 | TYROSINE KINASE | GABAERGIC INTERNEURONS | LONG-TERM POTENTIATION | NEUROSCIENCES | K+ CHANNELS | Animal experimentation | Usage | Neurons | Epilepsy | Physiological aspects | Cellular signal transduction | Research
Journal Article