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Neuron, ISSN 0896-6273, 10/2018, Volume 100, Issue 1, pp. 9 - 11
Cardiovascular dysfunction becomes recognized as a risk for dementia. In this issue of , report the age-related, brain endothelial-specific upregulation of... 
blood-brain barrier | caveolin-1 | dementia | acid sphingomyelinase | caveolae | permeability | Proteins | Phosphorylation | Blood-brain barrier | Neurodegeneration | Aging | Dementia disorders | Permeability | Age | Dementia | Sphingomyelin phosphodiesterase | Apoptosis
Journal Article
2015, 1st ed. 2015., ISBN 9781493929061
This book surveys healthy and diseased vascular systems in a multitude of model organisms and systems. It explores a plethora of functions, characteristics,... 
Medicine | Human physiology | Hematology | Systems biology | Angiology | Cell physiology
Web Resource
2015, 1st ed. 2015., ISBN 9781493929061
This book surveys healthy and diseased vascular systems in a multitude of model organisms and systems. It explores a plethora of functions, characteristics,... 
Medicine | Human physiology | Hematology | Systems biology | Angiology | Cell physiology
Web Resource
Blood, ISSN 0006-4971, 04/2014, Volume 123, Issue 15, pp. 2285 - 2286
In this issue of Blood, Yao and colleagues report that the morphogen sonic hedgehog (Shh) is driven by platelet-derived growth factor B (PDGF-BB) in vascular... 
ANGIOGENESIS | HEMATOLOGY | INTEGRITY | BLOOD-BRAIN-BARRIER | PERICYTES | Hedgehog Proteins - metabolism | Neovascularization, Physiologic - physiology | Signal Transduction - physiology | Animals | Proto-Oncogene Proteins c-sis - metabolism | Cell Movement - physiology | Index Medicus | Abridged Index Medicus
Journal Article
Acta Neuropathologica, ISSN 0001-6322, 6/2003, Volume 105, Issue 6, pp. 586 - 592
Journal Article
Neuropathology and Applied Neurobiology, ISSN 0305-1846, 02/2015, Volume 41, Issue 2, pp. 165 - 179
Journal Article
Neuron, ISSN 0896-6273, 10/2018, Volume 100, Issue 1, pp. 9 - 11
Cardiovascular dysfunction becomes recognized as a risk for dementia. In this issue of Neuron, Park et al. (2018) report the age-related, brain... 
blood-brain barrier | caveolin-1 | dementia | acid sphingomyelinase | caveolae | permeability | BLOOD-BRAIN-BARRIER | NEUROSCIENCES | ALZHEIMERS-DISEASE | Youth | Teenagers | Permeability | Dementia
Journal Article
Journal Article
Cancer Cell, ISSN 1535-6108, 04/2016, Volume 29, Issue 4, pp. 427 - 429
Medulloblastoma driven by Wnt/β-catenin and Sonic hedgehog pathway mutations show favorable and poor patient survival upon treatment, respectively. In this... 
ONCOLOGY | CELL BIOLOGY | Cerebellar Neoplasms - drug therapy | Animals | Signal Transduction | Humans | Hedgehog Proteins - metabolism | Medulloblastoma - drug therapy | beta Catenin - metabolism | Blood-Brain Barrier | Health aspects | Medulloblastoma | Index Medicus
Journal Article
Journal of Cerebral Blood Flow & Metabolism, ISSN 0271-678X, 3/2014, Volume 34, Issue 3, pp. 433 - 440
Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 8/2004, Volume 166, Issue 3, pp. 359 - 367
Journal Article
Acta Neuropathologica, ISSN 0001-6322, 5/2016, Volume 131, Issue 5, pp. 753 - 773
The homeostasis of the central nervous system is maintained by the blood–brain barrier (BBB). Angiopoietins (Ang-1/Ang-2) act as antagonizing molecules to... 
Tie2 signaling | Pathology | Neurosciences | Stroke | Vascular endothelial protein tyrosine phosphatase (VE-PTP) | Medicine & Public Health | Blood–brain barrier (BBB) | Angiopoietin-2 | Permeability | Endothelium | Pericytes - drug effects | Angiopoietin-2 - genetics | Humans | Microvessels - metabolism | Capillary Permeability - drug effects | Stroke - complications | Male | Pericytes - ultrastructure | Angiopoietin-2 - pharmacology | Angiopoietin-2 - metabolism | Blood-Brain Barrier - ultrastructure | Capillary Permeability - genetics | Stroke - pathology | Pericytes - pathology | Blood-Brain Barrier - physiology | Female | Receptor, TIE-2 - metabolism | Disease Models, Animal | Microvessels - cytology | Astrocytes - drug effects | Brain Edema - etiology | Brain Edema - pathology | Pericytes - metabolism | Cells, Cultured | Microvessels - drug effects | Mice, Transgenic | Signal Transduction - genetics | Stroke - drug therapy | Electric Impedance | Blood-Brain Barrier - drug effects | Nerve Tissue Proteins - genetics | Nerve Tissue Proteins - metabolism | Endothelium - drug effects | Stroke - metabolism | Animals | Endothelium - metabolism | Signal Transduction - drug effects | Receptor-Like Protein Tyrosine Phosphatases, Class 3 - metabolism | Mice | In Vitro Techniques | Astrocytes - metabolism | Drugs | Immunohistochemistry | Stroke (Disease) | Nervous system diseases | Drug delivery systems | Phosphatases | Vehicles | Index Medicus | Original Paper
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2013, Volume 8, Issue 8, pp. e70233 - e70233
Journal Article
Nature, ISSN 0028-0836, 12/2017, Volume 552, Issue 7684, pp. 248 - 252
Diabetic retinopathy is an important cause of blindness in adults(1,2), and is characterized by progressive loss of vascular cells and slow dissolution of... 
BLOOD-RETINAL BARRIER | ACID | ANGIOGENESIS | CHOLESTEROL | STABILITY | MULTIDISCIPLINARY SCIENCES | ENDOTHELIAL-CELLS | DEPENDENT ENDOCYTOSIS | MEMBRANES | VE-CADHERIN | ASSOCIATION | Retina - drug effects | Retina - metabolism | Cadherins - metabolism | Pericytes - drug effects | Humans | Capillary Permeability - drug effects | Male | Diabetic Retinopathy - pathology | Antigens, CD - metabolism | Vitreous Body - metabolism | Pericytes - pathology | Retina - enzymology | Intercellular Junctions - pathology | Female | Cell Membrane - drug effects | Disease Models, Animal | Ependymoglial Cells | Cell Survival - drug effects | Endothelial Cells - metabolism | Mice, Inbred C57BL | Solubility | Pancreatic Elastase - metabolism | Fatty Acids, Unsaturated - metabolism | Diabetic Retinopathy - metabolism | Disease Progression | Cell Movement - drug effects | Diabetic Retinopathy - prevention & control | Animals | Carrier Proteins - metabolism | Diabetic Retinopathy - enzymology | Intercellular Junctions - drug effects | Presenilin-1 - metabolism | Mice | Endothelial Cells - pathology | Epoxide Hydrolases - antagonists & inhibitors | Retina - pathology | Docosahexaenoic Acids - metabolism | Endothelial Cells - drug effects | Prevention | Physiological aspects | Enzymes | Diabetic retinopathy | Hydrolases | Epoxy compounds | Retinopathy | Pathogenesis | Cell junctions | Retina | Glial cells | Cell interactions | Kinases | Docosahexaenoic acid | Epoxide hydrolase | Vascularization | Proteins | Signal transduction | Hyperglycemia | N-Cadherin | Rodents | Inhibition | Growth factors | Edema | Abnormalities | Diabetes mellitus | Permeability | Cadherin | Cholesterol | Endothelial cells | Acids | Presenilin 1 | Nitric oxide | Blindness | Vitreous humour | Adults | Infiltration | Diabetes | Staphylococcal enterotoxin H | Index Medicus
Journal Article