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1. Future proofing 02
: Stuart Lipton / Richard Rogers, Chris Wise / Malcolm Smith
by Lipton, Stuart and Rappaport, Nina and Steffen, Andrew and Yale University. School of Architecture
2007, Edward P. Bass distinguished visiting architecture fellowship, ISBN 9780393732375, 192
Forecasting | Stratford (London, England) Forecasting | City planning
Book
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2. Full Text Pathologically activated therapeutics for neuroprotection
by Lipton, Stuart A
Nature Reviews Neuroscience, ISSN 1471-003X, 10/2007, Volume 8, Issue 10, pp. 803 - 808
Many drugs that have been developed to treat neurodegenerative diseases fail to gain approval for clinical use because they are not well tolerated in humans.... 
Excitatory Amino Acid Antagonists - metabolism | Neuroprotective Agents - therapeutic use | Receptors, N-Methyl-D-Aspartate - antagonists & inhibitors | Excitatory Amino Acid Antagonists - therapeutic use | Humans | Neurodegenerative Diseases - prevention & control | Receptors, N-Methyl-D-Aspartate - metabolism | Neurodegenerative Diseases - metabolism | Neurodegenerative Diseases - drug therapy | Neuroprotective Agents - metabolism | Animals | Receptors, N-Methyl-D-Aspartate - agonists | Memantine - metabolism | Memantine - therapeutic use | Drug Delivery Systems - methods | Physiological aspects | Complications and side effects | Nervous system diseases | Product development | Drug therapy | Nervous system agents
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3. Full Text Opinion - Pathologically activated therapeutics for neuroprotection
by Lipton, SA
NATURE REVIEWS NEUROSCIENCE, ISSN 1471-0048, 10/2007, Volume 8, Issue 10, pp. 803 - 808
Many drugs that have been developed to treat neurodegenerative diseases fail to gain approval for clinical use because they are not well tolerated in humans.... 
S-NITROSYLATION | NERVOUS-SYSTEM | MOLECULAR MECHANISMS | GLUTAMATE NEUROTOXICITY | FOCAL CEREBRAL-ISCHEMIA | ERYTHROPOIETIN RECEPTOR | NITRIC-OXIDE | MATRIX-METALLOPROTEINASE EXPRESSION | NMDA RECEPTOR INHIBITION | BLOOD-BRAIN-BARRIER | NEUROSCIENCES
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4. Full Text β-Amyloid-related mitochondrial fission and neuronal injury
by Cho, Dong-Hyung and Nakamura, Tomohiro and Fang, Jianguo and Cieplak, Piotr and Godzik, Adam and Gu, Zezong and Lipton, Stuart A
Science, ISSN 0036-8075, 04/2009, Volume 324, Issue 5923, pp. 102 - 105
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5. Full Text Oligomeric Aβ-induced synaptic dysfunction in Alzheimer's disease
by Tu, Shichun and Tu, Shichun and Okamoto, Shu-ichi and Okamoto, Shu-Ichi and Lipton, Stuart A and Lipton, Stuart A and Xu, Huaxi and Xu, Huaxi
Molecular Neurodegeneration, ISSN 1750-1326, 11/2014, Volume 9, Issue 1, pp. 48 - 48
Alzheimer's disease (AD) is a devastating disease characterized by synaptic and neuronal loss in the elderly. Compelling evidence suggests that soluble... 
Cognitive decline | Mitochondrial dysfunction | Tau hyperphosphorylation | Calcium | Aberrant neuronal network activity | Synaptic loss | NMDA receptors | PSD-95 | Alzheimer's disease | Aβ oligomers | POSTSYNAPTIC DENSITY-95 | AMYLOID PRECURSOR PROTEIN | NEUROSCIENCES | INDUCED NEURONAL DYSFUNCTION | TAU-HYPERPHOSPHORYLATION | S-NITROSYLATION | A beta oligomers | ABNORMAL MITOCHONDRIAL DYNAMICS | MOUSE MODEL | LONG-TERM POTENTIATION | SOLUBLE OLIGOMERS | TRANSGENIC MICE | Alzheimer Disease - physiopathology | Animals | Synapses - metabolism | Humans | Alzheimer Disease - metabolism | Amyloid beta-Peptides - metabolism | Synapses - pathology | Alzheimer Disease - pathology | Index Medicus
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6. Full Text Partnering with Big Pharma—What Academics Need to Know
by Lipton, Stuart A and Nordstedt, Christer
Cell, ISSN 0092-8674, 04/2016, Volume 165, Issue 3, pp. 512 - 515
Knowledge of the parameters of drug development can greatly aid academic scientists hoping to partner with pharmaceutical companies. Here, we discuss the three... 
Animals | Humans | Drug Design | Clinical Trials as Topic | Drug Evaluation, Preclinical | Pharmacokinetics | Drug Delivery Systems | Universities | Pharmaceutical industry | Medical colleges
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7. Full Text Paradigm shift in neuroprotection by NMDA receptor blockade: Memantine and beyond
by Lipton, Stuart A
Nature Reviews Drug Discovery, ISSN 1474-1776, 02/2006, Volume 5, Issue 2, pp. 160 - 170
Neuroprotective drugs tested in clinical trials, particularly those that block N-methyl-d-aspartate-sensitive glutamate receptors (NMDARs), have failed... 
NEURONAL APOPTOSIS | RETINAL GANGLION-CELLS | GLUTAMATE RECEPTORS | D-SERINE | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | ALZHEIMERS-DISEASE | NITRIC-OXIDE | PHARMACOLOGY & PHARMACY | RANDOMIZED CONTROLLED-TRIAL | CHANNEL BLOCKERS | METHYL-D-ASPARTATE | MEDIATED NEUROTOXICITY | Neuroprotective Agents - therapeutic use | Neurotoxins - pharmacology | Receptors, N-Methyl-D-Aspartate - antagonists & inhibitors | Memantine - pharmacology | Animals | Neuroprotective Agents - pharmacology | Memantine - analogs & derivatives | Humans | Excitatory Amino Acid Antagonists - pharmacology
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8. Prehistoric India to 1000 B.C.
by Piggott, Stuart
1950, Pelican books, Volume A205, 293
India History | India Antiquities
Book
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9. Full Text Protein S -Nitrosylation as a Therapeutic Target for Neurodegenerative Diseases
by Nakamura, Tomohiro and Lipton, Stuart A
Trends in Pharmacological Sciences, ISSN 0165-6147, 2015, Volume 37, Issue 1, pp. 73 - 84
At physiological levels, nitric oxide (NO) contributes to the maintenance of normal neuronal activity and survival, thus serving as an important regulatory... 
Advanced Basic Science | Parkinson's disease | protein S-nitrosylation | NitroMemantine | Alzheimer's disease | denitrosylation | transnitrosylation | THIOREDOXIN-MIMETIC PEPTIDES | NMDA RECEPTOR | OXIDATIVE STRESS | NITRIC-OXIDE PRODUCTION | INHIBITION | GLYCERALDEHYDE-3-PHOSPHATE DEHYDROGENASE | ALCOHOL-DEHYDROGENASE | PHARMACOLOGY & PHARMACY | NITROSOGLUTATHIONE | PARKINSONS-DISEASE | CELL-DEATH | Reactive Nitrogen Species - metabolism | Humans | Receptors, N-Methyl-D-Aspartate - metabolism | Protein Processing, Post-Translational | Glyceraldehyde-3-Phosphate Dehydrogenases - metabolism | Neurodegenerative Diseases - metabolism | Nitric Oxide - metabolism | Nitrosation | S-Nitrosothiols - metabolism | Second Messenger Systems | Post-translational modification | Cysteine | Thiols | Nitric oxide | Nervous system | Development and progression | Degeneration | Health aspects | Alzheimer’s disease | Protein S-nitrosylation | Parkinson’s disease
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10. Full Text Direct Reprogramming of Adult Human Fibroblasts to Functional Neurons under Defined Conditions
by Ambasudhan, Rajesh and Talantova, Maria and Coleman, Ronald and Yuan, Xu and Zhu, Saiyong and Lipton, Stuart A and Ding, Sheng
Cell Stem Cell, ISSN 1934-5909, 08/2011, Volume 9, Issue 2, pp. 113 - 118
Human induced pluripotent stem cells (hiPSCs) have been generated by reprogramming a number of different somatic cell types using a variety of approaches. In... 
SOMATIC-CELLS | STEM-CELLS | MIR-124 | DIRECT CONVERSION | HYBRIDIZATION | DIFFERENTIATION | MICRORNAS | EXPRESSION | CELL & TISSUE ENGINEERING | CELL BIOLOGY | Synapses - drug effects | Humans | Neurons - cytology | Cell Culture Techniques - methods | Cellular Reprogramming - drug effects | Synapses - metabolism | Cell Differentiation - drug effects | Fibroblasts - drug effects | Culture Media - pharmacology | Adult | Fibroblasts - cytology | Neurons - metabolism | Dermis - cytology | Neurons - drug effects | Cellular Reprogramming - genetics | Infant, Newborn | Fibroblasts - metabolism
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11. Full Text Nrf2/ARE-mediated antioxidant actions of pro-electrophilic drugs
by Satoh, Takumi and McKercher, Scott R and Lipton, Stuart A
Free Radical Biology and Medicine, ISSN 0891-5849, 12/2013, Volume 65, pp. 645 - 657
Living cells maintain a balance between oxidation and reduction, and perturbations of this redox balance are thought to contribute to various diseases. Recent... 
Keap1 | Pathologically activated therapeutic | Electrophilic counterattack | Glutathione | Electrophile | CARNOSIC ACID | OXIDATIVE-STRESS | BIOCHEMISTRY & MOLECULAR BIOLOGY | NEURONAL HT22 CELLS | STRESS-INDUCED DEATH | RETINAL GANGLION-CELLS | PIGMENT EPITHELIAL-CELLS | CYCLOPENTENONE PROSTAGLANDINS | ENDOCRINOLOGY & METABOLISM | ROSEMARY ROSMARINUS-OFFICINALIS | CENTRAL-NERVOUS-SYSTEM | HEME OXYGENASE-1 | Animals | Antioxidant Response Elements - physiology | Antioxidants - chemistry | Humans | Hydroquinones - chemistry | NF-E2-Related Factor 2 - metabolism | Drug Design | Prodrugs - chemistry | Antioxidants - pharmacology | Oxidative Stress - drug effects | Hydroquinones - pharmacology | Prodrugs - pharmacology | Drugs | Antioxidants | Enzymes | Nervous system diseases | Analysis | Quinone | Cysteine | Therapeutics | Homeopathy | Materia medica and therapeutics | Pathologically-Activated Therapeutic | Electrophilic Counterattack
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12. Full Text Cell death: protein misfolding and neurodegenerative diseases
by Nakamura, Tomohiro and Lipton, Stuart A
Apoptosis, ISSN 1360-8185, 4/2009, Volume 14, Issue 4, pp. 455 - 468
Several chronic neurodegenerative disorders manifest deposits of misfolded or aggregated proteins. Genetic mutations are the root cause for protein misfolding... 
Biochemistry, general | Biomedicine | Protein misfolding | S -Nitrosylation | Neurodegeneration | Oncology | Cancer Research | Ubiquitin-proteasome system | Molecular chaperone | Virology | Cell Biology | S-Nitrosylation | DISULFIDE-ISOMERASE FAMILY | NMDA RECEPTOR-ACTIVITY | NITRIC-OXIDE SYNTHASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | ENDOPLASMIC-RETICULUM STRESS | UBIQUITIN LIGASE ACTIVITY | CELL BIOLOGY | PARKINS PROTECTIVE FUNCTION | LONG-TERM POTENTIATION | LEWY-BODY FORMATION | METHYL-D-ASPARTATE | Neurodegenerative Diseases - pathology | Reactive Oxygen Species - metabolism | Models, Biological | Protein Disulfide-Isomerases - metabolism | Humans | Cell Death | Ubiquitin-Protein Ligases - metabolism | Neurodegenerative Diseases - metabolism | Nitric Oxide - metabolism | Nitrosation | Protein Folding | Ubiquitin | Methyl aspartate | Neurosciences | Ligases | Cell death | Nitric oxide | Stem cells | Nervous system | Degeneration | Biochemistry | Glutamate | Index Medicus
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13. Full Text Mitochondrial dynamics in cell death and neurodegeneration
by Cho, Dong-Hyung and Nakamura, Tomohiro and Lipton, Stuart A
Cellular and Molecular Life Sciences, ISSN 1420-682X, 10/2010, Volume 67, Issue 20, pp. 3435 - 3447
Mitochondria are highly dynamic organelles that continuously undergo two opposite processes, fission and fusion. Mitochondrial dynamics influence not only... 
Mitochondria dynamics | Neurodegenerative diseases | Opa1 | Cell death | Mitofusin 1/2 | Drp1 | DIFFERENTIATION-ASSOCIATED PROTEIN-1 | MARIE-TOOTH-DISEASE | ALZHEIMERS-DISEASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | PEROXISOMAL FISSION | MAMMALIAN-CELLS | CELL BIOLOGY | DOMINANT OPTIC ATROPHY | AMYLOID-BETA | CYTOCHROME-C RELEASE | HUNTINGTONS-DISEASE | PARKINSONS-DISEASE | Mitochondria - metabolism | Animals | Neurodegenerative Diseases - metabolism | Humans | Nerve Degeneration - metabolism | Cell Death | Neurosciences | Nervous system diseases |