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Journal Article
American Journal of Physiology: Regulatory, Integrative and Comparative Physiology, ISSN 0363-6119, 04/2017, Volume 312, Issue 4, pp. R485 - R485
Preeclampsia (PE) is a devastating disorder of pregnancy that classically presents with maternal hypertension and proteinuria after 20 wk of gestation. In... 
Journal Article
Hypertension, ISSN 0194-911X, 11/2014, Volume 64, Issue Suppl_1 Suppl 1, pp. A061 - A061
We recently reported that PVN-specific deletion of the NADPH oxidase subunit p22 attenuates oxidative stress in this brain region and prevents diet-induced... 
Journal Article
American Journal of Physiology, ISSN 0363-6119, 09/2014, Volume 307, Issue 5, p. R490
  Preeclampsia is a hypertensive, proteinuric disease that affects 5-10% of all pregnancies and is a leading cause of maternal and perinatal... 
Angiogenesis | Placenta | Cytokines | Preeclampsia | Prenatal development | Interferon
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 07/2011, Volume 31, Issue 7, pp. 1565 - 1572
OBJECTIVE—Interleukin 17A (IL17A) is involved in many inflammatory processes, but its role in atherosclerosis remains controversial. We examined the role of... 
atherosclerosis | nitric oxide | cytokines | vascular biology | superoxide | IL-17A | INTACT | FOAM CELLS | PLAQUE | MONOCYTE MIGRATION | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | T-CELLS | Antibodies, Neutralizing - administration & dosage | Inflammation - pathology | Spleen - immunology | Apolipoproteins E - deficiency | Atherosclerosis - genetics | Dendritic Cells - immunology | Humans | Middle Aged | Interleukin-17 - immunology | Male | Aorta - metabolism | Interferon-gamma - metabolism | Aortic Diseases - metabolism | Inflammation - metabolism | Ligation | Lymphocytes - immunology | Time Factors | Carotid Artery Diseases - pathology | Dietary Fats | Superoxides - metabolism | Female | Angiotensin II | Carotid Artery Diseases - metabolism | Aortic Diseases - pathology | Disease Models, Animal | Carotid Artery Diseases - immunology | Interleukin-17 - blood | Atherosclerosis - pathology | Atherosclerosis - immunology | Aortic Diseases - immunology | Mice, Inbred C57BL | Cells, Cultured | Interleukin-17 - genetics | Aorta - immunology | Inflammation - immunology | Interleukin-17 - deficiency | Atherosclerosis - metabolism | Mice, Knockout | Interleukin-17 - metabolism | Aortic Diseases - genetics | Animals | Apolipoproteins E - genetics | Carotid Artery Diseases - genetics | Inflammation - genetics | Carotid Arteries - surgery | Aged | Mice | Nitric Oxide - metabolism | Interferon-gamma | Reactive oxygen species | Apolipoprotein E | Interleukin 17 | Atherosclerosis
Journal Article
Circulation Research, ISSN 0009-7330, 07/2010, Volume 107, Issue 2, pp. 263 - 270
RATIONALE:We have previously found that T lymphocytes are essential for development of angiotensin II–induced hypertension; however, the mechanisms responsible... 
vascular inflammation | T cells | hypertension | central nervous system | CARDIAC & CARDIOVASCULAR SYSTEMS | AV3V | RATS | INFUSION | BLOOD-PRESSURE | RECEPTOR STIMULATION | REDUCTION | PERIPHERAL VASCULAR DISEASE | CENTRAL-NERVOUS-SYSTEM | DYSFUNCTION | HEMATOLOGY | EXTRACELLULAR-SUPEROXIDE DISMUTASE | CELL | Blood Pressure | Vasculitis - metabolism | Third Ventricle - injuries | Vasculitis - physiopathology | Homeodomain Proteins - metabolism | Antihypertensive Agents - administration & dosage | Receptors, Antigen, T-Cell, alpha-beta - immunology | Adoptive Transfer | Vasculitis - prevention & control | Genes, T-Cell Receptor beta | Third Ventricle - physiopathology | T-Lymphocytes - transplantation | Vasculitis - immunology | Vasculitis - genetics | Time Factors | T-Lymphocytes - drug effects | Hypertension - chemically induced | Superoxides - metabolism | Hypertension - prevention & control | Hypertension - genetics | Angiotensin II | Vasculitis - chemically induced | Disease Models, Animal | Administration, Oral | Mice, Inbred C57BL | Mice, Transgenic | Hypertension - immunology | Homeodomain Proteins - genetics | Hypertension - physiopathology | Hypertension - metabolism | Mice, Knockout | Hydralazine - administration & dosage | Animals | Lymphocyte Activation - drug effects | Third Ventricle - immunology | Genes, T-Cell Receptor alpha | T-Lymphocytes - immunology | Mice | Norepinephrine | Hypertension
Journal Article
JACC (Journal of the American College of Cardiology), ISSN 0735-1097, 2008, Volume 52, Issue 22, pp. 1803 - 1809
Journal Article
Biological Psychiatry, ISSN 0006-3223, 2012, Volume 71, Issue 9, pp. 774 - 782
Background Psychological stress is a significant risk factor for hypertension and also directly affects the immune system. We have previously reported that T... 
Psychiatry | T lymphocytes | Angiotensin II | inflammation | hypertension | vascular | psychological stress | PSYCHIATRY | RATS | NEUROSCIENCES | BLOOD-PRESSURE | RESTRAINT STRESS | II-INDUCED HYPERTENSION | PARAVENTRICULAR NUCLEUS | RENIN-ANGIOTENSIN SYSTEM | CENTRAL-NERVOUS-SYSTEM | DIFFERENTIAL CARDIOVASCULAR-RESPONSES | PSYCHOSOCIAL STRESS | Blood Pressure - immunology | Genes, RAG-1 - genetics | Vascular Diseases - complications | Stress, Psychological - immunology | Corticotropin-Releasing Hormone - biosynthesis | Inflammation - metabolism | Lymphocyte Activation - immunology | T-Lymphocytes - drug effects | Blood Pressure - physiology | Hypertension - genetics | Vascular Diseases - immunology | Angiotensin II - pharmacology | Maze Learning - physiology | Gene Expression Regulation - genetics | Mice, Inbred C57BL | Gene Expression Regulation - physiology | Hypertension - immunology | Inflammation - immunology | Vascular Diseases - physiopathology | Hypertension - physiopathology | Mice, Knockout | Gene Expression Regulation - drug effects | Stress, Psychological - genetics | Animals | Adoptive Transfer - methods | Lymphocyte Activation - drug effects | Hypertension - complications | T-Lymphocytes - immunology | Mice | Stress, Psychological - complications | Stress, Psychological - physiopathology | Hypertension | Medical colleges | Lymphocytes | Angiotensin | Inflammation | Stress (Psychology) | T cells | Psychological stress
Journal Article
American Journal of Physiology (Consolidated), ISSN 0002-9513, 04/2017, Volume 312, Issue 4, p. R485
--Preeclampsia (PE) is a devastating disorder of pregnancy that classically presents with maternal hypertension and proteinuria after 20 wk of gestation. In... 
Physiological aspects | Physiological research | Research | Prenatal influences | Preeclampsia
Journal Article
American Journal of Physiology - Regulatory Integrative and Comparative Physiology, ISSN 0363-6119, 09/2014, Volume 307, Issue 5, pp. R490 - R492
Journal Article