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Journal of Pharmacology and Experimental Therapeutics, ISSN 0022-3565, 02/2018, Volume 364, Issue 2, pp. 347 - 358
We describe a stress-derived type-2 diabetes model in male mice, and formulate new hypotheses on how the model was induced, how diabetes-like alterations were... 
Journal Article
PLOS ONE, ISSN 1932-6203, 03/2019, Volume 14, Issue 3, pp. e0213529 - e0213529
Plasmodium falciparum severe malaria causes more than 400,000 deaths every year. One feature of P. falciparum-parasitized erythrocytes (pRBC) leading to... 
SEQUESTRATION | PATHOGENESIS | APOPTOSIS | IN-VITRO | ACTIVATION | MULTIDISCIPLINARY SCIENCES | ENDOTHELIAL-CELLS | RHO GTPASES | PHENOTYPE | INFECTED ERYTHROCYTES | CEREBRAL MALARIA | Prevention | Complications and side effects | Usage | Nervous system diseases | Malaria | Genetic aspects | Scanning microscopy | Risk factors | Plasmodium falciparum | Permeability | G proteins | Endothelium
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2013, Volume 8, Issue 5, p. e65898
Overexpression of pro-inflammatory cytokines and cellular energy failure are associated with neuroinflammatory disorders, such as Alzheimer's disease.... 
TOXIN-INDUCED ACTIVATION | SPATIAL MEMORY PERFORMANCE | MULTIDISCIPLINARY SCIENCES | MOUSE | BETA PRECURSOR PROTEIN | RISK | LONG-TERM POTENTIATION | APOE | RHO-GTPASES | MODULATION | APOLIPOPROTEIN-E | Memory - drug effects | Apolipoprotein E4 - genetics | Humans | Hippocampus - drug effects | Alzheimer Disease - pathology | Brain - metabolism | Inflammation - metabolism | Adenosine Triphosphate - metabolism | Escherichia coli Proteins - pharmacology | Amyloid beta-Peptides - metabolism | Spatial Behavior - drug effects | Disease Models, Animal | Biomarkers - metabolism | Alzheimer Disease - physiopathology | Cytokines - metabolism | Brain - physiopathology | Alzheimer Disease - drug therapy | Spatial Behavior - physiology | Mice, Transgenic | Hippocampus - pathology | Cognition - drug effects | Enzyme Activation - drug effects | Bacterial Toxins - therapeutic use | Brain - drug effects | Hippocampus - metabolism | Animals | Escherichia coli Proteins - therapeutic use | Signal Transduction - drug effects | Bacterial Toxins - pharmacology | rho GTP-Binding Proteins - metabolism | Alzheimer Disease - metabolism | Mice | Hippocampus - physiopathology | Energy Metabolism - drug effects | Cytokines | Analysis | Atherosclerosis | Models | Genetic engineering | Research | G proteins | Alzheimer's disease | Neurophysiology | Brain | Neurosciences | Animal models | Energy levels | Memory | Interleukin | Cognitive ability | Spatial discrimination | Homeostasis | Cytotoxicity | Diabetic neuropathy | Accumulation | Proteins | E coli | Energy | Transgenic animals | Rodents | Modulation | Bacteria | Physiology | Behavior | Adenosine triphosphate | Apolipoprotein E4 | Neuromuscular diseases | Neurodegenerative diseases | Transgenic mice | Rac1 protein | Pharmacology | Metabolism | Apolipoproteins | Disease control | Energy balance | Brain research | Arteriosclerosis | β-Amyloid | Alzheimers disease | ATP | Spatial memory
Journal Article
International Journal of Molecular Sciences, ISSN 1661-6596, 07/2018, Volume 19, Issue 7, p. 1825
The Escherichia coli protein toxin cytotoxic necrotizing factor 1 (CNF1), which acts on the Rho GTPases that are key regulators of the actin cytoskeleton, is... 
Mitochondrial diseases | Myoclonic epilepsy with ragged red fibers syndrome | Actin cytoskeleton | Cytotoxic necrotizing factor 1 | Adenosine triphosphate | ACTIVATION | cytotoxic necrotizing factor 1 | BIOCHEMISTRY & MOLECULAR BIOLOGY | NECROTIZING FACTOR-1 | SYNAPTIC PLASTICITY | CHEMISTRY, MULTIDISCIPLINARY | FIBROBLASTS | actin cytoskeleton | mitochondrial diseases | MYOCLONIC EPILEPSY | MERRF | myoclonic epilepsy with ragged red fibers syndrome | adenosine triphosphate | MOUSE MODEL | RHO GTPASES | DEAMIDATION | MODULATION | Stress Fibers - drug effects | Humans | Middle Aged | Male | Adenosine Triphosphate - biosynthesis | Mitochondria - ultrastructure | Bacterial Toxins - isolation & purification | Electron Transport Complex IV - metabolism | Stress Fibers - ultrastructure | DNA, Mitochondrial - genetics | MERRF Syndrome - drug therapy | MERRF Syndrome - genetics | Mitochondria - genetics | Membrane Transport Proteins - genetics | Escherichia coli Proteins - pharmacology | Membrane Transport Proteins - metabolism | Fibroblasts - metabolism | Gene Expression | DNA, Mitochondrial - metabolism | MERRF Syndrome - pathology | Receptors, Cell Surface - metabolism | Mitochondria - metabolism | Electron Transport Complex IV - genetics | MERRF Syndrome - metabolism | Mitochondria - drug effects | Escherichia coli - chemistry | Escherichia coli Proteins - isolation & purification | Fibroblasts - pathology | Pilot Projects | Stress Fibers - metabolism | Bacterial Toxins - pharmacology | Fibroblasts - drug effects | Mutation | Primary Cell Culture | Receptors, Cell Surface - genetics | Regulators | Bacterial proteins | Cytotoxic necrotizing factor | Epilepsy | Impairment | Homeostasis | Cytotoxicity | Mitochondrial DNA | Cell interactions | Patients | Neurological diseases | Proteins | Energy balance | MERRF Syndrome | E coli | Actin | Morphology | Fibroblasts | Necrotizing factor 1 | Cytoskeleton | Deoxyribonucleic acid--DNA
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2015, Volume 10, Issue 10, p. e0140495
Epilepsy, one of the most common conditions affecting the brain, is characterized by neuroplasticity and brain cell energy defects. In this work, we... 
DENDRITIC SPINES | NEURONAL-ACTIVITY | TEMPORAL-LOBE EPILEPSY | HIGH-FREQUENCY OSCILLATIONS | MULTIDISCIPLINARY SCIENCES | ELECTROSHOCK SEIZURE THRESHOLD | RHO-GTPASES | SYNAPTIC PLASTICITY | ABSENCE EPILEPSY | WAVE-DISCHARGES | MITOCHONDRIAL FISSION | Biomarkers - metabolism | Seizures - prevention & control | Brain - physiopathology | Neuronal Plasticity - drug effects | Male | Seizures - metabolism | Mitochondria - metabolism | Mitochondria - drug effects | Cognition - drug effects | Brain - drug effects | Brain - metabolism | Seizures - physiopathology | Animals | Mitochondrial Dynamics - drug effects | Seizures - pathology | Aging - physiology | Bacterial Toxins - pharmacology | Adenosine Triphosphate - metabolism | Escherichia coli Proteins - pharmacology | Brain - pathology | Mice, Inbred DBA | Mice | Energy Metabolism - drug effects | Aging - metabolism | Physiological aspects | Causes of | Research | Seizures (Medicine) | G proteins | Muscle proteins | Brain | Neurosciences | Animal models | Spine | Cytotoxic necrotizing factor | Pathogenesis | Epilepsy | Cognitive ability | Homeostasis | Cytotoxicity | Proteins | Mitochondria | Energy | E coli | Actin | Rodents | Necrotizing factor 1 | Inbreeding | Alzheimer's disease | Seizures | Phenotypes | Neurodegenerative diseases | Cortex (visual) | Metabolism | Neurological diseases | Energy balance | Brain research | Pyramidal cells | Cytotoxic necrotizing factor 1 | Plasticity (visual) | Rett syndrome | Performance enhancement | Cytoskeleton | Mutation | ATP | Animal cognition
Journal Article
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2013, Volume 8, Issue 6
Medical and Surgical Sciences Department - DIMEC - University of Bologna, Bologna, Italy The correct affiliation for Gabriele Campana is: (2013) Correction:... 
Brain | Animal models | Energy levels | Neurodegenerative diseases | Cognitive ability | Inflammation | Alzheimers disease | Alzheimer's disease
Journal Article
Journal of Endocrinology, ISSN 0022-0795, 12/2012, Volume 215, Issue 3, pp. 413 - 424
The double postnatal stress model (brief maternal separation plus sham injection daily applied from birth to weaning) induces metabolic alterations similar to... 
MESSENGER-RNA | VISUAL-EVOKED-POTENTIALS | OPIOID RECEPTOR | BDNF | ADULT MALE-MICE | NEUROTROPHIC FACTOR | ENDOCRINOLOGY & METABOLISM | RATS | METABOLIC ALTERATIONS | TERM | EXPRESSION | Brain - drug effects | Stress, Physiological - physiology | Animals | Naloxone - pharmacology | Narcotic Antagonists - pharmacology | Male | Mice | Brain - physiology
Journal Article