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Clinical Oral Investigations, ISSN 1432-6981, 3/2019, Volume 23, Issue 3, pp. 1067 - 1075
The objective of the present study was to compare the clinical performance of screw-retained, monolithic, zirconia, and cemented porcelain-fused-to-metal (PFM)... 
Treatment outcome | Peri-implantitis | Dental restoration | Screw-retaining | Dentistry | Single-tooth implants | Cementation | SYSTEM | EXCESS | FIXED DENTAL PROSTHESES | GUIDELINES | RECONSTRUCTIONS | RESTORATIONS | BONE LOSS | FRACTURE-RESISTANCE | DENTISTRY, ORAL SURGERY & MEDICINE | OCCLUSION | Clinical trials | Zirconium | Zirconium oxide | Patient satisfaction | Implant dentures
Journal Article
International journal of molecular sciences, ISSN 1422-0067, 05/2019, Volume 20, Issue 9, p. 2179
Mathematical optimization framework allows the identification of certain nodes within a signaling network. In this work, we analyzed the complex... 
optimal drug combination | ERK signaling | efficient intervention points | optimal treatment strategies | optimal pharmacological modulation | optimal drug targeting
Journal Article
Basic Research in Cardiology, ISSN 0300-8428, 11/2016, Volume 111, Issue 6, pp. 1 - 4
To access, purchase, authenticate, or subscribe to the full-text of this article, please visit this link: http://dx.doi.org/10.1007/s00395-016-0582-1 
Medicine & Public Health | Cardiology | ARRHYTHMIAS | HYPERTROPHY | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | RYANODINE RECEPTOR | PHOSPHORYLATION | CALMODULIN | CARDIOVASCULAR-DISEASE | GENE-THERAPY | PROTEIN-KINASE-II | ATRIAL-FIBRILLATION | Heart failure
Journal Article
Journal Article
Nature Medicine, ISSN 1078-8956, 01/2009, Volume 15, Issue 1, pp. 75 - 83
The extracellular-regulated kinases ERK1 and ERK2 (commonly referred to as ERK1/2) have a crucial role in cardiac hypertrophy. ERK1/2 is activated by... 
MEDICINE, RESEARCH & EXPERIMENTAL | CARDIOMYOCYTE HYPERTROPHY | ACTIVATED PROTEIN-KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | HEART-FAILURE | MAP KINASES | NUCLEAR TRANSLOCATION | CELL BIOLOGY | MYOCARDIAL HYPERTROPHY | IN-VIVO | CHRONIC PRESSURE-OVERLOAD | BETA-GAMMA-SUBUNITS | SIGNAL-REGULATED KINASES-1/2 | Phosphorylation - physiology | MAP Kinase Signaling System - physiology | Humans | Protein Multimerization | Cardiomegaly - etiology | Cercopithecus aethiops | Threonine - metabolism | Phosphorylation - genetics | Mitogen-Activated Protein Kinase 1 - genetics | Threonine - genetics | GTP-Binding Protein beta Subunits - metabolism | Heart Failure - etiology | GTP-Binding Protein gamma Subunits - metabolism | Mitogen-Activated Protein Kinase 1 - physiology | Mitogen-Activated Protein Kinase 3 - genetics | Cells, Cultured | Mice, Transgenic | Heart Failure - metabolism | Mitogen-Activated Protein Kinase 3 - physiology | Animals | Amino Acid Substitution - physiology | Mitogen-Activated Protein Kinase 3 - metabolism | Models, Biological | Protein Binding | Signal Transduction - physiology | Mice | Cardiomegaly - genetics | COS Cells | Cardiomegaly - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Phosphorylation | Heart enlargement | Physiological aspects | Genetic aspects | Research | Gene expression | Protein kinases | Risk factors | Biochemistry | Cellular biology | Kinases | Cardiology | Substrates
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 10/2019
Journal Article
Journal of Physiology, ISSN 0022-3751, 06/2017, Volume 595, Issue 12, p. 4073
Stimulation of [beta]-adrenergic receptors ([beta]ARs) provides the most efficient physiological mechanism to enhance contraction and relaxation of the heart.... 
Heart failure | Heart | Physiological aspects | College teachers | G proteins | Protein kinases | Stress (physiology) | Fighting | Cycles | Calcium | Downstream effects | Raf protein | Stimulation | Activation | Fuels | Remodeling | Kinases | Contraction | Receptors | Control | Constrictions | Heart diseases | Muscles | Cardiomyocytes | Signaling | Side effects | Inhibitors | Reviews | Running | Death | Adrenergic receptors | Receptor mechanisms | Ventricle | Cardiac output | Receptors (physiology)
Journal Article
Journal Article
by Colleoni, Marco and Luo, Weixiu and Karlsson, Per and Chirgwin, Jacquie and Aebi, Stefan and Jerusalem, Guy and Neven, Patrick and Hitre, Erika and Graas, Marie-Pascale and Simoncini, Edda and Kamby, Claus and Thompson, Alastair and Loibl, Sibylle and Gavilá, Joaquín and Kuroi, Katsumasa and Marth, Christian and Müller, Bettina and O'Reilly, Seamus and Di Lauro, Vincenzo and Gombos, Andrea and Ruhstaller, Thomas and Burstein, Harold and Ribi, Karin and Bernhard, Jürg and Viale, Giuseppe and Maibach, Rudolf and Rabaglio-Poretti, Manuela and Gelber, Richard D and Coates, Alan S and Di Leo, Angelo and Regan, Meredith M and Goldhirsch, Aron and Vandebroek, An and Berliere, Martine and Mitine, Carine and Vuylsteke, Peter and Borms, Marleen and D'Hondt, Randal and Glorieux, Philippe and Mebis, Jeroen and Verhoeven, Didier and Coibion, Michael and Forget, Frederic and Duck, Lionel and Wyendaele, Wim and Barbeaux, Annelore and Salmon, Jean-Paul and Berteloot, Patrick and Vermeij, Joanna and Richard, Vincent and Cinieri, Saverio and Gianni, Lorenzo and Clerico, Mario and Pinotti, Graziella and Bernardo, Antonio and Biganzoli, Leo and Gennari, Alessandra and Graiff, Claudio and Amadori, Dino and Passalacqua, Rodolfo and Forbes, John and Francis, Prudence and Foo, Serene and Boyle, Frances and Redfern, Andrew and van der Westhuizen, Andre and Lewis, Craig and Sharma, Ravi and Sharma, Sharad and Beale, Philip and Byard, Ian and Begbie, Stephen and Sardelic, Frank and Abdi, Ehtesham and Clark, David and Chindewere, Aaron and Della-Fiorentina, Stephen and Asghari, Ray and Islam, Mohammed and Na Teo, Lee and White, Shane and Gilbert, Linda and Gardner, Katherine and Uhlmann, Catarina and Rauch, Daniel and Mannhart, Meinrad and Buser, Katharina and Dedes, Konstantin and Mueller, Andreas and Rageth, Christoph and Von Orelli, Stephanie and Senn, Hans Joerg and Pagani, Olivia and Pedrazzini, Augusto and Rochlitz, Christoph and Bodmer, Alexandre and Anchisi, Sandro and Zaman, Khalil and von Moos, Roger and Betticher, Daniel and ... and SOLE Investigators and Institute of Clinical Sciences, Department of Oncology and Sahlgrenska akademin and Göteborgs universitet and Gothenburg University and Sahlgrenska Academy and Institutionen för kliniska vetenskaper, Avdelningen för onkologi
The Lancet Oncology, ISSN 1470-2045, 01/2018, Volume 19, Issue 1, pp. 127 - 138
Journal Article
Nature Chemical Biology, ISSN 1552-4450, 2008, Volume 4, Issue 2, pp. 126 - 131
Journal Article
Proceedings of the National Academy of Sciences, ISSN 0027-8424, 04/2013, Volume 110, Issue 18, pp. 7440 - 7445
Extracellular signal-regulated kinases 1 and 2 (ERK1/2) are central mediators of cardiac hypertrophy and are discussed as potential therapeutic targets.... 
MAPK | GPCRs | Apoptosis | SIGNALING PATHWAYS | PRESSURE-OVERLOAD | ACTIVATED PROTEIN-KINASE | SUDDEN-DEATH | MULTIDISCIPLINARY SCIENCES | HEART-FAILURE | apoptosis | MYOCYTE APOPTOSIS | IN-VIVO | GENE-EXPRESSION | THERAPEUTIC TARGETS | STRESS | Biological Sciences
Journal Article