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1. Lu Qingxian gong nian pu
by Lu, Chenzheng and Li, Xuan and Wu, Guangyou, fl. 1868?
1868, 5,6,2,34 double leaves
Lu, Longqi, 1630-1693
Book
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2. Lu Qingxian gong song yang jiang yi
by Lu, Longqi, 1630-1693 and Hou, Quan, ed
1887
Si shu
Book
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3. Full Text Homeostatic Regulation of the Immune System by Receptor Tyrosine Kinases of the Tyro 3 Family
by Qingxian Lu and Greg Lemke
Science, ISSN 0036-8075, 7/2001, Volume 293, Issue 5528, pp. 306 - 311
Receptor tyrosine kinases and their ligands mediate cell-cell communication and interaction in many organ systems, but have not been known to act in this... 
T lymphocytes | Spleen | Receptors | Pain | Messenger RNA | B lymphocytes | Lymphocytes | Antibodies | Antigen presenting cells | Reports | Lymph nodes | ANTIBODIES | EXPRESSION ANALYSIS | LIGANDS | C-MER | AXL | CLONING | MULTIDISCIPLINARY SCIENCES | PROTEIN-S | SELECTION | GAS6 | Oncogene Proteins - genetics | Protein Biosynthesis | Immune System - physiology | Spleen - immunology | Dendritic Cells - immunology | Receptor Protein-Tyrosine Kinases - physiology | Homeostasis | Male | Lymphoid Tissue - growth & development | RNA, Messenger - metabolism | Oncogene Proteins - physiology | Lymphoid Tissue - immunology | Autoimmune Diseases - genetics | Lymphocytes - immunology | c-Mer Tyrosine Kinase | Female | Cell Differentiation | Proto-Oncogene Proteins | Protein S - biosynthesis | Autoimmune Diseases - pathology | Macrophages - immunology | Protein S - genetics | Lymphocyte Activation | Intercellular Signaling Peptides and Proteins | Autoimmune Diseases - immunology | Cell Transplantation | Lymphocytes - cytology | Spleen - cytology | Neural Cell Adhesion Molecules - genetics | Antigen-Presenting Cells - immunology | Proteins - genetics | Macrophages - metabolism | Animals | Receptor Protein-Tyrosine Kinases - genetics | Mice | Mutation | Neural Cell Adhesion Molecules - physiology | Research | B cells | T cells | Immune system | Proteins | Genetics | Rodents | Cells
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4. Full Text TAM receptors support neural stem cell survival, proliferation and neuronal differentiation
by Ji, Rui and Meng, Lingbin and Jiang, Xin and Naresh Kumar, Cvm and Ding, Jixiang and Li, Qiutang and Lu, Qingxian
PLoS ONE, ISSN 1932-6203, 12/2014, Volume 9, Issue 12, p. e115140
Tyro3, Axl and Mertk (TAM) receptor tyrosine kinases play multiple functional roles by either providing intrinsic trophic support for cell growth or regulating... 
NERVOUS-SYSTEM | AXL | MULTIDISCIPLINARY SCIENCES | APOPTOTIC CELLS | ADULT HIPPOCAMPAL NEUROGENESIS | TYROSINE KINASES | TRK RECEPTORS | NEUROTROPHINS | MER | NGF | TYRO-3 FAMILY | Cell Proliferation | Receptor Protein-Tyrosine Kinases - biosynthesis | Apoptosis - genetics | Recombinant Proteins | Proto-Oncogene Proteins - biosynthesis | Neurogenesis - genetics | Receptor, trkA - biosynthesis | c-Mer Tyrosine Kinase | Brain-Derived Neurotrophic Factor - biosynthesis | Proto-Oncogene Proteins - metabolism | Receptors, Nerve Growth Factor - biosynthesis | Cell Survival | Receptor, trkB - biosynthesis | Cells, Cultured | Proto-Oncogene Proteins - genetics | Hippocampus - cytology | Nerve Growth Factors - biosynthesis | Receptor Protein-Tyrosine Kinases - metabolism | Mice, Knockout | Hippocampus - metabolism | Animals | Receptor Protein-Tyrosine Kinases - genetics | Receptor, trkC - biosynthesis | Mice | Neural Stem Cells - metabolism | Brain | Nerve growth factor | Tubulins | Cell differentiation | Neurons | Stem cells | Cell proliferation | Neurosciences | Neurobiology | Central nervous system | Nervous system | Kinases | Neurogenesis | Neuronal-glial interactions | Axl protein | Proteins | Signal transduction | Receptors | Cell activation | Cell growth | Tubulin | Rodents | TrkA protein | Protein-tyrosine kinase receptors | TrkA receptors | Neurotrophins | Growth factors | Tyrosine | Cell survival | Immune response | Axonogenesis | Inflammation | Gene expression | Survival | Medicine | Brain-derived neurotrophic factor | Neural stem cells | TrkB receptors | Differentiation | Hippocampus | Apoptosis
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5. Full Text Systemic Autoimmunity in TAM Triple Knockout Mice Causes Inflammatory Brain Damage and Cell Death
by Li, Qiutang and Lu, Qingjun and Lu, Huayi and Tian, Shifu and Lu, Qingxian
PLoS ONE, ISSN 1932-6203, 06/2013, Volume 8, Issue 6, p. e64812
The Tyro3, Axl and Mertk (TAM) triply knockout (TKO) mice exhibit systemic autoimmune diseases, with characteristics of increased proinflammatory cytokine... 
INTERCELLULAR-ADHESION MOLECULE-1 | RECEPTOR TYROSINE KINASES | MRL-LPR MICE | MULTIPLE-SCLEROSIS | TYRO-3 FAMILY RECEPTORS | MULTIDISCIPLINARY SCIENCES | LUPUS-PRONE MICE | NECROSIS-FACTOR-ALPHA | CENTRAL-NERVOUS-SYSTEM | BLOOD-BRAIN | HUMAN ENDOTHELIAL-CELLS | Autoantibodies - blood | Tumor Necrosis Factor-alpha - blood | Microvessels - metabolism | Capillary Permeability - immunology | Mice, 129 Strain | Microvessels - immunology | Brain Damage, Chronic - genetics | Gene Knockdown Techniques | Autoimmune Diseases - genetics | Dentate Gyrus - immunology | Dentate Gyrus - blood supply | c-Mer Tyrosine Kinase | Inflammation Mediators - metabolism | Neurons - physiology | Female | Inclusion Bodies - metabolism | Autoimmune Diseases - pathology | Brain Damage, Chronic - pathology | Ubiquitinated Proteins - metabolism | Cytokines - metabolism | Endothelial Cells - metabolism | Mice, Inbred C57BL | Autoimmune Diseases - immunology | Cells, Cultured | Proto-Oncogene Proteins - genetics | Blood-Brain Barrier - metabolism | Mice, Knockout | CA3 Region, Hippocampal - blood supply | Endothelial Cells - immunology | Animals | CA3 Region, Hippocampal - immunology | Receptor Protein-Tyrosine Kinases - genetics | Lipopolysaccharides - pharmacology | Brain Damage, Chronic - immunology | T-Lymphocytes - immunology | Mice | CA3 Region, Hippocampal - pathology | Dentate Gyrus - pathology | Apoptosis | Ubiquitin | Dextran | Autoimmunity | Brain | Autoantibodies | Cytokines | Neurons | Cell death | Biochemistry | Permeability | T cells | Neurosciences | Multiple sclerosis | Disease | Laboratories | Cognitive ability | Fluorescence | Smooth muscle | Nervous system | Lymphocytes T | Kinases | Blood | Axl protein | Cell adhesion & migration | Fibers | Proteins | Blood-brain barrier | Neurodegeneration | Lymphocytes | Rodents | Animal tissues | Protein-tyrosine kinase receptors | Tumor necrosis factor-TNF | Lupus | Antigens | Immunoglobulins | Mortality | Blood vessels | Inflammation | Tumor necrosis factor-α | Ligands | Brain damage | Infiltration | Autoimmune diseases | Mossy fibers | Brain injury | Hippocampus | Animal cognition
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6. Secreted frizzled-related protein 5 suppresses aggressive phenotype and reverses docetaxel resistance in prostate cancer
by Xu, Q and Lu, Z and Wang, XG and Zhu, QX and Wu, HR
JOURNAL OF INVESTIGATIVE MEDICINE, ISSN 1081-5589, 08/2019, Volume 67, Issue 6, pp. 1009 - 1017
Secreted frizzled-related protein 5 (SFRP5) has been reported to be downregulated in prostate cancer. However, its biological role in this malignancy has not... 
TARGET | MEDICINE, RESEARCH & EXPERIMENTAL | ANTAGONIST | cell proliferation | SURVIVIN | PROLIFERATION | MEDICINE, GENERAL & INTERNAL | SFRP5 | GENES | cancer | CELL-CYCLE | EXPRESSION | PROGRESSION | BLOOD
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7. Full Text 14-3-3σ controls corneal epithelial cell proliferation and differentiation through the Notch signaling pathway
by Xin, Ying and Lu, Qingxian and Li, Qiutang
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 2010, Volume 392, Issue 4, pp. 593 - 598
14-3-3σ (also called stratifin) is specifically expressed in the stratified squamous epithelium and its function was recently shown to be linked to epidermal... 
Corneal epithelial | Proliferation | 14-3-3σ | Differentiation | Notch | differentiation | proliferation | corneal epithelial
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8. Full Text Mertk deficiency affects macrophage directional migration via disruption of cytoskeletal organization
by Tang, Yong and Wu, Shen and Liu, Qian and Xie, Jiayi and Zhang, Jingxue and Han, Dong and Lu, Qingxian and Lu, Qingjun
PLoS ONE, ISSN 1932-6203, 01/2015, Volume 10, Issue 1, p. e0117787
Mertk belongs to the Tyro3, Axl and Mertk (TAM) family of receptor tyrosine kinases, and plays a pivotal role in regulation of cytoskeletal rearrangement... 
Receptor Protein-Tyrosine Kinases - deficiency | Signal Transduction | Proto-Oncogene Proteins - genetics | Proto-Oncogene Proteins - deficiency | Gene Knockout Techniques | Cell Adhesion | Macrophages, Peritoneal - cytology | Microtubules - metabolism | Animals | Receptor Protein-Tyrosine Kinases - genetics | Cell Shape | c-Mer Tyrosine Kinase | Cytoskeleton - metabolism | Mice | Macrophages, Peritoneal - metabolism | Phagocytosis | Cell Movement | PHAGOCYTOSIS | ACTIVATION | LEADING-EDGE | MULTIDISCIPLINARY SCIENCES | APOPTOTIC CELLS | ALPHA-V-BETA-5 INTEGRIN | FOCAL ADHESION KINASE | TYRO-3 FAMILY | RECEPTOR TYROSINE KINASE | RETINAL-PIGMENT EPITHELIUM | MOTILITY | Macrophages | Phosphotransferases | Myosin | Cell culture | Phosphorylation | Motility | Laboratories | Immunocytochemistry | Kinases | Western blotting | Axl protein | Cell adhesion & migration | Proteins | Phagocytes | Signal transduction | Filaments | Actin | Extracellular matrix | Protein-tyrosine kinase receptors | Localization | Cell body | Tyrosine | Scanning electron microscopy | Polymerization | Electron microscopy | Photography | Spreading | Microtubules | Morphology | Regulatory mechanisms (biology) | Cytoskeleton | Photoreceptors | Cell size | Focal adhesion kinase | Cell migration | Peritoneum
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9. Full Text IKK2 inhibition attenuates laser-induced choroidal neovascularization
by Lu, Huayi and Lu, Qingxian and Gaddipati, Subhash and Kasetti, Ramesh Babu and Wang, Wei and Pasparakis, Manolis and Kaplan, Henry J and Li, Qiutang
PLoS ONE, ISSN 1932-6203, 01/2014, Volume 9, Issue 1, p. e87530
Choroidal neovascularization (CNV) is aberrant angiogenesis associated with exudative age-related macular degeneration (AMD), a leading cause of blindness in... 
CANCER-CELLS | ALPHA-INDUCED APOPTOSIS | PIGMENT EPITHELIAL-CELLS | INCREASED EXPRESSION | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | MACULAR DEGENERATION | NECROSIS-FACTOR-ALPHA | NF-KAPPA-B | TRANSCRIPTIONAL REGULATION | ENDOTHELIAL GROWTH-FACTOR | Animals | Choroidal Neovascularization - metabolism | Choroidal Neovascularization - pathology | Signal Transduction | I-kappa B Kinase - antagonists & inhibitors | Lasers | NF-kappa B - physiology | Choroidal Neovascularization - therapy | NF-kappa B - metabolism | Mice | Mice, Knockout | Macular degeneration | Platelet-derived growth factor | Cytokines | Blindness | Inflammation | Intermediate filament proteins | Vascular endothelial growth factor | Elderly people | Oxidative stress | Nestin | Transcription | Physicians | Gene regulation | Genes | IKK2 protein | Retina | Inflammatory response | Kinases | Risk factors | Vascularization | Defects | Angiogenesis | Clonal deletion | Interleukin 1 | Deletion | Inhibition | Growth factors | Age | NF-κB protein | Breeding | Flox | Gelatinase A | Medicine | Signaling | Inhibitors | Exudation | Aberration | Elderly | Chemokines | Monocyte chemoattractant protein 1 | Geriatrics | Heat shock | Cancer | Apoptosis
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10. Full Text Sperm from β1,4-galactosyltransferase-null mice are refractory to ZP3-induced acrosome reactions and penetrate the zona pellucida poorly
by Lu, Qingxian and Shur, Barry D
Development, ISSN 0950-1991, 1997, Volume 124, Issue 20, pp. 4121 - 4131
Galactosyltransferase | Fertilization | Mouse | ZP3 | Sperm | Zona pellucida
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11. Full Text Repression of Zeb1 and Hypoxia Cause Sequential Mesenchymal‐to‐Epithelial Transition and Induction of Aid, Oct4, and Dnmt1, Leading to Immortalization and Multipotential Reprogramming of Fibroblasts in Spheres
by Liu, Yongqing and Mukhopadhyay, Partha and Pisano, M. Michele and Lu, Xiaoqin and Huang, Li and Lu, Qingxian and Dean, Douglas C
STEM CELLS, ISSN 1066-5099, 07/2013, Volume 31, Issue 7, pp. 1350 - 1362
In this study, we demonstrate that sphere formation triggers immortalization and stable reprogramming of mouse fibroblasts. Cell contact signaling in spheres... 
Hypoxia | Adult stem cells | Reprogramming | Differentiation | PROLIFERATION | CANCER STEM-CELLS | DNA DEMETHYLATION | PLURIPOTENT | CELL & TISSUE ENGINEERING | CELL BIOLOGY | OXYGEN | GENE | ONCOLOGY | PATHWAY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | COMPONENT | GROWTH-FACTOR | HEMATOLOGY | EXPRESSION | Cell Hypoxia - physiology | Epithelial-Mesenchymal Transition - physiology | Homeodomain Proteins - metabolism | Multipotent Stem Cells - metabolism | Octamer Transcription Factor-3 - biosynthesis | Transcriptome | Stem Cells - cytology | Epithelial-Mesenchymal Transition - genetics | Stem Cells - metabolism | Gene Knockdown Techniques | DNA (Cytosine-5-)-Methyltransferases - metabolism | Octamer Transcription Factor-3 - genetics | Cell Differentiation - genetics | DNA Methylation | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Kruppel-Like Transcription Factors - metabolism | Kruppel-Like Transcription Factors - deficiency | Multipotent Stem Cells - physiology | Cell Differentiation - physiology | Fibroblasts - metabolism | Promoter Regions, Genetic | DNA (Cytosine-5-)-Methyltransferase 1 | Cytidine Deaminase - genetics | Cells, Cultured | Homeodomain Proteins - genetics | Animals | Multipotent Stem Cells - cytology | Cytidine Deaminase - metabolism | Octamer Transcription Factor-3 - metabolism | Stem Cells - physiology | Fibroblasts - cytology | Mice | Cell Hypoxia - genetics | Kruppel-Like Transcription Factors - genetics | Zinc Finger E-box-Binding Homeobox 1 | Methyltransferases | Gene expression | Analysis | Stem cells | Proteins | Spheres | Kinases
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12. Full Text Identification of 14-3-3σ Mutation Causing Cutaneous Abnormality in Repeated-Epilation Mutant Mouse
by Qiutang Li and Qingxian Lu and Gabriela Estepa and Inder M. Verma
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2005, Volume 102, Issue 44, pp. 15977 - 15982
Repeated-epilation (Er) mutation in the mouse is inherited as an autosomal and semidominant mutation. Major defects in heterozygous adults and homozygous... 
Proteins | Biological Sciences | Epidermal cells | Genes | Genomics | Keratinocytes | Epidermis | Skin | Mice | Genetic mutation | Embryos | Genetics | Epidermal development | IKK1 | Stratifin | stratifin | epidermal development | genetics
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13. Full Text Study of corneal epithelial progenitor origin and the Yap1 requirement using keratin 12 lineage tracing transgenic mice
by Kasetti, Ramesh Babu and Gaddipati, Subhash and Tian, Shifu and Xue, Lei and Kao, Winston W.-Y and Lu, Qingxian and Li, Qiutang
Scientific Reports, ISSN 2045-2322, 10/2016, Volume 6, Issue 1, p. 35202
Key issues in corneal epithelium biology are the mechanism for corneal epithelium stem cells to maintain the corneal epithelial homeostasis and wound healing... 
OCULAR SURFACE | MIGRATION | STEM-CELL DEFICIENCY | HIPPO SIGNALING PATHWAY | ORGAN SIZE CONTROL | MULTIDISCIPLINARY SCIENCES | GROWTH | DIFFERENTIATION | TRANSCRIPTION FACTOR | EXPRESSION | CANCER | Epithelium, Corneal - cytology | Keratin-12 - metabolism | Mice, Transgenic | Green Fluorescent Proteins - genetics | Stem Cells - cytology | Wound Healing | Phosphoproteins - metabolism | Stem Cells - metabolism | Cell Lineage | Animals | Epithelium, Corneal - metabolism | Mice | Adaptor Proteins, Signal Transducing - metabolism | Yes-associated protein | Keratin | Cornea | Wound healing | Transcription | Epithelial cells | Stem cells | Transgenic mice | Homeostasis | Stem cell transplantation | Epithelium
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14. Full Text IKK2 inhibition using TPCA-1-Loaded PLGA microparticles attenuates laser-induced choroidal neovascularization and macrophage recruitment
by Gaddipati, Subhash and Lu, Qingxian and Kasetti, Ramesh Babu and Miller, M. Clarke and Lu, Qingjun and Trent, John O and Kaplan, Henry J and Li, Qiutang
PLoS ONE, ISSN 1932-6203, 03/2015, Volume 10, Issue 3, pp. e0121185 - e0121185
The inhibition of NF-kappa B by genetic deletion or pharmacological inhibition of IKK2 significantly reduces laser-induced choroid neovascularization (CNV). To... 
CELLS | MICROSPHERES | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | MACULAR DEGENERATION | RETINOPATHY | MICE | MODEL | NF-KAPPA-B | RANIBIZUMAB | BETA | Lactic Acid - chemistry | Macrophages - pathology | Mice, Inbred C57BL | Drug Carriers - administration & dosage | Male | Thiophenes - administration & dosage | Macrophages - cytology | Drug Carriers - chemistry | Protein Kinase Inhibitors - administration & dosage | Protein Kinase Inhibitors - chemistry | Animals | Choroidal Neovascularization - drug therapy | Choroidal Neovascularization - pathology | I-kappa B Kinase - antagonists & inhibitors | Polyglycolic Acid - chemistry | Amides - chemistry | Female | Macrophages - drug effects | Mice | Thiophenes - chemistry | Amides - administration & dosage | Lactic Acid - administration & dosage | Polyglycolic Acid - administration & dosage | Disease Models, Animal | Macular degeneration | Employee recruitment | Neovascularization | Macrophages | Fluorescein | Diabetic retinopathy | Biodegradability | IKK2 protein | Retina | Microparticles | Vascularization | Recruitment | Angiogenesis | Lasers | Biocompatibility | Eye (anatomy) | Inhibition | Polymers | Vascular endothelial growth factor | Medical research | NF-κB protein | Solvents | Stability | Molecular chains | Injury prevention | Inhibitors | Infiltration | Monocyte chemoattractant protein 1 | Drug delivery systems | Toxicity | Medical services | Staining | Kinases | Experiments | Optokinetic response | Polylactide-co-glycolide | Nanoparticles | Clonal deletion | Eye injuries | Rodents | Deletion | Attenuation | Age | Biodegradation | Departments | Inflammation | Histology | Pharmacology | Breast cancer | Evaporation | Medicine | Eye diseases | In vivo methods and tests | Laboratory animals | Index Medicus
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15. Full Text TAM receptor deficiency affects adult hippocampal neurogenesis
by Ji, Rui and Ji, Rui and Meng, Lingbin and Meng, Lingbin and Li, Qiutang and Li, Qiutang and Lu, Qingxian and Lu, Qingxian
Metabolic Brain Disease, ISSN 0885-7490, 6/2015, Volume 30, Issue 3, pp. 633 - 644
The Tyro3, Axl and Mertk (TAM) subfamily of receptor protein tyrosine kinases functions in cell growth, differentiation, survival, and most recently found, in... 
Biochemistry, general | Neurology | Neurosciences | Biomedicine | TAM receptors | Neuronal stem cells | Metabolic Diseases | Oncology | Neuroinflammation | Neurogenesis | Neuronal stem cell differentiation | Microglia | INTERCELLULAR-ADHESION MOLECULE-1 | TYRO-3 FAMILY RECEPTORS | NECROSIS-FACTOR-ALPHA | PROTEIN-TYROSINE KINASE | NEUROSCIENCES | VASCULAR SMOOTH-MUSCLE | PHOSPHATIDYLINOSITOL 3-KINASE-DEPENDENT PATHWAY | NEURAL STEM-CELLS | BRAIN-BARRIER PERMEABILITY | ENDOCRINOLOGY & METABOLISM | CENTRAL-NERVOUS-SYSTEM | ARREST-SPECIFIC GENE | Hippocampus - metabolism | Receptor Protein-Tyrosine Kinases - deficiency | Animals | Age Factors | Neurogenesis - physiology | Humans | Signal Transduction - physiology | Hippocampus - cytology | Ophthalmology | Cell differentiation | Phosphotransferases | Stem cells | Neurophysiology
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