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Antioxidants & Redox Signaling, ISSN 1523-0864, 10/2011, Volume 15, Issue 7, pp. 1957 - 1997
Reactive oxygen species, such as superoxide and hydrogen peroxide, are generated in all cells by mitochondrial and enzymatic sources. Left unchecked, these... 
Comprehensive Invited Review | ISCHEMIA-REPERFUSION INJURY | GPX1 PRO198LEU POLYMORPHISM | ENDOTHELIAL PROGENITOR CELLS | SELENOCYSTEINE INSERTION-SEQUENCE | BREAST-CANCER RISK | MANGANESE SUPEROXIDE-DISMUTASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | ENDOCRINOLOGY & METABOLISM | MESSENGER-RNA STABILITY | NF-KAPPA-B | E-DEFICIENT MICE | CORONARY-ARTERY-DISEASE | Selenocysteine - metabolism | Neoplasms - etiology | Reactive Oxygen Species - metabolism | Oxidative Stress | Diabetes Mellitus - genetics | Glutathione Peroxidase - antagonists & inhibitors | Humans | Cardiovascular Diseases - therapy | Molecular Sequence Data | Cardiovascular Diseases - genetics | Neoplasms - therapy | Glutathione Peroxidase - chemistry | Neoplasms - genetics | Cell Death | Diabetes Mellitus - etiology | Amino Acid Sequence | Cardiovascular Diseases - etiology | Glutathione Peroxidase - metabolism | Gene Expression | Genetic Predisposition to Disease | Oxidation-Reduction | Risk Factors | Selenocysteine - chemistry | Enzyme Inhibitors - pharmacology | Gene Expression Regulation | Enzyme Inhibitors - therapeutic use | Glutathione Peroxidase - genetics | Polymorphism, Genetic | Animals | Diabetes Mellitus - therapy | Protein Conformation | Antioxidants | Active oxygen | Physiological aspects | Cellular signal transduction | Peroxidase | Research | Health aspects
Journal Article
JACC (Journal of the American College of Cardiology), ISSN 0735-1097, 2010, Volume 55, Issue 19, pp. 2096 - 2106
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 5, p. e10693
Background: Variability of gene expression in human may link gene sequence variability and phenotypes; however, non-genetic variations, alone or in combination... 
VARIANTS | MACROPHAGES | MULTIDISCIPLINARY SCIENCES | GENES | RISK | LOCI | SMOKING | EXPRESSION PROFILES | HAPLOTYPE | CORONARY-ARTERY-DISEASE | GENOME-WIDE ASSOCIATION | Smoking - adverse effects | Monocytes - cytology | Atherosclerosis - genetics | Humans | Middle Aged | Male | Monocytes - metabolism | Gene Expression Profiling | Cell Movement - genetics | Genetic Variation | Base Sequence | DNA Probes - metabolism | Adult | Female | Genetic Predisposition to Disease | Genome-Wide Association Study | Quantitative Trait, Heritable | Risk Factors | Gene Expression Regulation | Smoking - genetics | Chromosomes, Human, Pair 21 - genetics | Genome, Human - genetics | Phenotype | Polymorphism, Single Nucleotide - genetics | Aged | Immunity - genetics | Quantitative Trait Loci - genetics | Medical research | Genomics | Genes | Disease susceptibility | Gene expression | Mediation | Risk factors | Quantitative genetics | Analysis | Atherosclerosis | Physiological aspects | Medicine, Experimental | Genetic aspects | Cardiovascular diseases | Smoking | Attention deficit disorder | Pathogenesis | Variability | Colorectal cancer | Lipids | Cardiovascular disease | Genomes | Single-nucleotide polymorphism | Immunity | Medical schools | Body mass index | Risk assessment | Genetic analysis | Genetics | Blood pressure | Cytokines | Phenotypic variations | Health risks | Genetic diversity | Risk analysis | Cigarette smoking | Loci | Quantitative trait loci | Studies | Monocytes | Body mass | Arteriosclerosis | Body size | Chemokines
Journal Article
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 10/2017, Volume 70, Issue 18, pp. B259 - B260
Methods From 09/2008-05/2016, a total of 500 patients (age 75.6±8.3 years, male gender 60.4%, logistic EuroSCORE 25.3±17.0%, functional mitral regurgitation... 
Heart valve diseases | Care and treatment | Patient outcomes | Heart failure | Implantation | Mitral valve | Surgery
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 05/2009, Volume 284, Issue 18, pp. 11913 - 11921
Glutathione peroxidase-1 (GPx-1) is a selenocysteine-containing enzyme that plays a major role in the reductive detoxification of peroxides in cells. In... 
OVEREXPRESSION | OXIDATIVE STRESS | KINASE ACTIVITY | REACTIVE OXYGEN | ENDOTHELIAL DYSFUNCTION | BIOCHEMISTRY & MOLECULAR BIOLOGY | HYDROGEN-PEROXIDE | TUMOR-SUPPRESSOR PTEN | NF-KAPPA-B | SUPEROXIDE-DISMUTASE | KNOCKOUT MOUSE | Disulfides - metabolism | Mitochondria, Liver - genetics | Selenocysteine - metabolism | Oxidants - pharmacology | Adenoviridae | Uncoupling Agents - pharmacology | Selenoproteins - genetics | Mitochondria, Liver - metabolism | Humans | ErbB Receptors - genetics | Oxidants - metabolism | Adenosine Triphosphate - genetics | Adenosine Triphosphate - biosynthesis | Proto-Oncogene Proteins c-akt - genetics | Gene Knockdown Techniques | Oxidation-Reduction - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Transduction, Genetic - methods | Cell Line | Glutathione Peroxidase - metabolism | ErbB Receptors - metabolism | Catalase - genetics | Epidermal Growth Factor - genetics | Hydrogen Peroxide - pharmacology | Epidermal Growth Factor - metabolism | Enzyme Activation - drug effects | Glutathione Peroxidase - genetics | Selenoproteins - metabolism | Hydrogen Peroxide - metabolism | Catalase - metabolism | Selenocysteine - genetics | Signal Transduction - drug effects | Signal Transduction - physiology | Cell Proliferation - drug effects | Enzyme Activation - physiology | Index Medicus | Mechanisms of Signal Transduction
Journal Article