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41. Full Text Natriuretic Peptide Receptor B modulates the proliferation of the cardiac cells expressing the Stem Cell Antigen-1
by Rignault-Clerc, Stéphanie and Bielmann, Christelle and Liaudet, Lucas and Waeber, Bernard and Feihl, François and Rosenblatt-Velin, Nathalie
Scientific reports, ISSN 2045-2322, 02/2017, Volume 7, Issue 1, pp. 41936 - 41936
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Cell Proliferation | Signal Transduction | Receptors, Atrial Natriuretic Factor - metabolism | Mice, Inbred C57BL | Cells, Cultured | Homeobox Protein Nkx-2.5 - metabolism | Stem Cells - metabolism | Cyclic GMP-Dependent Protein Kinases - metabolism | Myocardium - cytology | Animals | Ataxin-1 - metabolism | Natriuretic Peptide, Brain - drug effects | Stem Cells - drug effects | Stem Cells - physiology | Mice | Ataxin-1 - genetics | Cell proliferation | Brain natriuretic peptide | Neonates | Peptides | Brain stem | Protein kinase G | Kinases | Regeneration | Signal transduction | Cell growth | Cell activation | Lymphocytes B | Protein kinase | Rodents | Stem cells | Fibroblasts | Heart diseases | Nkx2.5 protein | Index Medicus
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42. Full Text Chemo-manipulation of tumor blood vessels by a metal-based anticancer complex enhances antitumor therapy
by Riedel, Tina and Cavin, Sabrina and Van Den Bergh, Hubert and Krueger, Thorsten and Liaudet, Lucas and Ris, Hans-Beat and Dyson, Paul J and Perentes, Jean Y
Scientific reports, ISSN 2045-2322, 12/2018, Volume 8, Issue 1, pp. 10263 - 14
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Lung Neoplasms - drug therapy | Mesothelioma - pathology | Humans | Lung Neoplasms - pathology | Cisplatin - pharmacology | Mesothelioma - blood supply | Poly (ADP-Ribose) Polymerase-1 - antagonists & inhibitors | Mesothelioma - drug therapy | Drug Synergism | Xenograft Model Antitumor Assays | Animals | Mice, Nude | Female | Antineoplastic Agents - pharmacology | Mice | Mesothelioma, Malignant | Tumor Cells, Cultured | Drug Therapy, Combination | Endothelial Cells - pathology | Organometallic Compounds - pharmacology | Endothelial Cells - drug effects | Lung Neoplasms - blood supply | Blood vessels | Poly(ADP-ribose) polymerase | Cytotoxicity | Mesothelioma | Cisplatin | Endothelial cells | Poly(ADP-ribose) Polymerase 1 | Perfusion | Ribose | Xenografts | Pericytes | Oxygenation | Tumors | Cancer | Index Medicus
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43. Full Text Rapid Reversal of the Diabetic Endothelial Dysfunction by Pharmacological Inhibition of Poly(ADP-Ribose) Polymerase
by Soriano, Francisco Garcia and Pacher, Pál and Mabley, Jon and Liaudet, Lucas and Szabó, Csaba
Circulation research, ISSN 0009-7330, 10/2001, Volume 89, Issue 8, pp. 684 - 691
Diabetes | Nitric oxide | Apoptosis | Endothelium | Necrosis | Cardiac & Cardiovascular Systems | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Cardiovascular system | Biological and medical sciences | Miscellaneous | Medical sciences | Pharmacology. Drug treatments | Glycated Hemoglobin A - analysis | Streptozocin | Glycated Hemoglobin A - drug effects | Vasomotor System - drug effects | Diabetic Angiopathies - etiology | Endothelium, Vascular - drug effects | Male | Enzyme Inhibitors - administration & dosage | Diabetic Angiopathies - physiopathology | Vasomotor System - physiopathology | Adenosine Triphosphate - metabolism | Animal Diseases | Diabetes Mellitus, Experimental - chemically induced | Diabetes Mellitus, Experimental - complications | Aorta, Thoracic - drug effects | NADP - metabolism | Diabetes Mellitus, Experimental - physiopathology | Vasoconstrictor Agents - pharmacology | Vasodilator Agents - pharmacology | Administration, Oral | Endothelium, Vascular - physiopathology | Phenanthrenes - administration & dosage | Diabetic Angiopathies - drug therapy | Aorta, Thoracic - physiopathology | Poly(ADP-ribose) Polymerase Inhibitors | Disease Progression | Blood Glucose - drug effects | Animals | Mice | Mice, Inbred BALB C | In Vitro Techniques | Index Medicus
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44. Full Text Blocking mineralocorticoid receptor with spironolactone may have a wide range of therapeutic actions in severe COVID-19 disease
by Liaudet, Lucas and Szabo, Csaba
Critical care (London, England), ISSN 1466-609X, 06/2020, Volume 24, Issue 1, pp. 318 - 318
Life Sciences & Biomedicine | Critical Care Medicine | General & Internal Medicine | Science & Technology | COVID-19 | Severity of Illness Index | Pandemics | Coronavirus Infections - drug therapy | Humans | Pneumonia, Viral - drug therapy | Spironolactone - therapeutic use | Mineralocorticoid Receptor Antagonists - therapeutic use | Severe acute respiratory syndrome coronavirus 2 | Coronaviruses | Index Medicus
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45. Role of innate immunity in cardiac inflammation after myocardial infarction
by Liaudet, Lucas and Rosenblatt-Velin, Nathalie
Frontiers in bioscience (Scholar edition), ISSN 1945-0516, 01/2013, Volume 5, Issue 1, pp. 86 - 104
Heart | Myocardial infarction | NOD-like receptor | Oxidative stress | Cytokine | Innate immunity | Inflammasome | Inflammation | Review | Remodeling | Chemokine | High-mobility group box-1 | Toll-like receptor | Nuclear factor kappaB | Animals | Immunity, Innate | Myocarditis - immunology | Humans | Myocardial Infarction - immunology
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46. Full Text The role of endogenous and exogenous RasGAP-derived fragment N in protecting cardiomyocytes from peroxynitrite-induced apoptosis
by Khalil, Hadi and Rosenblatt, Nathalie and Liaudet, Lucas and Widmann, Christian
Free radical biology & medicine, ISSN 0891-5849, 08/2012, Volume 53, Issue 4, pp. 926 - 935
Peroxynitrite | Free radicals | Cardiomyocytes | Caspases | RasGAP | Akt | Apoptosis | Biochemistry & Molecular Biology | Endocrinology & Metabolism | Life Sciences & Biomedicine | Science & Technology | Cell Line | Gene Expression | Oxidants - pharmacology | Peptide Fragments - metabolism | ras GTPase-Activating Proteins - metabolism | Apoptosis - drug effects | Cell Survival | Humans | Caspase 3 - metabolism | Mutagens - pharmacology | Rats | Cisplatin - pharmacology | Peptide Fragments - physiology | ras GTPase-Activating Proteins - physiology | ras GTPase-Activating Proteins - genetics | Animals | Myocytes, Cardiac - drug effects | Myocytes, Cardiac - physiology | Peroxynitrous Acid - pharmacology | Proteolysis | Myocytes, Cardiac - metabolism | Mice | Proto-Oncogene Proteins c-akt - metabolism | Peptide Fragments - genetics | Fluorescence | Universities and colleges | Heart cells | Index Medicus
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47. Full Text Murine myocardial infarction model using permanent ligation of left anterior descending coronary artery
by Lugrin, Jérôme and Parapanov, Roumen and Krueger, Thorsten and Liaudet, Lucas
Journal of Visualized Experiments, ISSN 1940-087X, 08/2019, Volume 2019, Issue 150
Medicine | Ligation | Infarct | Ischemia | Mouse | Permanent | Coronary artery | Myocardial | Issue 150 | LAD | Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Index Medicus
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48. Full Text Poly(ADP-ribose) polymerases as modulators of mitochondrial activity
by Bai, Peter and Nagy, Lilla and Fodor, Tamás and Liaudet, Lucas and Pacher, Pal
Trends in endocrinology and metabolism, ISSN 1043-2760, 2014, Volume 26, Issue 2, pp. 75 - 83
Endocrinology & Metabolism | NAD | nuclear respiratory factor | hypoxia-inducible factor | AMP activated kinase | Sirtuin | poly(ADP-ribose) polymerase | Hypoxia-inducible factor | Poly(ADP-ribose) polymerase | Nuclear respiratory factor | Life Sciences & Biomedicine | Science & Technology | Mitochondrial Diseases - genetics | Neoplasms - metabolism | Animals | Poly(ADP-ribose) Polymerases - genetics | Neoplasms - genetics | Humans | Mitochondrial Diseases - metabolism | Transcription, Genetic | Mitochondria - metabolism | Signal Transduction - genetics | Mitochondria - physiology | Poly(ADP-ribose) Polymerases - physiology | Sugars | Monosaccharides | Index Medicus
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49. Full Text Hypertension: A Disease of the Microcirculation?
by Feihl, François and Liaudet, Lucas and Waeber, Bernard and Levy, Bernard I
Hypertension (Dallas, Tex. 1979), ISSN 0194-911X, 12/2006, Volume 48, Issue 6, pp. 1012 - 1017
Fundamental and applied biological sciences. Psychology | Cardiology. Vascular system | Arterial hypertension. Arterial hypotension | Biological and medical sciences | Hemodynamics. Rheology | Medical sciences | Clinical manifestations. Epidemiology. Investigative techniques. Etiology | Vertebrates: cardiovascular system | Blood and lymphatic vessels | Antihypertensive Agents - pharmacology | Humans | Hypertension - drug therapy | Antihypertensive Agents - therapeutic use | Microcirculation - drug effects | Muscle, Smooth, Vascular - physiopathology | Hypertension - physiopathology | Vascular Resistance - physiology | Microcirculation - physiopathology | Animals | Microcirculation - pathology | Arterioles - physiopathology | Muscle Contraction - physiology | Index Medicus
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50. Full Text Cannabidiol attenuates high glucose-induced endothelial cell inflammatory response and barrier disruption
by Mohanraj Rajesh and Partha Mukhopadhyay and Sándor Bátkai and György Haskó and Lucas Liaudet and Viktor R. Drel and Irina G. Obrosova and Pál Pacher
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 07/2007, Volume 293, Issue 1, pp. 610 - 619
Oxidative stress | Cannabinoids | Peroxynitrite | Inflammation | Endocannabinoids | Atherosclerosis | Cardiac & Cardiovascular Systems | Physiology | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Dose-Response Relationship, Drug | Endothelial Cells - immunology | Inflammation Mediators - immunology | Monocytes - drug effects | Cannabidiol - administration & dosage | Humans | Cells, Cultured | Monocytes - immunology | Cell Adhesion - drug effects | Cytokines - immunology | Endothelial Cells - drug effects | Glucose - administration & dosage | Molecules | Diabetes | Organic chemicals | Rodents | Coronary vessels | Cells | Index Medicus | atherosclerosis | cannabinoids | endocannabinoids | peroxynitrite | inflammation | oxidative stress
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51. Activation of poly(ADP-ribose) polymerase contributes to development of doxorubicin-induced heart failure
by Pacher, P and Liaudet, L and Bai, P and Virag, L and Mabley, J.G and Haskó, G and Szabó, C
The Journal of pharmacology and experimental therapeutics, ISSN 0022-3565, 2002, Volume 300, Issue 3, pp. 862 - 867
Life Sciences & Biomedicine | Pharmacology & Pharmacy | Science & Technology | L-Lactate Dehydrogenase - metabolism | Acute Disease | Ventricular Function, Left - genetics | Heart Failure - physiopathology | Male | Metalloendopeptidases - metabolism | Antibiotics, Antineoplastic | Heart Failure - pathology | Enzyme Activation - drug effects | Doxorubicin | Mice, Knockout | Poly(ADP-ribose) Polymerases - metabolism | Animals | Poly(ADP-ribose) Polymerases - genetics | Survival Analysis | Creatine Kinase - metabolism | Hemodynamics - drug effects | Mice | Mice, Inbred BALB C | Heart Failure - chemically induced | Index Medicus
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52. Full Text Left ventricular pressure-volume relationship in a rat model of advanced aging-associated heart failure
by Pál Pacher and Jon G. Mabley and Lucas Liaudet and Oleg V. Evgenov and Anita Marton and György Haskó and Márk Kollai and Csaba Szabó
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 11/2004, Volume 287, Issue 5, pp. 2132 - 2137
Diastolic dysfunction | Systolic dysfunction | Cardiac & Cardiovascular Systems | Physiology | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Blood Pressure | Heart - physiopathology | Diastole | Ventricular Function, Left | Rats, Inbred F344 | Rats | Male | Cardiac Output, Low - physiopathology | Systole | Cardiac Output, Low - etiology | Stroke Volume | Ventricular Remodeling | Animals | Aging | Ventricular Pressure | Index Medicus | diastolic dysfunction | systolic dysfunction
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53. Full Text Is nitric oxide overproduction the target of choice for the management of septic shock?
by Feihl, François and Waeber, Bernard and Liaudet, Lucas
Pharmacology & therapeutics (Oxford), ISSN 0163-7258, 2001, Volume 91, Issue 3, pp. 179 - 213
Shock | Sepsis | Animal models | Free radicals | Endotoxin | Nitric oxide | Life Sciences & Biomedicine | Pharmacology & Pharmacy | Science & Technology | Up-Regulation | Nitric Oxide - chemistry | Nitric Oxide - antagonists & inhibitors | Humans | Enzyme Inhibitors - pharmacology | Nitric Oxide Synthase - antagonists & inhibitors | Rats | Free Radicals - adverse effects | Inflammation | Nitric Oxide - adverse effects | Shock, Septic - physiopathology | Animals | Shock, Septic - drug therapy | Mice | Nitric Oxide Synthase - metabolism | Apoptosis | Disease Models, Animal | Index Medicus
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