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1. Full Text Topological data analysis for discovery in preclinical spinal cord injury and traumatic brain injury
by Nielson, Jessica L and Paquette, Jesse and Liu, Aiwen W and Guandique, Cristian F and Tovar, C. Amy and Inoue, Tomoo and Irvine, Karen-Amanda and Gensel, John C and Kloke, Jennifer and Petrossian, Tanya C and Lum, Pek Y and Carlsson, Gunnar E and Manley, Geoffrey T and Young, Wise and Beattie, Michael S and Bresnahan, Jacqueline C and Ferguson, Adam R
Nature communications, ISSN 2041-1723, 10/2015, Volume 6, Issue 1, pp. 8581 - 8581
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Computational Biology - methods | Data Interpretation, Statistical | Animals | Spinal Cord Injuries | Brain Injuries | Rats | Disease Models, Animal | Index Medicus
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2. Full Text BAP1 cancer syndrome: malignant mesothelioma, uveal and cutaneous melanoma, and MBAITs
by Carbone, Michele and Ferris, Laura K and Baumann, Francine and Napolitano, Andrea and Lum, Christopher A and Flores, Erin G and Gaudino, Giovanni and Powers, Amy and Bryant-Greenwood, Peter and Krausz, Thomas and Hyjek, Elizabeth and Tate, Rachael and Friedberg, Joseph and Weigel, Tracey and Pass, Harvey I and Yang, Haining
Journal of translational medicine, ISSN 1479-5876, 08/2012, Volume 10, Issue 1, pp. 179 - 179
MBAITs | Mesothelioma | Cancer syndrome | BAP1 | Melanoma | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Tumor Suppressor Proteins - physiology | Humans | Ubiquitin Thiolesterase - physiology | Mesothelioma - physiopathology | Uveal Neoplasms - physiopathology | Skin Neoplasms - physiopathology | Cohort Studies | Melanoma - physiopathology | Oncology, Experimental | Development and progression | Family | Research | Comparative analysis | Cancer | Proteins | Studies | Pathology | Automation | Genetic counseling | Surgery | Writing | Mutation | Manuscripts | Deoxyribonucleic acid--DNA | Index Medicus
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3. Full Text Confirmation of ProMisE: A simple, genomics‐based clinical classifier for endometrial cancer
by Talhouk, Aline and McConechy, Melissa K and Leung, Samuel and Yang, Winnie and Lum, Amy and Senz, Janine and Boyd, Niki and Pike, Judith and Anglesio, Michael and Kwon, Janice S and Karnezis, Anthony N and Huntsman, David G and Gilks, C. Blake and McAlpine, Jessica N
Cancer, ISSN 0008-543X, 03/2017, Volume 123, Issue 5, pp. 802 - 813
prognostic | risk‐stratification system | mismatch repair | endometrial carcinoma | histotype | polymerase‐ɛ (POLE) | The Cancer Genome Atlas (TCGA) | p53 | polymerase-ɛ (POLE) | risk-stratification system | Life Sciences & Biomedicine | Oncology | Science & Technology | Microsatellite Instability | Endometrial Neoplasms - classification | Humans | Risk Factors | Mutation, Missense | DNA Mismatch Repair - genetics | Tumor Suppressor Protein p53 - genetics | Disease-Free Survival | Poly-ADP-Ribose Binding Proteins | Endometrial Neoplasms - genetics | Adult | Endometrial Neoplasms - pathology | Female | Aged | Biomarkers, Tumor - genetics | Mutation | Neoplasm Staging | DNA Polymerase II - genetics | Pathology, Molecular | Clinical trials | Usage | Diagnosis | Endometrial cancer | Risk factors | Colorectal cancer | Immunohistochemistry | Classifiers | Prognosis | Carcinoma | p53 Protein | Genomics | Genomes | Subgroups | Proteins | Exonuclease | Missense mutation | Mismatch repair | Classification | Endometrium | Medical research | Risk groups | Feasibility studies | Health risks | Risk analysis | Survival | Polymerase | Medical prognosis | Risk management | Tumors | Cancer | Index Medicus | Abridged Index Medicus
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4. Full Text Cancer-Associated Mutations in Endometriosis without Cancer
by Anglesio, Michael S and Papadopoulos, Nickolas and Ayhan, Ayse and Nazeran, Tayyebeh M and Noë, Michaël and Horlings, Hugo M and Lum, Amy and Jones, Siân and Senz, Janine and Seckin, Tamer and Ho, Julie and Wu, Ren-Chin and Lac, Vivian and Ogawa, Hiroshi and Tessier-Cloutier, Basile and Alhassan, Rami and Wang, Yuxuan and Wang, Amy and Wang, Tian-Li and Cohen, Joshua D and Wong, Fontayne and Hasanovic, Adnan and Orr, Natasha and Zhang, Ming and Popoli, Maria and McMahon, Wyatt and Wood, Laura D and Mattox, Austin and Allaire, Catherine and Segars, James and Williams, Christina and Tomasetti, Cristian and Boyd, Niki and Kinzler, Kenneth W and Gilks, C. Blake and Diaz, Luis and Vogelstein, Bert and Yong, Paul J and Huntsman, David G and Shih, Ie-Ming
The New England journal of medicine, ISSN 0028-4793, 05/2017, Volume 376, Issue 19, pp. 1835 - 1848
Medicine, General & Internal | Life Sciences & Biomedicine | General & Internal Medicine | Science & Technology | Endometriosis - genetics | Proto-Oncogene Proteins p21(ras) - genetics | Humans | Middle Aged | Protein Phosphatase 2 - genetics | Endometriosis - pathology | Transcription Factors - genetics | DNA Mutational Analysis - methods | Exome | Phosphatidylinositol 3-Kinases - genetics | Class I Phosphatidylinositol 3-Kinases | Cell Transformation, Neoplastic - genetics | Polymerase Chain Reaction | Adult | Female | Mutation | Endometrium - pathology | Nuclear Proteins - genetics | Endometriosis | Research | Gene mutations | Oncology, Experimental | Cancer | Pathology | Womens health | Gynecology | Genomes | Tumors | Index Medicus | Abridged Index Medicus
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5. Full Text Closed-loop insulin delivery in suboptimally controlled type 1 diabetes: a multicentre, 12-week randomised trial
by Tauschmann, Martin and Thabit, Hood and Bally, Lia and Allen, Janet M and Hartnell, Sara and Wilinska, Malgorzata E and Ruan, Yue and Sibayan, Judy and Kollman, Craig and Cheng, Peiyao and Beck, Roy W and Acerini, Carlo L and Evans, Mark L and Dunger, David B and Elleri, Daniela and Campbell, Fiona and Bergenstal, Richard M and Criego, Amy and Shah, Viral N and Leelarathna, Lalantha and Hovorka, Roman and Alvarado, B and Ashanti, C and Baggott, J and Balakrishnan, K and Barber, N and Bath, L and Beasley, S and Beatson, C and Borgman, S and Bradshaw, S and Bugielski, B and Carlson, AB and Collett, E and Curtis, J and Demmitt, J and Donahue, D and Exall, J and Forshaw, R and Hayes, J and Heath, S and Hellmann, A and Huegel, V and Hyatt, J and James, L and Joseph, H and Joshee, P and Konerza, W and Lum, J and Madden, M and Martens, T and McCarthy, C and McDonald, M and Mikityuk, V and Miles, H and Miller, D and Mubita, W and Murphy, C and Olson, B and Pad, R and Patibandla, N and Riding, K and Shaju, A and Thomas, LA and Thomson, J and White, D and Yau, S and Yong, J and APCam11 Consortium and APCam 11 Consortium
The Lancet (British edition), ISSN 0140-6736, 10/2018, Volume 392, Issue 10155, pp. 1321 - 1329
Medicine, General & Internal | Life Sciences & Biomedicine | General & Internal Medicine | Science & Technology | Glycated Hemoglobin A - analysis | Infusion Pumps, Implantable | Humans | Child, Preschool | Insulin - administration & dosage | Male | Diabetes Mellitus, Type 1 - drug therapy | Hypoglycemia - prevention & control | Young Adult | Hypoglycemic Agents - administration & dosage | Adolescent | Blood Glucose Self-Monitoring | Adult | Female | Insulin Infusion Systems | Child | Type 1 diabetes | Insulin | Analysis | Diabetes therapy | Intervention | Therapy | Diabetes mellitus (insulin dependent) | Glucose | Coefficient of variation | Evidence-based medicine | Infusion | Hyperglycemia | Randomization | Hemoglobin | Teenagers | Sensors | Closed loop systems | Automation | Diabetes mellitus | Safety measures | Risk reduction | Hypoglycemia | Patients | Studies | Screening | Ketoacidosis | Diabetes | Risk management | Index Medicus | Abridged Index Medicus
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6. Full Text Variations in DNA elucidate molecular networks that cause disease
by Chen, Yanqing and Zhu, Jun and Lum, Pek Yee and Yang, Xia and Pinto, Shirly and MacNeil, Douglas J and Zhang, Chunsheng and Lamb, John and Edwards, Stephen and Sieberts, Solveig K and Leonardson, Amy and Castellini, Lawrence W and Wang, Susanna and Champy, Marie-France and Zhang, Bin and Emilsson, Valur and Doss, Sudheer and Ghazalpour, Anatole and Horvath, Steve and Drake, Thomas A and Lusis, Aldons J and Schadt, Eric E
Nature (London), ISSN 0028-0836, 03/2008, Volume 452, Issue 7186, pp. 429 - 435
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Obesity | Biological and medical sciences | Metabolic diseases | Miscellaneous | Medical sciences | Other metabolic disorders | Chromosomes, Mammalian - genetics | Phosphoprotein Phosphatases - metabolism | Apolipoprotein A-II - genetics | Male | Metabolic Syndrome - metabolism | Obesity - genetics | Lod Score | Adipose Tissue - metabolism | Female | Metabolic Syndrome - enzymology | Genetic Predisposition to Disease - genetics | Reproducibility of Results | Gene Regulatory Networks - genetics | Lipoprotein Lipase - genetics | Membrane Proteins - genetics | Ribosomal Proteins - genetics | Liver - metabolism | Linkage Disequilibrium | Obesity - metabolism | Macrophages - metabolism | Phenotype | Animals | Phosphoprotein Phosphatases - genetics | Metabolic Syndrome - genetics | Phosphoprotein Phosphatases - deficiency | Genetic Variation - genetics | Mice | Obesity - enzymology | Quantitative Trait Loci | Quantitative trait loci | Molecular neurobiology | Disease susceptibility | Genetic aspects | Research | Genetic variation | Integrated approach | Biomedical research | Gene expression | Genomics | Index Medicus | Lipoprotein Lipase | Liver | Apolipoprotein A-II | Metabolic Syndrome X | Macrophages | Gene Regulatory Networks | Life Sciences | Adipose Tissue | Phosphoprotein Phosphatases | Genetic Predisposition to Disease | Biochemistry, Molecular Biology | Variation (Genetics) | Chromosomes, Mammalian | Membrane Proteins | Molecular biology | Ribosomal Proteins
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7. Full Text Transcriptomic analysis of CIC and ATXN1L reveal a functional relationship exploited by cancer
by Wong, Derek and Lounsbury, Kohl and Lum, Amy and Song, Jungeun and Chan, Susanna and LeBlanc, Veronique and Chittaranjan, Suganthi and Marra, Marco and Yip, Stephen
Oncogene, ISSN 0950-9232, 01/2019, Volume 38, Issue 2, pp. 273 - 290
Biochemistry & Molecular Biology | Oncology | Genetics & Heredity | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Chromosome abnormalities | Gene mutations | Cellular control mechanisms | Genetic research | Development and progression | Genetic aspects | Research | DNA binding proteins | Health aspects | Cancer | Adenocarcinoma | Gene silencing | Stomach | Signal transduction | Transcription factors | Glioma | Ataxin | Derepression | Cell cycle | MAP kinase | Prostate | Index Medicus
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8. Full Text Oncogenic mutations in histologically normal endometrium: the new normal?
by Lac, Vivian and Nazeran, Tayyebeh M and Tessier‐Cloutier, Basile and Aguirre‐Hernandez, Rosalia and Albert, Arianne and Lum, Amy and Khattra, Jaswinder and Praetorius, Teresa and Mason, Madeline and Chiu, Derek and Köbel, Martin and Yong, Paul J and Gilks, Blake C and Anglesio, Michael S and Huntsman, David G
The Journal of pathology, ISSN 0022-3417, 10/2019, Volume 249, Issue 2, pp. 173 - 181
somatic mutations | endometrium | normal tissue | aging | digital PCR | Pathology | Oncology | Life Sciences & Biomedicine | Science & Technology | Predictive Value of Tests | Reproducibility of Results | Age Factors | Early Detection of Cancer - methods | Humans | Middle Aged | Endometrial Neoplasms - metabolism | Mutation Rate | Healthy Volunteers | Young Adult | Aging - genetics | Endometrial Neoplasms - genetics | DNA Mutational Analysis | Adult | Endometrial Neoplasms - pathology | Female | Mutation | Oncogenes | Aging - metabolism | Endometrium - metabolism | Genetic aspects | Skin | Endometrial cancer | Gene mutations | Analysis | Immunohistochemistry | Paraffins | Hyperplasia | Endometriosis | Tissue analysis | Malignancy | Paraffin | Tissues | Epithelium | Esophagus | Next-generation sequencing | Uterus | Aging | Fibroblast growth factor receptor 2 | Endometrium | PTEN protein | Cancer | Peritoneum | Index Medicus
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