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1. Full Text Common patterns and disease-related signatures in tuberculosis and sarcoidosis
by Jeroen Maertzdorf and January Weiner and Hans-Joachim Mollenkopf and Torsten Bauer and Antje Prasse and Joachim Müller-Quernheim and Stefan H. E. Kaufmann and TBornotTB Network
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 5/2012, Volume 109, Issue 20, pp. 7853 - 7858
In light of the marked global health impact of tuberculosis (TB), strong focus has been on identifying biosignatures. Gene expression profiles in blood cells... 
Datasets | MicroRNA | Tuberculosis | Cytokines | Genes | Lung diseases | Pulmonary tuberculosis | Signatures | Gene expression | Blood | PULMONARY | MARKERS | PERIPHERAL-BLOOD | INFECTION | MULTIDISCIPLINARY SCIENCES | DISCOVERY | Sarcoidosis - diagnosis | Diagnosis, Differential | Humans | Sarcoidosis - genetics | MicroRNAs - metabolism | Gene Expression Profiling | Biomarkers - blood | Sarcoidosis - blood | Tuberculosis - diagnosis | Tuberculosis - immunology | Tuberculosis - genetics | Microarray Analysis | Sarcoidosis - immunology | Tuberculosis - blood | Tuberculosis - metabolism | Sarcoidosis - metabolism | Cytokines - blood | Genetic aspects | Diagnosis | Research | Sarcoidosis | Signal transduction | Ribonucleic acid--RNA | Cells | Cadmium | Blood cells | Antimicrobial agents | Inflammation | Inflammatory diseases | Etiology | Chromium | miRNA | Interferon | Metalloproteinase | Immune system | Index Medicus | Biological Sciences
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2. Full Text Lung collagens perpetuate pulmonary fibrosis via CD204 and M2 macrophage activation
by Stahl, Mirjam and Schupp, Jonas and Jag̈er, Benedikt and Schmid, Michael and Zissel, Gernot and Mul̈ler-Quernheim, Joachim and Prasse, Antje
PLoS ONE, ISSN 1932-6203, 11/2013, Volume 8, Issue 11, pp. e81382 - e81382
Idiopathic pulmonary fibrosis is characterized by abundant collagen production and accumulation of alternatively activated macrophages (M2) in the lower... 
KAPPA-B ACTIVATION | SCAVENGER RECEPTOR | SERUM AMYLOID P | MEDIATED CELL-ADHESION | MULTIDISCIPLINARY SCIENCES | RECEPTOR CLASS-A | MONOCYTES | ALVEOLAR MACROPHAGES | SYSTEMIC-SCLEROSIS | METALLOPROTEINASES | EXPRESSION | Gene Expression | Lung - pathology | Phosphorylation | Collagen Type I - metabolism | Macrophage Activation - immunology | Humans | Collagen Type I - chemistry | Pulmonary Fibrosis - immunology | Collagen - chemistry | Phosphatidylinositol 3-Kinases - metabolism | Pulmonary Fibrosis - genetics | Case-Control Studies | Collagen - metabolism | Macrophages - metabolism | Scavenger Receptors, Class A - genetics | Chemokine CCL2 - metabolism | Lung - metabolism | Pulmonary Fibrosis - metabolism | Macrophages, Alveolar - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Lung - immunology | Macrophages - immunology | Macrophages, Alveolar - immunology | Scavenger Receptors, Class A - metabolism | Viral antibodies | Pulmonary fibrosis | Interleukins | Collagen | Antibodies | Respiratory tract diseases | Macrophages | Enzyme-linked immunosorbent assay | Flow cytometry | Collagen (type I) | Laboratories | Lung | Interleukin | Collagens | Stimulation | AKT protein | Interleukin 1 receptors | Kinases | Monomers | Respiratory tract | Signal transduction | Cell activation | Bronchoalveolar lavage | Rodents | Interleukin 1 | CCL18 protein | Extracellular matrix | Tumor necrosis factor-TNF | Alveoli | Immune response | Cytokines | Interleukin 1 receptor antagonist | Lung diseases | Bronchus | Patients | Polymerase chain reaction | Cytometry | Neutralizing | Fibrosis | Ligands | Monocyte chemoattractant protein 1 | Binding sites | Index Medicus
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3. Full Text Genome-wide association study identifies ANXA11 as a new susceptibility locus for sarcoidosis
by Krawczak, Michael and Schreiber, Stefan and Jacobs, Gunnar and Hofmann, Sylvia and Gaede, Karoline I and Schürmann, Manfred and Franke, Andre and Rosenstiel, Philip and Fischer, Annegret and Müller-Quernheim, Joachim and Nothnagel, Michael
Nature Genetics, ISSN 1061-4036, 09/2008, Volume 40, Issue 9, pp. 1103 - 1106
Sarcoidosis is a complex chronic inflammatory disorder with predominant manifestation in the lung. In the first genome-wide association study (>440,000 SNPs)... 
APOPTOSIS | DOMAIN | PROTEIN | SEARCH | ALG-2 | GENETICS & HEREDITY | ANNEXIN-XI | IL-15 | AFRICAN-AMERICANS | BTNL2 GENE | EXPRESSION | Amino Acid Sequence | Genetic Predisposition to Disease | Genome-Wide Association Study | Humans | Sarcoidosis - genetics | Models, Molecular | Chromosome Mapping | Annexins - genetics | Sequence Analysis, Protein | Validation Studies as Topic | Polymorphism, Single Nucleotide | Genome, Human | Chromosomes, Human, Pair 10 | Physiological aspects | Genetic aspects | Research | Single nucleotide polymorphisms | Sarcoidosis | Risk factors | Data analysis | Racial differences | Rheumatoid arthritis | Cell division | Genetics | Binding sites | Apoptosis | Index Medicus
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4. Full Text A randomized trial of everolimus‐based quadruple therapy vs standard triple therapy early after lung transplantation
by Gottlieb, Jens and Neurohr, Claus and Müller‐Quernheim, Joachim and Wirtz, Hubert and Sill, Bjoern and Wilkens, Heinrike and Bessa, Vasiliki and Knosalla, Christoph and Porstner, Martina and Capusan, Carmen and Strüber, Martin
American Journal of Transplantation, ISSN 1600-6135, 06/2019, Volume 19, Issue 6, pp. 1759 - 1769
Calcineurin inhibitor ( CNI ) therapy after lung transplantation increases risk of kidney failure. Early everolimus‐based quadruple low CNI immunosuppression... 
everolimus | immunosuppressant ‐ mechanistic target of rapamycin | immunosuppression/immune modulation | clinical research/practice | tacrolimus | lung transplantation/pulmonology | mTOR | cyclosporine A (CsA) | immunosuppressant ‐ calcineurin inhibitor | rejection | immunosuppressant - mechanistic target of rapamycin (mTOR) | immunosuppressant - mechanistic target of rapamycin | immunosuppressant - calcineurin inhibitor | Clinical trials | Transplantation of organs, tissues, etc | Tacrolimus | Analysis | Immunotherapy | Standards | Renal function | Epidermal growth factor receptors | Cyclosporins | Syngeneic grafts | Lung | Calcineurin | Lung transplantation | Calcineurin inhibitors | Prednisone | Glomerular filtration rate | Graft rejection | Immunosuppression | Biopsy | Cell cycle | Xenografts | Renal failure | Kidney transplantation | practice | immunosuppressant ‐ mechanistic target of rapamycin (mTOR) | ORIGINAL ARTICLES | immunosuppression | pulmonology | clinical research | lung transplantation | immune modulation | Original
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5. Full Text Serum CC-Chemokine Ligand 18 Concentration Predicts Outcome in Idiopathic Pulmonary Fibrosis
by Prasse, Antje and Probst, Corinna and Bargagli, Elena and Zissel, Gernot and Toews, Galen B and Flaherty, Kevin R and Olschewski, Manfred and Rottoli, Paola and Muller-Quernheim, Joachim
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 04/2009, Volume 179, Issue 8, pp. 717 - 723
Rationale Idiopathic pulmonary fibrosis (IPF) is a devastating lung disease with a poor prognosis. There is great effort to find predictors of outcome.... 
Serum | Biomarker | Pulmonary fibrosis | CCL18 | IPF | SURVIVAL | ACTIVATION | SURFACTANT PROTEIN-A | MACROPHAGES | INTERSTITIAL PNEUMONIA | biomarker | SYSTEMIC-SCLEROSIS | PROGNOSTIC VALUE | FIBROBLASTS | RESPIRATORY SYSTEM | pulmonary fibrosis | DISEASE | serum | CRITICAL CARE MEDICINE | Prognosis | Prospective Studies | Chemokines, CC - blood | Enzyme-Linked Immunosorbent Assay | Humans | Middle Aged | Male | Biomarkers - blood | Idiopathic Pulmonary Fibrosis - blood | Survival Analysis | Female | Aged | Idiopathic Pulmonary Fibrosis - physiopathology | Respiratory Function Tests | Index Medicus | Abridged Index Medicus
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6. Full Text Macrophage activation in acute exacerbation of idiopathic pulmonary fibrosis
by Schupp, Jonas Christian and Binder, Harald and Jäger, Benedikt and Cillis, Giuseppe and Zissel, Gernot and Müller-Quernheim, Joachim and Prasse, Antje
PLoS ONE, ISSN 1932-6203, 01/2015, Volume 10, Issue 1, pp. e0116775 - e0116775
Background Acute exacerbation (AE) of idiopathic pulmonary fibrosis (IPF) is a common cause of disease acceleration in IPF and has a major impact on mortality.... 
TUMOR-NECROSIS-FACTOR | INTERLEUKIN-1-BETA | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | LIPOPOLYSACCHARIDE | ACUTE LUNG INJURY | ALVEOLAR MACROPHAGES | MECHANISMS | SARCOIDOSIS | RESPIRATORY-DISTRESS-SYNDROME | FACTOR-ALPHA | Macrophage Activation - physiology | Tumor Necrosis Factor-alpha - metabolism | Humans | Middle Aged | Male | Case-Control Studies | Idiopathic Pulmonary Fibrosis - metabolism | Interleukins - metabolism | Idiopathic Pulmonary Fibrosis - pathology | Female | Aged | Chemokine CXCL1 - metabolism | Neutrophils - metabolism | Chemokines, CC - metabolism | Neutrophils - pathology | Pulmonary fibrosis | Tumor necrosis factor | Mortality | Respiratory tract diseases | Macrophages | Health aspects | Enzyme-linked immunosorbent assay | Respiratory therapy | Disease | Cytokines | Activation | IL-1β | Gene expression | Patients | Survival analysis | Respiratory distress syndrome | Medical prognosis | Fibrosis | Tomography | Tumor necrosis factor-TNF | TNF inhibitors | Acceleration | Chemokines | Index Medicus
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7. Full Text Functional toll-like receptor 9 expression and CXCR3 ligand release in pulmonary sarcoidosis
by Schnerch, Jasmin and Prasse, Antje and Vlachakis, Dimitrios and Schuchardt, Kathrin L and Pechkovsky, Dmitri V and Goldmann, Torsten and Gaede, Karoline I and Muller-Quernheim, Joachim and Zissel, Gernot
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 11/2016, Volume 55, Issue 5, pp. 749 - 757
Sarcoidosis is a granulomatous disease characterized by a T-helper type 1 (Th1) cell-dominated alveolitis. As a role of bacteria in the pathogenesis of... 
Toll-like receptors | Cxcr3 ligands | Sarcoidosis | Bronchoalveolar lavage | Alveolitis | MYCOBACTERIUM-TUBERCULOSIS | IMMUNE-RESPONSE | TH1 RESPONSES | HUMAN LUNG | BIOCHEMISTRY & MOLECULAR BIOLOGY | LYMPH-NODES | CPG-DNA | sarcoidosis | alveolitis | CELL BIOLOGY | RESPIRATORY SYSTEM | ALVEOLAR MACROPHAGES | BRONCHOALVEOLAR LAVAGE CELLS | PROPIONIBACTERIUM-ACNES | bronchoalveolar lavage | CXCR3 ligands | LYMPHOCYTES-T | Immunohistochemistry | Receptors, CXCR3 - metabolism | Humans | Cells, Cultured | Nod1 Signaling Adaptor Protein - metabolism | Flow Cytometry | Nod2 Signaling Adaptor Protein - metabolism | Biopsy | Sarcoidosis, Pulmonary - metabolism | Fluorescent Antibody Technique | Ligands | Bronchoalveolar Lavage Fluid - cytology | Oligodeoxyribonucleotides - pharmacology | Toll-Like Receptor 9 - metabolism | Bronchi - pathology | Chemokine CXCL10 - metabolism | Real-Time Polymerase Chain Reaction | Sarcoidosis, Pulmonary - pathology | Antigens | Cell culture | Disease | Values | Studies | Hypotheses | Tuberculosis | Lymphocytes | Rodents | Chemokines | Immune system | Index Medicus
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8. Full Text Psychometric properties of the German version of the Leicester Cough Questionnaire in sarcoidosis
by Schupp, Jonas Christian and Fichtner, Urs Alexander and Frye, Bjoèrn Christian and Heyduck-Weides, Katja and Birring, Surinder S and Windisch, Wolfram and Criée, Carl-Peter and Müller-Quernheim, Joachim and Farin, Erik
PLoS ONE, ISSN 1932-6203, 10/2018, Volume 13, Issue 10, pp. e0205308 - e0205308
Background Cough is one of the most common symptoms in general and pulmonary medicine with profound negative impact on health-related quality of life (HRQL).... 
FIT INDEXES | KOREAN VERSION | MULTIDISCIPLINARY SCIENCES | CLINICAL-TRIALS | DOUBLE-BLIND | VALIDATION | RELIABILITY | QUALITY-OF-LIFE | CYSTIC-FIBROSIS | RASCH ANALYSIS | VALIDITY | Cough - psychology | Cough - physiopathology | Sarcoidosis - psychology | Humans | Middle Aged | Male | Dyspnea - physiopathology | Factor Analysis, Statistical | Patients | Dyspnea - epidemiology | Germany - epidemiology | Dyspnea - psychology | Sarcoidosis - epidemiology | Psychometrics - standards | Cough - epidemiology | Sarcoidosis - physiopathology | Quality of Life | Adult | Female | Surveys and Questionnaires | Schools | Psychometrics | Usage | Research | Sarcoidosis | Analysis | Questionnaires | Mental disorders | Stigma | Consistency | Clinical trials | Cough | Quality of life | Asthma | Medicine | Studies | Validity | Properties (attributes) | Hospitals | Dyspnea | Chronic obstructive pulmonary disease | Quantitative psychology | Health surveys | Quality management | Index Medicus | Surveys
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9. Full Text Is serum level of CC chemokine ligand 18 a biomarker for the prediction of radiation induced lung toxicity (RILT)?
by Gkika, Eleni and Vach, Werner and Adebahr, Sonja and Schimeck-Jasch, Tanja and Brenner, Anton and Brunner, Thomas Baptist and Kaier, Klaus and Prasse, Antje and Müller-Quernheim, Joachim and Grosu, Anca-Ligia and Zissel, Gernot and Nestle, Ursula
PLoS ONE, ISSN 1932-6203, 09/2017, Volume 12, Issue 9, pp. e0185350 - e0185350
The CC chemokine ligand 18 (CCL18) is produced by alveolar macrophages in patients with fibrosing lung disease and its concentration is increased in various... 
CANCER PATIENTS | BREAST-CANCER | CYTOKINES | THERAPY | MULTIDISCIPLINARY SCIENCES | INDICATOR | TUMOR-ASSOCIATED MACROPHAGES | PNEUMONITIS | RADIOTHERAPY | CCL18 | PULMONARY-FIBROSIS | Prognosis | Chemokines, CC - blood | Humans | Middle Aged | Radiation Injuries - diagnosis | Male | Lung Diseases - therapy | Biomarkers - blood | Radiation Injuries - complications | Lung Diseases - etiology | Neoplasm Recurrence, Local - pathology | Lung Diseases - blood | Radiation Injuries - blood | Aged, 80 and over | Adult | Female | Aged | Lung cancer | Causes of | Physiological aspects | Genetic aspects | Diagnosis | Research | Chemokines | Biometrics | Pneumonia | Thymoma | Toxicity | Lung | Radiation | Medical services | Colorectal cancer | Oncology | Biology | Macrophages | Cancer therapies | Ovarian cancer | Consortia | Genotype & phenotype | Signal transduction | CCL18 protein | Bioindicators | Alveoli | Drug dosages | Enzyme-linked immunosorbent assay | Medical research | Radiation effects | Statistical analysis | Cytokines | Lung diseases | Thorax | Breast cancer | Radiation therapy | Patients | Esophagus | Medicine | Serum levels | Hospitals | Pneumonitis | Fibrosis | Irradiation | Biomarkers | Ligands | Tumors | Cancer | Index Medicus
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10. Full Text Whole-exome sequencing identifies rare genetic variations in German families with pulmonary sarcoidosis
by Kishore, Amit and Petersen, Britt-Sabina and Nutsua, Marcel and Müller-Quernheim, Joachim and Franke, Andre and Fischer, Annegret and Schreiber, Stefan and Petrek, Martin
Human Genetics, ISSN 0340-6717, 9/2018, Volume 137, Issue 9, pp. 705 - 716
Genome-wide and candidate gene studies for pulmonary sarcoidosis have highlighted several candidate variants among different populations. However, the genetic... 
Human Genetics | Gene Function | Molecular Medicine | Biomedicine | Metabolic Diseases | PATHWAYS | CELLS | VITAMIN-D METABOLISM | DISEASES | ANNOTATION | VARIANT ASSOCIATION | CALCIUM | GENETICS & HEREDITY | LINKAGE ANALYSIS | PRIORITIZATION | PHENOTYPES | Medical research | Gastrointestinal diseases | Genomics | Medicine, Experimental | Genetic aspects | Genomes | Protein-protein interactions | Phenotypes | Inflammatory bowel diseases | G protein-coupled receptors | Immune response | Sarcoidosis | Wnt protein | Calcium | Cytokines | Lung | Genetic diversity | Cadherin | Population genetics | Proteins | Signal transduction | Gene frequency | Intestine | γ-Interferon | Interleukin 10 | Protein interaction | Retinoic acid | Immune system | Index Medicus
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11. Full Text Immune response to Propionibacterium acnes in patients with sarcoidosis - in vivo and in vitro
by Schupp, Jonas Christian and Tchaptchet, Sandrine and Lützen, Niklas and Engelhard, Peggy and Müller-Quernheim, Joachim and Freudenberg, Marina A and Prasse, Antje
BMC Pulmonary Medicine, ISSN 1471-2466, 07/2015, Volume 15, Issue 1, pp. 75 - 75
Background: Propionibacterium acnes was found in lungs and lymph nodes of patients with sarcoidosis and may induce hypersensitivity type granuloma formation.... 
Immunoglobulin | Immune response | Sarcoidosis | Propionibacterium acnes | human | LYMPH-NODES | QUANTITATIVE-ANALYSIS | BLOOD MONONUCLEAR-CELLS | TUMOR-NECROSIS-FACTOR | GRANULOMA-FORMATION | INTERFERON-GAMMA | RESPIRATORY SYSTEM | PULMONARY SARCOIDOSIS | ALVEOLAR MACROPHAGES | BRONCHOALVEOLAR LAVAGE CELLS | JAPANESE PATIENTS | Bronchoscopy | Propionibacterium acnes - immunology | Gram-Positive Bacterial Infections - immunology | Sarcoidosis, Pulmonary - immunology | Enzyme-Linked Immunosorbent Assay | Immunoglobulins - immunology | Humans | Middle Aged | Antigens, Bacterial - immunology | Male | Immunity, Innate | Bronchoalveolar Lavage Fluid - microbiology | Propionibacterium acnes - isolation & purification | Adult | Antibodies, Bacterial - immunology | Female | Gram-Positive Bacterial Infections - complications | Sarcoidosis, Pulmonary - microbiology | Sarcoidosis, Pulmonary - complications | Medicine, Experimental | Medical research | Staphylococcus aureus | Index Medicus
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12. Full Text Identification of immune-relevant factors conferring sarcoidosis genetic risk
by Fischer, Annegret and Ellinghaus, David and Nutsua, Marcel and Hofmann, Sylvia and Montgomery, Courtney G and Iannuzzi, Michael C and Rybicki, Benjamin A and Petrek, Martin and Mrazek, Frantisek and Pabst, Stefan and Grohè, Christian and Grunewald, Johan and Ronninger, Marcus and Eklund, Anders and Padyukov, Leonid and Mihailovic-Vucinic, Violeta and Jovanovic, Dragana and Sterclova, Martina and Homolka, Jiri and Nöthen, Markus M and Herms, Stefan and Gieger, Christian and Strauch, Konstantin and Winkelmann, Juliane and Boehm, Bernhard O and Brand, Stephan and Büning, Carsten and Schürmann, Manfred and Ellinghaus, Eva and Baurecht, Hansjörg and Lieb, Wolfgang and Nebel, Almut and Müller-Quernheim, Joachim and Franke, Andre and Schreiber, Stefan and GenPhenReSa Consortium
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 09/2015, Volume 192, Issue 6, pp. 727 - 736
Rationale: Genetic variation plays a significant role in the etiology of sarcoidosis. However, only a small fraction of its heritability has been explained so... 
Association | BTNL2 | Immunochip | HLA | IL23 | CROHNS-DISEASE | SUSCEPTIBILITY LOCI | association | HLA ALLELES | VARIANT | INFLAMMATORY-BOWEL-DISEASE | POLYMORPHISMS | RESPIRATORY SYSTEM | AFRICAN-AMERICANS | BTNL2 GENE | CLASS-II | GENOME-WIDE ASSOCIATION | CRITICAL CARE MEDICINE | European Continental Ancestry Group - genetics | Genetic Predisposition to Disease | Genome-Wide Association Study | Europe | Humans | Middle Aged | Sarcoidosis - ethnology | Sarcoidosis - genetics | African Americans - genetics | Genotype | Male | Genetic Markers | Case-Control Studies | Sarcoidosis - immunology | Adult | Female | Aged | Polymorphism, Single Nucleotide | Index Medicus | Abridged Index Medicus | Original
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13. Full Text CC-Chemokine Ligand 18 Induces Epithelial to Mesenchymal Transition in Lung Cancer A549 Cells and Elevates the Invasive Potential
by Ploenes, Till and Scholtes, Ben and Krohn, Alexander and Burger, Meike and Passlick, Bernward and Müller-Quernheim, Joachim and Zissel, Gernot
PLoS ONE, ISSN 1932-6203, 01/2013, Volume 8, Issue 1, pp. e53068 - e53068
Lung cancer is one of the leading causes of cancer related death worldwide with more than a million deaths per year. The poor prognosis is due to its high... 
BREAST-CANCER | COLLAGEN PRODUCTION | METASTASIS | TUMOR PROGRESSION | MACROPHAGES | MULTIDISCIPLINARY SCIENCES | CCL18 | EXPRESSION | PULMONARY-FIBROSIS | FIBROBLASTS | PROTEIN S100A4 | Neoplasm Metastasis | Chemokines, CC - pharmacology | Neoplasm Invasiveness | Humans | Cell Line, Tumor | Epithelial-Mesenchymal Transition - drug effects | Lung Neoplasms - pathology | Cell Proliferation - drug effects | Chemotaxis - drug effects | Cisplatin - pharmacology | Gene Expression Regulation, Neoplastic - drug effects | Drug Resistance, Neoplasm - drug effects | Prognosis | Metastasis | Lung cancer, Non-small cell | Health aspects | Stem cells | Cell proliferation | Adenocarcinoma | Thoracic surgery | Transcription factors | Transcription | Mesenchyme | Lung cancer | Lung | Chemoresistance | Oncology | E-cadherin | Metastases | Fibroblasts | CCL18 protein | Growth factors | Cytokines | Internal medicine | Hematology | Invasiveness | Chemotaxis | Patients | Medicine | Serum levels | Pulmonary fibrosis | Medical prognosis | Ligands | Chemokines | Cancer | Index Medicus
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