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2001, Methods in molecular medicine, ISBN 0896037614, Volume 62, xi, 317
The use of molecular technologies to study Parkinson's disease in the laboratory is beginning to have a major impact in clinical medicine. In Parkinson's... 
Pathology, Molecular | Parkinson's disease | Laboratory manuals | Neurology
Book
Neurotherapeutics, ISSN 1933-7213, 10/2017, Volume 14, Issue 4, pp. 831 - 831
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2005, Volume 102, Issue 27, pp. 9691 - 9696
Journal Article
Neurotherapeutics, ISSN 1933-7213, 4/2019, Volume 16, Issue 2, pp. 287 - 298
Journal Article
Journal Article
2001, Methods in molecular medicine, ISBN 0896037614, Volume 62
Web Resource
2001, Methods in Molecular Medicine, ISBN 1592591426, Volume 62
Web Resource
PLoS ONE, ISSN 1932-6203, 01/2012, Volume 7, Issue 1, pp. 242 - 249
Apoptosis signal-regulating kinase 1 ASIK1) member of the mitogen-activated protein kinase 3 family, is activated by oxidative stress. The death-signaling... 
OXIDATIVE STRESS | MICROGLIAL ACTIVATION | ASK1 | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | NEURODEGENERATION | MICE | DOPAMINERGIC-NEURONS | SUBSTANTIA-NIGRA | PARKINSONS-DISEASE | CELL-DEATH | Oxidative Stress | Substantia Nigra - pathology | MAP Kinase Kinase Kinase 5 - deficiency | Neuroglia - pathology | Humans | Mice, Inbred C57BL | Neuroglia - enzymology | Cells, Cultured | Rats | Nerve Endings - metabolism | Substantia Nigra - metabolism | Rats, Sprague-Dawley | MPTP Poisoning - enzymology | Animals | Dopaminergic Neurons - metabolism | Behavior, Animal - drug effects | Mice | Enzyme Activation | MAP Kinase Kinase Kinase 5 - metabolism | Nerve Endings - pathology | Cytoprotection | Genetic aspects | Neurons | Protein kinases | Mitogens | Apoptosis | Brain | Oxidative stress | Phosphorylation | Parkinson's disease | Mesencephalon | Toxicity | Substantia nigra | Parkinsons disease | Activation | Kinases | Phosphatase | Medical schools | Neuronal-glial interactions | Proteins | Electron transport chain | Signal transduction | Mitochondria | Neurotoxicity | Methamphetamines | Rodents | Neostriatum | Inhibition | Movement disorders | NADH-ubiquinone oxidoreductase | Dopamine | Neurodegenerative diseases | MAP kinase | Inflammation | Microglia | PARK7 protein | Neurology | Signaling | Protein kinase | MPTP | Index Medicus
Journal Article
Diabetes, ISSN 0012-1797, 06/2019, Volume 68, Issue Supplement 1, p. 1813
Alzheimer’s disease is associated with many other disorders of healthful aging; one of the most prominent is the increased prevalence of diabetes in patients... 
Glucose tolerance | Adenylate cyclase | Animal models | Diabetes mellitus | Spatial discrimination learning | Aging | Glucose | Diabetes | Spatial memory
Journal Article
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, ISSN 0027-8424, 12/2018, Volume 115, Issue 51, pp. E12053 - E12062
Hyperphosphorylated alpha-synuclein in Lewy bodies and Lewy neurites is a characteristic neuropathological feature of Parkinson's disease (PD) and Dementia... 
CATALYTIC SUBUNIT | Parkinson's disease | PHOSPHORYLATION | PHOSPHOPROTEIN PHOSPHATASE 2A | PROTEIN PHOSPHATASE-2A | ALZHEIMERS-DISEASE | MULTIDISCIPLINARY SCIENCES | MOUSE | RISK | ALPHA-SYNUCLEIN | alpha-synuclein phosphorylation | neuroprotection | CARBOXYL METHYLATION | Dementia with Lewy bodies | TRANSGENIC MICE | Neurons - pathology | Phosphorylation | Parkinson Disease - drug therapy | Serotonin - pharmacology | Neuroprotection - drug effects | Brain - metabolism | Coffee - chemistry | Caffeine - pharmacology | Neurons - metabolism | Parkinson Disease - metabolism | Lewy Body Disease - drug therapy | Neurons - drug effects | Fatty Acids - metabolism | Disease Models, Animal | Parkinson Disease - pathology | Mice, Inbred C57BL | Lewy Body Disease - pathology | Mice, Transgenic | Brain - drug effects | Lewy Body Disease - metabolism | Animals | Protein Phosphatase 2 - metabolism | Brain - pathology | Serotonin - analogs & derivatives | Mice | alpha-Synuclein - metabolism | Physiological aspects | Amides | Coffee | Health aspects | Brain | Neuroprotection | Animal models | Toxicity | Phosphoprotein phosphatase | Parkinsons disease | Synuclein | Epidemiology | Accumulation | Proteins | Synergistic effects | Inoculation | Neurotoxicity | Neostriatum | Dementia disorders | Catalysis | Movement disorders | Caffeine | Phenotypes | Serotonin | Neurodegenerative diseases | Transgenic mice | Inflammation | Fatty acids | Lewy bodies | Axons | Protein phosphatase | Methylation | Dementia | Index Medicus | Biological Sciences | PNAS Plus | phosphorylation | α-synuclein | Parkinson’s disease
Journal Article
Science, ISSN 0036-8075, 6/2002, Volume 296, Issue 5576, pp. 2238 - 2243
Huntington's disease (HD) is an inherited neurodegenerative disease caused by expansion of a polyglutamine tract in the huntingtin protein. Transcriptional... 
Grade 1 | Brain | Grade 4 | Caudate nucleus | Neurons | Transgenic animals | Social interaction | DNA | Genes | Reports | Hippocampus | NERVOUS-SYSTEM | TFIID SUBUNIT | HUMAN BRAIN | MEDIATED TRANSCRIPTION | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | GLUTAMINE REPEATS | RECEPTOR GENE | BINDING PROTEIN | MAMMALIAN-CELLS | TRANSGENIC MICE | Corpus Striatum - embryology | Transcription Factors - chemistry | Peptides | Humans | Sp1 Transcription Factor - metabolism | Corpus Striatum - cytology | Corpus Striatum - metabolism | Brain - metabolism | DNA-Binding Proteins - metabolism | Transcription Factor TFIID | Nerve Tissue Proteins - chemistry | Cell Nucleus - metabolism | Transfection | Cell Death | Neurons - physiology | Transcription, Genetic | Nuclear Proteins - genetics | Cell Line | Promoter Regions, Genetic | Down-Regulation | Cells, Cultured | Gene Expression Regulation | Solubility | Rats | Mice, Transgenic | Nuclear Proteins - metabolism | Sp1 Transcription Factor - chemistry | DNA - metabolism | Nuclear Proteins - chemistry | TATA-Binding Protein Associated Factors | DNA-Binding Proteins - chemistry | Huntington Disease - metabolism | Nerve Tissue Proteins - genetics | Receptors, Dopamine D2 - genetics | Trinucleotide Repeat Expansion | Caudate Nucleus - metabolism | Nerve Tissue Proteins - metabolism | Transcription Factors - metabolism | Huntingtin Protein | Two-Hybrid System Techniques | Animals | Huntington Disease - genetics | Mice | Mutation | Genetic aspects | Research | Huntington's chorea | Nucleoproteins | Proteins | Neurology | Nervous system | Disease | Index Medicus
Journal Article
Neurotherapeutics, ISSN 1933-7213, 1/2014, Volume 11, Issue 1, pp. 2 - 2
Journal Article
Journal Article
Neurology, ISSN 0028-3878, 05/2014, Volume 82, Issue 18, pp. 1574 - 1575
Journal Article
Redox Biology, ISSN 2213-2317, 10/2018, Volume 19, pp. 62 - 73
DJ-1 is a highly conserved protein that protects neurons against oxidative stress and whose loss of function mutations are linked to recessively inherited... 
Oxidative stress | Autosomal recessive | Parkinson's disease | microRNA (miRNA) | miR-221 | DJ-1 |