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Development (Cambridge), ISSN 0950-1991, 11/2017, Volume 144, Issue 22, pp. 4047 - 4060
Journal Article
Proceedings of the National Academy of Sciences, ISSN 0027-8424, 07/2015, Volume 112, Issue 27, pp. 8421 - 8426
Cerebral cavernous malformation (CCM) is a disease of the central nervous system causing hemorrhage-prone multiple lumen vascular malformations and very severe... 
Cerebral cavernous malformation | Sulindac metabolites | catenin | Endothelial cells | Vascular pathology | cerebral cavernous malformation | beta-catenin | endothelial cells | SIGNALING CONTROLS | MULTIDISCIPLINARY SCIENCES | BLOOD-BRAIN-BARRIER | EFFECTOR | CEREBRAL CAVERNOUS MALFORMATIONS | vascular pathology | PATHWAY | RAP1 | DISEASE | JUNCTIONS | GROWTH | sulindac metabolites | Immunohistochemistry | Central Nervous System Neoplasms - metabolism | Endothelial Cells - metabolism | Central Nervous System Neoplasms - genetics | Intracellular Signaling Peptides and Proteins - metabolism | Signal Transduction - genetics | Reverse Transcriptase Polymerase Chain Reaction | Hemangioma, Cavernous, Central Nervous System - drug therapy | beta Catenin - metabolism | Hemangioma, Cavernous, Central Nervous System - genetics | Hemangioma, Cavernous, Central Nervous System - metabolism | beta Catenin - genetics | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Mice, Knockout | Intracellular Signaling Peptides and Proteins - deficiency | Sulindac - analogs & derivatives | Animals | Signal Transduction - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Central Nervous System Neoplasms - drug therapy | Intracellular Signaling Peptides and Proteins - genetics | Sulindac - pharmacology | Transforming Growth Factor beta - metabolism | Disease Models, Animal | Usage | Sulindac | Genetic aspects | Research | Drug therapy | Health aspects | Cerebrovascular disease | Biological Sciences | β-catenin | Other Medical Sciences not elsewhere specified | Other Medical Sciences | Medical and Health Sciences | Medicin och hälsovetenskap | Annan medicin och hälsovetenskap | Övrig annan medicin och hälsovetenskap
Journal Article
Nature, ISSN 0028-0836, 2013, Volume 498, Issue 7455, pp. 492 - 496
Journal Article
Nature Communications, ISSN 2041-1723, 2012, Volume 3, Issue 1, p. 1208
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2013, Volume 8, Issue 8, p. e70233
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 10/2014, Volume 124, Issue 10, pp. 4335 - 4350
Journal Article
European Journal of Immunology, ISSN 0014-2980, 09/2015, Volume 45, Issue 9, pp. 2517 - 2528
Chronic skin inflammation resulting from a defective epidermal barrier is a hallmark of atopic dermatitis (AD). We previously demonstrated that mice lacking... 
Dendritic epidermal T cells | Inflammation | T cells | Atopic dermatitis | Barrier function | T-Lymphocyte Subsets - immunology | Cell Proliferation | Immunoglobulins - immunology | Dermatitis - immunology | Receptors, Antigen, T-Cell, gamma-delta - genetics | Immunoglobulins - genetics | Lymphocyte Depletion | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Interleukin-7 - genetics | Receptor, Fibroblast Growth Factor, Type 1 - deficiency | Interleukin-2 - immunology | Minor Histocompatibility Antigens - genetics | Minor Histocompatibility Antigens - immunology | Receptor, Fibroblast Growth Factor, Type 2 - deficiency | Carrier Proteins - immunology | Disease Models, Animal | Nucleocytoplasmic Transport Proteins - immunology | Epidermis - pathology | Nerve Tissue Proteins - immunology | Signal Transduction | Dermatitis - genetics | Lymphocyte Activation | Nuclear Matrix-Associated Proteins - immunology | Gene Expression Regulation | Histocompatibility Antigens Class I - immunology | Mice, Transgenic | Receptors, Antigen, T-Cell, gamma-delta - immunology | Histocompatibility Antigens Class I - genetics | Nerve Tissue Proteins - genetics | Receptors, Antigen, T-Cell, gamma-delta - deficiency | Keratinocytes - immunology | Nucleocytoplasmic Transport Proteins - genetics | Carrier Proteins - genetics | Dermatitis - pathology | Interleukin-2 - genetics | Keratinocytes - pathology | Animals | Interleukin-7 - immunology | T-Lymphocyte Subsets - pathology | Epidermis - immunology | Nuclear Matrix-Associated Proteins - genetics | Receptor, Fibroblast Growth Factor, Type 1 - immunology | Receptor, Fibroblast Growth Factor, Type 2 - immunology | Mice | Chronic Disease | Receptor, Fibroblast Growth Factor, Type 2 - genetics
Journal Article
European Journal of Immunology, ISSN 0014-2980, 09/2015, Volume 45, Issue 9, p. 2517
  Chronic skin inflammation resulting from a defective epidermal barrier is a hallmark of atopic dermatitis (AD). We previously demonstrated that mice lacking... 
Genotype & phenotype | Lymphocytes | Dermatitis
Journal Article
Journal of Cell Science, ISSN 0021-9533, 04/2010, Volume 123, Issue 7, pp. 1073 - 1080
Journal Article
Journal Article
EUROPEAN JOURNAL OF IMMUNOLOGY, ISSN 0014-2980, 09/2015, Volume 45, Issue 9, pp. 2517 - 2528
Chronic skin inflammation resulting from a defective epidermal barrier is a hallmark of atopic dermatitis (AD). We previously demonstrated that mice lacking... 
Dendritic epidermal T cells | Barrier function | GROWTH-FACTOR RECEPTORS | Inflammation | FETAL THYMOCYTES | IMMUNOLOGY | T cells | GENE-MUTANT MICE | LANGERHANS CELLS | CUTTING EDGE | ATOPIC-DERMATITIS | Atopic dermatitis | SKIN | LIGAND | WOUND REPAIR
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2015, Volume 112, Issue 27, pp. 8421 - 8426
Cerebral cavernous malformation (CCM) is a disease of the central nervous system causing hemorrhage-prone multiple lumen vascular malformations and very severe... 
Journal Article
Journal Article