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Kidney International, ISSN 0085-2538, 01/2015, Volume 87, Issue 1, pp. 74 - 84
Journal Article
Nature Medicine, ISSN 1078-8956, 11/2007, Volume 13, Issue 11, pp. 1349 - 1358
Data providing direct evidence for a causative link between endothelial dysfunction, microvascular disease and diabetic end-organ damage are scarce. Here we... 
MEDICINE, RESEARCH & EXPERIMENTAL | POLY(ADP-RIBOSE) POLYMERASE INHIBITORS | ANTICOAGULANT PATHWAY | BIOCHEMISTRY & MOLECULAR BIOLOGY | MOUSE | ALPHA | CELL BIOLOGY | THROMBOMODULIN | INFLAMMATION | DYSFUNCTION | CELL | RECEPTOR-1 | SEVERE SEPSIS | Kidney Glomerulus - blood supply | Diabetic Nephropathies - enzymology | Protein C - physiology | Diabetes Mellitus, Experimental - enzymology | Humans | Diabetes Mellitus, Experimental - genetics | Podocytes - enzymology | Apoptosis - genetics | Endothelium, Vascular - enzymology | Thrombomodulin - physiology | Diabetes Mellitus, Experimental - prevention & control | Mice, Mutant Strains | Diabetic Nephropathies - prevention & control | Kidney Glomerulus - enzymology | Diabetic Nephropathies - pathology | Mice, Inbred C57BL | Cells, Cultured | Diabetic Nephropathies - genetics | Mice, Transgenic | Signal Transduction - genetics | Kidney Glomerulus - pathology | Podocytes - pathology | Protein C - biosynthesis | Animals | Microcirculation - pathology | Microcirculation - enzymology | Diabetes Mellitus, Experimental - pathology | Amino Acid Substitution - genetics | Cytoprotection - genetics | Endothelium, Vascular - pathology | Mice | Enzyme Activation - genetics | Cell Line, Transformed | Protein C - genetics | Proteins | Diabetes | Kidney diseases | Cellular biology | Rodents | Apoptosis
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2013, Volume 110, Issue 2, pp. 648 - 653
Journal Article
Nature Communications, ISSN 2041-1723, 03/2015, Volume 6, Issue 1, p. 6496
Endoplasmic reticulum (ER) stress is associated with diabetic nephropathy (DN), but its pathophysiological relevance and the mechanisms that compromise... 
KIDNEY-DISEASE | GLUCOSE-HOMEOSTASIS | XBP1 | UNFOLDED PROTEIN RESPONSE | GLOMERULAR PODOCYTE | ENDOPLASMIC-RETICULUM-STRESS | BOX-BINDING PROTEIN-1 | REGULATORY SUBUNITS | MULTIDISCIPLINARY SCIENCES | MICE | METABOLIC-DISORDERS | Transcription Factor CHOP - genetics | Streptozocin | Humans | Transcription Factors - deficiency | Diabetes Mellitus, Experimental - genetics | Receptor, Insulin - deficiency | Endoplasmic Reticulum - metabolism | Male | Endoplasmic Reticulum Stress - genetics | Activating Transcription Factor 6 - genetics | DNA-Binding Proteins - deficiency | X-Box Binding Protein 1 | Endoplasmic Reticulum - pathology | Class Ia Phosphatidylinositol 3-Kinase - deficiency | Receptor, Insulin - genetics | Diabetes Mellitus, Experimental - chemically induced | Class Ia Phosphatidylinositol 3-Kinase - genetics | Diabetes Mellitus, Experimental - metabolism | Databases, Factual | Diabetic Nephropathies - pathology | Podocytes - metabolism | Endoplasmic Reticulum - genetics | Signal Transduction | Diabetic Nephropathies - metabolism | Mice, Inbred C57BL | Diabetic Nephropathies - chemically induced | Gene Expression Regulation | Diabetic Nephropathies - genetics | Podocytes - pathology | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Activating Transcription Factor 6 - deficiency | Regulatory Factor X Transcription Factors | Mice, Knockout | Insulin - metabolism | Animals | Diabetes Mellitus, Experimental - pathology | Mice | Transcription Factor CHOP - metabolism
Journal Article
Blood, ISSN 0006-4971, 11/2018, Volume 132, Issue Supplement 1, pp. 137 - 137
Abstract Introduction Coagulation protease signaling via protease-activated receptors (PARs) is essential for maintenance of cellular homeostasis. Perturbed or... 
Journal Article
HAMOSTASEOLOGIE, ISSN 0720-9355, 06/2019, Volume 39, Issue 2, pp. 164 - 172
Nutrient excess in obesity drives metabolic reprogramming in multiple tissues involving extensive interorgan and intercellular crosstalk. Experimental and... 
protease-activated receptors | FACTOR XA | metabolic diseases | ADIPOSE INFLAMMATION | PLASMINOGEN-ACTIVATOR INHIBITOR-1 | DIABETIC-NEPHROPATHY | ARTERIAL THROMBOSIS | DIET-INDUCED OBESITY | TISSUE FACTOR EXPRESSION | THROMBIN GENERATION | RECEPTOR 2 | coagulation proteases | BODY-MASS INDEX | HEMATOLOGY
Journal Article
Scientific Reports, ISSN 2045-2322, 01/2014, Volume 4, Issue 1, p. 3718
Signal transduction is a process required to conduct information from a receptor to the nucleus. This process is vital for the control of cellular function and... 
CROSS-TALK | SIGNALING PATHWAYS | ROBUSTNESS | ACTIVATION | SPECIFICITY | PHOSPHORYLATION | MAP KINASE | MULTIDISCIPLINARY SCIENCES | FATE | CANCER | SYSTEMS BIOLOGY | Feedback, Physiological | Signal Transduction | Models, Biological | Cell Physiological Phenomena | Signal transduction | Transduction | Feedback | Nuclei | Information processing | Apoptosis
Journal Article
Blood, ISSN 0006-4971, 11/2012, Volume 120, Issue 21, pp. 3354 - 3354
Abstract Abstract 3354 Diabetic nephropathy (DN) is a multifactorial disease associated with substantial changes in the haemostatic system. A hallmark of... 
Journal Article
Journal of the American Society of Nephrology, ISSN 1046-6673, 2016, Volume 27, Issue 8, pp. 2270 - 2275
Journal Article
Development, ISSN 0950-1991, 06/2011, Volume 138, Issue 11, pp. 2235 - 2247
Journal Article
Journal Article