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Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 05/2018, Volume 38, Issue Suppl_1
Journal Article
Circulation. Cardiovascular Genetics, ISSN 1942-325X, 08/2017, Volume 10, Issue 4, pp. e001859 - e001859
Journal Article
Current Vascular Pharmacology, ISSN 1570-1611, 07/2015, Volume 13, Issue 3, pp. 277 - 279
Cardiovascular disease has become the predominant cause of human morbidity and mortality in the industrialized world, devouring extraordinary efforts to... 
BIOMARKERS | TARGET | CARDIOVASCULAR-DISEASE | PHARMACOLOGY & PHARMACY | PERIPHERAL VASCULAR DISEASE | MICE
Journal Article
Journal of Vascular Surgery, ISSN 0741-5214, 06/2018, Volume 67, Issue 6, pp. e236 - e236
Journal Article
Current vascular pharmacology, 2015, Volume 13, Issue 3, p. 277
Cardiovascular disease has become the predominant cause of human morbidity and mortality in the industrialized world, devouring extraordinary efforts to... 
MicroRNAs - therapeutic use | Neovascularization, Physiologic - drug effects | Neovascularization, Physiologic - genetics | Vascular Diseases - therapy | Humans | Cardiovascular Diseases - therapy | Angiogenesis Modulating Agents - pharmacology | Genetic Therapy - methods
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 02/2012, Volume 122, Issue 2, pp. 497 - 506
Journal Article
Thrombosis and Haemostasis, ISSN 0340-6245, 08/2019, Volume 119, Issue 8, pp. 1205 - 1211
Abstract Inflammation plays a pivotal role in the chronicity of atherosclerotic lesion development and progression. Myeloid cells are involved in all stages of... 
Theme Issue Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 03/2014, Volume 124, Issue 3, pp. 1083 - 1097
Genetic variation at the chromosome 9p21 risk locus promotes cardiovascular disease; however, it is unclear how or which proteins encoded at this locus... 
ENDOTHELIAL LIPASE | MEDICINE, RESEARCH & EXPERIMENTAL | SEQUENCE VARIANT | CHOLESTEROL EFFLUX | SUSCEPTIBILITY | MUSCLE-CELL APOPTOSIS | CHROMOSOME 9P21 | ABC TRANSPORTERS | CORONARY-HEART-DISEASE | EXPRESSION | MOUSE MODELS | Plaque, Atherosclerotic - genetics | Atherosclerosis - genetics | Coculture Techniques | Humans | Male | Atherosclerosis - enzymology | Plaque, Atherosclerotic - enzymology | Cyclin-Dependent Kinase Inhibitor p15 - genetics | Calreticulin - metabolism | Female | Plaque, Atherosclerotic - pathology | E2F4 Transcription Factor - metabolism | Macrophages - physiology | Atherosclerosis - pathology | Coronary Vessels - pathology | Gene Expression | Myocytes, Smooth Muscle - enzymology | Down-Regulation | Mice, Inbred C57BL | Retinoblastoma Protein - metabolism | Cells, Cultured | Mice, Knockout | E2F4 Transcription Factor - genetics | Animals | Retinoblastoma Protein - genetics | Carotid Arteries - pathology | Mice | Cyclin-Dependent Kinase Inhibitor p15 - metabolism | Quantitative Trait Loci | Necrosis - enzymology | Phagocytosis | Apoptosis | Physiological aspects | Development and progression | Enzyme inhibitors | Genetic variation | Identification and classification | Atherosclerosis | Studies | Heart attacks | Cardiovascular disease | Software | Genomes | Grants | Kinases | Laboratory animals | Gene expression | Experiments | Index Medicus | Abridged Index Medicus
Journal Article
Cardiovascular Research, ISSN 0008-6363, 03/2018, Volume 114, Issue 4, pp. 611 - 621
Journal Article
Nature Medicine, ISSN 1078-8956, 10/2016, Volume 22, Issue 10, pp. 1140 - 1150
Adenosine-to-inosine (A-to-I) RNA editing, which is catalyzed by a family of adenosine deaminase acting on RNA (ADAR) enzymes, is important in the... 
MEDICINE, RESEARCH & EXPERIMENTAL | ANGIOGENESIS | STABILIZATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | LUNG INJURY | CELL BIOLOGY | MESSENGER-RNA | GENE-EXPRESSION | ALU ELEMENTS | HUMAN TRANSCRIPTOME | BINDING PROTEIN HUR | TUMOR-GROWTH | ENZYME ADAR1 | Human Umbilical Vein Endothelial Cells | RNA-Binding Proteins - genetics | RNA Editing - genetics | Atherosclerosis - genetics | Humans | Middle Aged | Immunoblotting | Male | RNA, Messenger - metabolism | Gene Knockdown Techniques | Aged, 80 and over | Adult | Female | 3' Untranslated Regions | Real-Time Polymerase Chain Reaction | Adenosine Deaminase - genetics | RNA Processing, Post-Transcriptional - drug effects | ELAV-Like Protein 1 - genetics | Gene Expression Regulation | Inosine - metabolism | Gene Knock-In Techniques | Tumor Necrosis Factor-alpha - pharmacology | Hypoxia - genetics | RNA Processing, Post-Transcriptional - genetics | Sequence Analysis, RNA | Cathepsins - genetics | Carotid Artery Diseases - genetics | Coronary Artery Disease - genetics | Fluorescent Antibody Technique | Adenosine - metabolism | Aged | High-Throughput Nucleotide Sequencing | Aortic Aneurysm - genetics | Interferon-gamma - pharmacology | RNA Editing - drug effects | Adenosine | Atherosclerosis | Genetic aspects | Dosage and administration | Research | Gene expression | Drug therapy | Post-transcription | Gene regulation | Cysteine proteinase | Editing | Cardiovascular disease | Proteins | Angiogenesis | Control | Vascular diseases | Aorta | Heart diseases | HuR protein | RNA editing | Cytokines | Coronary artery | Inflammation | Cathepsin S | Adenosine deaminase | Metabolism | Coronary artery disease | Substrates | Endothelial cells | RNA-binding protein | Ribonucleic acids | 3' Untranslated regions | Arteriosclerosis | Hypoxia | Control stability | Interferon | mRNA stability | Index Medicus
Journal Article
Current vascular pharmacology, 05/2013
Journal Article
Thrombosis and Haemostasis, ISSN 0340-6245, 04/2019, Volume 119, Issue 4, pp. 542 - 552
Journal Article
PLoS Genetics, ISSN 1553-7390, 2014, Volume 10, Issue 3, pp. e1004263 - e1004263
Journal Article